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Francisco J. Chacón-Lozsán
MD student UCLA-Venezuela
European Society of Cardiology:
Heart Failure Association
Acute Cardiovascular Care Association
LinkedIn: http://ve.linkedin.com/in/chaconlozsanfrancisco
2013
Heart Failure (HF) Is a clinic condition which
the cardiac output in not adequate to supply
the tissue needs.
Framingham
Major Criteria

Minor Criteria

Major and minor criteria

•Orthopnea or nocturnal
paroxysmal dyspnea.
•Neck veins distension.
•Crackles.
•Cardiomegaly.
•Acute pulmonary edema.
•3 Cardiac murmur.
•CVP >6cmH2O
•Hepatic-Jugular reflux.

•Lower extremities
bilateral edema, nocturnal
cough or efforts dyspnea.
•Hepatomegaly
•Pulmonary vital capacity
reduced 50%
•HR>120/min

•Weigh loss >4,5Kg with
treatment.
AHA functional stratification of HF
Clinic presentation

Characteristics

Objectives

SBP >160mmHg

Pulmonary congestion without
systemic congestion. Many
with Ejection Fraction (EF)
preserved.

Objective: Volume
management. BP control.
Therapy: Vasodilator and loop
diuretics.

Normal BP or moderate high
BP (>160mmHg).

Gradual depression associated
to systemic congestion.
Radiologic pulmonary
congestion in patients with
advanced HF.

Objective: Volume
management.
Therapy: Vasodilator with or
without loop diuretics.

Low BP (>90mmHg)

Related to low cardiac output
with depression of renal
function.

Objective: Cardiac output.
Therapy: Cardiac inotropic
with vasodilator properties,
consider digoxin, vasodilators
and mechanical assistance.

Cardiogenic Shock.

Fast, complicated with MI, fast
myocarditis, acute valvular
disease.

Objective: Rise pump
function.
Therapy: vasoactive drugs,
Inotropic and mechanical
assistance.
Presentación clínica

Características

Objetivos

Acute pulmonary edema.

Abrupt, impaired by severe
hyperventilation. Patient
responses fast to vasodilators
and diuretics.

Objective: Volume
management.
Therapy: Vasodilators,
diuretics, ventilation,
morphine.

ACS with acute HF

Many patients have sings and
symptoms of HF that get better
resolving ischemia.

Objectives: Thrombolysis,
plaque stabilization, ischemic
correction.
Therapy: Reperfusion by PCI,
lysis, nitrates, antipatelet
agents.

Isolated Right HF IC or
intrinsic RV failure or valvular
disease.

Rapid of gradual, primary or
secondary to HBP or RV
pathology.

Objective: BP management.
Therapy: Nitrates,
phosphodiesterase inhibitors,
endoteline inhibitors, RV MI
reperfusion, valvular surgery.

HF post cardiac surgery.

Can be caused by inadequate
myocardial protection resulting
in cardiac damage.

Objective: Volume
management, rise CO.
Therapy: Use diuretic or
fluids, inotropic, mechanical
assistance.
ACE inhibitor (candesartan preferably)

+Beta-Blocker

If NYHA II-IV add MRA (spironolactone)

+Ivabradine (If using BB HR>70/min)

Still NYHA II-IV: Consider Pacemaker
• If QRS > 0,12sec use resynchronization.
• If QRS < 0,12sec use Automatic Implantable Defibrillator.

If still NYHA II-IV add Digoxin.
Heart failure quick guide 2013
Heart failure quick guide 2013
Heart failure quick guide 2013
Heart failure quick guide 2013
Heart failure quick guide 2013
Heart failure quick guide 2013
Heart failure quick guide 2013
Heart failure quick guide 2013

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Heart failure quick guide 2013

  • 1. Francisco J. Chacón-Lozsán MD student UCLA-Venezuela European Society of Cardiology: Heart Failure Association Acute Cardiovascular Care Association LinkedIn: http://ve.linkedin.com/in/chaconlozsanfrancisco 2013
  • 2. Heart Failure (HF) Is a clinic condition which the cardiac output in not adequate to supply the tissue needs.
  • 3. Framingham Major Criteria Minor Criteria Major and minor criteria •Orthopnea or nocturnal paroxysmal dyspnea. •Neck veins distension. •Crackles. •Cardiomegaly. •Acute pulmonary edema. •3 Cardiac murmur. •CVP >6cmH2O •Hepatic-Jugular reflux. •Lower extremities bilateral edema, nocturnal cough or efforts dyspnea. •Hepatomegaly •Pulmonary vital capacity reduced 50% •HR>120/min •Weigh loss >4,5Kg with treatment.
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  • 6. Clinic presentation Characteristics Objectives SBP >160mmHg Pulmonary congestion without systemic congestion. Many with Ejection Fraction (EF) preserved. Objective: Volume management. BP control. Therapy: Vasodilator and loop diuretics. Normal BP or moderate high BP (>160mmHg). Gradual depression associated to systemic congestion. Radiologic pulmonary congestion in patients with advanced HF. Objective: Volume management. Therapy: Vasodilator with or without loop diuretics. Low BP (>90mmHg) Related to low cardiac output with depression of renal function. Objective: Cardiac output. Therapy: Cardiac inotropic with vasodilator properties, consider digoxin, vasodilators and mechanical assistance. Cardiogenic Shock. Fast, complicated with MI, fast myocarditis, acute valvular disease. Objective: Rise pump function. Therapy: vasoactive drugs, Inotropic and mechanical assistance.
  • 7. Presentación clínica Características Objetivos Acute pulmonary edema. Abrupt, impaired by severe hyperventilation. Patient responses fast to vasodilators and diuretics. Objective: Volume management. Therapy: Vasodilators, diuretics, ventilation, morphine. ACS with acute HF Many patients have sings and symptoms of HF that get better resolving ischemia. Objectives: Thrombolysis, plaque stabilization, ischemic correction. Therapy: Reperfusion by PCI, lysis, nitrates, antipatelet agents. Isolated Right HF IC or intrinsic RV failure or valvular disease. Rapid of gradual, primary or secondary to HBP or RV pathology. Objective: BP management. Therapy: Nitrates, phosphodiesterase inhibitors, endoteline inhibitors, RV MI reperfusion, valvular surgery. HF post cardiac surgery. Can be caused by inadequate myocardial protection resulting in cardiac damage. Objective: Volume management, rise CO. Therapy: Use diuretic or fluids, inotropic, mechanical assistance.
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  • 11. ACE inhibitor (candesartan preferably) +Beta-Blocker If NYHA II-IV add MRA (spironolactone) +Ivabradine (If using BB HR>70/min) Still NYHA II-IV: Consider Pacemaker • If QRS > 0,12sec use resynchronization. • If QRS < 0,12sec use Automatic Implantable Defibrillator. If still NYHA II-IV add Digoxin.