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   Pain is a signal that the body has been
    damaged or something is wrong

   Reaction designed to protect you (makes you
    stop what you are doing what caused it

   Pain can either be acute or chronic
The Process
of Pain
Stimulus  Pain

receptors  Spinal

cord  Thalamus 

Cerebral cortex 

      “OUCH”
   Abnormality of the processing of pain by the
    neurologic system
   CRPS can be either type I (RSD) or type II
    (causalgia)
   Pain is disproportionate to the initial event
   An official diagnosis must first rule out any
    alternative diagnosis
   Frequently diagnosed associated with mild
    severe injuries/surgeries (commonly carpal tunnel
    release, Dupuytren’s release, and distal radial
    fracture)
   Can occur either before or after therapy referral
   Allodynia
   Hyperalgia
   Hyperpathia
   Swelling
   Stiffness
   Discoloration
   Abnormal hair/
    nail growth
   Hyperhydrosis
   Motor Dysfunction
   Bone Degeneration
CRPS Pain rated a 42
Most painful form of chronic pain
   Ask which areas are
    hypersensitive BEFORE
    touching the patient
   Pain assessment is
    important to determine
    client’s tolerance
   Postpone unnecessary
    tests to a time when they
    are not swollen, painful,
    and stiff
   Measuring edema: use
    warm water and as
    quickly as you can
   Phych eval
   Pain                                 Stress Loading
     Modalities (moist heat,              “load and carry”
       fluidotherapy, contrast           Joint Protection
       baths)                            Patient Education
   Edema                                    “To Improve, Move”
     Elevation with AROM,
       manual edema mobilization,
       compression, massage
   Sensation
     Desensitization
   Range of Motion
     PROM, stretching, blocking,
       tendon gliding, PNF patterns
   Splinting
     Resting position
No protocol works for all patients
  with CRPS. It is dependent on
  current pain level, symptoms,
   and tolerance (see handout)
OCCUPATIONAL THERAPY PHYSICAL THERAPY

i) to reduce clinical symptoms,    i) Increasing the degree of
and protect and support the        control over the pain and
affected limb in the most          improving the way the patient
functional and comfortable         copes with the syndrome
position by means of a splint.     ii) Extinguishing the source of
ii) to normalize sensitivity by    pain and treating any
carrying out an extensive          dysregulation
desensitization program            iii) Improving skills
iii) to encourage the functional
use of the limb within the pain
threshold.
iv) to encourage independence
   Communicate regarding progress/lack of progress
   Monitor symptoms and adjust treatment
    accordingly
   Communicate with therapist regarding goals
   Discuss maximal pain limits and which pain
    reduction techniques are most effective
   Record progress of home exercise program
   Provide adaptations and assistive devices for
    ADL’s and work related activities
   Modify/Adjust splints
Shows a trend that mirror therapy is effective with
 CRPS. Mirror therapy was shown to be effective in
 CRPS patients in Stage I and II but not effective in
  Stage III patients. It had an immediate analgesic
effect with a reduction in stiffness. In those patients
  which mirror therapy was not effective, all were
   lower extremity affected. 17 different outcome
     measures were used measuring symptoms,
functional levels, and the treatment itself. It is noted
  that Mirror therapy in CRPS II patients is worth
                 further exploration.

