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Vértigo
¿O mandé un ACV a la casa?

       Dr. Carlos Basaure V
         Emergenciólogo
Departamento Medicina de Urgencia
       Universidad de Chile
Vértigo
• Sensación de desorientación en el espacio
  más sensación de movimiento
• Mareos
• Debilidad
• Nauseas
• Lipotimia
Desafío


• ¿Es vértigo?
• ¿Es central o periférico?
Equilibrio




•Núcleo vestibular:
  • Integra todas las señales en el cerebelo
Sistema Vestibular
Nistagmo
Enfrentamiento

• ¿Es Vértigo?
    • Sensación de movimiento
    • Nauseas, vómitos, diaforesis, palidez.
Enfrentamiento
             Vértigo



Periférico             Central
               SVA     Hemorragia
     VPPB
                         Tumor
     Meniere            Isquemia
Enfrentamiento
             Vértigo



Periférico             Central
               SVA     Hemorragia
     VPPB
                         Tumor
     Meniere            Isquemia
Enfrentamiento
             Vértigo



Periférico             Central
               SVA     Hemorragia
     VPPB
                         Tumor
     Meniere            Isquemia
Enfrentamiento
               Vértigo



Periférico                 Central
                  SVA       Hemorragia
      VPPB
                              Tumor
      Meniere                Isquemia



 Neuritis Vestibular ACV Vertebrobasilar
            Esclerosis Múltiple
Sindrome Vestibular Agudo

•   SVA: vértigo de inicio súbito, nauseas, vómitos,
    marcha inestable y imposibilidad de mover la
    cabeza y nistagmo que dura días a semanas

