Prevent Diabetes

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arpanbhattacharya

Health & Medicine

EP Joslin, 1921 “…… it is proper at the present time to devote time not alone to treatment, but still more, as in the case of typhoid fever, to prevention. The results may not be quite so striking or as immediate, but they are sure to come and important …...”

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Type 1 Diabetes Mellitus is a form of diabetes mellitus that results from the autoimmune destruction of the insulin-producing beta cells in the pancreas. Genetic factors are believed to be a major com- ponent for the development of type 1diabetes, but the con- cordance rate for the development of diabetes in identical twins is only about 40%, suggesting that non-genetic factors play an important role in the expression of the disease. Viruses are one environmental factor that is implicated in the pathogenesis of type 1 diabetes mellitus. Shashikiran Umakanth made this presentation at the "First Endocrine Update Program” – ENDO EGYPT 2015, from 17-20 December 2015 in the Historic City of Luxor, Egypt. This endocrine update was organised by the Egyptian Association of Endocrinology , Diabetes and Atherosclerosis (EAEDA) in collaboration with the Endocrine Society, USA.

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This webinar will discuss the prevalence of pre-diabetes and it’s contributing factors and the initial efforts to translate the National Diabetes Prevention Program to public health. We will also look at new approaches to providing interventions. Learning objectives: Scope and scale of pre-diabetes and what factors contribute to it. Review initial efforts to translate the DPP to public health. New approaches to providing interventions. About The Presenter Dr. Marrero received a B.A. (1974), M.A. (1978) and Ph.D. (1982) in Social Ecology from the University of California, Irvine. He joined the IU School of Medicine in 1984 and became the J.O. Ritchey Professor of Medicine in 2004. He was a member of the Diabetes Research & Training Center and served as Director of the Diabetes Prevention and Control Division. He is currently the Director of the Diabetes Translational Research Center. Dr. Marrero is an expert in the field of clinical trails in diabetes and translation research which moves scientific advances obtained in clinical trails into the public health sector. He helped design the Diabetes Prevention Program and the TRIAD study, which evaluated strategies to improve diabetes care delivery in managed care settings. His research interests include strategies for promoting diabetes prevention, care settings, improving diabetes care practices used by primary care providers, and the use of technology to facilitate care and education. Dr. Marrero was twice awarded the Allene Von Son Award for Diabetes Patient Education Tools by the American Association of Diabetes Educators, nominated to Who’s Who in Medicine and Health care in 2000, served as Associate Editor for Diabetes Care (1997-2002) and is currently the Associate Editor for Diabetes Forecast. He was selected as Alumni of the Year for University of California Irvine in 2006 and The Outstanding Educator in Diabetes in 2008 by the American Diabetes Association. He is the current President of the American Diabetes Association.

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The Medicare Diabetes Prevention Program (MDPP) Model Expansion Medicare Learning Network (MLN) Call was held from 1:30 p.m. – 3:00 p.m. EST on November 30, 2016. During this call, CMS experts provided a high-level overview of the finalized policies in the CY 2017 Medicare Physician Fee Schedule (PFS) final rule (the CY 2017 Medicare PFS final rule includes the expansion of the MDPP Model beginning January 1, 2018), reviewed the steps necessary for enrollment into Medicare as an MDDPP supplier, and answered some of the audiences most pressing questions. - - - CMS Innovation Center http://innovation.cms.gov We accept comments in the spirit of our comment policy: http://newmedia.hhs.gov/standards/comment_policy.html CMS Privacy Policy http://cms.gov/About-CMS/Agency-Information/Aboutwebsite/Privacy-Policy.html

