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GUIDED BY : MAJ. SARAVANA ESWARI
CLINICAL TUTOR
PRESENTED BY : N/CDT PRIYANKA GIRI
IV YEAR BASIC B.SC NURSING
 Obstetrical emergencies are life-threatening
medical conditions that occur in pregnancy or during
or after labor and delivery.
 The main emergencies include-
I. Ectopic Pregnancy
II. Uterine Inversion
III. Obstetrical Shock
IV. Cord Prolepses
V. Amniotic Fluid Embolism
VI. Postpartum Hemorrhage
ECTOPIC
PREGNANCY
 “Any pregnancy where the fertilised ovum
gets implanted & develops in a site other
than normal uterine cavity”.
 Represent serious hazard to a woman’s
health and reproductive potential
 Requiring prompt recognition and early
intervention.
ECTOPIC PREGNANCY : is one in which
fertilized ovum is implanted & develops outside
normal uterine cavity
.
 Increased due to PID, use of IUCD, Tubal
surgeries, and ART
 Ranges from 1:25 to 1:250
 Average range is 1 in 100 normal pregnancies.
 Late marriages and late child bearing ->2%
 ART -> 5%
 Recurrence rate - 15% after 1st, 25% after 2
ectopics
 History of PID
 History of tubal ligation
 Contraception failure
 History of infertility
 IUD use
 Previous induced abortion
 Tubal reconstructive surgery
 Acute
 Unruptured
 Subacute
Acute eptopic pregnancy
 Fortunately less common (30%)
 Associated with cases of tubal rupture or tubal
abortion with massive intraperitoneal
hemorrhage.
 Classical triad is present in 50% of pt with rupture
ectopic.
 - PAIN:- most constant feature in 95% pt
- variable in severity and nature
 - AMENORRHOEA:- 60-80% of pt
- there may be delayed period or slight spotting at the
time of expected menses.
 - VAGINAL BLEEDING: - scanty dark brown
 Feeling of nausea,vomiting,fainting attack, syncope
attack(10%) due to reflex vasomotor disturbance.
 O/E:- patient is restless in agony, looks blanched,
pale, sweating with cold clammy skin.
Features of shock, tachycardia, hypotension.
 P/A:- abdomen tense, tender mostly in lower
abdomen, shifting dullness, rigidity may be
present.
 P/S:- minimal bleeding may be present
 P/V:- uterus may be bulky, deviated to opposite
side, fornix is tender, excitation pain on
movement of cervix.
POD may be full, uterus floats as if in water.
 PRINCIPLE:
“Resuscitation and laparotomy and not resuscitation
followed by laparotomy”.
 Antishock treatment:
• measures are to be taken energetically
• Simultaneous preparation for urgent laparotomy.
-ringer solution (crystalloid) is started
-arrangement for blood transfusion.
-Even if blood is not available laparotomy is to be
done desperately.
 Laparotomy;
Indication
1. Hemodynamically unstable.
2. Laparoscopy contraindicated,
3. Evidence of rupture.
The principle in laparotomy is "quick in quick
out".
 Acute pain related to distention of fallopian tube.
 Risk for Deficient Fluid Volume related to
bleeding from a ruptured ectopic pregnancy.
 Powerlessness related to early loss of
pregnancy secondary to ectopic pregnancy.
UTERINE
INVERSION
 When Uterus Turns Inside Out, It Is Called
Uterine Inversion.
 Uterine inversion is the folding of the fundus
into the uterine cavity in varying degrees.
Varies between 1 : 2000 to 1 : 35,000
deliveries
A. TYPES :
1) Incomplete Inversion:
 When fundus of uterus has turned inside out,
like toe of socks, but inverted fundus has not
descended through cervix
2) Complete Inversion:
 When the inverted fundus has passed
completely through cervix to lie within the vagina
or lie often outside the introitus.
B. DEGREES
 First degree: The uterus is partially turned out
 Second degree: The fundus has passed
through the cervix but not outside the vagina
 Third degree: The fundus is prolapsed outside
the vagina
 Fourth degree: The uterus, cervix and vagina
are completely turned inside out and are visible
Universally…
 First Degree : Incomplete Inversion
 Second Degree : Complete inversion in the
vagina
 Third Degree : Complete inversion outside the
introitus
 Manual removal of placenta
 Cord traction &/or fundal pressure
 Uterine anomalies
 Short umbilical cord
 Placenta Accreta
 Grand Multiparity
 Fetal macrosomia
 Rapid labor and delivery
Due to mismanagement of third stage of
labor…
 Faulty technique in manual removal of placenta
While separating retained placenta from the
wall, a portion may remain attached and as the
placenta is withdrawn, the fundus is also
withdrawn.
 Pulling the cord when the uterus is atonic while
combined with fundal pressure
 Crede’s Expression while the uterus is relaxed
 Large boggy mass appears at introitus
 With or without placenta attached
Other signs and symptoms are as follows –
 Severe and sustained hypogastric pain in 3rd stage
of labor
1. SHOCK
Extremely profound mainly neurogenic shock due to
A. Tension on the nerves due to stretching of the
infundibulo pelvic ligament.
B. Pressure on the ovaries as they are dragged with
the fundus through the cervical ring.
C. Peritoneal irritation
 Profound sweating with bradycardia,
 Hypotension
 Rarely cardiac arrest.
