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Tetralogy of Fallot, also called TOF, is a congenital heart disease. A congenital heart disease is one that a person is born with. TOF is the most common heart disease that causes cyanosis (bluish color of the skin).
Tetralogy of Fallot (TOF) - Symptoms, Tests and Treatment
Tetralogy of Fallot (TOF) - Symptoms, Tests and Treatment
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undergraduate course lectures in obstetrics and gynecology prepared by Dr Manal Behery Professor of OB&GYNE ,Faculty of medicine,Zagazig University
Hydrops fetails for undergranuate
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TAPVC defines the anomaly in which the pulmonary veins have no connection with the left atrium. Rather, the pulmonary veins connect directly to one of the systemic veins (TAPVC) or drain in to right atrium. A PFO or ASD is present essentially in those who survive after birth When pulmonary veins drain anomalously into the right atrium either because of complete absence of the interatrial septum or malattachment of the septum primum , then it is known as total anomalous pulmonary venous drainage. When some or all of the pulmonary veins drain anomalously in to RA or its tributaries without being abnormally connected, the terms partially anomalous pulmonary venous drainage (PAPVD) or totally anomalous pulmonary venous drainage (TAPVD) with normal pulmonary venous connections are used.
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Tetralogy of Fallot, also called TOF, is a congenital heart disease. A congenital heart disease is one that a person is born with. TOF is the most common heart disease that causes cyanosis (bluish color of the skin).
Tetralogy of Fallot (TOF) - Symptoms, Tests and Treatment
Tetralogy of Fallot (TOF) - Symptoms, Tests and Treatment
heartdoctor123456
undergraduate course lectures in obstetrics and gynecology prepared by Dr Manal Behery Professor of OB&GYNE ,Faculty of medicine,Zagazig University
Hydrops fetails for undergranuate
Hydrops fetails for undergranuate
Faculty of Medicine,Zagazig University,EGYPT
TAPVC defines the anomaly in which the pulmonary veins have no connection with the left atrium. Rather, the pulmonary veins connect directly to one of the systemic veins (TAPVC) or drain in to right atrium. A PFO or ASD is present essentially in those who survive after birth When pulmonary veins drain anomalously into the right atrium either because of complete absence of the interatrial septum or malattachment of the septum primum , then it is known as total anomalous pulmonary venous drainage. When some or all of the pulmonary veins drain anomalously in to RA or its tributaries without being abnormally connected, the terms partially anomalous pulmonary venous drainage (PAPVD) or totally anomalous pulmonary venous drainage (TAPVD) with normal pulmonary venous connections are used.
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this is the best notes of congenital heart disease in pediatric
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ETIOLOGY AND EPIDEMIOLOGY Congenital heart disease occurs in 8 per 1,000 births. The spectrum of lesions ranges from asymptomatic to fatal. Although most cases of congenital heart disease are multifactorial, some lesions are associated with chromosomal disorders, single gene defects, teratogens, or maternal metabolic disease (see Table139-2). Congenital heart defects can be divided into three pathophysiological groups (Table 143.1). 1. Left-to-right shunts 2. Right-to-left shunts 3. Obstructive, stenotic lesions Acyanotic congenital heart disease includes left-to-right shunts resulting in an increase in pulmonary blood flow (patent ductus arteriosus [PDA], ventricular septal defect [VSD], atrial septal defect [ASD]) and obstructive lesions (aortic stenosis, pulmonary stenosis, coarctation of the aorta), which usually have normal pulmonary blood flow. VENTRICULAR SEPTAL DEFECTEtiology and Epidemiology The ventricular septum is a complex structure that can be divided into four components. The largest component is the muscular septum. The inlet or posterior septum comprises endocardial cushion tissue. The subarterial or supracristal septum com prises conotruncal tissue. The membranous septum is below the aortic valve and is relatively small. VSDs occur when any of these components fail to develop normally (Fig. 143.1). VSD, the most common congenital heart defect, accounts for 25% of all congenital heart disease. Perimembranous VSD
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Copy and paste no references What changes in veins occur to create varicose veins? What is the disease process with superior vena cava syndrome? How does atherosclerosis cause an aneurysm? What are the differences between healthy vessel walls and those that promote clot formation? What are the causes of pulmonary emboli? What can trigger an immune response within the bloodstream that can result in an embolus? Describe thromboangiitis obliterans (Buerger disease). Describe Raynaud disease. What is the cause of the hypertrophy of the myocardium associated with hypertension? What pathologic change occurs to the kidney’s glomeruli as a result of hypertension? What is the primary mechanism of atherogenesis? What is the effect of oxidized low-density lipoproteins (LDLs) in atherosclerosis? Which inflammatory cytokines are released when endothelial cells are injured? What alteration occurs in injured endothelial cells which contributes to atherosclerosis? Differentiate between ischemia and infarction. Which of the lipids is manufactured by the liver and primarily contains cholesterol and protein? Which elevated lipid value may be protective of the development of atherosclerosis? What are the risk factors for developing coronary artery disease? Which risk factor increases the risk of developing coronary artery disease up to threefold? How does nicotine increases atherosclerosis? Which lab test is an indirect measure of atherosclerotic plaque? Cardiac cells can withstand ischemic conditions and still return to a viable state for how many minutes? Which form of angina occurs most often during sleep as a result of vasospasms of one or more coronary arteries? What is the expected electrocardiogram pattern for a person when a thrombus in a coronary artery lodges in the vessel and the infarction extends through the myocardium from the endocardium to the epicardium? How does angiotensin II increase the workload of the heart after a myocardial infarction? How long after a myocardial infarction is the scar tissue that forms most vulnerable to injury? What are the enzyme indicators of a myocardial infarction? A patient complains of sudden onset of severe chest pain that radiates to the back and worsens with respiratory movement and when lying down. These clinical manifestations are indicative of which disease? What is the significance of the pulsus paradoxus that occurs as a result of pericardial effusion? Biventricular dilation is the result of which form of cardiomyopathy? Which form of cardiomyopathy is characterized by ventricular dilation and grossly impaired systolic function, leading to dilated heart failure? The hallmark of which form of cardiomyopathy is a disproportionate thickening of the interventricular septum? Which form of cardiomyopathy is usually caused by an infiltrative disease of the myocardium, such as amyloidosis, hemochromatosis, or glycogen storage disease? Which are the clinical manifestations of a.
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The lecture is for medical student. It is from Dr RUSINGIZA Emmanuel, MD, senior lecture at UR( UNIVERSITY OF RWANDA) . It will help to understand heart diseases in newborn, infants and children.
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