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SENSORY DISORDERS
Nelia B. Perez RN, MSN
PCU – MJCN
Class 2015
CONTENTS
• Review Of Anatomy and Physiology
• Common Sensory Disorders
Ears
Anatomy

•The ear is responsible for
hearing and balance
•Consists of 3 regions
• External ear
• Middle ear
• Inner ear
Structure and Function
• External Ear
> auricle/pinna
- movable cartilage covered with skin
- Mastoid process= important Landmark
External Auditory Canal
- S-shaped pathway leading to the ME
- 2.5 to 3 cm. long in adult
- Its skeleton of bone and
cartilage is covered with
sensitive skin ( outer 1/3 is
cartilage, inner 2/3 consists of
bone)
- This canal lining is protected and
lubricated with cerumen
- Lymphatic drainage of
the external ear flows
into parotid , mastoid,
superficial cervical
nodes
MIDDLE EAR

air filled cavity in the
temporal bone
- >It contains the ossicles
( malleus, incus,stapes) that
transmit sound from the TM to
the oval window of the inner
ear
>
MIDDLE EAR

>Tympanic membrane (eardrum)
separates external and middle
ear.
• Translucent membrane
• Pearly, gray color
• Cone of light reflection when using
otoscope
• Oval and slightly concave shape,
pulled in at center by malleus
Middle ear

>Openings to

Outer ear covered by tympanic
membrane
Inner ear = oval and round
windows
Eustachian tube connects middle
ear to the nasopharnyx for air
passage (normally closed, opens
with swallowing/yawning)
Middle ear has 3 functions

1. Conducts sound vibration from
outer ear to inner ear
2. Protects the inner ear by
reducing the amplitude of loud
sounds
3. Eustachian tube allows
equalization of air pressure on
each side of the ear drum to
avoid rupture ( high altitudes)
Inner Ear

• Contains the Bony Labyrinth
which holds the sensory
organs for hearing and
equilibrium
1. Vestibule
2. Semicircular canals
3. Cochlea (contains the central
hearing apparatus)
Function of hearing
•

3 levels

1. Peripheral
> ear transmits sound and converts its
vibrations into electrical impulses
> The electrical impulses are conducted
by the auditory process of cranial nerve
VIII (Acoustic) to the brain stem
1. Amplitude=loudness
2. Frequency=pitch
Sound waves cause the eardrum
to vibrate
> Vibrations travel via the ossicles
thru the oval window > the
cochlea > to the round window
where they are dissipated
Vibrations in the basilar
membrane of the cochlea that
contain the organ of Corti
receptor hair cells > translate
the vibrations to electric
impulses
> The stimulated impulses go to
the brainstem via Acoustic nerve
(VIII)
2. Brain stem
- permits identification of sound
and locating the direction of a
sound in space.
- Sensitive to intensity and timing
from the ears
depending on head position
3. Cerebral cortex
- Intreprets the meaning of the
sound and begins the
appropriate response
Pathways of hearing

1. Air conduction (AC)– normal
pathway of hearing, the most
efficient
2. Bone conduction (BC)– bones
of the skull vibrate and
transmit vibrations to the
inner ear and acoustic nerve
Physical Examination
• The Auricle
1) inspect each auricle for size ,
shape, symmetry, color, position on
the head, deformities, nodules and
lesions
2) If ear pain, discharge or
inflammation is
present, move the auricle up and
down
3) Note tenderness of

pinna and mastoid area.
Press the tragus and
press firmly behind the
ear
Physical Examination
• Auricle
-Extends slightly outward from the skull
- Positioned in a nearly vertical plane
- The origin of the helix should be on a
horizontal line with corner of the eye
- It should have the same color as the
facial skin w/o moles, cysts & other
lesions
Otoscopic Exam
1) Tip the patient’s head to the
opposite side
2)Grasp the auricle firmly but gently,
while pulling it upward, backward
and slightly outward
3)Insert into the canal, sl down and
forward, the largest ear speculum
that the canal will accommodate
4) Observe the ff:
- patency of the ear canal
- describe the walls of the ear
canal. Note
any redness or swelling
- identify any discharge,
presence of cerumen or FB in the
ear canal
- tympanic membrane
Inspect using Otoscope

• External canal
• Color
• Swelling
• Lesions
• Discharge ; color and odor.
Clean or change speculum
before examining other ear.
Tympanic membrane

• Color – normal is shiny,
translucent, pearl-grey
• Landmarks ( umbo, handle of
malleus, light reflex)
• Position – flat, slightly pulled in at
the center and flutters when
person holds nose and swallows
• Integrity of membrane – intact
•Perform the
otoscope exam prior
to hearing tests.
Hearing Evaluation

1.Rough quantitative
test for hearing loss
2.Whisper test
3.Tuning fork
• Rough quantitative test for hearing
loss
- begins when the patient responds
to your questions and directions.
The patient responds without
excessive requests for repetition
- Speech with a monotonous tone and
erratic volume may indicate hearing
loss
WHISPER TEST

•Begins with the historyConversational tone
•The following tests may
indicate the presence of
hearing loss but not the
degree.
• Place your mouth at the side of
the patient’s head ( 2 ft.) from
her ear with the far ear covered
• Whisper test questions that can’t
be answered by yes or no
• Test consistently with loud,
medium and soft tones
• Repeat on the opposite ear
using another word, have the
client identify the words (Used
to detect high-tone loss)
• Normal Response to Voice test
• Correct identification of
whispered words bilaterally
TUNING FORK TESTS

• Measure hearing by air conduction
and bone conduction
• Frequency of fork is 256-1024
cycles/sec.
• To activate the tuning fork, hold
it by the stem and strike the tines
softly on the back of the hand
TUNING FORK TEST

• Weber test
> used when hearing is reported
as better in one ear than the
other ( bone conduction)
> with normal neurosensory
hearing and no conductive loss,
the sounds are equal in both
ears
> lateralization of the sound
to one ear indicates a
conductive loss on the same
side or a perceptive
loss/sensorineural loss on
the other side
• Weber Test
•

Rinne test – compares bone
conduction and air
conduction

1. Normally sound is heard 2X as
long by air conduction as by
bone conduction
2. Normal response ; positive
Rinne Test = AC>BC Bilaterally
Sound is heard longer by BC with
a conductive loss.
• Rinne Test
Weber test

Rinne test
Summary of any symptom should include
PQRSTU

• P= provocative or palliative
• Q= quality or quantity
• R= region or radiation
• S= severity scale
• T= timing (onset, duration,
frequency)
Subjective data