2. Hemorrhage - After detachment of placenta
3. Pulmonary embolism
4. If left uncared it may lead to ..
 Infection
 Uterine sloughing
 Peritoneal irritation
 Commonly unmet,
 The prognosis is extremely gloomy.
 Even if patient survives, infection ,sloughing of
the uterus and chronic inversion with ill health
may occur.
 Do not employ any method to expel the placenta
when the uterus is relaxed
 Patient should not be instructed to change her
position.
 Pulling the cord simultaneously with fundal
pressure should be avoided
 Manual removal of placenta should be done in
proper manner.
 1) Starting from the edge of placenta ,
 2) The placenta is separated by
a) keeping the back of the hand in contact with
the uterine wall.
b) with slicing movement of the hand.
 Delay in treatment increases the mortality, So
number of steps are taken immediately and
simultaneously.
Before shock develops :
 When one is on the spot when the inversion
happens TRY IMMEDIATE MANUAL
REPLACEMENT, even without anesthesia if
not easily available.
Principle :
 “ The part of the uterus which has come
down last, should go back first ”.
 If the diagnosis is made immediately after the
inversion has occurred, then that same degree of
relaxation of myometrium and cervix (which is
required for the inversion to occur) will allow
uterine replacement easily…
1. The gloved hand is lubricated with suitable
antiseptic cream and placed inside the vagina.
2. The uterine fundus with or without the attached
placenta, is cupped in the palm of the hand. The
fingers and thumb of the hand are extended to
identify margins of the cervix.
 3. The whole uterus is
lifted upwards towards
and beyond umbilicus.
 4. Additional pressure is
exerted with the
fingertips systematically
and sequentially to push
and squeeze the uterine
wall back through the
cervix.
5. Sustained pressure for 3-5 mins to achieve
complete replacement
6. Apply counter support by the other hand placed
on the abdomen
7. Once the fundus has been replaced keep the
hand in the uterus while rapid infusion of
oxytocin is given to contract the uterus.
Initially, bimanual compression aids in control of
further hemorrhage until uterine tone is
recovered.
8. When the uterus is felt contracting, the hand is
slowly withdrawn.
If placenta is attached, it is to be removed only
after the uterus becomes contracted.
If the placenta is partially attached, it should be
peeled out before replacement of uterus.
OBSTETRICAL
SHOCK
 Critical condition, life threatening medical
emergency.
 Shock results from acute , generalized,
inadequate perfusion of tissues; below that
needed to deliver the oxygen and nutrients for
normal function.
 Prompt recognition and management can
improve maternal and fetal outcome.
Major causes of shock include –
 1. Hypovoluemic
Hemorrhage(occult /overt) , hyperemesis,diarrhoea,
diabetic acidosis, peritonitis, burns.
 2. Septic
sepsis, endotoxaemia.
 3.Cardiogenic
cardiomyopathies , obstructive structural , obstructive
non structural , dysrrhythmias, regurgitant lesions.
 4.Distributive
Neurogenic- spinal injury, regional anesthesia,
 5.Anaphylaxis.
 Requires teamwork—Senior anesthetist,
obstetrician, physician and hematologist are to
be summoned immediately.
 Obstetrical units should have established
protocols for dealing with shock.
 Active management should start as soon as it is
suspected or expected
 Aiming for prompt restoration of tissue perfusion
and oxygenation.
 Resuscitation follows---ABC
A Airway—
Patent airway is assured and high pressure
oxygen (15 l/min)using mask/intra tracheal
intubation and anaesthesia machine.
B Breathing—
Ventilation checked and supported if needed .
C Circulation—
1. Insert two wide bore cannulas
2. Restore blood volume and reverse hypotension
with crystalloids/colloids.
3. Initial request for4-6 units of blood should be
sent. Blood may be transfused
 Monitor response to therapy - Pulse , BP, SPO2
/pulse oxymetry, urine output & pH .
 Position of patient - Head down and left lateral
 Avoid aortocaval compression
 Which further worsen the hypotension.
 Antenatal
Ruptured ectopic pregnancy, Incomplete abortion,
MTP, Uterine perforation during evacuation , APH,
Uterine rupture, Abdominal wall hematoma, Non
obstetrical intra abdominal bleeding.
 Intra natal
Uterine rupture.
 Post natal
PPH(primary, secondary) Atonic, Traumatic, Retained
tissue, Thrombosis, Acute uterine inversion .
Nonhaemorrhagic hypovolaemic shock, Burns,
Hyperemesis gravidarum, Acute Diarrhea
 The diagnosis of underlying cause and definitive
treatment is initiated once resuscitation is under
way.
 Surgical/ obstetrical—
ectopic pregnancy, abortion, uterine perforation,
APH, uterine rupture. PPH, inversion of uterus.
 Blood transfusion
 The failure of heart to provide adequate output,
leads to tissue under perfusion.
 Back pressure on lungs leads to Pulmonary edema.
 Pregnancy puts progressive strain on cardiac
function as pregnancy progresses , the peak being
between 32-34 wks.
 Pre existing cardiac disease further increases the
risk.
 Cardiac related death are 2nd most common
causes of death in pregnancy and commoner than
the direct leading cause , thromboembolism.
 Early diagnosis of cardiac lesion.
 Surgical correction of operable cardiac lesion,
before pregnancy is planned.
 Medical control of decompensated cardiac
lesion by cardiac correction before pregnancy is
planned.
