2. LEARNING OBJECTIVES
On completion of the course students will be able
to:
explain common symptoms of cardiac disease
conduct a step -wise approach in cardiovascular
examination
identify the normal and abnormal cardiac findings
interpret cardiac findings
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3. INTRODUCTION: OVER VIEW OF CARDIAC
ANATOMY
The right ventricle occupies most of the anterior
cardiac surface.
The inferior border of the right ventricle lies below
the junction of the sternum and the xiphoid
process.
The right ventricle narrows superiorly and meets
the pulmonary artery at the level of the base of the
heart.
The left ventricle, behind the right ventricle and to
the left, forms the left lateral margin of the heart.
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4. Point of maximum impulse is located in the left
border of the heart.
is usually found in the 5th interspaces 7 cm to 9
cm lateral to the midsternal line.
it is about 1 to 2.5 cm in diameter.
right atrium of the heart is found anteriorly and
accessible for physical examination.
The left atrium of the heart is mostly posterior
and cannot be examined directly.
Circulation through the heart includes the cardiac
chambers, valves, blood, blood vessels.
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7. Because of their positions, the tricuspid and mitral
valves are called atrioventricular valves.
The aortic and pulmonic valves are called semilunar
valves because each of their leaflets is shaped like a
half moon.
As the heart valves close, the heart sounds arise
from vibrations emanating from the leaflets, the
adjacent cardiac structures, and the flow of blood.
Systole is the period of ventricular contraction.
During systole Pressure in the left ventricle rises
from less than 5 mm Hg in its resting state to a
normal peak of 120 mm Hg.04/03/2011 menbit@ymail.com 7
8. Diastole is the period of ventricular relaxation.
During diastole ventricular pressure falls further to
below 5 mm Hg, and blood flows from atrium to
ventricle.
The mitral valve is closed, preventing blood from
regurgitating back into the left atrium.
during diastole the aortic valve is closed, preventing
regurgitation of blood from the aorta back into the
left ventricle.
Closure of the atrioventricular valves produce the
first heart sound, S1.
Closure of the semilunar valves produce the second
heart sound, S2.
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9. SPLITTING OF HEART SOUNDS
Right ventricular and pulmonary arterial pressures
are significantly lower than corresponding pressures
on the left side.
right-sided events usually occur slightly later than
those on the left.
So instead of a single heart sound, you may hear
two discernible components, the first from left-
sided aortic valve closure, or A2, and the second
from right-sided closure of the plutonic valve, or P2.
Consider the second heart sound and its two
components, A2 and P2, which come from closure
of the aortic and pulmonic valves respectively.04/03/2011 menbit@ymail.com 9
10. During expiration, these two components are
fused into a single sound, S2.
During inspiration, however, A2 and P2 separate
slightly, and S2 may split into its two audible
components.
Current explanations of inspiratory splitting cite
increased capacitance in the pulmonary vascular
bed during inspiration, which prolongs ejection of
blood from the right ventricle, delaying closure of
the pulmonic valve, or P2.
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12. Of the two components of the second heart
sound, A2 is normally louder, reflecting the high
pressure in the aorta.
It is heard throughout the precordium.
P2, in contrast, is relatively soft, reflecting the lower
pressure in the pulmonary artery.
It is heard best in its own area—the 2nd and 3rd
left interspaces close to the sternum. It is here that
you should search for splitting of the second heart
sound.
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13. CON…
S1 also has two components, an earlier mitral and
a later tricuspid sound.
The mitral sound, its principal component, is much
louder, again reflecting the high pressures on the left
side of the heart.
It can be heard throughout the precordium and is
loudest at the cardiac apex.
The softer tricuspid component is heard best at the
lower left sternal border, and it is here that you may
hear a split S1.
