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The Diabetic
Foot in
Primary Care
Andre Sookdar
Class of 2013
Objectives
• Epidemiology
• Clinical Presentation
• Prevention
• Management
• Insulin Initiation
Definition
WHO
• The foot of a Diabetic Patient that has the
potential risk of pathologic consequences
including infection, ulceration and/or
destruction of deep tissues associated with
neurologic abnormalities, various degrees
of peripheral vascular disease and/or
metabolic complications of diabetes in the
lower limb.
Definition
Any foot pathology that results from Diabetes
or its long-term complications
(Boulton. 2002). Diabetes, 30 : 36, 2002
Epidemiology
• WHO estimates approx 60,000 persons in
T&T were diabetic in 2000
• Projected increase to 125,000 by 2030
• MOH estimates 1 in 5 adults are diabetic;
as much as 175,000
• 450 children with Type 1 DM
• More prevalent in the East Indian
community, but 33% of African attendees
of the public health services are both
Diabetic and Hypertensive
• Cause for about 25% Hospital Admissions
Epidemiology
• More than 450 non traumatic lower limb
amputations in 2010
• DM foot problems account for 14% of
admissions, 29% of bed occupancy
• 50% of persons who had lower limb
amputations develop depression; 20% die
within 2 years
• V Naraynsingh et al - 822 clinic patients
who had amputations between 2000-2004
reviewed; 515 (80%) due to DM
Risk Factors
• Age
• Duration of DM
>10yrs
• Gender M>W
• Poor glycemic
control
• Social situation and
support
• Obesity
• Alcohol
• Smoking
• Depression or
Mental illness
• Previous Ulcer
• Trauma
• Retinopathy
• Nephropathy
• Willful self neglect
Pathology
Neuropathy
• Sensory: lack of sensation  Repetitive
Trauma
• Motor: Changes in Foot anatomy 
Pressure Points
• Autonomic: Lack of sweat  Dry Skin
Distended veins  AV Shunting
Osteoarthropathy: Changes in foot structure
Charcot’s foot
Pathology
Callus: separates dermis  Ulcer Formation
Infection: Disruption of skin barrier, warmth
and moisture
Peripheral Vascular Disease: reduced blood
flow  decreased O2 supply  increased
risk of infection and poor healing
Diabetic Foot Assessment
• History
• Examination
• Investigations
• Risk Assessment
History
General Hx
• Medical Hx
• Surgical Hx
• Drug Hx
• Allergies
Foot History
• PC for Foot
• Neuropathic vs
Ischaemic Pain
• Daily activities &
use
• Foot Care
• Callus Formation
• Deformities
• Prev Surgeries
• Skin & Nail
Ulcer History
• Site, size, shape, duration, odor, type
• Precipitating event or Trauma
• Recurrence
• Infection
• Hospitalization & Treatment
• Wound Care
• Patient Compliance
• Previous Foot Trauma or Surgery
• ? Charcot’s Foot
Examination
• General Examination
• Inspection
• Palpation
• Neurological Assessment
• Footwear Assessment
General Examination
• Cardiovascular
• Respiratory
• Abdominal
• Eyes : Visual Acuity, Fundi
• Systemic Signs of Infection
Inspection
• Skin: dry, fissures, hair loss, dilated veins,
ulcers, bullae, fungal infections
Necrobiosis Lipoidica Diabeticorum,
Diabetic Dermopathy
Inspection
• Corns and Calluses
• Nails: Thickened or Atrophic, Ingrown
,Colour of nail bed, Discharge, Fungal
infections
• Oedema: poor fitting shoes, impedes
healing
Indicator of CV, Renal status, venous
insufficiency, infection, Gout, Trauma,
DVT, lymphoedema and many more
Inspection
• Deformity: abnormal pressure distribution
• Pes Cavus
• Fibrofatty padding depletion
• Hammer toes
• Claw Toes
• Hallux Valgus
• Charcot Foot
• Iatrogenic
Inspection
• Colour
• Red – Cellulitis, Critical ischaemia,
Osteomyelitis, Gout, Burn
• Blue – Cardiac Failure, Venous insuffiency
• Black – Necrosis, Ischaemia, Emboli,
Bruise, Melanoma, Henna, Dye
Palpation
• Pulses – Dorsalis Pedis, Posterior Tibial
Presence of either makes ischaemia
unlikely
If neither are present  Doppler
Dependent Rubor  PVD
• Temperature
Hot  Infection, Charcot, Bony or Soft
tissue Trauma, Gout, DVT
Cold  Ischaemia (acute and chronic),
Cardiac Failure
• Oedema
• Crepitus  Gas Gangrene
Neurologic Assessment
• Motor Neuropathy
• Classically, high medial longitudinal arch
 prominent metatarsal heads and
pressure points over the plantar forefoot
• Assess dorsiflexion for foot drop (common
peroneal nerve palsy)
• Autonomic Neuropathy
• Dry skin, fissures, distended veins
• Stocking distribution
Neurologic Assessment
• Sensory Neuropathy
• Monofilament test buckles @10g
• Vibration 128 Hz tuning fork
• Temperature
• Light Touch
• Pain
• Eyes closed, non-touch
Footwear Assessment
• Examine both
shoes and socks
• Length, breadth,
depth
• Heel height
• Lace/strap vs slip-
on
• Shoe lining
• Foreign bodies
• Wear and tear
• Snug fit, loose or
tight?