 Avoiding Pregnancy/MTP at 6-8 wks if cardiac
condition is not under control.
 Management of pregnancy in such patients by
the expert team of cardiologist and obstetrician .
Definition –
A serious allergic reaction that is rapid in onset
and may result in death.
Aetiology –
 Pharmacological agents ,
 Insect stings,
 Foods,
 Latex
 1.Cutaneous -- (80%) flushing , pruritis ,
urticaria ,rhinitis , conjunctival erythema,
lacrymation
 2.Cardiovascular -- cardiovascular collapse ,
hypotension, vasodilatation, pale , cold clammy
skin , nausea , vomiting.
 3.Respiratory -- airway oedema , stridor ,
wheezing , dyspnoea , cough , chest/throat
tightness , hypoxia—confusion , increased
airway resistance.
 4. Gastrointestinal -
nausea , vomiting , abdominal pain .
 5. C N S -
Hypotension causes collapse with/without
unconsciousness , dizziness , incontinence,
confusion and throbbing headache .
1. Basic shock management -> ABC
2. Circulatory management
3. Primary (Special aspect)
- Stop administration of suspected substance and call for
help.
- IV 1ml injection of diluted
Adrenaline (1:1000)
- Early intra tracheal
- Supine/trendelenberg position with raised legs increases
venous return.
- Start vasopressor drugs and monitor BP.
Rapid infusion for plasma volume expansion , with
crystalloids
4. Secondary
- Atropine may be given if significant bradycardia.
- If bronchospasm – nebulise /I V
Amino/Derriphyllin or Beta 2 agonist such as
Salbutamol , Inhaled Ipravent may be particularly
useful for treatment of bronchospasm in patients
on B-blockers.
- Antihistamines - IV Chlorpheniramine.
- Corticosteroids -Dexamthesone.
Referral to critical care unit.
 Decreased cardiac output related to fluid volume
loss.
 Impaired gas exchange related to change in the
alveolar capillary membrane.
 Ineffective breathing pattern related to
bronchospasm.
CORD
PROLAPSE
 Umbilical cord prolapse occurs when the
umbilical cord comes out of the uterus with or
before the presenting part of the fetus.
 The overall incidence of cord prolapse ranges
from 0.1–0.6%.
In the case of breech presentation, the incidence
is higher at 1%.
The incidence is higher when there is a greater
percentage of multiple gestation.
 Mother
-Multiparity
-CPD
-Pelvic tumours
 Placenta & Cord
-P. previa
-Long cord
-Rupture of membranes
 Liquor
- Polyhydramnious
 Fetal
-prematurity
-Multiple gestation
-Anencephaly
-Malpresentation
(breach, Transverse lie,
Oblique)
 Iatrogenic prolapse
-ARM
-Placement of forceps or
a scalp electrode
 Appearance of loop of umbilical cord
 Pulsation of cord on V/E
 Suspect in unexplained fetal distress
- Variable deceleration
- Prolonged bradycardia
 Fetal survival depends on swift action
 Call for help – midwifery colleagues
 Factors to consider:
-Viability of fetus
-Severe fetal abnormalities
 Emergency delivery for a normally formed and
mature fetus
 Emergency LSCS
 Take measures to optimize fetal well-
being(maternal positioning)
 Multidisciplinary approach
 Teamwork
 Vaginal delivery
-Depends on descent of head & rate of progress;
parity
 Instrumental delivery
-Depends on skill levels & confidence; descent of
head & rate of progress.
 Caesarean section
-Take measure to ensure to optimise fetal wellbeing
-Multidisciplinary approach
-Teamwork
 Elevation of the presenting part:
-Digital pressure
-Kneeling on all fours, buttocks uppermost, or
-Exaggerated Sims (left lateral)
-Fill bladder with 500mls saline
-Tocolysis
 DO
 Replace the cord into
the vagina
 Monitor the fetal HR
 Inform the woman
 DON’T
 Replace the cord
inside the uterus
 Handle the cord
excessively
 Call for help
 Organise delivery
 Relieve pressure on the cord
 Deliver
 Fetal : impaired gas exchange related to
insufficient oxygen delivery secondary to cord
occlusion
 Risk for injury related to early cord steps into
presenting part.
 Fear related to perceived grave danger to fetus
and self obstetric emergency.
AMNIOTIC FLUID
EMBOLISM
 An amniotic fluid embolism is rare but serious
condition that occur when amniotic fluid, fetal
material, such as hair, enters the maternal
bloodstream.
 Very uncommon, 1/20,000 births.
 Though rare it comprises 10% of all maternal
death.
 Advance maternal age
 Multiparity
 Meconium
 Cervical laceration
 Intrauterine fetal death
 Sudden fetal expulsion
 Polyhyramnios
 Uterine rupture
 Maternal history of allergy or atopy
 Macrosomia
 Sudden shortness of breath
 Excess fluid in the lungs
 Sudden low blood pressure
 Sudden circulatory failure
Life-threatening problems
with
 Blood clotting
(disseminated intravascular
coagulopathy)
 Altered mental status
 Nausea or vomiting
 Chills
 Rapid heart rate
 Fetal distress
 Seizures
 Coma
NON SPECIFIC SPECIFIC
 Complete blood count
 Coagulation parameters
 ABG
 Chest x-ray
 Electrocardiogram
 Echocardiogram
 Cervical histology
 Serum tryptase
 Management is symptomatic and supportive.