• Earaches
• Tinnitus
• Vertigo
• Dizziness
• Discharge
• Hearing loss
HISTORY
Always ask the following:
• Tinnitus –ringing in the ears
causes:
a.Outer ear- cerumen, foreign body,polyp
in the external auditory canal
b. Middle ear – inflammation ,otosclerosis
c. Internal ear- fever, suppuration of the
labyrinth, SY,acoustic nerve
tumor
internal ear – fracture at the
base of the skull, meniere
syndrome
d.Drugs
quinine, salicylates,
aminoglycosides, gentamicin
•Ear pain ( Otalgia )
- pain may arise from
inflammation of structure in
the ear or be referred from
other pharyngeal sites
including the thyroid
Causes:
Auricletrauma,hematoma,frostbite,burn,
eczema,
lnsect bites, impetigo, herpes
zoster
External auditory canalotitis externa ,carbuncle,
eczema, hard cerumen, FB,
herpes zoster
Middle earacute otits media, acute
mastoiditis
Referred pain- unerrupted lower
third molar, carious
teeth, tonsillitis, carcinoma of
pharynx, trigeminal neuralgia ,
subacute thyroiditis
• Dizziness
- patient has a sense of disturbed
relation to space
- described as being unsteady, weak,
light headed or having the feeling of
turning
Causes:
Endocrine
hypothyroidism,pregnancy,
hypoparathyroidism
Idiopathic
multisystem atrophy
Infectious
tabes dorsalis, meningitis,
encephalitis, brain abscess
Metabolic/ nutritional
pellagra, Vit.B12 def.,fluid &
electrolyte imbalance
Mechanical/trauma
skull fracture, otosclerosis, eye muscle imbalance
glaucoma
Neoplastic
Brain tumors
Neurologic
migraine, peripheral neuropathy
Psychosocial
anxiety disorder
Vascular
hypertension, orthostatic hypotension
• Vertigo
- persistent stimulation of the semicircular
canals or vestibular nucleus when the
head is at rest
- It gives a hallucination of motion
- When the eyes open, the pts.surrounding
seems to be whirling or spinning
- When the eyes closed, the pt.continues to feel in motion
Causes:
Peripheral labyrinthine System
- otitis media with effusion,
otosclerosis,
temporal bone fracture
Central labyrinthine system
- migraine, cerebellar
hemorrhage, intracranial abscess
Cranial V111 infections
- Acute meningitis,
tuberculous meningitis,
tumors
Brainstem nuclei
- encephalitis, brain abscess,
hemorrhage, multiple
sclerosis
• Hearing loss
a. Conductive- seen in people with
external or middle ear problem
Causes:
-obstruction of external auditory canal
(FB, impacted cerumen)
- Disorder of the eardrum & middle ear
( perforated TM, pus/blood in the ME )
- Overgrowth of bone with fixation of
the stapes ((Otosclerosis)
b. Sensorineural hearing loss ( Perceptive)
- involves the inner ear
Causes:
- disorders of the cochlea or the acoustic nerve (CN 8)
- Aging ( Presbycusis ) due to nerve degeneration
- Trauma
- Drug toxicity
- Tumors
- infections
- Heredity/congenital deafness
EAR SIGNS
• EXTERNAL EAR
a) Malformations of the Pinna
microtia – smaller than normal
macrotia – unusually large
lop or bat ear- pinna may protude
at R angle
aztec or cagot ear – failure of
development of the lobule
Macrotia or large ear
Before Surgery

After Surgery
Before Surgery

After surgery
Lop or Bat ear
- pinna may protrude
at right angle
Lop or Bat Ears
satyr ear- pointed pinna
cauliflower ear- untreated
hematomas heal as nodular and
bulbous irregularities of the helix
and and antihelix
- result of blunt trauma and
necrosis of the underlying
cartilage
Cauliflower Ears
b)

Pinna nodule
Darwin tubercle- harmless developmental
eminence in the upper 3rd
of the posterior helix
Gouty tophus – small, whitish uric acid
crystals along the
peripheral margins of the
auricles, olecranon bursa,
tendon sheaths
- nodules are painless hard,
and irregular
Gouty deposits
b)External acoustic meatus
Cerumen Impaction
- due to excessive production of
wax or a narrowed meatus leads
to partial or complete obstruction
of the canal
- complete obstruction leads to
partial deafness acc. by tinnitus
or dizziness
Otorrhea( ear discharge)
yellow discharge- melted cerumen
serous discharge- eczema in the meatal
wall, early ruptured acute OM
bloody discharge- temporal bone fracture
purulent discharge- chronic external otitis,
chronic suppurative OM,
cholesteatoma, TB, polyps
Foreign body
Insect invaders
Polyps
Furuncle
• Tympanic membrane
Retracted Tympanic membrane
:
- Seen in Serous Otitis media
- more concave TM
- accentuated bony landmarks
- distorted light reflex
Normal Tympanic Membrane

Retracted Tympanic Membrane
Bulging Tympanic membrane:
- seen in Acute suppurative
otitis media
- more conical
- loss of bony landmarks
- distorted light reflex
Normal Tympanic Membrane

Bulging Tympanic Membrane
Perforated Tympanic
membrane:
- previous suppurative middle
ear infection has eroded thru
the membrane producing
holes
- perforation appears as oval
holes thru which the darkened
middle ear cavity is seen
Perforated Tympanic Membrane
Perforated Tympanic Membrane
COMMON DISORDERS OF THE EAR
• Otitis Externa
a) Acute external otitis
-due to Ps.aeruginosa, staph, strep, proteus
- pain maybe mild or severe accentuated by
movement of the pinna
- swimmers’ ear
- preauricular, postauricular , Ant cervical LN
b) Chronic external otitis
- commonly due to bacteria
and fungal
- pruritus is the main
complain instead of pain
- aural discharge maybe
present
• Otitis Media
a) Chronic suppurative otitis media
- ass. with permanent
perforation of the eardrum
-hearing is always impaired
- painless aural discharge
- pain and vertigo indicates
development of complications
like brain abscess
b) Cholesteatoma
- collection of desquamated
epithelial cells in the middle ear
- foul smelling discharge, marginal
perforation,hearing loss, pearly
gray mass
superior part of tympanic
membrane
- eustachian tube dysfunction causes
retraction of tympanic membrane
• Vertiginous disorder
a) Acute Labyrinthitis
- most frequent cause of vertigo
- patient gradually develop a sense
of whirling that reaches a climax in
24-48 hrs. disappear gradually in 36 wks.
- N/V may occur at the height of
symptoms
- no accompanying tinnitus or hearing
loss
b) Benign

Paroxysmal positional Vertigo
(BPPV)
- Calcium deposits in the labyrinth
( otoliths)
are dislodged and move in response
to gravity eliciting a feeling of
motion
- More common in older individuals
- Sudden onset, often when rolling
over in bed or arising in the morning
- No headaches/fever but with nausea
and inability to stand
- Avoid any head motion to lessen
symptoms
Thank You
Nose, Throat and Mouth
Nose

• First segment of the
respiratory system
• Warms, moistens and filters
inhaled air
• Sensory organ for smell
• Resonance of laryngeal sound
External parts
• Bridge – frontal and maxillary bones
• Tip
• Nares – anterior openings of the nos
• Columella - divides the nares
• Ala nasi –lateral outside wing of the
nose bilaterally
• Upper 1/3 nose is bone; rest is
cartilage
Internal

• Nasal cavity
-floor of the nose ( hard and soft
palate)
- roof of the nose ( frontal and
sphenoid bone)
• Nasal hair
• Nasal Septum-divides cavity into
2 passages
• Nasal turbinates
Internal