The earlier louder mitral component may mask the
tricuspid sound, however, and splitting is not always
detectable. Splitting of S1 does not vary with
respiration.04/03/2011 menbit@ymail.com 13
18. Common symptoms
Dyspnea:
is a state of shortness of breath on exertion and /or rest
is graded based on the New York Heart Association
Class (NHAC):
Class I: No limitation of physical activity .No symptoms
with ordinary exertion
Class II: Slight limitation of physical activity Ordinary
activity causes symptoms
Class III: Marked limitation of physical activity less than
ordinary activity causes symptoms . Asymptomatic at
rest.
Class IV: Inability to carry out any physical activity
without discomfort .Symptomatic at rest.04/03/2011 menbit@ymail.com 18
19. Paroxysmal Nocturnal Dyspnea
shortness of breath that occurs during sleep
Orthopnea
Shortness of breath that occurs during recumbent
position
Palpitation
subjective unpleasant perception of one’s own heart
beat.
Syncope
Sudden episode of fainting
Chest pain
Body swelling
Cough04/03/2011 menbit@ymail.com 19
20. Peripheral symptoms
1. Symptoms of Arterial occlusion:
pain, loss of function, altered cutaneous
sensation, gangrene, pain around calf muscle on
walking which gets relieved with rest
2. Symptoms of Venous insufficiency:
Swelling and pain of the affected body area.
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22. 1. Peripheral manifestation
Observe the following general conditions :
I. Face
Malar flush (thin face, purple cheeks) may be found
in mitral stenosis.
Lips for (cyanosis).
II. Eyes
Pallor of the conjunctiva ,palms and nail bed indicates
anemia.
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23. III. Hands
Clubbing of fingers : Cyanotic congenital heart
disease, Infective endocarditis
Peripheral cyanosis
Splinter hemorrhages: - vertical linear hemorrhages
beneath the nails.
Osler's nodes: - Tender lumps in pulp of fingertips which
may be found in endocarditis
Jane way lesions:- are painless red macules on the wrist
and palm which may be seen in patients with acute
infective endocarditis.
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24. 2. JUGULAR VENOUS PRESSURE (JVP):-
Systemic venous pressure is much lower than arterial
pressure because:
much of the force of ventricular contraction is dissipated as
blood passes through the arterial tree and the capillary bed.
Walls of veins contain less smooth muscle, which reduces
venous vascular tone and makes veins more distensible.
blood volume and the capacity of the right heart to eject
blood into the pulmonary arterial system.
Cardiac disease may alter these variables, producing
abnormalities in central venous pressure.
For example, venous pressure falls when left ventricular
output or blood volume is significantly reduced
it rises when the right heart fails or when increased pressure
in the pericardial sac impedes the return of blood to the right
atrium.
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25. • These venous pressure changes are reflected in the
height of the venous column of blood in the internal
jugular veins, termed the jugular venous pressure.
• Pressure in the jugular veins reflects right atrial
pressure, giving clinicians an important clinical indicator
of cardiac function and right heart hemodynamics.
• The JVP is best estimated from the internal jugular vein,
usually on the right side, since the right internal jugular
vein has a more direct anatomic channel into the right
atrium.
• The internal jugular veins lie deep to the sternomastoid
muscles in the neck and are not directly visible.
• carefully distinguish these venous pulsations from
pulsations of the carotid artery.04/03/2011 menbit@ymail.com 25
27. STEPS
Raise the head of the bed or examining table to
about 30°.
Raise the head slightly on a pillow to relax the
sternomastoid muscles.
Turn the patient’s head slightly away from the side
you are inspecting.
Use tangential lighting and examine both sides of
the neck and find the internal jugular venous
pulsations.
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28. Look for pulsations in the suprasternal
notch, between the attachments of the
sternomastoid muscle on the sternum and
clavicle, or just posterior to the sternomastoid.
Identify the highest point of pulsation in the right
internal jugular vein.
Extend a long rectangular object or card
horizontally from this point and a centimeter ruler
vertically from the sternal angle, making an exact
right angle.
