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Extrapulmonary tuberculosis
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Chronic myeloid leukemia (CML)
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Lung consolidation
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Hodgkins & Non-Hodgkins Lymphomas
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Carlos Cerna
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Balasubramanian Thiagarajan
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paras suthar
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Pathogenesis of tuberculosis
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Pulmonary tuberculosis ppt
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Mycobacterium Tuberculosis
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Mohsin Javed
TB Tuberculosis Extra-pulmonary TB. As of 2017, about two billion people worldwide are infected with Mycobacterium tuberculosis, the causative pathogen of tuberculosis disease, commonly known as ‘TB’. However, for the vast majority, (90-95%) of infected individuals, the infection is contained by the immune system and cannot multiply. In other words, the TB disease remains latent, or dormant, as opposed to active, which usually causes symptoms and can easily be transmitted to others. When the host’s immune system becomes compromised, e.g. due to HIV or malnutrition and aging, TB can reactivate, and become very serious, especially if the infection spreads through the body. Moreover, people with active TB can easily infect 10-15 other people via close contact within a year. Mycobacteria are slender, rod-shaped, and need high levels of oxygen to survive, i.e., “strict aerobes”. They possess a waxy cell wall that is capable of retaining dyes even when exposed to alcohol. Thus they are referred to as “acid-fast”, appearing as bright- red colored rods when a Ziehl–Neelsen stain is used. The wall also makes them incredibly hardy and allows them to resist weak disinfectants and survive on dry surfaces for months. M. tuberculosis is usually transmitted via inhalation, which is how they gain entry into the lungs. Although we breathe in all sorts of viruses and bacteria all the time, we have defenses that take care of most of them. For one, the air that we breathe in is turbulent in the upper airways and drives most bacteria against mucus which is then cleared pretty quickly. Ultimately, though, TB can avoid the mucus traps and make its way to the deep airways and alveoli where we have macrophages that eat up foreign cells, digest and destroy them. With TB, they recognize foreign proteins on their cell surface, and phagocytize them, or essentially package them into a space called a phagosome. In most cases, the macrophage then fuses the phagosome with a lysosome, which has hydrolytic enzymes that can pretty much break down any biochemical molecule. TB’s tricky, though, and once inside the macrophage, they produce a protein that inhibits this fusion, which allows the mycobacterium to survive. It doesn’t just survive, though, it proliferates and creates a localized infection. At this point, somebody has developed primary tuberculosis, which means that they have signs of infection soon after being exposed to TB.
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