• Superior, middle, inferior
turbinates- 3 parallel bony
projections on lateral walls of
each cavity
• Meatus- cleft/ groove
underlying each turbinate.
•Inspired air enters thru the
nares > passes thru the
vestibule> to the choanae
which are posterior
openings > leading to the
nasopharynx
Internal
• Olfactory receptors
- roof of the nasal cavity & upper part of septum above
the superior turbinate.
-merge into the olfactory nerve (I) > goes to the
temporal lobe of the brain
• Kiesselbach plexus
- a vascular network located superficially on the anterior
superior portion of the septum
- site of most anterior nosebleeds
SINUSES
•

Paranasal sinuses
- air-filled paired extensions of the nasal cavities
within the bones of the skull
- lined with mucous membranes and cilia that move
secretions along excretory pathways
- sinus openings are narrow, susceptible to occlusion>
resulting in inflammation /sinusitis.
- drained into the medial meatus
• Purpose
• Serve as resonators for sound
• Provide mucous for the nasal
cavity
Types:
1. Frontal sinuses
2. Maxillary sinuses
3. Ethmoid sinuses
4. Sphenoid sinuses
Frontal & Maxillary sinuses are
accessible to examination
Physical Examination
• Nose – Inspect and
palpate
• INSPECT for:
•
•
•
•
•
•

Symmetry, deformity
Inflammation
Skin lesions
Color
Nasal flaring
discharges
• Palpate
- ridge & soft tissues of the nose
- note any displacement of the
bone,
cartilage
- note for tenderness & any mass
- The nasal structures should be
firm and stable to palpation
- if with injury, palpate gently
•Test for sense of smell (CN
1)
•Evaluate the patency of the
nose
- nasal breathing should be
noiseless and easy thru the
open nares
Nasal Cavity
 Use the nasal speculum and good
light source to inspect the nasal
cavity
a) Nasal mucosa
- inspect for color, discharge,
lesions, masses
- it should appear deep pink
( pinker than the buccal mucosa) &
glistening
b) nasal septum
- In normal adult, the nasal
septum is seldom precisely a
midline structure
- No perforations, bleeding or
crusting should be apparent
- a film of clear discharge is
often apparent on the nasal
septum
c) Nasal Turbinates
- only the inferior and
middle turbinates will be
visible
- it should be the same
color as the surrounding
area and have a firm
consistency
• Paranasal Sinuses: Inspect and
Palpate
• Press thumbs over frontal &
maxillary sinuses ( palpate the
cheeks and supraorbital ridges)
• No tenderness or swelling over the
soft tissue should be present
•Transillumination test
a) Frontal & Maxillary sinuses
b) nasal septum
- Best perform in a dark room
- Look for a bright light in the
supraorbital ridge
and maxilla
- Look for deviation, perforation,
masses in the
transilluminated septum
SYMPTOMS

• Loss of smell ( anosmia )
- lesion of CN 1 or nasal
obstruction
- commonly due to closed head
trauma
- invariably accompanied by a
perceived change in taste of food
( bland & unpalatable)
• Abnormal smell/ taste
(dysgeusia)
- this is a common complaint in
patients who have loss of smell
- if it is paroxysmal and
associated with behavioral
symptoms, it suggests complex
partial seizures
SIGNS

SKIN
LESIONS
Basal Cell Carcinoma
SIGNS
• Discharge
- Describe discharge as to its
character
( watery, mucoid, purulent ,
bloody)

- color ( greenish, whitish, bloody)
- bilateral or unilateral
•
•

Running Nose
. 1.Unilateral
- Choanal atresia
- Foreign body- foul purulent
discharge
- neoplasm – bloody discharge
- Head injury or surgery – clear spinal
fluid
2. Bilateral
- allergy
- infection ( upper respiratory)
Foreign Body
. Unilateral
- Choanal atresia
- Foreign body
- neoplasm
- Head injury or surgery
• Epistaxis ( nosebleed)
-Kiesselbach plexus – most
common site of bleeding
anteriorly
- Back 3rd of the Inferior
Meatus – most common site
posteriorly
Causes:
1. Local
- coughing
- sneezing
- nose pricking
- fracture
- foreign bodies
2. Generalized
- Congenital – hereditary
telangiectasia
- inflammatory/immune – wegener
granulomatosis
- infectious – typhoid fever, dengue,
diphtheria
- Metabolic/toxic – aspirin, scurvy
- Mechanical – change in atmospheric
pressure ( mountain climbing, flying),
exertion
- Neoplastic – nasopharyngeal Ca
leukemia
- vascular- hemophilia,
thrombocytopeni
- trauma- nasal and maxillary fracture
- Elevated venous pressure- Cor
pulmonale
Congestive Heart failure
- Elevated arterial pressure – HPN,
coarctation of aorta
• Nasal septum
a) Deviation
- the cartilagenous and bony septum
may deviate as a hump, spur, shelf to
enroach on one nasal chamber
occlusion causing obstruction
b) Perforation
- a hole in the nasal septum
(transillumination test) is
commonly caused by chronic
infection, nasal surgery,
repeated trauma in picking off
crusts,
cocaine abuse
- rarely due to SY, TB
Nasal Septum Perforation
Nasal Syndromes
• Acute Rhinitis ( infectious) ( common
cold)
- Rhinoviruses infect the mucous
membranes of the nose & sinuses
causing inflammation and inc. nasal
secretions
- Watery nasal discharge, sneezing,
discharge becomes purulent acc. by
fever and body malaise
-Symptoms 3-10 days
-Severe local pain suggest a
complication-bacterial
sinusitis
• Allergic rhinosinusitis
- itching of the nose & eyes,
rhinorrhea, lacrimation,
sneezing
- headache is common
- maybe seasonal or perennial
- common allergens are pollens,
molds, house dust, mites,
coachroach, animal danders
• Vasomotor Rhinitis
- nonallergic mucosal edema and
rhinorrhea ass. with vasodilatation of the
nasal vessels, mucosal edema & inc.
mucous production
- due to chronic environmental irritants
( dust , smoke, strong odor, cold air),
pregnancy, estrogens, progesterone
• Suppurative Paranasal Sinusitis
- due to Strep. pneumonia, H.
influenza
- severe pain in the face occuring 714 days after signs & symptoms of an
acute URTI
- pain & pressure without fever
suggest sinus obstruction requiring
decongestants
• Cavernous Sinus Thrombosis
-This is the most feared
complication of nasal
infections. It can cause
blindness or death
- Infection spreads from the
nose>angular veins> cavernous
sinus> septic thrombosis
-patient

eyes

complains of pain deep in the

- Both eyes are involved,
immobilization of the globes,
periorbital edema, chemosis
- May involve CN 3,4, &6
- Sudden chills, high fever, prostated,
comatose, death within 2-3 days
THANK YOU
THANK YOU
Mouth
• First segment of the digestive
system
• Airway for the respiratory system
• ORAL CAVITY
• Lips
• Palate

1. Hard
2. Soft
3. Uvula – hangs down from the soft
palate
• Cheeks- side walls of cavity
• Tongue
1. Papillae- rough, bumpy elevations
on dorsal
2. Frenulum
3. Taste buds