•   25% de estos son ACV cerebeloso o de tronco

•   TC tiene sens. baja (16%) para fosa posterior

•   RNM no esta presente en todas partes

•   Menos de a mitad tiene disartria o dismetría
Enfrentamiento
• ¿Periférico o central?
 • Periférico: episódico, intenso, cambios de
    posición, tinítus, hipoacusia
  • Central: ataxia, lateropulsión, edad
    avanzada, FA, HTA, DM, E Coronaria.
  • Asociación a trauma (cuello, craneo)
It has clearly been shown that isolated vertigo can be the             The abnormal jerk nystagmus of inner ear disease consists
 , and            only initial symptom of cerebellar and other posterior circula-        of slow and quick components. The eyes slowly “drift” in the
asion-            tion bleeds, transient ischemic attacks (TIAs), and infarc-            direction of the diseased, hypoactive ear, then quickly jerk
ediate    Table 12-1 Characteristics of Peripheral and physicians often
                  tion.11-13 One study showed that emergency                             back to the intended direction of gaze. Positional nystagmus,
  laby-           did not make Vertigo diagnosis in patients with validated
                         Central the correct                                             induced by rapidly changing the position of the head, strongly
quires            strokes or TIAs that presented with only vertigo.6 Risk factor         suggests an organic vestibular disorder. The characteristics of
denti-     CHARACTERISTIC and symptom patterns can beCENTRAL helpful in
                  assessment            PERIPHERAL                extremely              nystagmus are one of the most valuable tools for distinguishing
                  deciding which patients warrant imaging and admission. Older           peripheral from central causes of vertigo (Table 12-2).
e 12-1
           Onset age, male sex, hypertension, coronary artery disease, diabetes
                                        Sudden                   Gradual or sudden          Positional Testing. If nystagmus is not present at rest, positional
l and             mellitis, and atrial fibrillation put patients at higher risk. In
           Intensity                    Severe                   Mild                    testing can be helpful in determining its existence and char-
                  addition, frequent episodes lastingor
           Duration                     Usually seconds only minutes weeks,
                                                                 Usually or prolonged    acteristics. In the Hallpike maneuver, the patient is moved
                  episodes of a day minutes; are more often associated with
                                            or more                months                quickly from an upright seated position to a supine position,
                                         6,11,12
                  central processes. occasionally retrospective study showed
                                                 A recent          (continuous) but      and the head is turned to one side and extended (to a head-
                  emergency physicians often failed to chartcan be seconds
                                            hours, days             triggers and dura-   down posture) approximately 30° from the horizontal plane off
                  tion of dizziness, information that could potentially lead to
                                            (intermittent)         or minutes with       the end of the stretcher. The eyes should be observed for
                  increased likelihood of a more serious cause of symptoms.14
                                                                   vascular causes       nystagmus and the patient queried for the occurrence of symp-
                     Past Medical History. Many medications have direct vestibulo-
           Direction of                 One direction            Horizontal, rotary,
 orma-            toxicity. The most commonly encounteredor verticalare the aminogly-
 Does        nystagmus                      (usually
                  cosides, anticonvulsants, alcohols, quinine, quinidine, and
                                            horizontorotary),      (different
e or a            minocycline. In addition, caffeine and nicotine can have wide-
                                            never vertical         directions in                      Distinguishing Characteristics of Nystagmus
  indi-           ranging autonomic effects that may exacerbate vestibular               Table 12-2 with Central and Peripheral Vertigo
                                                                   different
 r, and           symptoms. The history of past and present illnesses should be
                                                                   positions)
                  explored, with specific questioning about the existence of dia-
 ms of     Effect of headdrug or alcohol use,by the risk factors mentioned
                                        Worsened and             Little change,           CHARACTERISTIC        CENTRAL                    PERIPHERAL
                  betes,
 ertigo      position
                  earlier.                  position, often        associated with        Direction             Any direction              Horizontal or
 effect                                     single critical        more than one                                                             horizontorotary
ave an                                      position               position               Laterality            Unilateral or bilateral    Bilateral
                  Physical Examination
           Associated                   None                     Usually present
 on of                                                                                    Position testing
y, dis-      neurologic
                  Vital Signs. In some cases, pulses and blood pressure should be           effects:
             findings
                  checked in both arms. Most patients with subclavian steal                 Latency             Short                      Long
 entral                                                                                     Duration            Sustained                  Transient
           Associated auditory
                  syndrome, whichMay canpresent,
                                         also be cause vertebrobasilar artery insuffi-
                                                                 None
                                                                                            Intensity           Mild                       Mild to severe
             findings
                  ciency, have pulseincluding  or systolic blood pressure differences
 often            between the two arms.     tinnitus
                                                                                            Fatigability        Nonfatigable               Fatigable
  have                                                                                    Effect of visual      Not suppressed, may        Suppressed
                     Head and Neck. Carotid or vertebral artery bruits suggest ath-         fixation               be enhanced
spells.           erosclerosis. The neck is auscultated along the course of the
ble to    evidence suggesting seizures, syncope, or imbalance unrelated
e able    to feelings of vertigo.
 iately      The time of onset and the duration of vertigo are important
  addi-   clues to the cause. Episodic vertigo that is severe, lasts several
 cover    hours, and has symptom-free intervals between episodes sug-
Enfrentamiento
• ¿SVA es Central o periférico?
 • HINTS
                    HINTS to Diagnose Stroke in the Acute Vestibular Syndrome : Three-Step Bedside
                  Oculomotor Examination More Sensitive Than Early MRI Diffusion-Weighted Imaging
                       Jorge C. Kattah, Arun V. Talkad, David Z. Wang, Yu-Hsiang Hsieh and David E.
                                                      Newman-Toker


                          Stroke. 2009;40:3504-3510; originally published online September 17, 2009;
                                           doi: 10.1161/STROKEAHA.109.551234
                       Stroke is published by the American Heart Association, 7272 Greenville Avenue, Dallas, TX 75231
                                     Copyright © 2009 American Heart Association, Inc. All rights reserved.
                                                Print ISSN: 0039-2499. Online ISSN: 1524-4628




                 The online version of this article, along with updated information and services, is located on the
                                                        World Wide Web at:
                                               http://stroke.ahajournals.org/content/40/11/3504
Enfrentamiento
• Head Impulse (HI) :
 • Evalúa el reflejo vestíbulo-ocular
 • Se fija la mirada en un punto y se gira la
    cabeza en 30º de manera aleatoria
  • Si esta presente, indica lesión vestibular
Head Impulse
                      (HI)