 Targets- Maintaining oxygenation ,hemodynamic
support and correction of coagulopathy
 Immediate Resuscitation- ABC
 Airway and breathing
 Administer 100% oxygen via a non- rebreathing
reservoir face mask
 Prompt assessment, with control of the airway
and ventilation of the lungs with tracheal
intubation may be essential.
 Circulation
 2 large bore IV lines,
 Send blood for coagulation profile,
 CBC, crossmatch,
 Arrange 6units blood.
 Left lateral tilt/Manual uterine displacement.
 Hemodynamic support would include preload
optimization and vasopressors.
 Fluid resuscitation with crystalloid/colloid to
optimize filling.
 Infusion of an inotrope may be required to
maintain a mean arterial blood pressure and
achieve an adequate urine output.
 An arterial line for continuous blood pressure
monitoring is essential, and the use of a non-
invasive cardiac output monitor may be helpful.
 Continuously monitor the fetus.
 Uterine tone –Pharmacologic agents such as
oxytocin, ergometrine and prostaglandins
carboprost and misoprost.
 Coagulation:
 Use of plasma, cryoprecipitate, and platelets to
be guided by clinical condition of the patient and
laboratory investigations.
 Recombinant factor VII may be used, but one
should be careful as this can cause thrombotic
complications
 Antifibrinolytics, like e-aminocaproic acid and
tranexamic acid, might be helpful but evidence
is lacking.
POSTPARTUM
HEMORRHAGE
 More than 500 ml of blood loss following normal
vaginal delivery of the fetus or 1000ml following
Cesarean section.
 Clinically the amount of blood loss from or into
the genital tract which will adversely affect the
general condition of the patient
 Hemorrhage leading to fall in hematocrit by
10%.
 Incidence – 1- 4 %
 1] Primary
 2] Secondary
 Primary – bleeding occurs following delivery of
the baby up to 24 hours
 Primary is two types:
 A] Third Stage hemorrhage
 B] True Post Partum hemorrhage
A] Third Stage hemorrhage:
 Bleeding occurs before the expulsion of
placenta
 Example- Placenta accreta, increta and percreta
& retained placenta
B] True Postpartum hemorrhage:
 Occurs after the expulsion of placenta
 Secondary or Delayed or Late Postpartum
hemorrhage:
 Bleeding occurs following delivery of the baby
after 24 hours up to 6 weeks
 Tone
 Tissue
 Trauma
 Thrombin
Causes:
 1] Atonic
 2] Traumatic
 3] Mixed
 4] Retained Placenta
 5] Coagulopathy
 Contributes for 80 % of PPH
 Commonest cause of PPH
 Cause – Faulty retraction of the uterus
 Etiology:
 1] Grand Multipara
 2] Over- distension of uterus – Multiple
pregnancy, Hydramnios, big baby
 3] Anemia
 4] Prolonged Labor
 5] Anaesthesia – Halothane. Ether,
Cyclopropane
 6] Uterine fibroid
 7] Precipitate labor
 8] Malformations of uterus – septate uterus,
bicornuate uterus
 9] Ante partum hemorrhage
 10] Initiation & augmentation of delivery with
oxytocin
 1] Cervix – lacerations
 2] Vaginal laceration
 3] Perineum injury
 4] Paraurethral injury
 5] Uterine rupture
 Retained Placenta
Placenta accreta, increta and percreta
Succentuirate placenta.
 Abruptio Placenta,
 Jaundice,
 Thrombocytopenia
 Purpura,
 HELLP syndrome
 Vaginal bleeding may be revealed or concealed
 Alteration in pulse, Blood pressure and Pulse
pressure
 Flabby uterus in atonic uterus
UTEROTONIC DRUGS
 Routine oxytocin administration in third stage of
labour can reduce the risk of PPH by more than
40%
 The routine prophylaxis with oxytocins results in
a reduced need to use these drugs
therapeutically
 Management of the third stage of labour should
therefore include the administration of oxytocin
after the delivery of the anterior shoulder.
 Early recognition of PPH is a very important
factor in management.
 An established plan of action for the
management of PPH is of great value when the
preventative measures have failed
DRUG DOSE SIDE EFFECTS CONTRAINDICATION
OXYTOCIN 10 units IM/IMM
5units IV bolus
10 to 20 units/ litre
Usually none painful
contractions nausea,
Vomiting,
(water intoxication)
Hypersensitivity to drug
METHYLERGONOVINE
MALEATE
0.25mg IM/0.125mg IV
repeat every 5 mins as
needed max 5 doses
Peripheral vasospasm
Hypertension nausea,
Vomiting
Hypertension,
Hypersensitivity to drug
CARBOPROST
(15-METHYL PGF2alpha)
0.25 IM/IMM repeat every
15 mins as needed max 8
doses
Flushing, Diarrhea,
Bronchospasm, Nausea,
Vomiting, Restlessness,
Oxygen desaturation
Active Cardiac,
Pulmonary renal or
Hepatic disease,
Hypersensitivity to drug
VASOPRESSIN 20 units diluted in 100ml
normal saline=(0.2
units/ml)
Injected 1 ml at bleeding
site avoid intravascular
injection
Acute hypertension,
Bronchospasm, Nausea,
Vomiting, Abdominal
cramps, Angina,
headache, Vertigo, Death
with intravascular
injection
Coronary artery diseases
Hypersensitivity to drug
 Fluid volume deficit related to vaginal bleeding
 Ineffective tissue perfusion related to vaginal
bleeding
 Anxiety / fear related to changes in
circumstances or the threat of death
 Risk of infection due to bleeding
 Risk of shock : hypovolemic related to bleeding
.