• Teeth – 32 permanent
• Salivary glands
1. Parotid- largest of the glands,
located in the cheeks, front of
the ear. Stenson’s duct opens in
buccal mucosa
2. Submandibular- walnut size,
beneath the mandible at the
angle of the jaw. Wharton’s duct
either side of the frenulum
3. Sublingual –smallest, almond
shape, under tongue
Throat
 Area behind the mouth & nose
 Oropharynx – separated from the
mouth by a fold of tissue on each
side called anterior tonsillar pillars
 Tonsils – lymphoid tissue behind
pillars
• Posterior pharyngeal wall located
behind the tonsils
• Nasopharynx continues from the
oropharynx but it is above it and
behind the nasal cavity.
-It holds the adenoids and the
eustachian tube openings.
Physical Examination
• Preparation for examination
a) Face the patient with both of
you seated at the same level
b) Remove any dentures to see
the mucosa underneath
c) Hold the tongue blade in the
left hand and penlight in the right
hand
d) A good light source is needed
INSPECT AND PALPATE

Use gloves, tongue depressor, light
• Lips
• Teeth
• Gums
• Tongue
• Buccal mucosa
• Mouth ( roof and floor of the
mouth)
• Lips
- remove lipstick
- should be pink , smooth surface,
free of lesions.
- distinct border between the lips
and facial skin should not be
interrupted by lesions
- Vertical and horizontal symmetry
both at rest and with movements
Rest
r
e
s
t

Movement
- Inspect the inner surface of
the lips by retracting them
with a tongue blade
Retraction of the Upper Lip

Retraction of the lower Lip
• Teeth
- ask patient to clench his/her
teeth , smile and observe the
occlusion of the teeth.
- facial nerve is also tested
- Make sure teeth are firmly
anchored, probing each with a
tongue blade
- Generally ivory white in color with
32 permanent teeth in adults
Proper Occlusion of Teeth
• Buccal mucosa
- with mouth open, using a tongue
blade,
inspect for color, pigmentation,
nodules, white patches
- normally pinkish red, smooth, moist
- orifice of the stensen duct should
appear as a whitish yellow or whitish
pink protrusion in alignment with the
2nd upper molar
Retraction of the cheek
to view the Buccal Mucosa

Buccal Mucosa with prominent
Papilla of Stensen Duct
• Gums
- using a tongue blade, gums
should have pink appearance with
clearly defined tight margin at
each tooth
- gum surface beneath dentures
should be free of inflammation,
swelling or bleeding
- Using gloves, palpate gums for
tenderness, mass, induration,
thickening
• Tongue
- should fit well in the floor of the
mouth
- ask the patient to extend the tongue
while you inspect for color, lesions,
deviation, tremor, limitation of
movement
- Ask the patient to touch the tongue tip
to the hard palate area directly behind
the upper central incisors. There
should be no difficulty.
- Inspect the dorsum of the tongue
it should appear dull red ,moist,
glistening
note also for any swelling,
coating, ulcerations
- Inspect the ventral surface of the
tongue
it should be pink and smooth with
large veins bet. the frenulum and
fimbriated folds
-

Wharton ducts should be
apparent on each side of
the frenulum
Mouth

>Roof of the mouth
- hard and soft palate
Floor of the mouth
- tongue
Take note of the smell coming
from the oral cavity
Ask the patient to tilt his head to
inspect the palate and uvula
Uvula , soft palate, bilateral fauces
Throat

Tonsils
- usually blend into the pink
surface of the pharynx
- surface of the tonsils have
crypts where cellular debris and
food collect
- in normal adult, tonsils seldom
protrude beyond the faucial
pillars
Posterior wall of the pharynx
-It should be smooth and glistening
pink mucosa with some irregular
spots of lymphatic tissue and
small blood vessels
-Test CN 9 and 10
touch the posterior wall of the
pharynx on each side
(+) gag reflex
Larynx
- immediately behind and
below the oral cavity
- it is on the anterior wall of
the pharynx
- it is viewed in the laryngeal
mirror held behind it
SIGNS
• Lips
Cyanotic Lips
Chapped dry lips
> Cheilitis
- dry cracked lips due to
dehydration from wind
chapping, dentures , braces, or
excessive lip licking
- angular cheilitis due to
candidiasis
Chapped Lips with Cheilitis
Cheilosis ( angular
stomatitis)
- ulcerations of skin at the
corners of the mouth due to
crusting 2ndary to riboflavin
deficiency or ill fitting
dentures
Cheilosis (Angular Stomatitis)
Cleft lip
- due to incomplete fusion
of the frontonasal process
with the 2 maxillary
processes
Cleft Lip
Retraction of the Lower Lip showing
white scars Traumatized Lip (Green
arrows)
• Hard palate

Maxillary Torus
Maxillary torus
- bony protuberance at the
midline
- no clinical significance
Cleft palate
- a midline opening in the hard
palate
- congenital failure of the
fusion of the maxillary process
- usually ass. with cleft palate
Cleft Palate
• Tonsils

Enlarged tonsils
- Grading tonsillar enlargement
• Grade size 1+ visible
• …………….2+ ½ way b/t tonsillar
pillars and uvula
• …………….3+ touching the uvula
• …………….4+ touching each
other
• Uvula

Deviation of the uvula
• Posterior pharyngeal wall

After tonsillectomy
Posterior Pharyngeal Wall
With a yellow Pseudocyst

Posterior Pharyngeal Wall with
White removable mass of mucus
Acute viral pharyngitis
- mucosa of oropharynx shows lymphoid
tissue are elevated but noo edema
- sore throat, rhinorrhea, malaise,
myalgia
Streptococal or staphylococcal
pharyngitis
- Pharyngeal mucosa is bright red,
swollen, edematous studded with white
or yellow follicles
- Tonsils maybe enlarged
Pharyngeal diptheria
- patch of white membrane in the
tonsils.
- pharyngeal mucosa bleeds on
surface, reddened , reddened,
swollen ,edematous
Candidiasis
- shining raised white patches on
posterior pharynx, buccal mucosa and
tongue
• Tongue

Lingual Deviation
Tongue-tie or shortened
frenulum
Folliate Papillae(Green)
Circumvallate Papillae(blue)

Elongated filiform Papillae
Large reddened fungiform Papillae

Circumvallate Papillae
• Gums

Gingival Fibrous Nodule
At the mucogingival junction
Bleeding gums
local causes:
traumatic – toothbrush,
laceration, dental caries, tartar
on the teeth
infection – pyorrhea alveolaris,
stomatitis
neoplasm – epulis, papilloma of
gums
General causes:
Scurvy, syphilis
Metal poisoning –phosporous, lead,
mercury
Blood dyscrasia – hemophilia,
leukemia,
thrombocytopenia
Deep red or purple gums
- tender , swollen, spongy
and easily bleeds
- due to scurvy ( ascorbic
acid deficiency)
• Teeth