Prueba POSITIVA
Periférico
Bueno
Head Impulse
                      (HI)


Prueba POSITIVA
Periférico
Bueno
Enfrentamiento

• Nistagmo (N)
 • Multidireccional, no agotable, alternante
Nistagmo “Horizontal”



   Horizontal Periférico Bueno
Nistagmo “Horizontal”



   Horizontal Periférico Bueno
Nistagmo “vertical”



  Vertical   Central   Malo
Nistagmo “vertical”



  Vertical   Central   Malo
Enfrentamiento

• Test of Skew (TS)
 • Se cubre un ojo y se pide que fije la
    mirada en un punto comandos ojos.
 • Central si el ojo examinado esta
    desalineado de manera vertical
Test of Skew (TS)




  Desalineamiento Central
Test of Skew (TS)




  Desalineamiento Central
Enfrentamiento
    Sindrome Vestibular Agudo
•   Util:
     •      Múltiples episodios → ACV
     •      Cefalea o Cervicalgia → LR 3.2
     •      Sint Neurológicos: ataxia de tronco o lateropulsión
     •      Head Impulse: Normal LR 18.4 para ACV
     •      Nistagmo: alternante S30% E98%
     •      Test Skew: anormal → ACV S38% E98%
     •      RNM: S83% para ACV
Enfrentamiento
Inútil:
•   Diferenciar tipo de vértigo
•   Onset del vértigo
•   Provocación con movimiento
•   Proporcionalidad de síntomas (distinto → periférico)
•   Hipoacusia
•   Parton y vector del nistagmo
•   TC Cerebro sin cte. S16% para ACV
Enfrentamiento
HINTS
•   INFARCT
•   Impulso Normal
•   Fast-Phase Alternating
•   Refixation on Cover Test
•   Si 1 de 3 anormal S100% E96% para CENTRAL
Vértigos Periféricos
•   VPPB:
    •   Intenso, corta duración, sintomas asociados,
        nistagmo reproducible con cambios de
        posición
• Meniere:
 • Horas de duración, tinítus, hipoacusia,
        periodos de ataques frecuentes y periodos
        largos asintomáticos, sin nistagmo posicional
Sindrome Vestibular Agudo

HINTS

•   Head Impulse: anormal
                                                      SVA Periférico
•   Nistagmo: horizontal, agotable, unidireccional   Neuritis Vestibular
•   Test Skew: normal
FIN
Vertigo
Vertigo
Vertigo
Vertigo