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Obstetrical Emergencies

  • 1. GUIDED BY : MAJ. SARAVANA ESWARI CLINICAL TUTOR PRESENTED BY : N/CDT PRIYANKA GIRI IV YEAR BASIC B.SC NURSING
  • 2.  Obstetrical emergencies are life-threatening medical conditions that occur in pregnancy or during or after labor and delivery.  The main emergencies include- I. Ectopic Pregnancy II. Uterine Inversion III. Obstetrical Shock IV. Cord Prolepses V. Amniotic Fluid Embolism VI. Postpartum Hemorrhage
  • 4.  “Any pregnancy where the fertilised ovum gets implanted & develops in a site other than normal uterine cavity”.  Represent serious hazard to a woman’s health and reproductive potential  Requiring prompt recognition and early intervention.
  • 5. ECTOPIC PREGNANCY : is one in which fertilized ovum is implanted & develops outside normal uterine cavity .
  • 6.
  • 7.
  • 8.  Increased due to PID, use of IUCD, Tubal surgeries, and ART  Ranges from 1:25 to 1:250  Average range is 1 in 100 normal pregnancies.  Late marriages and late child bearing ->2%  ART -> 5%  Recurrence rate - 15% after 1st, 25% after 2 ectopics
  • 9.  History of PID  History of tubal ligation  Contraception failure  History of infertility  IUD use  Previous induced abortion  Tubal reconstructive surgery
  • 10.  Acute  Unruptured  Subacute Acute eptopic pregnancy  Fortunately less common (30%)  Associated with cases of tubal rupture or tubal abortion with massive intraperitoneal hemorrhage.
  • 11.  Classical triad is present in 50% of pt with rupture ectopic.  - PAIN:- most constant feature in 95% pt - variable in severity and nature  - AMENORRHOEA:- 60-80% of pt - there may be delayed period or slight spotting at the time of expected menses.  - VAGINAL BLEEDING: - scanty dark brown  Feeling of nausea,vomiting,fainting attack, syncope attack(10%) due to reflex vasomotor disturbance.
  • 12.  O/E:- patient is restless in agony, looks blanched, pale, sweating with cold clammy skin. Features of shock, tachycardia, hypotension.  P/A:- abdomen tense, tender mostly in lower abdomen, shifting dullness, rigidity may be present.  P/S:- minimal bleeding may be present  P/V:- uterus may be bulky, deviated to opposite side, fornix is tender, excitation pain on movement of cervix. POD may be full, uterus floats as if in water.
  • 13.  PRINCIPLE: “Resuscitation and laparotomy and not resuscitation followed by laparotomy”.  Antishock treatment: • measures are to be taken energetically • Simultaneous preparation for urgent laparotomy. -ringer solution (crystalloid) is started -arrangement for blood transfusion. -Even if blood is not available laparotomy is to be done desperately.
  • 14.  Laparotomy; Indication 1. Hemodynamically unstable. 2. Laparoscopy contraindicated, 3. Evidence of rupture. The principle in laparotomy is "quick in quick out".
  • 15.  Acute pain related to distention of fallopian tube.  Risk for Deficient Fluid Volume related to bleeding from a ruptured ectopic pregnancy.  Powerlessness related to early loss of pregnancy secondary to ectopic pregnancy.
  • 17.  When Uterus Turns Inside Out, It Is Called Uterine Inversion.  Uterine inversion is the folding of the fundus into the uterine cavity in varying degrees.
  • 18. Varies between 1 : 2000 to 1 : 35,000 deliveries
  • 19. A. TYPES : 1) Incomplete Inversion:  When fundus of uterus has turned inside out, like toe of socks, but inverted fundus has not descended through cervix 2) Complete Inversion:  When the inverted fundus has passed completely through cervix to lie within the vagina or lie often outside the introitus.
  • 20. B. DEGREES  First degree: The uterus is partially turned out  Second degree: The fundus has passed through the cervix but not outside the vagina  Third degree: The fundus is prolapsed outside the vagina  Fourth degree: The uterus, cervix and vagina are completely turned inside out and are visible
  • 21. Universally…  First Degree : Incomplete Inversion  Second Degree : Complete inversion in the vagina  Third Degree : Complete inversion outside the introitus
  • 22.  Manual removal of placenta  Cord traction &/or fundal pressure  Uterine anomalies  Short umbilical cord  Placenta Accreta  Grand Multiparity  Fetal macrosomia  Rapid labor and delivery
  • 23. Due to mismanagement of third stage of labor…  Faulty technique in manual removal of placenta While separating retained placenta from the wall, a portion may remain attached and as the placenta is withdrawn, the fundus is also withdrawn.  Pulling the cord when the uterus is atonic while combined with fundal pressure  Crede’s Expression while the uterus is relaxed
  • 24.  Large boggy mass appears at introitus  With or without placenta attached Other signs and symptoms are as follows –  Severe and sustained hypogastric pain in 3rd stage of labor 1. SHOCK Extremely profound mainly neurogenic shock due to A. Tension on the nerves due to stretching of the infundibulo pelvic ligament. B. Pressure on the ovaries as they are dragged with the fundus through the cervical ring. C. Peritoneal irritation
  • 25.  Profound sweating with bradycardia,  Hypotension  Rarely cardiac arrest. 2. Hemorrhage - After detachment of placenta 3. Pulmonary embolism 4. If left uncared it may lead to ..  Infection  Uterine sloughing  Peritoneal irritation
  • 26.  Commonly unmet,  The prognosis is extremely gloomy.  Even if patient survives, infection ,sloughing of the uterus and chronic inversion with ill health may occur.