• What other shoes
does the patient
wear?
• Sock size, seams,
tightness, holes,
absorbency?
• Cardboard cutout
test
Investigations
• Laboratory
• Radiological
• Vascular
• Neurological
• Foot Pressures
Laboratory
• CBC
• RFT
• LFT
• RBS, HbA1C
• Lipid Profile
• CRP
• Wound Cultures
• Blood and Urine Cultures
Radiological
• Plain Films
• Osteomyelitis
• Fractures
• Dislocations
• Charcot foot
• Foreign Body
• Gas
• CT
• Technetium bone scan – early detection
• MRI – Soft tissue
Vascular
• Doppler; pulses, Ankle Brachial Pressure
Index
• <1  ischaemia
• Patients with arterial calcification 
elevated systolic pressure, hence the
pressure index may be >1 in spite of
ischaemia
• Investigate Popliteal and Femoral Arteries
Neurological
• Neurothesiometer
• Varying vibratory stimulus
Foot Pressures
• Plantar pressure measurement devices
• Ink and paper
Classification
• University of Texas Wound Classification
System of Diabetic Foot Ulcers
• Wagner
• Edmonds
Edmonds Classification
• Based on natural progression
• Stage 1: Normal or Low Risk Foot
• Stage 2: High-Risk Foot
• Stage 3: Ulcerated Foot
• Stage 4: Infected Foot
• Stage 5: Necrotic Foot
• Stage 6: Unsalvageable Foot
Edmonds Classification
• Stage 1 – The foot is not at risk
Sensation and pulses good
No deformities, calluses or swelling
• Stage 2 – One or more risk factors for
ulceration
Neuropathy and Ischaemia are the
main risk factors
Deformity, oedema and callus may not
lead to ulceration unless one or both of the
main risk factors are present
Edmonds Classification
• Stage 3 – Skin breakdown occurs usually
as an ulcer, but injuries such as grazes,
bruises and blisters can eventually
become ulcers
• Stage 4 – Infection can complicate both
the neuropathic and ischaemic foot
• Stage 5 – Necrosis can further lead to
tissue destruction
• Stage 6 – The foot cannot be saved
Edmonds Classification
Exceptions to this classification include
• Charcot’s foot
• Neuropathic fractures
• Painful neuropathy
Management
• Regular inspection and examination
• Multidisciplinary team
• Patient education
• Assess risk of foot
• Non ulcer pathology
• Ulcers and related pathology
Patient Education
• Optimum Glycemic control
• Management of co-morbid conditions
• Stop Smoking
• Warning signs
Foot Care
• Daily Routine and Inspection
• Between toes and below foot
• Nail Care: trim wet, straight across, proper
clippers (NO KNIVES)
• Skin Care: Moisture, Callus
• Footwear: Proper fit, clean
• Avoid excessive heat (Radiators, Hot
water, hot pitch)
• Avoid OTC Corn/Callus medications
• NEVER WALK BAREFOOT
Non Ulcer Pathology
• Calluses & Nails – Podiatrist
• Skin pathology
• Foot deformities – Surgical / Orthopedic
consult
Ulcer Pathology
• Treat the Cause(s) and co-morbid factors
• Psychosocial Factors
• Relief of mechanical pressure and protect
ulcer from stress
• Local Wound Care
• Treatment of Infection: Abx (Broad
Spectrum, multiple), Drainage,
Debridement
• Moisture control: Dressings
• Outpatient or Inpatient Care
Ulcer Pathology
Ulcer Pathology
• Low Threshold for Referral
• Stage 3 and above associated with poor
control
Conclusion
• Diabetic foot is a serious complication
• Associated with poor control
• Prevention requires vigilance and patient
education
• Treated by a multidisciplinary team
Thank You
• Questions?