Results of these studies were not statistically analyzed
May be safe and effective
                                                          186 Patients
Assumes that avoiding use of a limb                        Referred
    due to pain will result in loss of
    function
“Graded exposure”
Discussion of possible pain increase
Traction & translation of joints                                  106 Patients Included
                                            80 Patients           4 Patients Stopped 2
Passive Stretching                           Excluded             Male 2Female
Functional use immediately after                                  2 arm/hand
                                                                  2 leg/foot
Desensitization
Max of 5 45 min sessions over 3
    months with evaluation of
                                         Arm/hand 39              Leg/foot 63
    treatment 3 months after last        patients                 patients
    treatment                            18 full recovery         31 full recovery
                                         19 partial recovery      27 partial recovery
Focuses on FUNCTIONAL                    2 patients lost to       5 no change
    improvement only                     follow-up
   Completely individual
   The sooner treatment begins, the quicker
    improvements are noted
   The longer treatment is delayed, the more
    likely it is to require long-term treatment
No, there is a lack of evidence in all areas of
CRPS and more research needs to be done
 to find the most effective treatments for
                these patients.
   Cooper, C. (2007). Fundamentals of hand therapy. Mosby-Elsevier: St. Louis, MO.
   Ek, J., Gijn, J., Samwel, H., Egmond, J., Klomp, F., & Dongen, R. (2009). Pain exposure
    physical therapy may be a safe and effective treatment for longstanding complex regional
    pain syndrome type 1: a case series. Clinical Rehabilitation, 23, 1059–1066. doi:
    10.1177/0269215509339875
   Ezendam, D., Bongers, R. & Jannik, M. (2009). Systematic review of the effectiveness of
    mirror therapy in upper extremity function. Disability and Rehabilitation, 31(26), 2135–
    2149. doi: 10.3109/09638280902887768
   Geertzen, J. & Harden, R. (2006). Physical and Occupational Therapies in Complex Regional
    Pain Syndrome Type I. Joumal of Neuropathic Pain & Symptom Palliation, 2(3), 51-55. doi:
    doi:10.1300/J426v02n03_11
   Kishner, S., Rothaermel, B., Munshi, S., Malalis, J. & Gunduz, O. (2011). Complex regional
    pain syndrome. Turkish Journal of Physical Medicine and Rehabilitation, 57, 156-164. doi:
    10.4274/tftr.09327
   Maihofer, C., Seifert, F., & Markovic, K. (2010). Complex regional pain syndromes: new
    pathophysiological concepts and therapies. European Journal of Neurology, 17, 649–660.
    doi: doi:10.1111/j.1468-1331.2010.02947.x
   Mos, M., Sturkenboom, M., & Huygen, F. (2009). Current understandings of complex
    regional pain syndrome. Pain Practice, 9(2), 86-99. doi: 10.1111/j.1533-2500.2009.00262.x
   Perez, R., Zollinger, P., Dijkstra, P., Thomassen-Hilgersom, I., Zuurmond, W., Rosenbrand,
    K. & Geertzen, J. (2010). REevseiadrche anrticclee based guidelines for complex regional
    pain syndrome type 1. BMC Neurology, 10(20), 1-14.