  • 27.  Do not employ any method to expel the placenta when the uterus is relaxed  Patient should not be instructed to change her position.  Pulling the cord simultaneously with fundal pressure should be avoided  Manual removal of placenta should be done in proper manner.
  • 28.  1) Starting from the edge of placenta ,  2) The placenta is separated by a) keeping the back of the hand in contact with the uterine wall. b) with slicing movement of the hand.
  • 29.  Delay in treatment increases the mortality, So number of steps are taken immediately and simultaneously. Before shock develops :  When one is on the spot when the inversion happens TRY IMMEDIATE MANUAL REPLACEMENT, even without anesthesia if not easily available.
  • 30. Principle :  “ The part of the uterus which has come down last, should go back first ”.
  • 31.  If the diagnosis is made immediately after the inversion has occurred, then that same degree of relaxation of myometrium and cervix (which is required for the inversion to occur) will allow uterine replacement easily… 1. The gloved hand is lubricated with suitable antiseptic cream and placed inside the vagina. 2. The uterine fundus with or without the attached placenta, is cupped in the palm of the hand. The fingers and thumb of the hand are extended to identify margins of the cervix.
  • 32.  3. The whole uterus is lifted upwards towards and beyond umbilicus.  4. Additional pressure is exerted with the fingertips systematically and sequentially to push and squeeze the uterine wall back through the cervix.
  • 33. 5. Sustained pressure for 3-5 mins to achieve complete replacement 6. Apply counter support by the other hand placed on the abdomen 7. Once the fundus has been replaced keep the hand in the uterus while rapid infusion of oxytocin is given to contract the uterus. Initially, bimanual compression aids in control of further hemorrhage until uterine tone is recovered.
  • 34. 8. When the uterus is felt contracting, the hand is slowly withdrawn. If placenta is attached, it is to be removed only after the uterus becomes contracted. If the placenta is partially attached, it should be peeled out before replacement of uterus.
  • 35.
  • 36.
  • 38.  Critical condition, life threatening medical emergency.  Shock results from acute , generalized, inadequate perfusion of tissues; below that needed to deliver the oxygen and nutrients for normal function.  Prompt recognition and management can improve maternal and fetal outcome.
  • 39. Major causes of shock include –  1. Hypovoluemic Hemorrhage(occult /overt) , hyperemesis,diarrhoea, diabetic acidosis, peritonitis, burns.  2. Septic sepsis, endotoxaemia.  3.Cardiogenic cardiomyopathies , obstructive structural , obstructive non structural , dysrrhythmias, regurgitant lesions.  4.Distributive Neurogenic- spinal injury, regional anesthesia,  5.Anaphylaxis.
  • 40.  Requires teamwork—Senior anesthetist, obstetrician, physician and hematologist are to be summoned immediately.  Obstetrical units should have established protocols for dealing with shock.  Active management should start as soon as it is suspected or expected  Aiming for prompt restoration of tissue perfusion and oxygenation.
  • 41.  Resuscitation follows---ABC A Airway— Patent airway is assured and high pressure oxygen (15 l/min)using mask/intra tracheal intubation and anaesthesia machine. B Breathing— Ventilation checked and supported if needed .
  • 42. C Circulation— 1. Insert two wide bore cannulas 2. Restore blood volume and reverse hypotension with crystalloids/colloids. 3. Initial request for4-6 units of blood should be sent. Blood may be transfused
  • 43.  Monitor response to therapy - Pulse , BP, SPO2 /pulse oxymetry, urine output & pH .  Position of patient - Head down and left lateral  Avoid aortocaval compression  Which further worsen the hypotension.
  • 44.  Antenatal Ruptured ectopic pregnancy, Incomplete abortion, MTP, Uterine perforation during evacuation , APH, Uterine rupture, Abdominal wall hematoma, Non obstetrical intra abdominal bleeding.  Intra natal Uterine rupture.  Post natal PPH(primary, secondary) Atonic, Traumatic, Retained tissue, Thrombosis, Acute uterine inversion . Nonhaemorrhagic hypovolaemic shock, Burns, Hyperemesis gravidarum, Acute Diarrhea
  • 45.  The diagnosis of underlying cause and definitive treatment is initiated once resuscitation is under way.  Surgical/ obstetrical— ectopic pregnancy, abortion, uterine perforation, APH, uterine rupture. PPH, inversion of uterus.  Blood transfusion
  • 46.  The failure of heart to provide adequate output, leads to tissue under perfusion.  Back pressure on lungs leads to Pulmonary edema.  Pregnancy puts progressive strain on cardiac function as pregnancy progresses , the peak being between 32-34 wks.  Pre existing cardiac disease further increases the risk.  Cardiac related death are 2nd most common causes of death in pregnancy and commoner than the direct leading cause , thromboembolism.
