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Asallam Alekkum 
Dr. Gaurav Garg, Lecturer 
College of Dentistry, Al Zulfi
Diagnosis ?
Diagnosis is a process of determining the nature of 
a disease. 
very important for proper treatment. 
Differential diagnosis is the process of 
differentiating between similar diseases. 
For correct differential diagnosis: 
 Proper knowledge of the disease 
 Skill and Art on how to apply proper diagnostic 
methods
A good clinician has to be a good 
diagnostician. 
Qualities of a good diagnostician- 
Knowledge 
Interest 
Intuition 
Curiosity 
Patience
As stated by Grossman, are phenomenon or 
signs of a departure from the normal, and are 
indicative of an illness. 
Types: 
1. Subjective Symptoms are those which are 
experienced and reported to the clinician by the 
patient. 
2. Objective Symptoms are those, which are 
obtained by the clinician through various tests.
Lingering tooth sensitivity to cold liquids. 
Lingering tooth sensitivity to hot liquids. 
Tooth sensitivity to sweets. 
Tooth pain to biting pressure. 
Tooth pain that is referred from a tooth to another 
area, such as the neck, temple, or the ear. 
Spontaneous toothache, such as that experienced 
while reading a magazine, watching television, etc. 
Constant or intermittent tooth pain. 
Severe tooth pain. 
Throbbing tooth pain. 
Tooth pain that may occur in response to postural 
changes, such as when going from a standing to a 
reclining position.
Good case history: 
 Chief complaint 
 Past medical history 
 Past dental history 
 Also includes vital signs, history of presenting illness. 
• It is essential to gather collective information 
regarding signs, symptoms, and history for a 
successful outcome of any treatment procedure. 
Thorough clinical examination 
Relevant investigations / diagnostic tests
Hardly exists any contraindication except: 
 Uncontrolled Diabetes 
 A very recent Myocardial infarction 
Aid the dental clinician in deciding whether a 
prior medical consultation or pre-medication 
(chemo-prophylaxis) would be required
Cardiovascular: 
 High & moderate risk of Endocarditis 
 Pathologic heart Murmers 
 Hypertension 
 Unstable angina 
 Recent myocardial infarction 
 Arrythmias 
 Poorly managed congestive heart failure. 
Pulmonary: 
 COPD 
 Asthma 
 Tuberculosis
GI & Renal: 
 End stage renal disease , Hemodialysis 
 Viral hepatitis( B,C,D,E) 
 Alcoholic liver disease 
 Peptic ulcer disease 
Endocrine & Hematologic: 
 STDs, HIV & AIDS 
 Diabetes mellitus 
 Adrenal insufficiency 
 Hyperthyroidism 
 Pregnancy 
 Bleeding disorders 
 Cancer & leukemia 
 Osteoarthritis, rheumatoid arthritis, SLE. 
NeurologicDisorders
Side effects of medications: 
 Stomatitis, xerostomia, petechiae, ecchymosis, lichenoid 
lesions & mucosal & gingival bleeding. 
Lymphoma or Tuberculous involvement of cervical & 
submandibular lymph nodes 
Immunocompromised & patients with uncontrolled Diabetes 
Patients with iron deficiency anaemia, pernicious anemia & 
leukemia frequently exhibits paraesthesia of oral soft tissues 
complicating making a diagnosis when other dental pathosis 
is also present in the same area of the oral cavity.
Sickle cell anemia has the complicating factor of bone pain, which 
mimics odontogenic pain & loss of trabecular bone pattern on 
radiographs which can be confused with radiographic lesions of 
endodontic origin. 
Multiple myeloma can result in unexplained mobility of teeth. 
Radiation therapy to head & neck region can result in increased 
sensitivity of teeth & osteoradionecrosis 
Trigeminal neuralgia, referred pain from cardiac angina, and 
multiple sclerosis can also mimic dental pain. 
Acute maxillary Sinusitis- a condition that may mimics tooth pain in 
maxillary posterior quadrant. In this situation the teeth in the 
quadrant will be extremely sensitive to cold & percussion, thus 
mimicking the signs & symptoms of Pulpitis.
Chief complaint is the best starting point for a 
correct diagnosis 
PAIN is one of the most common chief 
complaints encountered
In order to attain a detailed knowledge regarding pain, following 
questions may be necessary: 
1. Type of pain : 
 Grossman has stated Pulpal pain to be of the following two 
varieties : 
 Sharp, piercing and lancinating -- a painful response usually 
associated with the excitation of the A-DELTA nerve fibers. This 
pain usually reflects REVERSIBLE state. 
 Dull, borinq, gnawing and excruciating-- a painful response 
usually associated with the excitation of C-nerve fibers. 
 Usually reflects an irreversible state of pulpitis.
2. The duration of pain: 
 When the pain is of a shorter duration (1 
minute)-Reversible Pulpitis - Excellent chance 
of recovery without the need for endodontic 
treatment 
 Whereas when the pain is of a longer duration, 
it is considered to be Irreversible Pulpitis
3. The localization of pain: 
 Sharp piercing pain can usually be localized and 
responds to cold 
 Dull pain usually referred / spread over a larger area 
responds more abnormally to heat 
 Patients may report that their dental pain is 
exacerbated while lying down or bending over 
 This occurs because of the increase in blood pressure 
to the head, which therefore increases the pressure on 
the confined pulp
4. Factors which provoke/ relieve pain: 
 Response to a provoking factor (e.g. on 
mastication) indicates pulp vitality, but 
stimulation causing extended severe pain 
suggests irreversible pulpitis. 