References
1. Edmonds ME, Foster AVM, Sanders LJ.
A Practical Manual of Diabetic Foot Care
2nd Ed. Blackwell Publishing 2008
2. Radwan M. The Diabetic Foot: An
Overview [Internet] cited 1st June 2012
Available from:
www.mansdf.edu.eg/Videos_presentation
s/DF-overview.pdf
3. National Institute of Health. Feet can last
a Lifetime NIH and CDC. 2010

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The diabetic foot in primary care andre sookdar

  • 1. The Diabetic Foot in Primary Care Andre Sookdar Class of 2013
  • 2. Objectives • Epidemiology • Clinical Presentation • Prevention • Management • Insulin Initiation
  • 3. Definition WHO • The foot of a Diabetic Patient that has the potential risk of pathologic consequences including infection, ulceration and/or destruction of deep tissues associated with neurologic abnormalities, various degrees of peripheral vascular disease and/or metabolic complications of diabetes in the lower limb.
  • 4. Definition Any foot pathology that results from Diabetes or its long-term complications (Boulton. 2002). Diabetes, 30 : 36, 2002
  • 5. Epidemiology • WHO estimates approx 60,000 persons in T&T were diabetic in 2000 • Projected increase to 125,000 by 2030 • MOH estimates 1 in 5 adults are diabetic; as much as 175,000 • 450 children with Type 1 DM • More prevalent in the East Indian community, but 33% of African attendees of the public health services are both Diabetic and Hypertensive • Cause for about 25% Hospital Admissions
  • 6. Epidemiology • More than 450 non traumatic lower limb amputations in 2010 • DM foot problems account for 14% of admissions, 29% of bed occupancy • 50% of persons who had lower limb amputations develop depression; 20% die within 2 years • V Naraynsingh et al - 822 clinic patients who had amputations between 2000-2004 reviewed; 515 (80%) due to DM
  • 7. Risk Factors • Age • Duration of DM >10yrs • Gender M>W • Poor glycemic control • Social situation and support • Obesity • Alcohol • Smoking • Depression or Mental illness • Previous Ulcer • Trauma • Retinopathy • Nephropathy • Willful self neglect
  • 8. Pathology Neuropathy • Sensory: lack of sensation  Repetitive Trauma • Motor: Changes in Foot anatomy  Pressure Points • Autonomic: Lack of sweat  Dry Skin Distended veins  AV Shunting Osteoarthropathy: Changes in foot structure Charcot’s foot
  • 9. Pathology Callus: separates dermis  Ulcer Formation Infection: Disruption of skin barrier, warmth and moisture Peripheral Vascular Disease: reduced blood flow  decreased O2 supply  increased risk of infection and poor healing
  • 10.
  • 11. Diabetic Foot Assessment • History • Examination • Investigations • Risk Assessment
  • 12. History General Hx • Medical Hx • Surgical Hx • Drug Hx • Allergies Foot History • PC for Foot • Neuropathic vs Ischaemic Pain • Daily activities & use • Foot Care • Callus Formation • Deformities • Prev Surgeries • Skin & Nail
  • 13.
  • 14. Ulcer History • Site, size, shape, duration, odor, type • Precipitating event or Trauma • Recurrence • Infection • Hospitalization & Treatment • Wound Care • Patient Compliance • Previous Foot Trauma or Surgery • ? Charcot’s Foot
  • 15. Examination • General Examination • Inspection • Palpation • Neurological Assessment • Footwear Assessment
  • 16. General Examination • Cardiovascular • Respiratory • Abdominal • Eyes : Visual Acuity, Fundi • Systemic Signs of Infection
  • 17. Inspection • Skin: dry, fissures, hair loss, dilated veins, ulcers, bullae, fungal infections Necrobiosis Lipoidica Diabeticorum, Diabetic Dermopathy
  • 18. Inspection • Corns and Calluses • Nails: Thickened or Atrophic, Ingrown ,Colour of nail bed, Discharge, Fungal infections • Oedema: poor fitting shoes, impedes healing Indicator of CV, Renal status, venous insufficiency, infection, Gout, Trauma, DVT, lymphoedema and many more
  • 19.
  • 20.
  • 21. Inspection • Deformity: abnormal pressure distribution • Pes Cavus • Fibrofatty padding depletion • Hammer toes • Claw Toes • Hallux Valgus • Charcot Foot • Iatrogenic
  • 22.
  • 23.
  • 24. Inspection • Colour • Red – Cellulitis, Critical ischaemia, Osteomyelitis, Gout, Burn • Blue – Cardiac Failure, Venous insuffiency • Black – Necrosis, Ischaemia, Emboli, Bruise, Melanoma, Henna, Dye
  • 25.
  • 26. Palpation • Pulses – Dorsalis Pedis, Posterior Tibial Presence of either makes ischaemia unlikely If neither are present  Doppler Dependent Rubor  PVD • Temperature Hot  Infection, Charcot, Bony or Soft tissue Trauma, Gout, DVT Cold  Ischaemia (acute and chronic), Cardiac Failure • Oedema • Crepitus  Gas Gangrene
  • 27.
