Cardiac Anatomy and Physiology
•Download as PPT, PDF•
47 likes•7,935 views
Presentation to participants undertaking the: Critical Care Transition Program at ACT Health, 2008
![Overview ,[object Object],[object Object]](data:image/gif;base64,R0lGODlhAQABAIAAAAAAAP///yH5BAEAAAAALAAAAAABAAEAAAIBRAA7)
Recommended
More Related Content
What's hot
What's hot (20)
Viewers also liked
Viewers also liked (20)
Similar to Cardiac Anatomy and Physiology
Similar to Cardiac Anatomy and Physiology (20)
More from Jamie Ranse
More from Jamie Ranse (20)
Cardiac Anatomy and Physiology
- 1. Cardiac Anatomy and Physiology
- 28. Assessment
- 29. Assessment
- 30. Assessmen t
- 31. Pneumothorax Myocardial Infarction Respiratory Infection Angina Musculoskeletal Pericarditis Aortic Dissection Trauma Anxiety Pulmonary Embolism Oesophageal Reflux / Spasm Causes of chest pain
- 33. Diabetes High Blood Pressure Physical Inactivity Over 40 Vascular Disease High Cholesterol Previous MI Obesity Smoking Family History Unhealthy Dietary Habits Risk Factors
- 37. lleregies A M P L E edications revious medical, surgical and family history ast meal vents Assessment
- 38. osition: Where is the Pain? P Q R S T A A A uality: What does the pain feel like? [sharp, dull, burning] adiation: Does the pain move anywhere? everity: Rate the pain on a scale between 0 and 10 iming: When did the pain start? Is it continuous? lleviating factors: What makes it better? ggravating factors: What makes it worse? ssociated symptoms: e.g., nausea / pins and needles Assessment
- 39. Inspect Palpate Percussion Auscultation Assessment
Editor's Notes
- 4 chambers Separated by septum and valves
- 4 chambers Separated by septum and valves
- 4 chambers Separated by septum and valves
- 4 chambers Separated by septum and valves
- RCA is dominant in 84% of people Dominant artery usually does not perfuse largest % of myocardium Arise from aorta left branches into two large vessels right branches into single vessel from aortic sinus Can dilate meeting increased demand
- Contributes approximately 30% of CO alterations in atrial contraction effect CO
- Pulse – pulse deficit listen to apex and feel radial – if there is a difference this indicates a pulse deficit
- S2 split – A2 = aortic valve closure, P2 = pulmonic valve closure. On inspiration, venous return to the heart is impeded and pulmonic valve closure is delayed resulting in a split sound. Can be normal in some people. Get patient to hold breath to hear this better S3 – left ventricular failure: and is caused by blood from the left atrium slamming into an already overfilled ventricle during early diastolic filling S4 - left ventricular hypertrophy: blood trying to enter a stiff, non-compliant left ventricle during atrial contraction
- Inspect skin colour, scars, etc.. Palpate pulse Percussion lungs / heart boarders Auscultation heart murmurs
- B 2 via HM or NRB C FBC: Hb anaemia, WCC infection UEC: electrolyte imbalance CP: coagulation ?PE CK / Troponin: muscle / cardiac enzymes ABG: ventilation / perfusion status F Observations 5mins apart G Analgesia / medications
- B 2 via HM or NRB C FBC: Hb anaemia, WCC infection UEC: electrolyte imbalance CP: coagulation ?PE CK / Troponin: muscle / cardiac enzymes ABG: ventilation / perfusion status F Observations 5mins apart G Analgesia / medications
- Anginine Vasodilator, decreases preload and afterload therefore decreasing the workload of the heart, dilates coronary arteries Aspirin Platelet aggregation inhibitor a study of 17,000 people showed a reduced re-infarction rate of 50% Morphine Analgesia, decreases anxiety, Clopidogrel Platelet aggregation inhibitor GTN Infusion Blood pressure control, reduces pain