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Edwards
Edwards
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Want to learn about Williams Syndrome? Well...here is a slide show that tells you the cover story behind Williams Syndrome. Enjoy.
Williams Syndrome
Williams Syndrome
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Myzak
Want to learn about Williams Syndrome? Well...here is a slide show that tells you the cover story behind Williams Syndrome. Enjoy.
Williams Syndrome
Williams Syndrome
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Williams Syndrome
Myzak
Williams Syndrome susquenita
Williams Syndrome susquenita
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Zellweger Syndrome is one class of Cerebrohepatorenal syndromes. It is a rare hereditary disorder and caused by a gene mutation that affects the way peroxisomes function.
Zellweger's syndrome explained
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Based in the Bay Area, Insight Memory Care Choices is guided by licensed assisted living administrator Sandra Wallace. The Insight Memory Care Choices team provides client-driven solutions focused on ensuring that loved ones living with dementia find the communities best suited to their needs.
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Genetic factors are actually the main category of hearing losses. On top of that, hearing loss is considered the most common birth defect in the developed world.
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After leading an investment firm near Chicago, Leslie (Les) Goldstein is with a new Broker Dealer, Arete Wealth Management. Outside of his work as a financial fiduciary, Les takes on charitable projects, such as fundraising for two family members in wheelchairs who have Freidreich’s ataxia (FA). FA is a rare disease, with symptoms beginning from ages 5 to 18, that inhibits many types of muscular functioning and brings on other complications. About one American in 50,000 has FA. Symptoms range from losing coordination legs and arms and lack of energy to severe heart conditions and diabetes requiring insulin. Patients also may develop curvature of the spine, impaired hearing and vision, and slurred speech. However, the disease leaves mental capacity unaffected. The disease is genetic, caused by abnormal or mutated copies of the FXN gene, which hampers the essential energy production of cells, particularly nerve cells. A child only develops FA if both parents have this genetic defect. Currently, FA has no cure; care is limited to managing symptoms. However, researchers supported by the nonprofit Freidreich’s Ataxia Research Alliance are pursuing some 20 avenues of inquiry.
Freidreich’s Ataxia Hinders Coordination, Muscle and Heart Function
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My tomorrow's presentation for my Pediatrics report, tackling Edward's syndrome, also known as Trisomy 13. :)
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Edward’s syndrome (trisomy 18)
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How can a digital marketing consultant help your business? In this resource we'll count the ways. 24 additional marketing resources are bundled for free.
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Zellweger Syndrome is one class of Cerebrohepatorenal syndromes. It is a rare hereditary disorder and caused by a gene mutation that affects the way peroxisomes function.
Zellweger's syndrome explained
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Hallermann–Streiff syndrome is a congenital disorder that affects growth, cranial development, hair growth and dental development. There are fewer than 200 people with the syndrome worldwide
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Based in the Bay Area, Insight Memory Care Choices is guided by licensed assisted living administrator Sandra Wallace. The Insight Memory Care Choices team provides client-driven solutions focused on ensuring that loved ones living with dementia find the communities best suited to their needs.
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Genetic factors are actually the main category of hearing losses. On top of that, hearing loss is considered the most common birth defect in the developed world.
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After leading an investment firm near Chicago, Leslie (Les) Goldstein is with a new Broker Dealer, Arete Wealth Management. Outside of his work as a financial fiduciary, Les takes on charitable projects, such as fundraising for two family members in wheelchairs who have Freidreich’s ataxia (FA). FA is a rare disease, with symptoms beginning from ages 5 to 18, that inhibits many types of muscular functioning and brings on other complications. About one American in 50,000 has FA. Symptoms range from losing coordination legs and arms and lack of energy to severe heart conditions and diabetes requiring insulin. Patients also may develop curvature of the spine, impaired hearing and vision, and slurred speech. However, the disease leaves mental capacity unaffected. The disease is genetic, caused by abnormal or mutated copies of the FXN gene, which hampers the essential energy production of cells, particularly nerve cells. A child only develops FA if both parents have this genetic defect. Currently, FA has no cure; care is limited to managing symptoms. However, researchers supported by the nonprofit Freidreich’s Ataxia Research Alliance are pursuing some 20 avenues of inquiry.
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Edward’s syndrome (trisomy 18)
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Down syndrome (DS or DNS), also known as trisomy 21, is a genetic disorder caused by the presence of all or part of a third copy of chromosome 21. It is typically associated with physical growth delays, characteristic facial features and mild to moderate intellectual disability. The average IQ of a young adult with Down syndrome is 50, equivalent to the mental ability of an 8- or 9-year-old child, but this can vary widely.