Measure the vertical distance in centimeters above
the sternal angle where the horizontal object
crosses the ruler.04/03/2011 menbit@ymail.com 28
32. This distance, measured in centimeters above the
sternal angle or the atrium, is the JVP.
normally Level of sternal angle is about 5 cm above
the level of mid right atrium and JVP is less than 8 cm
above right atrium.
Venous pressure measured at greater than 3 cm
above the sternal angle, or more than 8 cm in total
distance above the right atrium, is considered
elevated above normal.
Increased pressure suggests right sided heart failure
, constrictive pericarditis, tricuspid stenosis, or
superior vena cava obstruction.
Unilateral distention of the external jugular vein is
usually due to local kinking or obstruction.
Occasionally, even bilateral distention has a local
cause.04/03/2011 menbit@ymail.com 32
33. IJV VS CAROTID ARTERY PULSATION
INTERNAL JUGULAR VEIN
PULSATIONS
Rarely palpable
Soft, rapid,
Pulsations eliminated by light
pressure
Level of the pulsations
changes with
position, dropping as the
patient becomes more
upright.
Level of the pulsations usually
descends with inspiration.
CAROTID ARTERY PULSATIONS
Palpable
A more vigorous thrust with a
single outward component
Pulsations not eliminated by
pressure
Level of the pulsations
unchanged by position
Level of the pulsations not
affected by inspiration
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34. 3. THE CAROTID PULSE
provides valuable info.
useful for detecting stenosis or insufficiency of the
aortic valve.
pt. lay down with the head of the bed still elevated
to about 30°.
Then place your left index and middle fingers on the
right carotid artery in the lower third of the
neck, press posteriorly, and feel for pulsation.
Never press both carotids at the same time. This
may decrease blood flow to the brain and induce
syncope.04/03/2011 menbit@ymail.com 34
36. ASSESS:
I. amplitude
This correlate reasonably well with the pulse
pressure.
Small, thready, or weak pulse in cardiogenic shock;
bounding pulse in aortic insufficiency.
II. contour of the pulse wave, namely the speed of the
upstroke, the duration of its summit, and the speed
of the down stroke.
The normal upstroke is smooth, rapid, and follows
S1 almost immediately.
The down stroke is less abrupt than the upstroke.
Delayed carotid upstroke occurs in aortic stenosis
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37. III. BRUITS.
Detect thrills, that feel like the throat of a purring cat.
in the presence of a thrill, you should listen over both
carotid arteries with the diaphragm of your
stethoscope for a bruit, a murmur-like sound of
vascular rather than cardiac origin.
Ask the patient to hold breathing for a moment so that
breath sounds do not obscure the vascular sound.
A carotid bruit with or without a thrill in a middle-aged
or older person suggests but does not prove arterial
narrowing.
Note: An aortic murmur may radiate to the carotid
artery and sound like a bruit
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38. 4. HERATPositions used:
1. Supine, with the head elevated 30°
Inspect and palpate the precordium:
the 2nd interspaces;
the right ventricle;
the left ventricle,
the apical impulse (diameter, location, amplitude,
duration).
2. Left lateral decubitus
Palpate the apical impulse if not previously detected.
Listen at the apex with the bell of the stethoscope.
Used for Low-pitched extra sounds (S 3, opening snap,
diastolic rumble of mitral stenosis)
04/03/2011 menbit@ymail.com 38
39. Con…
3. Sitting, leaning forward, after full exhalation
Listen along the left sternal border and at the
apex.
Soft decrescendo diastolic murmur of aortic
insufficiency.
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40. INSPECTION AND PALPATION
Look at PMI. , aortic area , pulmonic area, and left
ventricular area.
the ventricular movements of a left-sided S3 or S4.
Then Palpate all the above areas .
Begin with general palpation of the chest wall.
First palpate for impulses using your fingerpads.
Hold them flat on the body surface, using light
pressure for an S3 or S4, and firmer pressure for S1
and S2.
Ventricular impulses may heave or lift your fingers.