  • 47.  Early diagnosis of cardiac lesion.  Surgical correction of operable cardiac lesion, before pregnancy is planned.  Medical control of decompensated cardiac lesion by cardiac correction before pregnancy is planned.  Avoiding Pregnancy/MTP at 6-8 wks if cardiac condition is not under control.  Management of pregnancy in such patients by the expert team of cardiologist and obstetrician .
  • 48. Definition – A serious allergic reaction that is rapid in onset and may result in death. Aetiology –  Pharmacological agents ,  Insect stings,  Foods,  Latex
  • 49.  1.Cutaneous -- (80%) flushing , pruritis , urticaria ,rhinitis , conjunctival erythema, lacrymation  2.Cardiovascular -- cardiovascular collapse , hypotension, vasodilatation, pale , cold clammy skin , nausea , vomiting.  3.Respiratory -- airway oedema , stridor , wheezing , dyspnoea , cough , chest/throat tightness , hypoxia—confusion , increased airway resistance.
  • 50.  4. Gastrointestinal - nausea , vomiting , abdominal pain .  5. C N S - Hypotension causes collapse with/without unconsciousness , dizziness , incontinence, confusion and throbbing headache .
  • 51. 1. Basic shock management -> ABC 2. Circulatory management 3. Primary (Special aspect) - Stop administration of suspected substance and call for help. - IV 1ml injection of diluted Adrenaline (1:1000) - Early intra tracheal - Supine/trendelenberg position with raised legs increases venous return. - Start vasopressor drugs and monitor BP. Rapid infusion for plasma volume expansion , with crystalloids
  • 52. 4. Secondary - Atropine may be given if significant bradycardia. - If bronchospasm – nebulise /I V Amino/Derriphyllin or Beta 2 agonist such as Salbutamol , Inhaled Ipravent may be particularly useful for treatment of bronchospasm in patients on B-blockers. - Antihistamines - IV Chlorpheniramine. - Corticosteroids -Dexamthesone. Referral to critical care unit.
  • 53.  Decreased cardiac output related to fluid volume loss.  Impaired gas exchange related to change in the alveolar capillary membrane.  Ineffective breathing pattern related to bronchospasm.
  • 55.  Umbilical cord prolapse occurs when the umbilical cord comes out of the uterus with or before the presenting part of the fetus.
  • 56.  The overall incidence of cord prolapse ranges from 0.1–0.6%. In the case of breech presentation, the incidence is higher at 1%. The incidence is higher when there is a greater percentage of multiple gestation.
  • 57.  Mother -Multiparity -CPD -Pelvic tumours  Placenta & Cord -P. previa -Long cord -Rupture of membranes  Liquor - Polyhydramnious  Fetal -prematurity -Multiple gestation -Anencephaly -Malpresentation (breach, Transverse lie, Oblique)  Iatrogenic prolapse -ARM -Placement of forceps or a scalp electrode
  • 58.  Appearance of loop of umbilical cord  Pulsation of cord on V/E  Suspect in unexplained fetal distress - Variable deceleration - Prolonged bradycardia
  • 59.  Fetal survival depends on swift action  Call for help – midwifery colleagues  Factors to consider: -Viability of fetus -Severe fetal abnormalities  Emergency delivery for a normally formed and mature fetus
  • 60.  Emergency LSCS  Take measures to optimize fetal well- being(maternal positioning)  Multidisciplinary approach  Teamwork
  • 61.  Vaginal delivery -Depends on descent of head & rate of progress; parity  Instrumental delivery -Depends on skill levels & confidence; descent of head & rate of progress.  Caesarean section -Take measure to ensure to optimise fetal wellbeing -Multidisciplinary approach -Teamwork
  • 62.  Elevation of the presenting part: -Digital pressure -Kneeling on all fours, buttocks uppermost, or -Exaggerated Sims (left lateral) -Fill bladder with 500mls saline -Tocolysis
  • 63.
  • 64.  DO  Replace the cord into the vagina  Monitor the fetal HR  Inform the woman  DON’T  Replace the cord inside the uterus  Handle the cord excessively
  • 65.  Call for help  Organise delivery  Relieve pressure on the cord  Deliver
  • 66.  Fetal : impaired gas exchange related to insufficient oxygen delivery secondary to cord occlusion  Risk for injury related to early cord steps into presenting part.  Fear related to perceived grave danger to fetus and self obstetric emergency.
  • 68.  An amniotic fluid embolism is rare but serious condition that occur when amniotic fluid, fetal material, such as hair, enters the maternal bloodstream.
  • 69.  Very uncommon, 1/20,000 births.  Though rare it comprises 10% of all maternal death.
  • 70.  Advance maternal age  Multiparity  Meconium  Cervical laceration  Intrauterine fetal death  Sudden fetal expulsion  Polyhyramnios  Uterine rupture  Maternal history of allergy or atopy  Macrosomia
  • 71.  Sudden shortness of breath  Excess fluid in the lungs  Sudden low blood pressure  Sudden circulatory failure Life-threatening problems with  Blood clotting (disseminated intravascular coagulopathy)  Altered mental status  Nausea or vomiting  Chills  Rapid heart rate  Fetal distress  Seizures  Coma
  • 72. NON SPECIFIC SPECIFIC  Complete blood count  Coagulation parameters  ABG  Chest x-ray  Electrocardiogram  Echocardiogram  Cervical histology  Serum tryptase
  • 73.  Management is symptomatic and supportive.  Targets- Maintaining oxygenation ,hemodynamic support and correction of coagulopathy  Immediate Resuscitation- ABC  Airway and breathing  Administer 100% oxygen via a non- rebreathing reservoir face mask  Prompt assessment, with control of the airway and ventilation of the lungs with tracheal intubation may be essential.