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Vertigo

  • 1. Vértigo ¿O mandé un ACV a la casa? Dr. Carlos Basaure V Emergenciólogo Departamento Medicina de Urgencia Universidad de Chile
  • 2. Vértigo • Sensación de desorientación en el espacio más sensación de movimiento • Mareos • Debilidad • Nauseas • Lipotimia
  • 3. Desafío • ¿Es vértigo? • ¿Es central o periférico?
  • 4. Equilibrio •Núcleo vestibular: • Integra todas las señales en el cerebelo
  • 6.
  • 7.
  • 9. Enfrentamiento • ¿Es Vértigo? • Sensación de movimiento • Nauseas, vómitos, diaforesis, palidez.
  • 10. Enfrentamiento Vértigo Periférico Central SVA Hemorragia VPPB Tumor Meniere Isquemia
  • 11. Enfrentamiento Vértigo Periférico Central SVA Hemorragia VPPB Tumor Meniere Isquemia
  • 12. Enfrentamiento Vértigo Periférico Central SVA Hemorragia VPPB Tumor Meniere Isquemia
  • 13. Enfrentamiento Vértigo Periférico Central SVA Hemorragia VPPB Tumor Meniere Isquemia Neuritis Vestibular ACV Vertebrobasilar Esclerosis Múltiple
  • 14. Sindrome Vestibular Agudo • SVA: vértigo de inicio súbito, nauseas, vómitos, marcha inestable y imposibilidad de mover la cabeza y nistagmo que dura días a semanas • 25% de estos son ACV cerebeloso o de tronco • TC tiene sens. baja (16%) para fosa posterior • RNM no esta presente en todas partes • Menos de a mitad tiene disartria o dismetría
  • 15. Enfrentamiento • ¿Periférico o central? • Periférico: episódico, intenso, cambios de posición, tinítus, hipoacusia • Central: ataxia, lateropulsión, edad avanzada, FA, HTA, DM, E Coronaria. • Asociación a trauma (cuello, craneo)
  • 16. It has clearly been shown that isolated vertigo can be the The abnormal jerk nystagmus of inner ear disease consists , and only initial symptom of cerebellar and other posterior circula- of slow and quick components. The eyes slowly “drift” in the asion- tion bleeds, transient ischemic attacks (TIAs), and infarc- direction of the diseased, hypoactive ear, then quickly jerk ediate Table 12-1 Characteristics of Peripheral and physicians often tion.11-13 One study showed that emergency back to the intended direction of gaze. Positional nystagmus, laby- did not make Vertigo diagnosis in patients with validated Central the correct induced by rapidly changing the position of the head, strongly quires strokes or TIAs that presented with only vertigo.6 Risk factor suggests an organic vestibular disorder. The characteristics of denti- CHARACTERISTIC and symptom patterns can beCENTRAL helpful in assessment PERIPHERAL extremely nystagmus are one of the most valuable tools for distinguishing deciding which patients warrant imaging and admission. Older peripheral from central causes of vertigo (Table 12-2). e 12-1 Onset age, male sex, hypertension, coronary artery disease, diabetes Sudden Gradual or sudden Positional Testing. If nystagmus is not present at rest, positional l and mellitis, and atrial fibrillation put patients at higher risk. In Intensity Severe Mild testing can be helpful in determining its existence and char- addition, frequent episodes lastingor Duration Usually seconds only minutes weeks, Usually or prolonged acteristics. In the Hallpike maneuver, the patient is moved episodes of a day minutes; are more often associated with or more months quickly from an upright seated position to a supine position, 6,11,12 central processes. occasionally retrospective study showed A recent (continuous) but and the head is turned to one side and extended (to a head- emergency physicians often failed to chartcan be seconds hours, days triggers and dura- down posture) approximately 30° from the horizontal plane off tion of dizziness, information that could potentially lead to (intermittent) or minutes with the end of the stretcher. The eyes should be observed for increased likelihood of a more serious cause of symptoms.14 vascular causes nystagmus and the patient queried for the occurrence of symp- Past Medical History. Many medications have direct vestibulo- Direction of One direction Horizontal, rotary, orma- toxicity. The most commonly encounteredor verticalare the aminogly- Does nystagmus (usually cosides, anticonvulsants, alcohols, quinine, quinidine, and horizontorotary), (different e or a minocycline. In addition, caffeine and nicotine can have wide- never vertical directions in Distinguishing Characteristics of Nystagmus indi- ranging autonomic effects that may exacerbate vestibular Table 12-2 with Central and Peripheral Vertigo different