 Thus pain, which is recorded as the complaint 
is considered to conclude an acute or chronic, 
reversible or irreversible condition of the pulp.
Characterized by pain which is of a :- 
 Shorter duration 
 Localized 
 May be piercing/ lancinating in nature 
 More responsive to cold than heat 
 Caused by a specific irritant & disappears as 
soon as it is removed
 Abnormal dental pain, which responds to heat 
 Which occurs on changing the position of the 
head, awakening the patient from sleep 
 Dull pain of Longer duration, which occurs 
during mastication in a Cariously exposed 
tooth
History Slight sensitivity or 
occlusal pain 
Constant or intermittent pain 
Pain Momentary & 
immediate, sharp in 
nature & quickly 
disappears thereafter 
Continuous, Delayed onset, 
throbbing, persists for minutes to 
hours after removal of stimulus 
Location of pain May be localized & is 
not reffered 
Pain is not localized. If it is 
localized, its only after periapical 
involvement. Pain is reffered. 
Lying down No difference Pain increases 
Thermal test Responds Marked & prolonged 
E.P.T. Early response Early, delayed or mixed response 
Percussion Negative Negative in early stages. Later 
positive when periapex is 
involved 
Radiography Negative May show widening of PDL space
The clinician should 
look for: 
 Facial asymmetry 
 Localized 
swellings 
 Lymphadenopathy 
 Changes in color, 
bruises/ scars, 
similar signs of 
disease, trauma or 
of any various 
treatment.
Begins with a general evaluation of the oral 
structures. 
The occlusion is checked (for any 
derangements if any) 
The lips, cheeks, vestibules and mucosa are 
examined for any evident abnormalities. 
Several tests are employed in order to 
determine the condition of teeth and supporting 
structures.
Simplest and the easiest of the diagnostic tests 
Acc. To Grossman the prime objective of visual & 
tactile inspector is evaluation of the “3 C’s” viz: 
 Color 
 Contour 
 Consistency of hard and soft tissues. 
Soft tissue: 
Color- the normal color of gingiva is coral pink. 
Change 
from this is easily visualized in inflammatory 
conditions. 
Contour- change in normal contour (eg, of 
scalloped gingiva) occurs with a swelling. 
Consistency- On inspection (most commonly 
gingiva) appears healthy, firm, resilient, while a soft, 
fluctuant or spongy tissue is more indicative of a 
pathological state.
Hard tissue: 
A similar parameter of the visual and tactile 
inspection., ie, of the “C’s’is employed for 
the dental tissues as well. 
 Color- Normal teeth show life like 
translucency & sparkle that is missing in 
pulpless teeth which appear more or less 
opaque. 
Note: This discoloration however could be 
due to a variety of other reasons like old 
amalgam restorations, tetracycline stains 
etc. 
 Contour- This examination should also 
include the visualization of contours of 
affected teeth, such as fractured teeth, wear 
facets, improperly contoured restorations, or 
altered crown contours as these factors can 
have a marked effect on the respective 
pulps. 
 Consistency- Change in the consistency of 
hard dental tissues is related to the 
presence of caries, external and internal 
resorption
The visual and tactile 
inspection is usually carried 
out with a mouth mirror, 
explorer and a periodontal 
probe under dry conditions 
with good illumination source. 
A general visual examination 
of the entire mouth should be 
made to ascertain whether the 
tooth requiring treatment is a 
strategic tooth
Before percussing the involved tooth, instructions 
are to be given to the patient to raise his/ her 
hand or make an audible sound in order to let the 
clinician know when and whether the tooth feels 
“TENDER”, “DIFFERENT” or painful on 
percussion. 
Before percussing the teeth with the handle of the 
instrument (a mouth mirror etc), the quadrant of 
the involved tooth is percussed using the index 
finger with quick blows of low intensity. 
The teeth should be tapped (with the index finger) 
in a random fashion so that the patient cannot 
“anticipate” when the tooth will be percussed 
When no response is elicited on digital 
percussion, then the handle of an instrument is to 
be used/employed.
Percussion is done in both vertical and horizontal 
directions. 
Change the sequence of percussion in successive 
tests to eliminate bias. 
The force of percussion should only be strong enough 
for the patient to differentiate between a sound tooth 
and a tooth with inflamed periodontal ligament. 
The proprioceptive fibers in an inflamed periodontal 
ligament will, when percussed, help the patient and the 
clinician locate the source of pain.
Positive response is indicative of periodontitis (pericementitis) 
which could be due to: 
 Teeth undergoing rapid orthodontic movement. 
 High points in recent restorations. 
 Lateral periodontal abscess. 
 Partial/total Pulpal necrosis. 
Negative response may be seen in cases of 
 Chronic periapical inflammation 
 Usually 
* Dull note- Signifies abscess formation. 
* Sharp note- denotes Inflammation. 
* Metallic note- Ankylosis.