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42. Thrills may accompany loud, harsh, or echoing
murmurs as in:
aortic stenosis
patent ductus arteriosus
ventricular septal defect, and
less commonly, mitral stenosis.
They are palpated more easily in patient positions
that accentuate the murmur.
at normal sized individual, s2,s3,s4,opening snap ,
systolic ejection click are not appreciated through
inspection and palpation
A palpable S2 suggests systemic hypertension.04/03/2011 menbit@ymail.com 42
44. Apical impulse characteristics
I. LOCATION.
Located usually in 5th interspaces 7-9 cm from the mid sternal
line
the apical impulse may be displaced upward and to the left by
pregnancy or a high left diaphragm.
Lateral displacement from cardiac enlargement in congestive
heart failure, cardiomyopathy, and ischemic heart disease.
Displacement in deformities of the thorax and mediastinal
shift.
II. DIAMETER.
In the supine patient, it usually measures less than 2.5 cm and
occupies only one interspace.
Note: In the left lateral decubitus position, a diameter greater
than 3 cm indicates left ventricular enlargement
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45. III. AMPLITUDE.
Estimate the amplitude of the impulse.
It is usually small and feels brisk and tapping.
Increased amplitude may also reflect hyperthyroidism, severe
anemia, pressure overload of the left ventricle (e.g., aortic
stenosis), or volume overload of the left ventricle (e.g., mitral
regurgitation)
IV. DURATION.
To assess duration, listen to the heart sounds as you feel the
apical impulse.
Normally it lasts through the first two thirds of systole, and
often less.
NOTE: A sustained, high-amplitude impulse that is normally
located suggests left ventricular hypertrophy from pressure
overload (as in hypertension).
A sustained low-amplitude (hypokinetic) impulse may indicate
dilated cardiomyopathy.
04/03/2011 menbit@ymail.com 45
47. PERCUSSION
In most cases, palpation has replaced percussion in
the estimation of cardiac size.
But When you cannot feel the apical
impulse, percussion may suggest where to search
for it.
percuss from resonance toward cardiac dullness in
the 3rd, 4th, 5th, and possibly 6th interspaces.
NOTE:
A markedly dilated failing heart may have a
hypokinetic apical impulse that is displaced far to
the left.
A large pericardial effusion may make the impulse
undetectable
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48. AUSCULTATION
AUSCULTTATION TIPS
The diaphragm is better for picking up the relatively
high-pitched sounds of S1 and S2, the murmurs of
aortic and mitral regurgitation, and pericardial friction
rubs.
Listen throughout the precordium with the diaphragm,
pressing it firmly against the chest.
The bell is more sensitive to the low-pitched sounds of
S3 and S4 and the murmur of mitral stenosis.
Apply the bell lightly, with just enough pressure to
produce an air seal with its full rim.
Low-pitched sounds such as S3 and S4 may disappear
with high pressure.04/03/2011 menbit@ymail.com 48
49. Ask the patient to roll partly onto the left side into
the left lateral decubitus position, bringing the left
ventricle close to the chest wall
This position accentuates or brings out a left-sided
S3 and S4 and mitral murmurs, especially mitral
stenosis. You may otherwise miss them.
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50. Ask the patient to sit up, lean forward, exhale
completely, and stop breathing in expiration.
This position accentuates or brings out aortic
murmurs resulted from aortic regurgitation.
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51. HEART MURMURS.
Are abnormal heart sounds
are longer than heart sounds
created by :
Restricted forward flow Of blood through stenotic
valve.
Backward Flow of blood through regurgitant valve
Abnormal opening in heart chambers
Over flow of blood through normal valves
innocently with any detectable cardiac structure
abnormality.
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52. GENERAL CLASSIFICATION
I. INNOCENT
murmur with no detectable physiologic disorder
II. PHYSIOLOGIC
related to demand supply disharmonization (over
flow) e.g. anemia, pregnancy ,fever etc.