Malocclusion of teeth
Periodontitis ( Pyorrhea
Alveolaris)
- lower teeth are involved
- with purulent and retracted
gums
Epulis
- fibrous tumor arising from
periosteum and emerges from
between the teeth.
• Larynx
> hoarseness
acute laryngitis – most common
cause of hoarseness
> laryngeal edema
signs of obstruction – hoarseness,
dyspnea and stridor
Laryngeal spasm
- acute obstruction of the upper
airways accompanied by hoarse
brassy cough, dyspnea in children
- due to allergy, infection, FB,
neoplasm
Laryngeal paralysis
- Due to immobile vocal cords
• Halitosis ( fetor Oris) bad breath
- Poor hygiene
- Dental or tonsillar infections
- Atrophic rhinitis
- Putrefaction of food in the
stomach from pyloric obstruction
- Infected sputum form lung
abscess and bronchiectasis
THANK YOU
Sensory Disorders

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Sensory Disorders

  • 1. SENSORY DISORDERS Nelia B. Perez RN, MSN PCU – MJCN Class 2015
  • 2. CONTENTS • Review Of Anatomy and Physiology • Common Sensory Disorders
  • 4. Anatomy •The ear is responsible for hearing and balance •Consists of 3 regions • External ear • Middle ear • Inner ear
  • 5. Structure and Function • External Ear > auricle/pinna - movable cartilage covered with skin - Mastoid process= important Landmark External Auditory Canal - S-shaped pathway leading to the ME - 2.5 to 3 cm. long in adult
  • 6. - Its skeleton of bone and cartilage is covered with sensitive skin ( outer 1/3 is cartilage, inner 2/3 consists of bone) - This canal lining is protected and lubricated with cerumen
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  • 8. - Lymphatic drainage of the external ear flows into parotid , mastoid, superficial cervical nodes
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  • 10. MIDDLE EAR air filled cavity in the temporal bone - >It contains the ossicles ( malleus, incus,stapes) that transmit sound from the TM to the oval window of the inner ear >
  • 11. MIDDLE EAR >Tympanic membrane (eardrum) separates external and middle ear. • Translucent membrane • Pearly, gray color • Cone of light reflection when using otoscope • Oval and slightly concave shape, pulled in at center by malleus
  • 12. Middle ear >Openings to Outer ear covered by tympanic membrane Inner ear = oval and round windows Eustachian tube connects middle ear to the nasopharnyx for air passage (normally closed, opens with swallowing/yawning)
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  • 15. Middle ear has 3 functions 1. Conducts sound vibration from outer ear to inner ear 2. Protects the inner ear by reducing the amplitude of loud sounds 3. Eustachian tube allows equalization of air pressure on each side of the ear drum to avoid rupture ( high altitudes)
  • 16. Inner Ear • Contains the Bony Labyrinth which holds the sensory organs for hearing and equilibrium 1. Vestibule 2. Semicircular canals 3. Cochlea (contains the central hearing apparatus)
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  • 18. Function of hearing • 3 levels 1. Peripheral > ear transmits sound and converts its vibrations into electrical impulses > The electrical impulses are conducted by the auditory process of cranial nerve VIII (Acoustic) to the brain stem 1. Amplitude=loudness 2. Frequency=pitch
  • 19. Sound waves cause the eardrum to vibrate > Vibrations travel via the ossicles thru the oval window > the cochlea > to the round window where they are dissipated
  • 20. Vibrations in the basilar membrane of the cochlea that contain the organ of Corti receptor hair cells > translate the vibrations to electric impulses > The stimulated impulses go to the brainstem via Acoustic nerve (VIII)
  • 21. 2. Brain stem - permits identification of sound and locating the direction of a sound in space. - Sensitive to intensity and timing from the ears depending on head position 3. Cerebral cortex - Intreprets the meaning of the sound and begins the appropriate response
  • 22. Pathways of hearing 1. Air conduction (AC)– normal pathway of hearing, the most efficient 2. Bone conduction (BC)– bones of the skull vibrate and transmit vibrations to the inner ear and acoustic nerve
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  • 25. Physical Examination • The Auricle 1) inspect each auricle for size , shape, symmetry, color, position on the head, deformities, nodules and lesions 2) If ear pain, discharge or inflammation is present, move the auricle up and down
  • 26. 3) Note tenderness of pinna and mastoid area. Press the tragus and press firmly behind the ear
  • 27. Physical Examination • Auricle -Extends slightly outward from the skull - Positioned in a nearly vertical plane - The origin of the helix should be on a horizontal line with corner of the eye - It should have the same color as the facial skin w/o moles, cysts & other lesions
  • 28. Otoscopic Exam 1) Tip the patient’s head to the opposite side 2)Grasp the auricle firmly but gently, while pulling it upward, backward and slightly outward 3)Insert into the canal, sl down and forward, the largest ear speculum that the canal will accommodate
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  • 32. 4) Observe the ff: - patency of the ear canal - describe the walls of the ear canal. Note any redness or swelling - identify any discharge, presence of cerumen or FB in the ear canal - tympanic membrane
  • 33. Inspect using Otoscope • External canal • Color • Swelling • Lesions • Discharge ; color and odor. Clean or change speculum before examining other ear.
  • 34. Tympanic membrane • Color – normal is shiny, translucent, pearl-grey • Landmarks ( umbo, handle of malleus, light reflex) • Position – flat, slightly pulled in at the center and flutters when person holds nose and swallows • Integrity of membrane – intact
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  • 36. •Perform the otoscope exam prior to hearing tests.
  • 37. Hearing Evaluation 1.Rough quantitative test for hearing loss 2.Whisper test 3.Tuning fork
  • 38. • Rough quantitative test for hearing loss - begins when the patient responds to your questions and directions. The patient responds without excessive requests for repetition - Speech with a monotonous tone and erratic volume may indicate hearing loss
  • 39. WHISPER TEST •Begins with the historyConversational tone •The following tests may indicate the presence of hearing loss but not the degree.
  • 40. • Place your mouth at the side of the patient’s head ( 2 ft.) from her ear with the far ear covered • Whisper test questions that can’t be answered by yes or no • Test consistently with loud, medium and soft tones
  • 41. • Repeat on the opposite ear using another word, have the client identify the words (Used to detect high-tone loss) • Normal Response to Voice test • Correct identification of whispered words bilaterally
  • 42. TUNING FORK TESTS • Measure hearing by air conduction and bone conduction • Frequency of fork is 256-1024 cycles/sec. • To activate the tuning fork, hold it by the stem and strike the tines softly on the back of the hand
  • 43. TUNING FORK TEST • Weber test > used when hearing is reported as better in one ear than the other ( bone conduction) > with normal neurosensory hearing and no conductive loss, the sounds are equal in both ears