Employs the usage of the (index finger) 
fingertip, supplemented with a light digital 
pressure to examine tissue consistency 
and pain response. 
The importance of this test other than as 
an aid in locating the swelling over an 
involved tooth, is in determining the 
following: 
 Whether the tissue is fluctuant and 
enlarged sufficiently for incision and 
drainage. 
 The presence, intensity and location of 
pain. 
 The presence and location of 
Adenopathy 
 The presence of bony Crepitus.
To evaluate the integrity of the 
attachment apparatus 
surrounding the tooth. 
Moving the involved tooth 
laterally in socket using handles 
of two instruments or more 
preferably using two index 
fingers. 
The test for Depressibility is 
similar and is performed by 
applying pressure in an apical 
direction on the occlusal/incisal 
aspect of tooth and observing 
vertical movement if any.
GROSSMAN AND COHEN: 
GRADE I. [First degree] - Noticeable/ barely discernable movement of the 
teeth within its sockets. 
GRADE-II. [Second degree] - .Lateral/ horizontal mobility within a range of 
1 mm or less. 
GRADE III [Third degree]-Movement greater than 1 mm or when the tooth 
can be depressed into the socket. 
MILLER: 
 0 - Non mobile/ mobility within physiologic limits. 
 1 - Mobility within range of 0-0.5mm. 
 2 - Mobility within range of 0.5-1.5mm with lateral movements. 
 3 - Mobility more than 1.5 mm with lateral movements and can be 
Intruded/depressed into the socket.
Periodontal examination- a must 
Furcation involvement. 
A lateral canal - portal of entry for 
toxins- Pulpal degeneration. 
Thermal and electric pulp tests 
must be performed along with 
periodontal examination to 
distinguish between disease of 
Pulpal and Periodontal origin.
Assess the state of the pulp. 
Based on the Hydrodynamic Theory as postulated by Brannstrom (1963). 
 Heat Test 
 Cold Test 
A response to cold - a vital pulp regardless of whether it is normal or 
abnormal- Grossman. 
A heat test does not confirm vitality. 
An abnormal response to a heat test however exhibits presence of a 
pulpal or peri apical disorder requiring endodontic treatment. 
when a reaction to cold occurs the patient can quickly point out to the 
painful tooth, unlike in a heat test situation where the response could be 
localized, diffused or even referred to a different site- Grossman. 
The results should be co-related with other tests to ensure validity.
Explain the manner and procedure to the patient and 
also the kind of sensation he/she may experience. 
First on control teeth- placing the stimuli on the Inciso- 
Labial (anterior) surface or the Occluso-Buccal 
(posterior) surface. 
 Guides the clinician to evaluate the difference in 
response, the affected tooth provides. 
 Helps the patient understand better the nature of the 
stimulus he/she would experience. 
Exposed dentinal surfaces and restored surfaces should 
be avoided.
Isolation of the quadrant to be tested 
The preferred temperature, however, for 
performing a heat test (according to Cohen) is 
65.5ºC or 150F.
The heat test can be performed using different techniques 
such as: 
1. Hot air 
2. Hot water 
3. A hot burnisher 
4. Hot gutta-percha 
5. Hot compound 
6. Polishing of crown with a rubber cup
Isolating the quadrant with the tooth 
to be tested. 
Cold application can be performed in 
any of the following ways viz. 
A stream of cold air from a 3-way 
syringe directed against the crown of 
previously dried tooth. 
Use of ethyl chloride spray (which 
evaporates rapidly ) absorbing heat 
and cooling the tooth surface (- 
55ºC). 
Ice Stick 
CO2 snow
The patient’s response to heat and cold tests are identical 
because the neural fibers in the pulp transmit only the sensation of 
pain (Hydrodynamic theory -Brannstorm). 
There are four possible reactions, that the patient may experience, 
1. No response - may be non vital or vital but giving a false-negative 
response due to excessive calcifications, immature apex, 
recent trauma, patient medication etc. 
2. Painful response- which subsides when stimulus is removed 
from the tooth- Reversible- pulpitis. 
3. Moderate, transient response- Normal. 
4. Painful response- which lingers after removal of stimulus- 
Irreversible Pulpitis.
•Bender IB et al (1989), J Am. Dent. Assoc : 118; 305-310 
•Pulpal Diagnosis, Endodontic Topics 2003, 5, 12-25 
•Jack Lin et al (2007) JOE ; 33: 11
Historically, the E.P. tester has been used in dentistry 
as early as 1867. 
Designed to stimulate a response by electrical 
excitation of the neural elements within the pulp. 
Does not provide any information regarding the 
vascular supply to the tooth. 
Considered advantageous when compared with the 
thermal tests since the quantitative readings are 
obtained which can be compared with that of a later 
test (when conducted).
Test should be first described to the patient . 
Teeth to be tested should be isolated with cotton rolls, saliva 
ejector and air dried. 
Check the E.P. tester for proper functioning. 
Apply an electrolyte on the tooth surface (Nicholls-colloidal 
graphite, Grossman-tooth paste). 
Avoid contact of the electrolyte or electrode with any restorations 
or the adjacent gingival tissue as this could lead to a false 
response. 