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In this lecture on circulatory shock, Dr. Faiza, an Assistant Professor of Physiology, delves into the profound implications of inadequate blood flow throughout the body, leading to tissue damage. The session begins by defining circulatory shock and elucidating its physiological causes, including decreased cardiac output, diminished blood volume, decreased vascular tone, obstruction to blood flow, excess metabolic rate, and abnormal perfusion patterns. Dr. Faiza categorizes shock into various types, such as cardiogenic shock, hypovolemic shock, neurogenic shock, obstructive shock, anaphylactic shock, and septic shock, and explores the pathophysiological basis of each. Furthermore, the lecture examines the stages of shock, from the non-progressive phase where compensatory mechanisms aim for full recovery to the progressive phase where shock worsens steadily without intervention, potentially leading to irreversible damage. Dr. Faiza discusses therapeutic interventions for shock, including replacement therapy, blood and plasma transfusion, sympathomimetic drugs, and other adjunctive treatments like head-down positioning, oxygen therapy, and glucocorticoids.
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Delve into valuable content elucidating the anatomy and physiology of the respiratory system, in line with the PCI syllabus for pharmacy and PharmD students.
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In this captivating lecture, Dr. Faiza, an esteemed Assistant Professor of Physiology, embarks on a comprehensive journey into the multifaceted realm of local blood flow regulation. With meticulous detail and clarity, she unravels the intricate mechanisms that govern the dynamic balance between tissue perfusion and vascular resistance, shedding light on both acute and chronic aspects of blood flow control. Beginning with an exploration of acute influences, Dr. Faiza elucidates how physiological factors such as metabolic demand, myogenic responses, and autoregulatory mechanisms orchestrate rapid adjustments in local blood flow. Students gain insight into the vasodilator theory, which elucidates the role of metabolic byproducts and vasoactive metabolites in promoting vasodilation, alongside the oxygen demand theory, which underscores the interplay between tissue oxygenation and nutrient demand. Delving deeper, the lecture delves into the fascinating realm of humoral control, where hormones and vasoactive substances wield significant influence over vascular tone. Dr. Faiza navigates through the intricacies of endothelial-derived factors like nitric oxide (NO) and histamine, alongside hormonal regulators such as angiotensin II and vasopressin. Through vivid explanations and illustrative examples, she unveils the intricate dance of vasodilation and vasoconstriction orchestrated by these biochemical messengers. Moreover, the lecture extends its gaze to the extrinsic control of blood flow, where sympathetic innervation exerts a profound impact on vascular dynamics. Dr. Faiza elucidates the role of sympathetic tone in modulating vascular resistance, highlighting its implications in diverse physiological and pathological scenarios. From skin perfusion to coronary circulation, students gain a nuanced understanding of how neural inputs shape local blood flow patterns. Enriched with clinical insights and contemporary research findings, this lecture equips students with a holistic understanding of blood flow regulation. By exploring concepts like active and reactive hyperemia, vascular remodeling, and collateral circulation, Dr. Faiza fosters a deeper appreciation for the adaptive responses of the cardiovascular system to changing physiological demands and pathological insults.
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These are simplified slides discussing the regulation of cardiac output and venous return. Learning objectives: 1. Comprehend the determinants of cardiac output and factors affecting cardiac output 2. Comprehend the factors affecting stroke volume and heart rate and total peripheral resistance 3. Identify the factors regulating venous return 4. Discuss the causes of high and low output cardiac failure 5. Enlist the functions of veins and recognise the significance of venous reservoirs Study Resources: 1. Chapter 20, Guyton and Hall Textbook of Medical Physiology, 14th edition 2. Chapter 30 and 32, Ganong’s Review of Medical Physiology, 26th edition 3. Chapter 10, Human Physiology by Lauralee Sherwood, 9th edition 4. Physiology, Cardiac Output - StatPearls https://www.ncbi.nlm.nih.gov/books/NBK470455/
Cardiac Output, Venous Return, and Their Regulation
Cardiac Output, Venous Return, and Their Regulation
MedicoseAcademics
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Shazia Iqbal 2024 - Bioorganic Chemistry.pdf
Circulatory Shock, types and stages, compensatory mechanisms
Circulatory Shock, types and stages, compensatory mechanisms
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ANATOMY AND PHYSIOLOGY OF RESPIRATORY SYSTEM.pptx
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Cardiac Output, Venous Return, and Their Regulation
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