  • 28. Neurologic Assessment • Motor Neuropathy • Classically, high medial longitudinal arch  prominent metatarsal heads and pressure points over the plantar forefoot • Assess dorsiflexion for foot drop (common peroneal nerve palsy) • Autonomic Neuropathy • Dry skin, fissures, distended veins • Stocking distribution
  • 29. Neurologic Assessment • Sensory Neuropathy • Monofilament test buckles @10g • Vibration 128 Hz tuning fork • Temperature • Light Touch • Pain • Eyes closed, non-touch
  • 30.
  • 31. Footwear Assessment • Examine both shoes and socks • Length, breadth, depth • Heel height • Lace/strap vs slip- on • Shoe lining • Foreign bodies • Wear and tear • Snug fit, loose or tight? • What other shoes does the patient wear? • Sock size, seams, tightness, holes, absorbency? • Cardboard cutout test
  • 32. Investigations • Laboratory • Radiological • Vascular • Neurological • Foot Pressures
  • 33. Laboratory • CBC • RFT • LFT • RBS, HbA1C • Lipid Profile • CRP • Wound Cultures • Blood and Urine Cultures
  • 34. Radiological • Plain Films • Osteomyelitis • Fractures • Dislocations • Charcot foot • Foreign Body • Gas • CT • Technetium bone scan – early detection • MRI – Soft tissue
  • 35.
  • 36. Vascular • Doppler; pulses, Ankle Brachial Pressure Index • <1  ischaemia • Patients with arterial calcification  elevated systolic pressure, hence the pressure index may be >1 in spite of ischaemia • Investigate Popliteal and Femoral Arteries
  • 38. Foot Pressures • Plantar pressure measurement devices • Ink and paper
  • 39.
  • 40. Classification • University of Texas Wound Classification System of Diabetic Foot Ulcers • Wagner • Edmonds
  • 41. Edmonds Classification • Based on natural progression • Stage 1: Normal or Low Risk Foot • Stage 2: High-Risk Foot • Stage 3: Ulcerated Foot • Stage 4: Infected Foot • Stage 5: Necrotic Foot • Stage 6: Unsalvageable Foot
  • 42. Edmonds Classification • Stage 1 – The foot is not at risk Sensation and pulses good No deformities, calluses or swelling • Stage 2 – One or more risk factors for ulceration Neuropathy and Ischaemia are the main risk factors Deformity, oedema and callus may not lead to ulceration unless one or both of the main risk factors are present
  • 43. Edmonds Classification • Stage 3 – Skin breakdown occurs usually as an ulcer, but injuries such as grazes, bruises and blisters can eventually become ulcers • Stage 4 – Infection can complicate both the neuropathic and ischaemic foot • Stage 5 – Necrosis can further lead to tissue destruction • Stage 6 – The foot cannot be saved
  • 44. Edmonds Classification Exceptions to this classification include • Charcot’s foot • Neuropathic fractures • Painful neuropathy
  • 45. Management • Regular inspection and examination • Multidisciplinary team • Patient education • Assess risk of foot • Non ulcer pathology • Ulcers and related pathology
  • 46. Patient Education • Optimum Glycemic control • Management of co-morbid conditions • Stop Smoking • Warning signs
  • 47. Foot Care • Daily Routine and Inspection • Between toes and below foot • Nail Care: trim wet, straight across, proper clippers (NO KNIVES) • Skin Care: Moisture, Callus • Footwear: Proper fit, clean • Avoid excessive heat (Radiators, Hot water, hot pitch) • Avoid OTC Corn/Callus medications • NEVER WALK BAREFOOT
  • 48.
  • 49. Non Ulcer Pathology • Calluses & Nails – Podiatrist • Skin pathology • Foot deformities – Surgical / Orthopedic consult
  • 50. Ulcer Pathology • Treat the Cause(s) and co-morbid factors • Psychosocial Factors • Relief of mechanical pressure and protect ulcer from stress • Local Wound Care • Treatment of Infection: Abx (Broad Spectrum, multiple), Drainage, Debridement • Moisture control: Dressings • Outpatient or Inpatient Care
  • 51.
  • 53. Ulcer Pathology • Low Threshold for Referral • Stage 3 and above associated with poor control
  • 54.
  • 55. Conclusion • Diabetic foot is a serious complication • Associated with poor control • Prevention requires vigilance and patient education • Treated by a multidisciplinary team
  • 57. References 1. Edmonds ME, Foster AVM, Sanders LJ. A Practical Manual of Diabetic Foot Care 2nd Ed. Blackwell Publishing 2008 2. Radwan M. The Diabetic Foot: An Overview [Internet] cited 1st June 2012 Available from: www.mansdf.edu.eg/Videos_presentation s/DF-overview.pdf 3. National Institute of Health. Feet can last a Lifetime NIH and CDC. 2010