Retract the patient’s cheek or lip with free hand, away from the 
tooth electrode.
* Bender IB et al (1989), J Am. Dent. Assoc : 118; 305- 
310 
* Pulpal Diagnosis, Endodontic Topics 2003, 5, 12-25
Intensity of stimulus is comfortable to the patients. 
The digital display of many E.P.Testers provide 
instant, easy and reliable information. 
In some E.P. Testers, a red indicator light flashes 
on and off when maximum stimulus is reached. 
Gives a quantitative reading and can be compared 
with the normal reading of control tooth.
Cannot be used on patients having cardiac pace maker. 
Some E.P.T equipments are very expensive. 
E.P.T is not useful for recently erupted teeth with immature 
apex. This may be because the relationship between the 
odontoblasts and the nerve fibers of the pulp has yet to 
develop (Nicholls). 
Recently traumatized teeth cannot be tested. 
No indication is given regarding state of the vascular supply 
which would give a more reliable measure of the vitality of 
the pulp. 
Readings from posterior teeth with partially vital pulps may 
be misleading.
A. False Positive Responses: When the pulp is necrotic but 
patient gives a positive response. 
B. False Negative Responses: When the pulp is vital, but the 
patient is unresponsive to the E.P.T. 
Reasons for False Positive Response: 
 Conductor / electrode in contact with a metallic restoration or 
gingiva along with the current to reach the attachment apparatus. 
 Patient anxiety. 
 Liquefaction necrosis-This may conduct the current to the 
attachment apparatus and the patient may slowly raise his hand 
near the highest range. 
 Failure to isolate or dry the teeth 
 In multi-rooted teeth where the pulp may be partially necrotic.
Reasons for a False Negative Response: 
 Patient heavily pre-medicated with analgesics, narcotics, 
alcohol, 
tranquilizers. 
 Inadequate contact with enamel. 
 Recently traumatized tooth. 
 Excessive calcification in the canal. 
 Recently erupted tooth with an immature apex. This 
according to Nicholls, may be because the relationship 
between the odontoblasts and nerve fibers of the pulp has 
yet to develop. 
The results obtained / tabulated through the E.P. T 
conducted should not be thoroughly relied upon. These 
results should be co-related with those obtained with other 
vitality tests such as the thermal tests etc.
Laser Doppler flowmetery 
Pulse Oximetery
Restricted to patients who are in pain at the time of the test and 
when the usual tests have failed to help identify or localize the 
offending tooth. 
The objective is to anesthetize a single tooth at a time until the 
pain disappears and is localized to specific tooth. 
infiltrate the posterior most tooth in the suspected zone. 
If pain persists anesthetize the next tooth mesial to it and continue 
to do so until the pain disappears. 
If the source of pain cannot be differentiated ie. ,maxillary / 
mandibular, then mandibular block is implemented. 
further localization of the affected tooth is done by an 
intraligamentary injection, once the anesthetic has spent itself. 
one of the last resorts in localizing the offending tooth.
last resort 
The cavity is prepared by drilling through DEJ of an 
unanesthetized tooth at a slow speed and without a water coolant. 
Sensitivity and pain elicited by the patient is an indication of the 
pulp vitality. 
A Sedative cement is then placed in the prepared cavity and the 
search for the cause of pain may be continued. 
On the contrary, if no pain/sensitivity is recorded, the cavity 
preparation may be continued until the pulp chamber is reached 
and if the pulp is noticed to be necrotic, routine endodontic 
treatment could be performed.
Light from a fiberoptic is 
applied from the buccal 
surface to illuminate the 
tooth to detect the 
fractured lines when 
present
An orangewood 
stick is placed on 
the occiusal/incisal 
aspect (on each 
cusp in case of 
posteriors) of the 
tooth and the 
patient is asked to 
bite. 
To identify the 
fractured tooth 
/cracked tooth 
syndrome.
1. Remove the filling from the suspected tooth and 
place 2% Iodine in the cavity preparation. 
* The iodine stains the fracture line dark. 
2. Mix a dye with zinc-oxide eugenol and place it in the 
cavity preparation after filling has been removed. 
* The dye will seep out and color the fracture line. 
3. Have a patient chew a disclosing tablet after taking 
out the filling in the suspected fractured tooth. 
* The line will be stained.
Can localize the 
endodontic lesion to the 
specific tooth. 
Aids in the differential 
diagnosis between a 
periodontal and an 
endodontic lesion. 
Placing a gutta percha 
point through the 
sinus/fistula tract and 
take a radiograph.
 Provides information on 
the extent of caries in to 
the pulp 
 No. of root canals and 
accessories 
 The course & shape of 
the canals 
 Length of the root 
 Calcifications 
 Resorptions 
 PDL status
 Nature of periapical 
area & alveolar bone 
 Root fractures 
 Differentiation of 
pathosis 
 Location of 
perforations
 Post obturation 
evaluation 
 Evaluate healing 
after RCT 
 Medico legal 
records
State of pulpal health can not 
be ascertained 
P/A pathology is evident only 
after much destruction(33%) 
Vertical root fracture can not 
be diagnosed 
Bony trabculae misinterpreted 
for horizontal root # 
Extend of caries is usually 
less than the actual extent as 
is true for P/A pathology
Xeroradiography 
Radiovisiography (RVG) 
Cone beam computerized tomography (CBCT) 
Magnetic Resonance Imaging (MRI) 
Ultrasound imaging
REFERENCES: 
1. COHEN-Pathways of the Pulp (Ninth Edition) 
2. GROSSMAN- Endodontjc Practice (Eleventh Edition) 
3. INGLE-Endodontjcs (Fourth Edition) 
4. WEINE-Endodontic Therapy (Fifth Edition) 
5. Technical equipment for assessment of dental pulp status(Endodontic 
Topics 2004, 7, 2–13). 