  • 74.  Circulation  2 large bore IV lines,  Send blood for coagulation profile,  CBC, crossmatch,  Arrange 6units blood.  Left lateral tilt/Manual uterine displacement.  Hemodynamic support would include preload optimization and vasopressors.  Fluid resuscitation with crystalloid/colloid to optimize filling.
  • 75.  Infusion of an inotrope may be required to maintain a mean arterial blood pressure and achieve an adequate urine output.  An arterial line for continuous blood pressure monitoring is essential, and the use of a non- invasive cardiac output monitor may be helpful.  Continuously monitor the fetus.  Uterine tone –Pharmacologic agents such as oxytocin, ergometrine and prostaglandins carboprost and misoprost.
  • 76.  Coagulation:  Use of plasma, cryoprecipitate, and platelets to be guided by clinical condition of the patient and laboratory investigations.  Recombinant factor VII may be used, but one should be careful as this can cause thrombotic complications  Antifibrinolytics, like e-aminocaproic acid and tranexamic acid, might be helpful but evidence is lacking.
  • 78.  More than 500 ml of blood loss following normal vaginal delivery of the fetus or 1000ml following Cesarean section.  Clinically the amount of blood loss from or into the genital tract which will adversely affect the general condition of the patient  Hemorrhage leading to fall in hematocrit by 10%.  Incidence – 1- 4 %
  • 79.  1] Primary  2] Secondary  Primary – bleeding occurs following delivery of the baby up to 24 hours  Primary is two types:  A] Third Stage hemorrhage  B] True Post Partum hemorrhage
  • 80. A] Third Stage hemorrhage:  Bleeding occurs before the expulsion of placenta  Example- Placenta accreta, increta and percreta & retained placenta B] True Postpartum hemorrhage:  Occurs after the expulsion of placenta
  • 81.  Secondary or Delayed or Late Postpartum hemorrhage:  Bleeding occurs following delivery of the baby after 24 hours up to 6 weeks
  • 82.  Tone  Tissue  Trauma  Thrombin
  • 83. Causes:  1] Atonic  2] Traumatic  3] Mixed  4] Retained Placenta  5] Coagulopathy
  • 84.  Contributes for 80 % of PPH  Commonest cause of PPH  Cause – Faulty retraction of the uterus  Etiology:  1] Grand Multipara  2] Over- distension of uterus – Multiple pregnancy, Hydramnios, big baby  3] Anemia
  • 85.  4] Prolonged Labor  5] Anaesthesia – Halothane. Ether, Cyclopropane  6] Uterine fibroid  7] Precipitate labor  8] Malformations of uterus – septate uterus, bicornuate uterus  9] Ante partum hemorrhage  10] Initiation & augmentation of delivery with oxytocin
  • 86.  1] Cervix – lacerations  2] Vaginal laceration  3] Perineum injury  4] Paraurethral injury  5] Uterine rupture  Retained Placenta Placenta accreta, increta and percreta Succentuirate placenta.
  • 87.  Abruptio Placenta,  Jaundice,  Thrombocytopenia  Purpura,  HELLP syndrome
  • 88.  Vaginal bleeding may be revealed or concealed  Alteration in pulse, Blood pressure and Pulse pressure  Flabby uterus in atonic uterus
  • 89.
  • 90. UTEROTONIC DRUGS  Routine oxytocin administration in third stage of labour can reduce the risk of PPH by more than 40%  The routine prophylaxis with oxytocins results in a reduced need to use these drugs therapeutically  Management of the third stage of labour should therefore include the administration of oxytocin after the delivery of the anterior shoulder.
  • 91.  Early recognition of PPH is a very important factor in management.  An established plan of action for the management of PPH is of great value when the preventative measures have failed
  • 92. DRUG DOSE SIDE EFFECTS CONTRAINDICATION OXYTOCIN 10 units IM/IMM 5units IV bolus 10 to 20 units/ litre Usually none painful contractions nausea, Vomiting, (water intoxication) Hypersensitivity to drug METHYLERGONOVINE MALEATE 0.25mg IM/0.125mg IV repeat every 5 mins as needed max 5 doses Peripheral vasospasm Hypertension nausea, Vomiting Hypertension, Hypersensitivity to drug CARBOPROST (15-METHYL PGF2alpha) 0.25 IM/IMM repeat every 15 mins as needed max 8 doses Flushing, Diarrhea, Bronchospasm, Nausea, Vomiting, Restlessness, Oxygen desaturation Active Cardiac, Pulmonary renal or Hepatic disease, Hypersensitivity to drug VASOPRESSIN 20 units diluted in 100ml normal saline=(0.2 units/ml) Injected 1 ml at bleeding site avoid intravascular injection Acute hypertension, Bronchospasm, Nausea, Vomiting, Abdominal cramps, Angina, headache, Vertigo, Death with intravascular injection Coronary artery diseases Hypersensitivity to drug
  • 93.  Fluid volume deficit related to vaginal bleeding  Ineffective tissue perfusion related to vaginal bleeding  Anxiety / fear related to changes in circumstances or the threat of death  Risk of infection due to bleeding  Risk of shock : hypovolemic related to bleeding
  • 94. .