  • 44. > lateralization of the sound to one ear indicates a conductive loss on the same side or a perceptive loss/sensorineural loss on the other side
  • 46. • Rinne test – compares bone conduction and air conduction 1. Normally sound is heard 2X as long by air conduction as by bone conduction 2. Normal response ; positive Rinne Test = AC>BC Bilaterally Sound is heard longer by BC with a conductive loss.
  • 49. Summary of any symptom should include PQRSTU • P= provocative or palliative • Q= quality or quantity • R= region or radiation • S= severity scale • T= timing (onset, duration, frequency)
  • 50. Subjective data • Earaches • Tinnitus • Vertigo • Dizziness • Discharge • Hearing loss
  • 51. HISTORY Always ask the following: • Tinnitus –ringing in the ears causes: a.Outer ear- cerumen, foreign body,polyp in the external auditory canal b. Middle ear – inflammation ,otosclerosis c. Internal ear- fever, suppuration of the labyrinth, SY,acoustic nerve tumor
  • 52. internal ear – fracture at the base of the skull, meniere syndrome d.Drugs quinine, salicylates, aminoglycosides, gentamicin
  • 53. •Ear pain ( Otalgia ) - pain may arise from inflammation of structure in the ear or be referred from other pharyngeal sites including the thyroid
  • 54. Causes: Auricletrauma,hematoma,frostbite,burn, eczema, lnsect bites, impetigo, herpes zoster External auditory canalotitis externa ,carbuncle, eczema, hard cerumen, FB, herpes zoster
  • 55. Middle earacute otits media, acute mastoiditis Referred pain- unerrupted lower third molar, carious teeth, tonsillitis, carcinoma of pharynx, trigeminal neuralgia , subacute thyroiditis
  • 56. • Dizziness - patient has a sense of disturbed relation to space - described as being unsteady, weak, light headed or having the feeling of turning Causes: Endocrine hypothyroidism,pregnancy, hypoparathyroidism
  • 57. Idiopathic multisystem atrophy Infectious tabes dorsalis, meningitis, encephalitis, brain abscess Metabolic/ nutritional pellagra, Vit.B12 def.,fluid & electrolyte imbalance
  • 58. Mechanical/trauma skull fracture, otosclerosis, eye muscle imbalance glaucoma Neoplastic Brain tumors Neurologic migraine, peripheral neuropathy Psychosocial anxiety disorder Vascular hypertension, orthostatic hypotension
  • 59. • Vertigo - persistent stimulation of the semicircular canals or vestibular nucleus when the head is at rest - It gives a hallucination of motion - When the eyes open, the pts.surrounding seems to be whirling or spinning - When the eyes closed, the pt.continues to feel in motion
  • 60. Causes: Peripheral labyrinthine System - otitis media with effusion, otosclerosis, temporal bone fracture Central labyrinthine system - migraine, cerebellar hemorrhage, intracranial abscess
  • 61. Cranial V111 infections - Acute meningitis, tuberculous meningitis, tumors Brainstem nuclei - encephalitis, brain abscess, hemorrhage, multiple sclerosis
  • 62. • Hearing loss a. Conductive- seen in people with external or middle ear problem Causes: -obstruction of external auditory canal (FB, impacted cerumen) - Disorder of the eardrum & middle ear ( perforated TM, pus/blood in the ME ) - Overgrowth of bone with fixation of the stapes ((Otosclerosis)
  • 63. b. Sensorineural hearing loss ( Perceptive) - involves the inner ear Causes: - disorders of the cochlea or the acoustic nerve (CN 8) - Aging ( Presbycusis ) due to nerve degeneration - Trauma - Drug toxicity - Tumors - infections - Heredity/congenital deafness
  • 64. EAR SIGNS • EXTERNAL EAR a) Malformations of the Pinna microtia – smaller than normal macrotia – unusually large lop or bat ear- pinna may protude at R angle aztec or cagot ear – failure of development of the lobule
  • 65. Macrotia or large ear Before Surgery After Surgery
  • 67. Lop or Bat ear - pinna may protrude at right angle
  • 68. Lop or Bat Ears
  • 69. satyr ear- pointed pinna cauliflower ear- untreated hematomas heal as nodular and bulbous irregularities of the helix and and antihelix - result of blunt trauma and necrosis of the underlying cartilage
  • 71. b) Pinna nodule Darwin tubercle- harmless developmental eminence in the upper 3rd of the posterior helix Gouty tophus – small, whitish uric acid crystals along the peripheral margins of the auricles, olecranon bursa, tendon sheaths - nodules are painless hard, and irregular
  • 73. b)External acoustic meatus Cerumen Impaction - due to excessive production of wax or a narrowed meatus leads to partial or complete obstruction of the canal - complete obstruction leads to partial deafness acc. by tinnitus or dizziness
  • 74. Otorrhea( ear discharge) yellow discharge- melted cerumen serous discharge- eczema in the meatal wall, early ruptured acute OM bloody discharge- temporal bone fracture purulent discharge- chronic external otitis, chronic suppurative OM, cholesteatoma, TB, polyps
  • 76. • Tympanic membrane Retracted Tympanic membrane : - Seen in Serous Otitis media - more concave TM - accentuated bony landmarks - distorted light reflex
  • 78. Bulging Tympanic membrane: - seen in Acute suppurative otitis media - more conical - loss of bony landmarks - distorted light reflex
  • 79. Normal Tympanic Membrane Bulging Tympanic Membrane
  • 80. Perforated Tympanic membrane: - previous suppurative middle ear infection has eroded thru the membrane producing holes - perforation appears as oval holes thru which the darkened middle ear cavity is seen
  • 83. COMMON DISORDERS OF THE EAR • Otitis Externa a) Acute external otitis -due to Ps.aeruginosa, staph, strep, proteus - pain maybe mild or severe accentuated by movement of the pinna - swimmers’ ear - preauricular, postauricular , Ant cervical LN
  • 84. b) Chronic external otitis - commonly due to bacteria and fungal - pruritus is the main complain instead of pain - aural discharge maybe present
  • 85. • Otitis Media a) Chronic suppurative otitis media - ass. with permanent perforation of the eardrum -hearing is always impaired - painless aural discharge - pain and vertigo indicates development of complications like brain abscess
  • 86. b) Cholesteatoma - collection of desquamated epithelial cells in the middle ear - foul smelling discharge, marginal perforation,hearing loss, pearly gray mass superior part of tympanic membrane - eustachian tube dysfunction causes retraction of tympanic membrane
  • 87. • Vertiginous disorder a) Acute Labyrinthitis - most frequent cause of vertigo - patient gradually develop a sense of whirling that reaches a climax in 24-48 hrs. disappear gradually in 36 wks. - N/V may occur at the height of symptoms - no accompanying tinnitus or hearing loss
  • 88. b) Benign Paroxysmal positional Vertigo (BPPV) - Calcium deposits in the labyrinth ( otoliths) are dislodged and move in response to gravity eliciting a feeling of motion - More common in older individuals - Sudden onset, often when rolling over in bed or arising in the morning - No headaches/fever but with nausea and inability to stand - Avoid any head motion to lessen symptoms
  • 91. Nose • First segment of the respiratory system • Warms, moistens and filters inhaled air • Sensory organ for smell • Resonance of laryngeal sound
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  • 93. External parts • Bridge – frontal and maxillary bones • Tip • Nares – anterior openings of the nos • Columella - divides the nares • Ala nasi –lateral outside wing of the nose bilaterally • Upper 1/3 nose is bone; rest is cartilage