III. PATHOLOGIC
as aresult of tangible cardiac disorder .e.g. valvular
lesions
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53. CHARACTERISTIC OF MUMURS
ALL MURMURS ARE DESCRIBED AS FOLLOWS:
TIMING
SHAPE
QUALITY
LOCATION
RADIATION
PITCH
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54. 1. TIMING
a systolic murmur, falling between S1 and S2, or a
diastolic murmur, falling between S2 and S1.
Murmurs that coincide with the carotid upstroke
are systolic.
Classified as:
systolic,
diastolic,
continuous
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55. SYSTOLIC MURMURS
I. A midsystolic murmur
Begins after S1 and stops before S2.
most often related to blood flow across the stenotic
semilunar (aortic and pulmonic) valves.
II. A pansystolic (holosystolic) murmur
Starts with S1 and stops at S2, without a gap between
murmur and heart sounds.
often occur with regurgitant (backward) flow across the
atrioventricular valves.
III. A late systolic murmur
Starts in mid- or late systole and persists up to S2
This is the murmur of mitral valve prolapse.04/03/2011 menbit@ymail.com 55
56. DIASTOLIC MURMURS
I. An early diastolic murmur
Starts right after S2, without a discernible gap, and
then usually fades into silence before the next S1.
related to incompetent semilunar valves.
II. A middiastolic murmur
Starts a short time after S2.
Related to turbulent flow of blood across the
atrioventricular valves.
III. A late diastolic (presystolic) murmur
Starts late in diastole and typically continues up to S1.
Related to turbulent flow of blood across the
atrioventricular valves.04/03/2011 menbit@ymail.com 56
57. CONTINUOUS MURMUR.
have both systolic and diastolic components.
starts in systole and continues without pause
through S2 into but not necessarily throughout
diastole.
related to patent ductus arteriosus , ventricvular
septal defect.
Note: like cardiac murmurs pericardial friction rubs
continues through both phases .04/03/2011 menbit@ymail.com 57
58. 2. SHAPE.
The shape or configuration of a murmur is determined
by its intensity over time.
I. crescendo (grows louder)
The presystolic murmur of mitral stenosis
II. decrescendo(grows softer)-
The early diastolic murmur of aortic regurgitation
III. crescendo-decrescendo(first rises in intensity, then
falls).
The midsystolic murmur of aortic stenosis and innocent
flow murmurs
IV. plateau(has the same intensity throughout).
The pansystolic murmur of mitral regurgitation
04/03/2011 menbit@ymail.com 58
59. 3. LOCATION OF MAXIMAL INTENSITY.
This is determined by the site where the murmur
originates.
For example, a murmur best heard in the 2nd right
interspace usually originates at or near the aortic
valve.
4. RADIATION
This reflects the intensity of the murmur.
Explore the area around a murmur and determine
where else you can hear it.
A loud murmur of aortic stenosis often radiates into
the neck (in the direction of arterial flow).
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60. 5. INTENSITY.
This is usually graded on a 6-point scale and expressed
as a fraction.
The numerator describes the intensity of the murmur
wherever it is loudest, and the denominator indicates
the scale you are using.
Intensity is influenced by the thickness of the chest wall
and the presence of intervening tissue.
An identical degree of turbulence would cause a louder
murmur in a thin person than in a very muscular or
obese one.
Emphysematous lungs may diminish the intensity of
murmurs.
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61. GRADING OF MURMURS
Grade 1 = Very faint, heard only after listener has
“tuned in”; may not be heard in all positions
Grade 2 = Quiet, but heard immediately after placing
the stethoscope on the chest
Grade 3 = moderately loud
Grade 4 = Loud, with palpable thrill
Grade 5 = Very loud, with thrill. May be heard when
the stethoscope is partly off the chest
Grade 6 = Very loud, with thrill. May be heard with
stethoscope entirely off the chest
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62. 6. PITCH
This is categorized as high, medium, or low.
7. QUALITY.
This is described in terms such as blowing, harsh,
echoing, and musical.
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