6. Pulpal diagnosis (Endodontic Topics 2003, 5, 12–25). 
7. Classification, diagnosis and clinical manifestations of apical 
periodontitis(Endodontic Topics 2004, 8, 36–54)

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Diagnostic procedures in endodontics

  • 1. Asallam Alekkum Dr. Gaurav Garg, Lecturer College of Dentistry, Al Zulfi
  • 3. Diagnosis is a process of determining the nature of a disease. very important for proper treatment. Differential diagnosis is the process of differentiating between similar diseases. For correct differential diagnosis:  Proper knowledge of the disease  Skill and Art on how to apply proper diagnostic methods
  • 4. A good clinician has to be a good diagnostician. Qualities of a good diagnostician- Knowledge Interest Intuition Curiosity Patience
  • 5. As stated by Grossman, are phenomenon or signs of a departure from the normal, and are indicative of an illness. Types: 1. Subjective Symptoms are those which are experienced and reported to the clinician by the patient. 2. Objective Symptoms are those, which are obtained by the clinician through various tests.
  • 6. Lingering tooth sensitivity to cold liquids. Lingering tooth sensitivity to hot liquids. Tooth sensitivity to sweets. Tooth pain to biting pressure. Tooth pain that is referred from a tooth to another area, such as the neck, temple, or the ear. Spontaneous toothache, such as that experienced while reading a magazine, watching television, etc. Constant or intermittent tooth pain. Severe tooth pain. Throbbing tooth pain. Tooth pain that may occur in response to postural changes, such as when going from a standing to a reclining position.
  • 7. Good case history:  Chief complaint  Past medical history  Past dental history  Also includes vital signs, history of presenting illness. • It is essential to gather collective information regarding signs, symptoms, and history for a successful outcome of any treatment procedure. Thorough clinical examination Relevant investigations / diagnostic tests
  • 8. Hardly exists any contraindication except:  Uncontrolled Diabetes  A very recent Myocardial infarction Aid the dental clinician in deciding whether a prior medical consultation or pre-medication (chemo-prophylaxis) would be required
  • 9. Cardiovascular:  High & moderate risk of Endocarditis  Pathologic heart Murmers  Hypertension  Unstable angina  Recent myocardial infarction  Arrythmias  Poorly managed congestive heart failure. Pulmonary:  COPD  Asthma  Tuberculosis
  • 10. GI & Renal:  End stage renal disease , Hemodialysis  Viral hepatitis( B,C,D,E)  Alcoholic liver disease  Peptic ulcer disease Endocrine & Hematologic:  STDs, HIV & AIDS  Diabetes mellitus  Adrenal insufficiency  Hyperthyroidism  Pregnancy  Bleeding disorders  Cancer & leukemia  Osteoarthritis, rheumatoid arthritis, SLE. NeurologicDisorders
  • 11. Side effects of medications:  Stomatitis, xerostomia, petechiae, ecchymosis, lichenoid lesions & mucosal & gingival bleeding. Lymphoma or Tuberculous involvement of cervical & submandibular lymph nodes Immunocompromised & patients with uncontrolled Diabetes Patients with iron deficiency anaemia, pernicious anemia & leukemia frequently exhibits paraesthesia of oral soft tissues complicating making a diagnosis when other dental pathosis is also present in the same area of the oral cavity.
  • 12. Sickle cell anemia has the complicating factor of bone pain, which mimics odontogenic pain & loss of trabecular bone pattern on radiographs which can be confused with radiographic lesions of endodontic origin. Multiple myeloma can result in unexplained mobility of teeth. Radiation therapy to head & neck region can result in increased sensitivity of teeth & osteoradionecrosis Trigeminal neuralgia, referred pain from cardiac angina, and multiple sclerosis can also mimic dental pain. Acute maxillary Sinusitis- a condition that may mimics tooth pain in maxillary posterior quadrant. In this situation the teeth in the quadrant will be extremely sensitive to cold & percussion, thus mimicking the signs & symptoms of Pulpitis.
  • 13. Chief complaint is the best starting point for a correct diagnosis PAIN is one of the most common chief complaints encountered
  • 14. In order to attain a detailed knowledge regarding pain, following questions may be necessary: 1. Type of pain :  Grossman has stated Pulpal pain to be of the following two varieties :  Sharp, piercing and lancinating -- a painful response usually associated with the excitation of the A-DELTA nerve fibers. This pain usually reflects REVERSIBLE state.  Dull, borinq, gnawing and excruciating-- a painful response usually associated with the excitation of C-nerve fibers.  Usually reflects an irreversible state of pulpitis.
