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POSTPARTUM
HEMORRHAGE
DEFINITION
 Hemorrhage occurring after the
delivery of the baby
 Hemorrhage occurring within 24
hours of delivery – Primary
postpartum hemorrhage
 Hemorrhage occurring anytime
after 24 hours of delivery- Secondary
postpartum hemorrhage
PRIMARY POSTPARTUM HEMORRHAGE
 Blood loss of more than 500ml from the genital
tract in the first 24 hours of childbirth.
 For caesarean section, a blood loss more than
1000ml is considered significant.
Incidence of about 5% of all deliveries.
 A more practical definition is a haematocrit drop of
10% or a hemorrhage that requires immediate
transfusion.
CAUSES
1 . Atonic postpartum hemorrhage
2 . Traumatic postpartum hemorrhage
3 . Coagulopathy
4 . Retained placenta and placental fragments
5 . Morbidly adherent placenta
6 . Uterine inversion
ATONIC POSTPARTUM HEMORRHAGE
 Most common cause (90%)
 Bleeding occurs because the blood vessels have
not been obliterated by contraction and
retraction of the uterine muscle fibres.
PREDISPOSING FACTORS
 Grand multiparity
 Over distended uterus due to multiple
pregnancy hydramnios or macrosomia
 Previous history of postpartum hemorrhage
 Antepartum hemorrhage
 Fibroids of uterus
 General malnutrition
 Prolonged labour leading to uterine exhustion
 Precipitate labour
TRAUMATIC POSTPARTUM
HEMORRHAGE
 It occurs due to genital tract injuries.
 Include lacerations of the cervix , vagina and
perineum
 Vulvovaginal hematomas may be a concealed
cause
PREDISPOSING FACTORS
 Instrumental delivery
 Vaginal birth after caesarian
 Face to pubis delivery
 Precipitate labour
 Macrosomia
COAGULOPATHY
 Disseminated intravascular coagulation and
hypofibrinogenemia should be considered in all
patients at high risk for coagulopathy.
PREDISPOSING FACTORS
• Abruption
• Sepsis
• Intrauterine death
• Amniotic fluid embolism
PREVENTION OF PPH
 Anemia should be corrected in the antenatal
period.
 In high risk patients PPH should be anticipated
and institutional delivery should be arranged.
 Blood should be arranged in high risk patients
 Use of partogram during labour will help to
avoid prolonged labour.
 Dehydration should be promptly corrected
 Premature attempts to express a placenta that
has not yet seperated are to be avoided.
 After delivery completeness of placenta is
checked for.
 The genital tract should be exposed in case of
instrumental delivery.
 Oxytocin infusion can be continued and vital
signs monitored closely
 Every hospital should have a protocol for
managing PPH
SECONDARY POSTPARTUM
HEMORRHAGE
• Hemorrhage occurring after the first 24 hours of
delivery and within 6 weeks
AETIOLOGY
• Sepsis
• Retained placental fragments
• Poor healing of the uterine incisions in previous
caesarian section.
• Choriocarcinoma
SHEEHAN SYNDROME
• Described by Sheehan and colleagues in 1983
• Rare consequence of severe PPH
• There is anterior pituitary necrosis and pituitary
failure
• There is failure of lactation, amenorrhoea,
hypothyroidism and adrenocorticalinsufficiency.
Postpartum hemorrhage1

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Postpartum hemorrhage1

  • 2. DEFINITION  Hemorrhage occurring after the delivery of the baby  Hemorrhage occurring within 24 hours of delivery – Primary postpartum hemorrhage  Hemorrhage occurring anytime after 24 hours of delivery- Secondary postpartum hemorrhage
  • 3. PRIMARY POSTPARTUM HEMORRHAGE  Blood loss of more than 500ml from the genital tract in the first 24 hours of childbirth.  For caesarean section, a blood loss more than 1000ml is considered significant. Incidence of about 5% of all deliveries.  A more practical definition is a haematocrit drop of 10% or a hemorrhage that requires immediate transfusion.
  • 4. CAUSES 1 . Atonic postpartum hemorrhage 2 . Traumatic postpartum hemorrhage 3 . Coagulopathy 4 . Retained placenta and placental fragments 5 . Morbidly adherent placenta 6 . Uterine inversion
  • 5. ATONIC POSTPARTUM HEMORRHAGE  Most common cause (90%)  Bleeding occurs because the blood vessels have not been obliterated by contraction and retraction of the uterine muscle fibres.
  • 6. PREDISPOSING FACTORS  Grand multiparity  Over distended uterus due to multiple pregnancy hydramnios or macrosomia  Previous history of postpartum hemorrhage  Antepartum hemorrhage  Fibroids of uterus  General malnutrition  Prolonged labour leading to uterine exhustion  Precipitate labour
  • 7. TRAUMATIC POSTPARTUM HEMORRHAGE  It occurs due to genital tract injuries.  Include lacerations of the cervix , vagina and perineum  Vulvovaginal hematomas may be a concealed cause
  • 8. PREDISPOSING FACTORS  Instrumental delivery  Vaginal birth after caesarian  Face to pubis delivery  Precipitate labour  Macrosomia
  • 9. COAGULOPATHY  Disseminated intravascular coagulation and hypofibrinogenemia should be considered in all patients at high risk for coagulopathy. PREDISPOSING FACTORS • Abruption • Sepsis • Intrauterine death • Amniotic fluid embolism
  • 10. PREVENTION OF PPH  Anemia should be corrected in the antenatal period.  In high risk patients PPH should be anticipated and institutional delivery should be arranged.  Blood should be arranged in high risk patients  Use of partogram during labour will help to avoid prolonged labour.  Dehydration should be promptly corrected  Premature attempts to express a placenta that has not yet seperated are to be avoided.
  • 11.  After delivery completeness of placenta is checked for.  The genital tract should be exposed in case of instrumental delivery.  Oxytocin infusion can be continued and vital signs monitored closely  Every hospital should have a protocol for managing PPH
  • 12. SECONDARY POSTPARTUM HEMORRHAGE • Hemorrhage occurring after the first 24 hours of delivery and within 6 weeks AETIOLOGY • Sepsis • Retained placental fragments • Poor healing of the uterine incisions in previous caesarian section. • Choriocarcinoma
  • 13. SHEEHAN SYNDROME • Described by Sheehan and colleagues in 1983 • Rare consequence of severe PPH • There is anterior pituitary necrosis and pituitary failure • There is failure of lactation, amenorrhoea, hypothyroidism and adrenocorticalinsufficiency.