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Crps ppt

  • 2. Pain is a signal that the body has been damaged or something is wrong  Reaction designed to protect you (makes you stop what you are doing what caused it  Pain can either be acute or chronic
  • 3. The Process of Pain Stimulus  Pain receptors  Spinal cord  Thalamus  Cerebral cortex  “OUCH”
  • 4. Abnormality of the processing of pain by the neurologic system  CRPS can be either type I (RSD) or type II (causalgia)  Pain is disproportionate to the initial event  An official diagnosis must first rule out any alternative diagnosis  Frequently diagnosed associated with mild severe injuries/surgeries (commonly carpal tunnel release, Dupuytren’s release, and distal radial fracture)  Can occur either before or after therapy referral
  • 5. Allodynia  Hyperalgia  Hyperpathia  Swelling  Stiffness  Discoloration  Abnormal hair/ nail growth  Hyperhydrosis  Motor Dysfunction  Bone Degeneration
  • 6.
  • 7. CRPS Pain rated a 42 Most painful form of chronic pain
  • 8. Ask which areas are hypersensitive BEFORE touching the patient  Pain assessment is important to determine client’s tolerance  Postpone unnecessary tests to a time when they are not swollen, painful, and stiff  Measuring edema: use warm water and as quickly as you can  Phych eval
  • 9. Pain  Stress Loading  Modalities (moist heat,  “load and carry” fluidotherapy, contrast  Joint Protection baths)  Patient Education  Edema  “To Improve, Move”  Elevation with AROM, manual edema mobilization, compression, massage  Sensation  Desensitization  Range of Motion  PROM, stretching, blocking, tendon gliding, PNF patterns  Splinting  Resting position
  • 10. No protocol works for all patients with CRPS. It is dependent on current pain level, symptoms, and tolerance (see handout)
  • 11. OCCUPATIONAL THERAPY PHYSICAL THERAPY i) to reduce clinical symptoms, i) Increasing the degree of and protect and support the control over the pain and affected limb in the most improving the way the patient functional and comfortable copes with the syndrome position by means of a splint. ii) Extinguishing the source of ii) to normalize sensitivity by pain and treating any carrying out an extensive dysregulation desensitization program iii) Improving skills iii) to encourage the functional use of the limb within the pain threshold. iv) to encourage independence
  • 12. Communicate regarding progress/lack of progress  Monitor symptoms and adjust treatment accordingly  Communicate with therapist regarding goals  Discuss maximal pain limits and which pain reduction techniques are most effective  Record progress of home exercise program  Provide adaptations and assistive devices for ADL’s and work related activities  Modify/Adjust splints
  • 13. Shows a trend that mirror therapy is effective with CRPS. Mirror therapy was shown to be effective in CRPS patients in Stage I and II but not effective in Stage III patients. It had an immediate analgesic effect with a reduction in stiffness. In those patients which mirror therapy was not effective, all were lower extremity affected. 17 different outcome measures were used measuring symptoms, functional levels, and the treatment itself. It is noted that Mirror therapy in CRPS II patients is worth further exploration. Results of these studies were not statistically analyzed
  • 14. May be safe and effective 186 Patients Assumes that avoiding use of a limb Referred due to pain will result in loss of function “Graded exposure” Discussion of possible pain increase Traction & translation of joints 106 Patients Included 80 Patients 4 Patients Stopped 2 Passive Stretching Excluded Male 2Female Functional use immediately after 2 arm/hand 2 leg/foot Desensitization Max of 5 45 min sessions over 3 months with evaluation of Arm/hand 39 Leg/foot 63 treatment 3 months after last patients patients treatment 18 full recovery 31 full recovery 19 partial recovery 27 partial recovery Focuses on FUNCTIONAL 2 patients lost to 5 no change improvement only follow-up
  • 15. Completely individual  The sooner treatment begins, the quicker improvements are noted  The longer treatment is delayed, the more likely it is to require long-term treatment
  • 16. No, there is a lack of evidence in all areas of CRPS and more research needs to be done to find the most effective treatments for these patients.
  • 17.
  • 18. Cooper, C. (2007). Fundamentals of hand therapy. Mosby-Elsevier: St. Louis, MO.  Ek, J., Gijn, J., Samwel, H., Egmond, J., Klomp, F., & Dongen, R. (2009). Pain exposure physical therapy may be a safe and effective treatment for longstanding complex regional pain syndrome type 1: a case series. Clinical Rehabilitation, 23, 1059–1066. doi: 10.1177/0269215509339875  Ezendam, D., Bongers, R. & Jannik, M. (2009). Systematic review of the effectiveness of mirror therapy in upper extremity function. Disability and Rehabilitation, 31(26), 2135– 2149. doi: 10.3109/09638280902887768  Geertzen, J. & Harden, R. (2006). Physical and Occupational Therapies in Complex Regional Pain Syndrome Type I. Joumal of Neuropathic Pain & Symptom Palliation, 2(3), 51-55. doi: doi:10.1300/J426v02n03_11  Kishner, S., Rothaermel, B., Munshi, S., Malalis, J. & Gunduz, O. (2011). Complex regional pain syndrome. Turkish Journal of Physical Medicine and Rehabilitation, 57, 156-164. doi: 10.4274/tftr.09327  Maihofer, C., Seifert, F., & Markovic, K. (2010). Complex regional pain syndromes: new pathophysiological concepts and therapies. European Journal of Neurology, 17, 649–660. doi: doi:10.1111/j.1468-1331.2010.02947.x  Mos, M., Sturkenboom, M., & Huygen, F. (2009). Current understandings of complex regional pain syndrome. Pain Practice, 9(2), 86-99. doi: 10.1111/j.1533-2500.2009.00262.x  Perez, R., Zollinger, P., Dijkstra, P., Thomassen-Hilgersom, I., Zuurmond, W., Rosenbrand, K. & Geertzen, J. (2010). REevseiadrche anrticclee based guidelines for complex regional pain syndrome type 1. BMC Neurology, 10(20), 1-14.