  • 15. 2. The duration of pain:  When the pain is of a shorter duration (1 minute)-Reversible Pulpitis - Excellent chance of recovery without the need for endodontic treatment  Whereas when the pain is of a longer duration, it is considered to be Irreversible Pulpitis
  • 16. 3. The localization of pain:  Sharp piercing pain can usually be localized and responds to cold  Dull pain usually referred / spread over a larger area responds more abnormally to heat  Patients may report that their dental pain is exacerbated while lying down or bending over  This occurs because of the increase in blood pressure to the head, which therefore increases the pressure on the confined pulp
  • 17. 4. Factors which provoke/ relieve pain:  Response to a provoking factor (e.g. on mastication) indicates pulp vitality, but stimulation causing extended severe pain suggests irreversible pulpitis.  Thus pain, which is recorded as the complaint is considered to conclude an acute or chronic, reversible or irreversible condition of the pulp.
  • 18. Characterized by pain which is of a :-  Shorter duration  Localized  May be piercing/ lancinating in nature  More responsive to cold than heat  Caused by a specific irritant & disappears as soon as it is removed
  • 19.  Abnormal dental pain, which responds to heat  Which occurs on changing the position of the head, awakening the patient from sleep  Dull pain of Longer duration, which occurs during mastication in a Cariously exposed tooth
  • 20. History Slight sensitivity or occlusal pain Constant or intermittent pain Pain Momentary & immediate, sharp in nature & quickly disappears thereafter Continuous, Delayed onset, throbbing, persists for minutes to hours after removal of stimulus Location of pain May be localized & is not reffered Pain is not localized. If it is localized, its only after periapical involvement. Pain is reffered. Lying down No difference Pain increases Thermal test Responds Marked & prolonged E.P.T. Early response Early, delayed or mixed response Percussion Negative Negative in early stages. Later positive when periapex is involved Radiography Negative May show widening of PDL space
  • 21.
  • 22.
  • 23. The clinician should look for:  Facial asymmetry  Localized swellings  Lymphadenopathy  Changes in color, bruises/ scars, similar signs of disease, trauma or of any various treatment.
  • 24.
  • 25. Begins with a general evaluation of the oral structures. The occlusion is checked (for any derangements if any) The lips, cheeks, vestibules and mucosa are examined for any evident abnormalities. Several tests are employed in order to determine the condition of teeth and supporting structures.
  • 26.
  • 27. Simplest and the easiest of the diagnostic tests Acc. To Grossman the prime objective of visual & tactile inspector is evaluation of the “3 C’s” viz:  Color  Contour  Consistency of hard and soft tissues. Soft tissue: Color- the normal color of gingiva is coral pink. Change from this is easily visualized in inflammatory conditions. Contour- change in normal contour (eg, of scalloped gingiva) occurs with a swelling. Consistency- On inspection (most commonly gingiva) appears healthy, firm, resilient, while a soft, fluctuant or spongy tissue is more indicative of a pathological state.
  • 28. Hard tissue: A similar parameter of the visual and tactile inspection., ie, of the “C’s’is employed for the dental tissues as well.  Color- Normal teeth show life like translucency & sparkle that is missing in pulpless teeth which appear more or less opaque. Note: This discoloration however could be due to a variety of other reasons like old amalgam restorations, tetracycline stains etc.  Contour- This examination should also include the visualization of contours of affected teeth, such as fractured teeth, wear facets, improperly contoured restorations, or altered crown contours as these factors can have a marked effect on the respective pulps.  Consistency- Change in the consistency of hard dental tissues is related to the presence of caries, external and internal resorption
  • 29. The visual and tactile inspection is usually carried out with a mouth mirror, explorer and a periodontal probe under dry conditions with good illumination source. A general visual examination of the entire mouth should be made to ascertain whether the tooth requiring treatment is a strategic tooth
  • 30. Before percussing the involved tooth, instructions are to be given to the patient to raise his/ her hand or make an audible sound in order to let the clinician know when and whether the tooth feels “TENDER”, “DIFFERENT” or painful on percussion. Before percussing the teeth with the handle of the instrument (a mouth mirror etc), the quadrant of the involved tooth is percussed using the index finger with quick blows of low intensity. The teeth should be tapped (with the index finger) in a random fashion so that the patient cannot “anticipate” when the tooth will be percussed When no response is elicited on digital percussion, then the handle of an instrument is to be used/employed.
  • 31. Percussion is done in both vertical and horizontal directions. Change the sequence of percussion in successive tests to eliminate bias. The force of percussion should only be strong enough for the patient to differentiate between a sound tooth and a tooth with inflamed periodontal ligament. The proprioceptive fibers in an inflamed periodontal ligament will, when percussed, help the patient and the clinician locate the source of pain.
  • 32. Positive response is indicative of periodontitis (pericementitis) which could be due to:  Teeth undergoing rapid orthodontic movement.  High points in recent restorations.  Lateral periodontal abscess.  Partial/total Pulpal necrosis. Negative response may be seen in cases of  Chronic periapical inflammation  Usually * Dull note- Signifies abscess formation. * Sharp note- denotes Inflammation. * Metallic note- Ankylosis.