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Advanced Pharmacology Diabetes is a group of metabolic diseases characterized by hyperglycemia resulting from defects in insulin secretion, insulin action, or both. ( McCance & Huether 2019). Per The American Diabetic Association (ADA) each year 1.5 million Americans are diagnosed with diabetes. There are different types of diabetes in this paper we will differentiate between the various types and focus on type 2 diabetes. Comparison of different types of diabetes including drug treatment Type 1 Diabetes: caused by Beta-cell destruction leading to absolute insulin deficiency ( pancreas stops producing insulin) . This is the most common form of diabetes. Patients with type 1 diabetes are insulin-dependent. About 75% of them develop before the age of 30. Patients are prone to diabetic ketoacidosis. Type 1 diabetes is secondary to other diseases such as pancreatitis, it is also said to be caused by genetic predisposition and environmental factors. It is most common in people of African and Asian descent. Clinical manifestations of type 1 diabetes include Polydipsia, polyuria, polyphagia, weight loss, and fatigue. Patients with type 1 diabetes take different types of insulin. Rapid-acting (Lispro ), short-acting ( regular insulin ) intermediate-acting ( NPH), long-acting (glargine). Type 2 Diabetes; is caused by the progressive loss of b-cell insulin secretion frequency on the background of insulin resistance ( insulin becomes resistant, may be produced but does not do its job). Patients with type 2 diabetes are usually not insulin dependent but might be insulin requiring depending on the severity of hyperglycemia. Not ketosis-prone but may form ketones under stress. obesity in the abdominal region. Mostly occurs in those 40years of age and above . has a strong genetic predisposition and is often associated with dyslipidemia and hypertension. Prevalence is highest among American Indians and Alaska Natives and lowest among non-Hispanic whites. Clinical manifestations of type 2 diabetes are nonspecific but the most common ones are; recurrent infections ( boils and carbuncles, skin infections, and prolonged wound healing ), genital pruritus, visual changes, paresthesias, fatigue, and Acanthosis nigricans ( brown to black pigmentation in body folds associated with insulin resistance ). Treatment of diabetes type two involves lifestyle modifications like weight loss which results in improved insulin sensitivity, and glucose tolerance, and preserves beta-cell function. In cases where diet and exercise yield unsatisfactory results, first line of drugs for type 2 diabetes is Sulfonylurea, and in some cases insulin is added if the oral hypoglycemic agents do not work. they are nine classes of oral hypoglycemic agents with metformin being the drug of choice. Gestational Diabetes Mellitus ( GDM); Any degree of glucose intolerance with onset or first recognition of symptoms during pregnancy. Caused by insulin resistance combined with inad ...

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BETH MARTINS Explain the difference between the types of diabetes, including type 1, type 2, gestational, and juvenile diabetes Type I Diabetes is hyperglycemia due to insulin deficiency. This is caused by autoimmune destruction of B cells that are within the islets of Langerhans (Chetan, Thrower,& Narendran, 2019). Some signs surrounding Type I DM is polydipsia, polyuria, and weight loss. This is more common in children, but can be diagnosed at any age throughout life. Type I diabetes used to be called juvenile diabetes,but this is now known to not be the correct way to view this disease. Type 2 Diabetes is impaired insulin secretion and abnormal suppression of postprandial (Hurtado & Vella, 2019). Type 2 DM the pancreas resists insulin, or does not produce enough insulin on its own. Often Type I and Type 2 DM are diagnosed incorrectly or mistaken for each other as signs and symptoms are similar. Being overweight or reduced amount of exercise is though to be a contributing factor as well as genetics, but this is till not fully understood. Gestational diabetes is characterized by spontaneous hyperglycemia during pregnancy. Some risk factors include family history of diabetes, obesity during pregnancy, and advanced maternal age (Plows, Stanley, Baker, 2019). B cell dysfunction which lacks the ability to release insulin when the body needs it is what causes gestational diabetes. Consider one type of drug used to treat the type of diabetes you selected, including proper preparation and administration of this drug. Then, reflect on dietary considerations related to treatment. Patients who have Type 2 DM have both insulin resistance and impaired insulin secretion (Rosenthal & Burchum, 2021). The liver, muscle and adipose tissue are insulin resistant and increased blood sugar causes B cell function to fail. Insulin secretion decreases and B cells must increase how hard they must work to work against insulin resistance within the tissues (Rosenthal & Burchum, 2021). The medication I chose os Metformin from the class of Biguanide medications for treatment of Type 2 DM. Metformin uses 3 mechanisms to help lower blood sugar. Inhibits glucose production in the liver Reduces glucose absorption in the gut Sensitizes insulin receptors in fat and skeletal muscle Metformin is taken by mouth and is absorbed by the small intestines and excreted by the kidneys. Metformin is known to be a great choice for those who skip meals as it will not make blood sugar levels become dangerously low. Those with DM Type 2 should eat healthy carbs such as fruits and vegetables and not indulge in sugary drinks. Protein and polyunsaturated fats should be a staple in the everyday diet. Alcohol should be avoided, and the patient should try to eat several small “meals” per day tp keep blood sugar steady. If blood sugar becomes low having a sugary snack with a complex carbohydrate such as peanut butter and crackers should be on hand. Short-term and long-t.

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