  • 94. Internal • Nasal cavity -floor of the nose ( hard and soft palate) - roof of the nose ( frontal and sphenoid bone) • Nasal hair • Nasal Septum-divides cavity into 2 passages • Nasal turbinates
  • 95. Internal • Superior, middle, inferior turbinates- 3 parallel bony projections on lateral walls of each cavity • Meatus- cleft/ groove underlying each turbinate.
  • 96. •Inspired air enters thru the nares > passes thru the vestibule> to the choanae which are posterior openings > leading to the nasopharynx
  • 97. Internal • Olfactory receptors - roof of the nasal cavity & upper part of septum above the superior turbinate. -merge into the olfactory nerve (I) > goes to the temporal lobe of the brain • Kiesselbach plexus - a vascular network located superficially on the anterior superior portion of the septum - site of most anterior nosebleeds
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  • 99. SINUSES • Paranasal sinuses - air-filled paired extensions of the nasal cavities within the bones of the skull - lined with mucous membranes and cilia that move secretions along excretory pathways - sinus openings are narrow, susceptible to occlusion> resulting in inflammation /sinusitis. - drained into the medial meatus
  • 100. • Purpose • Serve as resonators for sound • Provide mucous for the nasal cavity Types: 1. Frontal sinuses 2. Maxillary sinuses 3. Ethmoid sinuses 4. Sphenoid sinuses Frontal & Maxillary sinuses are accessible to examination
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  • 104. • Nose – Inspect and palpate • INSPECT for: • • • • • • Symmetry, deformity Inflammation Skin lesions Color Nasal flaring discharges
  • 105. • Palpate - ridge & soft tissues of the nose - note any displacement of the bone, cartilage - note for tenderness & any mass - The nasal structures should be firm and stable to palpation - if with injury, palpate gently
  • 106. •Test for sense of smell (CN 1) •Evaluate the patency of the nose - nasal breathing should be noiseless and easy thru the open nares
  • 107. Nasal Cavity  Use the nasal speculum and good light source to inspect the nasal cavity a) Nasal mucosa - inspect for color, discharge, lesions, masses - it should appear deep pink ( pinker than the buccal mucosa) & glistening
  • 108. b) nasal septum - In normal adult, the nasal septum is seldom precisely a midline structure - No perforations, bleeding or crusting should be apparent - a film of clear discharge is often apparent on the nasal septum
  • 109. c) Nasal Turbinates - only the inferior and middle turbinates will be visible - it should be the same color as the surrounding area and have a firm consistency
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  • 111. • Paranasal Sinuses: Inspect and Palpate • Press thumbs over frontal & maxillary sinuses ( palpate the cheeks and supraorbital ridges) • No tenderness or swelling over the soft tissue should be present
  • 112. •Transillumination test a) Frontal & Maxillary sinuses b) nasal septum - Best perform in a dark room - Look for a bright light in the supraorbital ridge and maxilla - Look for deviation, perforation, masses in the transilluminated septum
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  • 117. SYMPTOMS • Loss of smell ( anosmia ) - lesion of CN 1 or nasal obstruction - commonly due to closed head trauma - invariably accompanied by a perceived change in taste of food ( bland & unpalatable)
  • 118. • Abnormal smell/ taste (dysgeusia) - this is a common complaint in patients who have loss of smell - if it is paroxysmal and associated with behavioral symptoms, it suggests complex partial seizures
  • 121. SIGNS • Discharge - Describe discharge as to its character ( watery, mucoid, purulent , bloody) - color ( greenish, whitish, bloody) - bilateral or unilateral
  • 123.
  • 124. . 1.Unilateral - Choanal atresia - Foreign body- foul purulent discharge - neoplasm – bloody discharge - Head injury or surgery – clear spinal fluid 2. Bilateral - allergy - infection ( upper respiratory)
  • 126. . Unilateral - Choanal atresia - Foreign body - neoplasm - Head injury or surgery
  • 127. • Epistaxis ( nosebleed) -Kiesselbach plexus – most common site of bleeding anteriorly - Back 3rd of the Inferior Meatus – most common site posteriorly
  • 128. Causes: 1. Local - coughing - sneezing - nose pricking - fracture - foreign bodies
  • 129. 2. Generalized - Congenital – hereditary telangiectasia - inflammatory/immune – wegener granulomatosis - infectious – typhoid fever, dengue, diphtheria - Metabolic/toxic – aspirin, scurvy
  • 130. - Mechanical – change in atmospheric pressure ( mountain climbing, flying), exertion - Neoplastic – nasopharyngeal Ca leukemia - vascular- hemophilia, thrombocytopeni
  • 131. - trauma- nasal and maxillary fracture - Elevated venous pressure- Cor pulmonale Congestive Heart failure - Elevated arterial pressure – HPN, coarctation of aorta
  • 132. • Nasal septum a) Deviation - the cartilagenous and bony septum may deviate as a hump, spur, shelf to enroach on one nasal chamber occlusion causing obstruction
  • 133. b) Perforation - a hole in the nasal septum (transillumination test) is commonly caused by chronic infection, nasal surgery, repeated trauma in picking off crusts, cocaine abuse - rarely due to SY, TB
  • 135. Nasal Syndromes • Acute Rhinitis ( infectious) ( common cold) - Rhinoviruses infect the mucous membranes of the nose & sinuses causing inflammation and inc. nasal secretions - Watery nasal discharge, sneezing, discharge becomes purulent acc. by fever and body malaise
  • 136. -Symptoms 3-10 days -Severe local pain suggest a complication-bacterial sinusitis
  • 137. • Allergic rhinosinusitis - itching of the nose & eyes, rhinorrhea, lacrimation, sneezing - headache is common - maybe seasonal or perennial - common allergens are pollens, molds, house dust, mites, coachroach, animal danders
  • 138. • Vasomotor Rhinitis - nonallergic mucosal edema and rhinorrhea ass. with vasodilatation of the nasal vessels, mucosal edema & inc. mucous production - due to chronic environmental irritants ( dust , smoke, strong odor, cold air), pregnancy, estrogens, progesterone
  • 139. • Suppurative Paranasal Sinusitis - due to Strep. pneumonia, H. influenza - severe pain in the face occuring 714 days after signs & symptoms of an acute URTI - pain & pressure without fever suggest sinus obstruction requiring decongestants
  • 140. • Cavernous Sinus Thrombosis -This is the most feared complication of nasal infections. It can cause blindness or death - Infection spreads from the nose>angular veins> cavernous sinus> septic thrombosis
  • 141. -patient eyes complains of pain deep in the - Both eyes are involved, immobilization of the globes, periorbital edema, chemosis - May involve CN 3,4, &6 - Sudden chills, high fever, prostated, comatose, death within 2-3 days
  • 144. Mouth • First segment of the digestive system • Airway for the respiratory system • ORAL CAVITY • Lips • Palate 1. Hard 2. Soft 3. Uvula – hangs down from the soft palate
  • 145. • Cheeks- side walls of cavity • Tongue 1. Papillae- rough, bumpy elevations on dorsal 2. Frenulum 3. Taste buds • Teeth – 32 permanent