  • 33. Employs the usage of the (index finger) fingertip, supplemented with a light digital pressure to examine tissue consistency and pain response. The importance of this test other than as an aid in locating the swelling over an involved tooth, is in determining the following:  Whether the tissue is fluctuant and enlarged sufficiently for incision and drainage.  The presence, intensity and location of pain.  The presence and location of Adenopathy  The presence of bony Crepitus.
  • 34. To evaluate the integrity of the attachment apparatus surrounding the tooth. Moving the involved tooth laterally in socket using handles of two instruments or more preferably using two index fingers. The test for Depressibility is similar and is performed by applying pressure in an apical direction on the occlusal/incisal aspect of tooth and observing vertical movement if any.
  • 35. GROSSMAN AND COHEN: GRADE I. [First degree] - Noticeable/ barely discernable movement of the teeth within its sockets. GRADE-II. [Second degree] - .Lateral/ horizontal mobility within a range of 1 mm or less. GRADE III [Third degree]-Movement greater than 1 mm or when the tooth can be depressed into the socket. MILLER:  0 - Non mobile/ mobility within physiologic limits.  1 - Mobility within range of 0-0.5mm.  2 - Mobility within range of 0.5-1.5mm with lateral movements.  3 - Mobility more than 1.5 mm with lateral movements and can be Intruded/depressed into the socket.
  • 36. Periodontal examination- a must Furcation involvement. A lateral canal - portal of entry for toxins- Pulpal degeneration. Thermal and electric pulp tests must be performed along with periodontal examination to distinguish between disease of Pulpal and Periodontal origin.
  • 37. Assess the state of the pulp. Based on the Hydrodynamic Theory as postulated by Brannstrom (1963).  Heat Test  Cold Test A response to cold - a vital pulp regardless of whether it is normal or abnormal- Grossman. A heat test does not confirm vitality. An abnormal response to a heat test however exhibits presence of a pulpal or peri apical disorder requiring endodontic treatment. when a reaction to cold occurs the patient can quickly point out to the painful tooth, unlike in a heat test situation where the response could be localized, diffused or even referred to a different site- Grossman. The results should be co-related with other tests to ensure validity.
  • 38. Explain the manner and procedure to the patient and also the kind of sensation he/she may experience. First on control teeth- placing the stimuli on the Inciso- Labial (anterior) surface or the Occluso-Buccal (posterior) surface.  Guides the clinician to evaluate the difference in response, the affected tooth provides.  Helps the patient understand better the nature of the stimulus he/she would experience. Exposed dentinal surfaces and restored surfaces should be avoided.
  • 39.
  • 40. Isolation of the quadrant to be tested The preferred temperature, however, for performing a heat test (according to Cohen) is 65.5ºC or 150F.
  • 41. The heat test can be performed using different techniques such as: 1. Hot air 2. Hot water 3. A hot burnisher 4. Hot gutta-percha 5. Hot compound 6. Polishing of crown with a rubber cup
  • 42.
  • 43. Isolating the quadrant with the tooth to be tested. Cold application can be performed in any of the following ways viz. A stream of cold air from a 3-way syringe directed against the crown of previously dried tooth. Use of ethyl chloride spray (which evaporates rapidly ) absorbing heat and cooling the tooth surface (- 55ºC). Ice Stick CO2 snow
  • 44. The patient’s response to heat and cold tests are identical because the neural fibers in the pulp transmit only the sensation of pain (Hydrodynamic theory -Brannstorm). There are four possible reactions, that the patient may experience, 1. No response - may be non vital or vital but giving a false-negative response due to excessive calcifications, immature apex, recent trauma, patient medication etc. 2. Painful response- which subsides when stimulus is removed from the tooth- Reversible- pulpitis. 3. Moderate, transient response- Normal. 4. Painful response- which lingers after removal of stimulus- Irreversible Pulpitis.
  • 45. •Bender IB et al (1989), J Am. Dent. Assoc : 118; 305-310 •Pulpal Diagnosis, Endodontic Topics 2003, 5, 12-25 •Jack Lin et al (2007) JOE ; 33: 11
  • 46.
  • 47. Historically, the E.P. tester has been used in dentistry as early as 1867. Designed to stimulate a response by electrical excitation of the neural elements within the pulp. Does not provide any information regarding the vascular supply to the tooth. Considered advantageous when compared with the thermal tests since the quantitative readings are obtained which can be compared with that of a later test (when conducted).
  • 48. Test should be first described to the patient . Teeth to be tested should be isolated with cotton rolls, saliva ejector and air dried. Check the E.P. tester for proper functioning. Apply an electrolyte on the tooth surface (Nicholls-colloidal graphite, Grossman-tooth paste). Avoid contact of the electrolyte or electrode with any restorations or the adjacent gingival tissue as this could lead to a false response. Retract the patient’s cheek or lip with free hand, away from the tooth electrode.
  • 49. * Bender IB et al (1989), J Am. Dent. Assoc : 118; 305- 310 * Pulpal Diagnosis, Endodontic Topics 2003, 5, 12-25
  • 50. Intensity of stimulus is comfortable to the patients. The digital display of many E.P.Testers provide instant, easy and reliable information. In some E.P. Testers, a red indicator light flashes on and off when maximum stimulus is reached. Gives a quantitative reading and can be compared with the normal reading of control tooth.