r, and symptoms. The history of past and present illnesses should be positions) explored, with specific questioning about the existence of dia- ms of Effect of headdrug or alcohol use,by the risk factors mentioned Worsened and Little change, CHARACTERISTIC CENTRAL PERIPHERAL betes, ertigo position earlier. position, often associated with Direction Any direction Horizontal or effect single critical more than one horizontorotary ave an position position Laterality Unilateral or bilateral Bilateral Physical Examination Associated None Usually present on of Position testing y, dis- neurologic Vital Signs. In some cases, pulses and blood pressure should be effects: findings checked in both arms. Most patients with subclavian steal Latency Short Long entral Duration Sustained Transient Associated auditory syndrome, whichMay canpresent, also be cause vertebrobasilar artery insuffi- None Intensity Mild Mild to severe findings ciency, have pulseincluding or systolic blood pressure differences often between the two arms. tinnitus Fatigability Nonfatigable Fatigable have Effect of visual Not suppressed, may Suppressed Head and Neck. Carotid or vertebral artery bruits suggest ath- fixation be enhanced spells. erosclerosis. The neck is auscultated along the course of the ble to evidence suggesting seizures, syncope, or imbalance unrelated e able to feelings of vertigo. iately The time of onset and the duration of vertigo are important addi- clues to the cause. Episodic vertigo that is severe, lasts several cover hours, and has symptom-free intervals between episodes sug-
  • 17. Enfrentamiento • ¿SVA es Central o periférico? • HINTS HINTS to Diagnose Stroke in the Acute Vestibular Syndrome : Three-Step Bedside Oculomotor Examination More Sensitive Than Early MRI Diffusion-Weighted Imaging Jorge C. Kattah, Arun V. Talkad, David Z. Wang, Yu-Hsiang Hsieh and David E. Newman-Toker Stroke. 2009;40:3504-3510; originally published online September 17, 2009; doi: 10.1161/STROKEAHA.109.551234 Stroke is published by the American Heart Association, 7272 Greenville Avenue, Dallas, TX 75231 Copyright © 2009 American Heart Association, Inc. All rights reserved. Print ISSN: 0039-2499. Online ISSN: 1524-4628 The online version of this article, along with updated information and services, is located on the World Wide Web at: http://stroke.ahajournals.org/content/40/11/3504
  • 18. Enfrentamiento • Head Impulse (HI) : • Evalúa el reflejo vestíbulo-ocular • Se fija la mirada en un punto y se gira la cabeza en 30º de manera aleatoria • Si esta presente, indica lesión vestibular
  • 19. Head Impulse (HI) Prueba POSITIVA Periférico Bueno
  • 20. Head Impulse (HI) Prueba POSITIVA Periférico Bueno
  • 21. Enfrentamiento • Nistagmo (N) • Multidireccional, no agotable, alternante
  • 22. Nistagmo “Horizontal” Horizontal Periférico Bueno
  • 23. Nistagmo “Horizontal” Horizontal Periférico Bueno
  • 24. Nistagmo “vertical” Vertical Central Malo
  • 25. Nistagmo “vertical” Vertical Central Malo
  • 26. Enfrentamiento • Test of Skew (TS) • Se cubre un ojo y se pide que fije la mirada en un punto comandos ojos. • Central si el ojo examinado esta desalineado de manera vertical
  • 27. Test of Skew (TS) Desalineamiento Central
  • 28. Test of Skew (TS) Desalineamiento Central
  • 29. Enfrentamiento Sindrome Vestibular Agudo • Util: • Múltiples episodios → ACV • Cefalea o Cervicalgia → LR 3.2 • Sint Neurológicos: ataxia de tronco o lateropulsión • Head Impulse: Normal LR 18.4 para ACV • Nistagmo: alternante S30% E98% • Test Skew: anormal → ACV S38% E98% • RNM: S83% para ACV
  • 30. Enfrentamiento Inútil: • Diferenciar tipo de vértigo • Onset del vértigo • Provocación con movimiento • Proporcionalidad de síntomas (distinto → periférico) • Hipoacusia • Parton y vector del nistagmo • TC Cerebro sin cte. S16% para ACV
  • 31. Enfrentamiento HINTS • INFARCT • Impulso Normal • Fast-Phase Alternating • Refixation on Cover Test • Si 1 de 3 anormal S100% E96% para CENTRAL
  • 32. Vértigos Periféricos • VPPB: • Intenso, corta duración, sintomas asociados, nistagmo reproducible con cambios de posición • Meniere: • Horas de duración, tinítus, hipoacusia, periodos de ataques frecuentes y periodos largos asintomáticos, sin nistagmo posicional
  • 33. Sindrome Vestibular Agudo HINTS • Head Impulse: anormal SVA Periférico • Nistagmo: horizontal, agotable, unidireccional Neuritis Vestibular • Test Skew: normal
  • 34. FIN

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