Notas del editor

  1. Pain starts at the source of an injury or inflammation and the body's automatic response is to stimulate pain receptors and they release chemicals. These chemicals, carrying the message “Ouch, that hurts,” go to the spinal cord. The spinal cord then carries the message from its receptors all the way up to the brain, where it is received by the thalamus and sent to the cerebral cortex, the part of the brain that processes the message. Your brain perceives that pain, and sends the pain message back to the area of your body that hurts
  2. Risk factor age 20-35/women more than menType I CRPS develops after an initiating noxious event Type II develops after a nerve injuryClients may be referred to OT/PT for associated diagnosis and develop CRPS during course of treatment or the may be originally referred because CRPS is suspected. Has no apparent cause, but can be provoked by inappropriate and aggressive medical or therapeutic treatment.Diagnosis has no one single test, but thermography and xray can be helpful
  3. Allodynia- pain from sources that don’t typically cause painHyperalgia- increased response to painful stimuliHyperpathia- pain that continues after stimulus is removes (worse distally, asymmetrical)Swelling can become permanent, thick, and can lead to joint stiffnessStiffness including contractures and nodulesDiscoloration usually cyanotic in color, mottling, or redness, with abnormal temperatureHyperhydrosis- abnormal sweating along nerve distribution or atypical placesMotor dysfunction can include tremor, dystonia, increased muscle tone, loss of strength and endurance
  4. Rule out The differential diagnosis includes but is not limited to:Acute fracture or trauma Blood clot or DVT CellulitisChronic vascular insufficiency Fibromyalgia Septic arthritis Septic tenosynovitisScleroderma Peripheral neuropathy plexitisAllergic reaction Localized joint inflammation Tendonitis Bursitis Pain assessment: location, type, aggravating factors, alleviating factors, duration, limitations due to painEdema: cold water is not tolerated well being in the dependant position for long periods of time can increase swellingStage 1 (acute) less than 3 monthsStage 2 (dystrophic) 3-6 monthsStage 3 (atrophic)- after 6 monthsOften recommended for psych evals if any of the signs are present
  5. Fluidotherapy (with lowered temp of 98 degrees)Ice is not often tolerated wellDesensitization (5-6 times per day as tolerated) paired with TENS to help with pain if not tolerated wellSplinting in resting position at night can help the limb to rest and decrease potential contractures wrist in neutral, MCP flexion and IP extension Stress loading is the most recognized therapeutic treatment for CRPS Starts with scrubbing a table or rolling a ball for 3-5 min (3x/day) up to 10 min Follow with distraction (carrying weights (1lb-5lbs for up to 10 min)Joint protection and energy conservation assistive devicesPatient Education educate about symptoms and symptom management encourage use as much as possible encourage with examples of progress to ensure they don’t dwell on limitations
  6. OT (found to be effective for CRPS patients)i) The practitioner will decide whether the patient should bemeasured for a supportive splint. This could be a resting splint forthe entire hand and forearm, or for part thereof (wrist or thumb,for instance). Patients are instructed individually on how to wearthe splint. The aim of wearing an orthotic device is to minimizesymptoms and prevent overstrainiii) Various play activities, dexterity techniques and/or everyday activitiesiv) particularly with regard to self-care, productivity and relaxation. The strategies can be targeted at restoring the necessary skills, at learning to do things in another way (with one hand, for instance), or at advising the patient on devices he or she could use or sources of additional support and care that are availablePT (found to be effective for CRPS patients)i) giving him or her information and support (recording anddiscussing a program of daily activities) or relaxation exercisesii) for example by performing exercises to attenuate pain, desensitization, or the use of a sling or splintiii) , for example by practicing compensatoryskills, training skills, and posture and movement instruction. Thepatient's need for (and interest in) help will determine thespecific exercises carried out at a later stage
  7. Lit review identified 717 studies and 15 studies fit criteria. (6 focused on mirror therapy with CRPS I & II)
  8. 1st- 106 patients functional improvement in 95 full functional improvement in 49 reduction in pain in 75 increase in pain with increase in function in 23 4 dropped out due to painEffective only when neglecting pain“graded exposure” a sort of “happy medium” between “no pain, no gain” and aggressive therapyAfter patients had failure with all other interventions and analgesics, pain meds were stopped and patients agreed to this therapy after in depth discussion of what was involved and the risks. no response to pain by therapists functional use after therapy (walking with arms swinging, opening bottle,) Lessened use of mobility devices the week afterWhen the pain is ignored it leads to the shrinkage of the somatotopic areas of the extremity in the somatosensory cortexFUNCTIONAL improvement only, not pain
  9. Early diagnosis ( <3 mo.) with PROPER treatment, success rate is highest, the best prognosisIf left untreated, can lead to lifetime of severe, intractable, chronic painFirst 3-6 months after onset: 80-90% recovery rate6 months to 2 years 70-80%, after 2 years: 20%