  • 51. Cannot be used on patients having cardiac pace maker. Some E.P.T equipments are very expensive. E.P.T is not useful for recently erupted teeth with immature apex. This may be because the relationship between the odontoblasts and the nerve fibers of the pulp has yet to develop (Nicholls). Recently traumatized teeth cannot be tested. No indication is given regarding state of the vascular supply which would give a more reliable measure of the vitality of the pulp. Readings from posterior teeth with partially vital pulps may be misleading.
  • 52. A. False Positive Responses: When the pulp is necrotic but patient gives a positive response. B. False Negative Responses: When the pulp is vital, but the patient is unresponsive to the E.P.T. Reasons for False Positive Response:  Conductor / electrode in contact with a metallic restoration or gingiva along with the current to reach the attachment apparatus.  Patient anxiety.  Liquefaction necrosis-This may conduct the current to the attachment apparatus and the patient may slowly raise his hand near the highest range.  Failure to isolate or dry the teeth  In multi-rooted teeth where the pulp may be partially necrotic.
  • 53. Reasons for a False Negative Response:  Patient heavily pre-medicated with analgesics, narcotics, alcohol, tranquilizers.  Inadequate contact with enamel.  Recently traumatized tooth.  Excessive calcification in the canal.  Recently erupted tooth with an immature apex. This according to Nicholls, may be because the relationship between the odontoblasts and nerve fibers of the pulp has yet to develop. The results obtained / tabulated through the E.P. T conducted should not be thoroughly relied upon. These results should be co-related with those obtained with other vitality tests such as the thermal tests etc.
  • 54. Laser Doppler flowmetery Pulse Oximetery
  • 55.
  • 56. Restricted to patients who are in pain at the time of the test and when the usual tests have failed to help identify or localize the offending tooth. The objective is to anesthetize a single tooth at a time until the pain disappears and is localized to specific tooth. infiltrate the posterior most tooth in the suspected zone. If pain persists anesthetize the next tooth mesial to it and continue to do so until the pain disappears. If the source of pain cannot be differentiated ie. ,maxillary / mandibular, then mandibular block is implemented. further localization of the affected tooth is done by an intraligamentary injection, once the anesthetic has spent itself. one of the last resorts in localizing the offending tooth.
  • 57. last resort The cavity is prepared by drilling through DEJ of an unanesthetized tooth at a slow speed and without a water coolant. Sensitivity and pain elicited by the patient is an indication of the pulp vitality. A Sedative cement is then placed in the prepared cavity and the search for the cause of pain may be continued. On the contrary, if no pain/sensitivity is recorded, the cavity preparation may be continued until the pulp chamber is reached and if the pulp is noticed to be necrotic, routine endodontic treatment could be performed.
  • 58.
  • 59. Light from a fiberoptic is applied from the buccal surface to illuminate the tooth to detect the fractured lines when present
  • 60.
  • 61. An orangewood stick is placed on the occiusal/incisal aspect (on each cusp in case of posteriors) of the tooth and the patient is asked to bite. To identify the fractured tooth /cracked tooth syndrome.
  • 62. 1. Remove the filling from the suspected tooth and place 2% Iodine in the cavity preparation. * The iodine stains the fracture line dark. 2. Mix a dye with zinc-oxide eugenol and place it in the cavity preparation after filling has been removed. * The dye will seep out and color the fracture line. 3. Have a patient chew a disclosing tablet after taking out the filling in the suspected fractured tooth. * The line will be stained.
  • 63. Can localize the endodontic lesion to the specific tooth. Aids in the differential diagnosis between a periodontal and an endodontic lesion. Placing a gutta percha point through the sinus/fistula tract and take a radiograph.
  • 64.
  • 65.  Provides information on the extent of caries in to the pulp  No. of root canals and accessories  The course & shape of the canals  Length of the root  Calcifications  Resorptions  PDL status
  • 66.  Nature of periapical area & alveolar bone  Root fractures  Differentiation of pathosis  Location of perforations
  • 67.  Post obturation evaluation  Evaluate healing after RCT  Medico legal records
  • 68.
  • 69.
  • 70. State of pulpal health can not be ascertained P/A pathology is evident only after much destruction(33%) Vertical root fracture can not be diagnosed Bony trabculae misinterpreted for horizontal root # Extend of caries is usually less than the actual extent as is true for P/A pathology
  • 71.
  • 72.
  • 73. Xeroradiography Radiovisiography (RVG) Cone beam computerized tomography (CBCT) Magnetic Resonance Imaging (MRI) Ultrasound imaging
  • 74. REFERENCES: 1. COHEN-Pathways of the Pulp (Ninth Edition) 2. GROSSMAN- Endodontjc Practice (Eleventh Edition) 3. INGLE-Endodontjcs (Fourth Edition) 4. WEINE-Endodontic Therapy (Fifth Edition) 5. Technical equipment for assessment of dental pulp status(Endodontic Topics 2004, 7, 2–13). 6. Pulpal diagnosis (Endodontic Topics 2003, 5, 12–25). 7. Classification, diagnosis and clinical manifestations of apical periodontitis(Endodontic Topics 2004, 8, 36–54)