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  • 147. • Salivary glands 1. Parotid- largest of the glands, located in the cheeks, front of the ear. Stenson’s duct opens in buccal mucosa 2. Submandibular- walnut size, beneath the mandible at the angle of the jaw. Wharton’s duct either side of the frenulum 3. Sublingual –smallest, almond shape, under tongue
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  • 149. Throat  Area behind the mouth & nose  Oropharynx – separated from the mouth by a fold of tissue on each side called anterior tonsillar pillars  Tonsils – lymphoid tissue behind pillars
  • 150. • Posterior pharyngeal wall located behind the tonsils • Nasopharynx continues from the oropharynx but it is above it and behind the nasal cavity. -It holds the adenoids and the eustachian tube openings.
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  • 153. • Preparation for examination a) Face the patient with both of you seated at the same level b) Remove any dentures to see the mucosa underneath c) Hold the tongue blade in the left hand and penlight in the right hand d) A good light source is needed
  • 154. INSPECT AND PALPATE Use gloves, tongue depressor, light • Lips • Teeth • Gums • Tongue • Buccal mucosa • Mouth ( roof and floor of the mouth)
  • 155. • Lips - remove lipstick - should be pink , smooth surface, free of lesions. - distinct border between the lips and facial skin should not be interrupted by lesions - Vertical and horizontal symmetry both at rest and with movements
  • 157. - Inspect the inner surface of the lips by retracting them with a tongue blade
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  • 159. Retraction of the Upper Lip Retraction of the lower Lip
  • 160. • Teeth - ask patient to clench his/her teeth , smile and observe the occlusion of the teeth. - facial nerve is also tested - Make sure teeth are firmly anchored, probing each with a tongue blade - Generally ivory white in color with 32 permanent teeth in adults
  • 162. • Buccal mucosa - with mouth open, using a tongue blade, inspect for color, pigmentation, nodules, white patches - normally pinkish red, smooth, moist - orifice of the stensen duct should appear as a whitish yellow or whitish pink protrusion in alignment with the 2nd upper molar
  • 163. Retraction of the cheek to view the Buccal Mucosa Buccal Mucosa with prominent Papilla of Stensen Duct
  • 164. • Gums - using a tongue blade, gums should have pink appearance with clearly defined tight margin at each tooth - gum surface beneath dentures should be free of inflammation, swelling or bleeding - Using gloves, palpate gums for tenderness, mass, induration, thickening
  • 165. • Tongue - should fit well in the floor of the mouth - ask the patient to extend the tongue while you inspect for color, lesions, deviation, tremor, limitation of movement - Ask the patient to touch the tongue tip to the hard palate area directly behind the upper central incisors. There should be no difficulty.
  • 166. - Inspect the dorsum of the tongue it should appear dull red ,moist, glistening note also for any swelling, coating, ulcerations - Inspect the ventral surface of the tongue it should be pink and smooth with large veins bet. the frenulum and fimbriated folds
  • 167. - Wharton ducts should be apparent on each side of the frenulum
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  • 169. Mouth >Roof of the mouth - hard and soft palate Floor of the mouth - tongue Take note of the smell coming from the oral cavity Ask the patient to tilt his head to inspect the palate and uvula
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  • 171. Uvula , soft palate, bilateral fauces
  • 172. Throat Tonsils - usually blend into the pink surface of the pharynx - surface of the tonsils have crypts where cellular debris and food collect - in normal adult, tonsils seldom protrude beyond the faucial pillars
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  • 174. Posterior wall of the pharynx -It should be smooth and glistening pink mucosa with some irregular spots of lymphatic tissue and small blood vessels -Test CN 9 and 10 touch the posterior wall of the pharynx on each side (+) gag reflex
  • 175. Larynx - immediately behind and below the oral cavity - it is on the anterior wall of the pharynx - it is viewed in the laryngeal mirror held behind it
  • 176. SIGNS
  • 180. > Cheilitis - dry cracked lips due to dehydration from wind chapping, dentures , braces, or excessive lip licking - angular cheilitis due to candidiasis
  • 181. Chapped Lips with Cheilitis
  • 182. Cheilosis ( angular stomatitis) - ulcerations of skin at the corners of the mouth due to crusting 2ndary to riboflavin deficiency or ill fitting dentures
  • 184. Cleft lip - due to incomplete fusion of the frontonasal process with the 2 maxillary processes
  • 186. Retraction of the Lower Lip showing white scars Traumatized Lip (Green arrows)
  • 188. Maxillary torus - bony protuberance at the midline - no clinical significance
  • 189. Cleft palate - a midline opening in the hard palate - congenital failure of the fusion of the maxillary process - usually ass. with cleft palate
  • 192. - Grading tonsillar enlargement • Grade size 1+ visible • …………….2+ ½ way b/t tonsillar pillars and uvula • …………….3+ touching the uvula • …………….4+ touching each other
  • 194. • Posterior pharyngeal wall After tonsillectomy
  • 195. Posterior Pharyngeal Wall With a yellow Pseudocyst Posterior Pharyngeal Wall with White removable mass of mucus
  • 196. Acute viral pharyngitis - mucosa of oropharynx shows lymphoid tissue are elevated but noo edema - sore throat, rhinorrhea, malaise, myalgia Streptococal or staphylococcal pharyngitis - Pharyngeal mucosa is bright red, swollen, edematous studded with white or yellow follicles - Tonsils maybe enlarged
  • 197. Pharyngeal diptheria - patch of white membrane in the tonsils. - pharyngeal mucosa bleeds on surface, reddened , reddened, swollen ,edematous Candidiasis - shining raised white patches on posterior pharynx, buccal mucosa and tongue
  • 201. Large reddened fungiform Papillae Circumvallate Papillae
  • 202. • Gums Gingival Fibrous Nodule At the mucogingival junction
  • 203. Bleeding gums local causes: traumatic – toothbrush, laceration, dental caries, tartar on the teeth infection – pyorrhea alveolaris, stomatitis neoplasm – epulis, papilloma of gums
  • 204. General causes: Scurvy, syphilis Metal poisoning –phosporous, lead, mercury Blood dyscrasia – hemophilia, leukemia, thrombocytopenia
  • 205. Deep red or purple gums - tender , swollen, spongy and easily bleeds - due to scurvy ( ascorbic acid deficiency)
  • 207. Periodontitis ( Pyorrhea Alveolaris) - lower teeth are involved - with purulent and retracted gums Epulis - fibrous tumor arising from periosteum and emerges from between the teeth.
  • 208. • Larynx > hoarseness acute laryngitis – most common cause of hoarseness > laryngeal edema signs of obstruction – hoarseness, dyspnea and stridor
  • 209. Laryngeal spasm - acute obstruction of the upper airways accompanied by hoarse brassy cough, dyspnea in children - due to allergy, infection, FB, neoplasm Laryngeal paralysis - Due to immobile vocal cords
  • 210. • Halitosis ( fetor Oris) bad breath - Poor hygiene - Dental or tonsillar infections - Atrophic rhinitis - Putrefaction of food in the stomach from pyloric obstruction - Infected sputum form lung abscess and bronchiectasis