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Presented BY…….
Dr. Rezuan
Internee Doctor
Jahurul Islam Medical College & Hospital,
Bangladesh
Burn
It may be defined as injuries
resulting from the application of dry
heat or chemical substances to the
external surface of the body resulting
in more or less destruction of tissues.
INTRODUCTION….
 Throughout the country, an
estimated 1% of the population
suffers from different types of
burns each year.
OThe incidence of burn
injury varies greatly
between cultures.
Human skin can tolerate tempratures up
to 42 – 44* C But above these, the
higher the temperature the more severe
the tissue destruction
Exposure
10 seconds at 50ºC redness
10 seconds at 60ºC very superficial burn
10 seconds at 70ºC full thickness burn
1 second at > 70ºC partial thickness burn
1 minute at 55-65ºC partial thickness burn
5 minutes at 50ºC full thickness burn
SCALD
It is the injuries produced by
application by moist heat (like as hot water,
oil) to the body .
TRAITS BURN (Dry Heat) SCALD (Moist Heat)
Cause Flame , Hot Metal Boiling Water,milk,oil
etc
Site At or Above the site
of contact
At or Below the site
of contact
Singeing of hair Present Absent
Depth Often deep upto skin
or more
Usually depth is less
Healing Delayed Comperatively early
Scar Thick Thin
Contracture Common Not common
The common causes of burns in
Bangladesh are as follows:
A. Flame burn: 75% of
incidences occur due to flame injuries -
CAUSES OF BURN….
1. Accidental:- this occurs
mainly in the home during
household work like cooking,
particularly from gas leakage or
burst stoves
2. Trapped in a burning house.
It includes..
3. Using fire pots for warming in
the winter, especially old people
and children.
4. Warming the lower part of the
body after delivery specially
women in remote village areas.
B. Others: About 20% burns
are electric burns.
The cause of electric burn is
mainly accidental.
5. Homicidal and suicidal:
Pouring kerosene oil, diesel, gasoline
or other inflammable liquids onto a
human body
 Fluid Shift
 Period of inflammatory response
 Vessels adjacent to burn injury
dilate > increase capillary
hydrostatic pressure & increase
capillary permeability
Patho-phisiology…
The increase in permeability is such
that large protein molecules can also
now escape with ease.
The damaged collagen and these
extravesated proteins increase the
oncotic pressure within the burned
tissue, further increasing the flow of
water from the intravascular to the
extravascular space.
 Continuous leak of plasma from
intravascular space
 Associated imbalance of fluid,
electrolytes & acid-base occur
 Hemoconcentration
 Lasts 24 – 36 hours
SYSTEMIC CHANGES
Cardiac
 decreased cardiac output
Pulmonary
 Alveolar damage
 Gas exchange
 Can progress to
respiratory failure
Gastrointestinal
 Curling ulcer formation
 Blood flow to splancnic
circulation
 Metabolic
 Hypermetabolic state
 Immunologic
 Increased risk of infection
■ Burns produce an inflammatory
reaction
■ This leads to vastly increased
vascular permeability
■ Water, solutes and proteins move
from the intra- to the extravascular
space
The shock reaction after burns :
■ The volume of fluid lost is
directly proportional to the area
of the burn
■ Above 15% of surface area
the loss of fluid produces
shock
(Cont..)
Burn
Pathology of Burn
(Jackson’s Zone of Injury)
Zone of Hyperaemia
Zone of Stasis
Zone of Coagulation
Necrosis
Severity is determined by :
 Depth of burn
 Extend of Burn calculated in
percent of total body surface
 location of burn
 patient risk factor
Assessing the area of a burn :
■ The rule of nines is adequate for
a first approximation only
■ The patient’s whole hand is 1%
TBSA, and is a useful guide in
small burns
■ The Lund and Browder chart is
useful in larger burns
Burn size needs to be formally
assessed in a controlled
environment.
This allows the area to be
exposed and any soot or debris
washed off.
The best measurement is
to cut a piece of clean
paper as the size of the
patient’s whole hand
(digits and palm), which
represents 1% TBSA,
and match this to the
area.
In the case of smaller burns
which states that –
 each upper limb is
9% TBSA,
 each lower limb 18%,
 the torso 18% each
side
 the head & neck
9%,
 Perineum 1%
rule of nines
Before going to the Classification,
1st we take a look about the layers
of skin…..
The human skin is an intricate
organ. It has two layers-dermis and
epidermis.The outer most, known
as the epidermis, Below the
epidermis lies the dermis.
Classification Of Burn:
Layers Of Skin:
 Epidermis :
○ Stratum Corneum
○ Stratum Lucidum
○ Stratum Granulosum
○ Stratum Spinosum
○ Stratum Basale
 Dermis
○ Papillary Dermis
○ Reticular Dermis
Nowthe Classification of Burn……….
 A. According to Depth Of burn:
 Superficial Partial Thickness Burn:
Involve epidermis + Papillary Dermis
 Deep Partial Thickness Burn:
Involve epidermis + Upto Reticular Dermis
 Full thickness Burn:
Involve the whole dermis
B. Traditionally :
 1St Degree Burn : Only Epidermis
 2nd Degree Burn: Epidermis and part
of Dermis
 3rd Degree Burn: Full thickness Skin
Superficial partial thickness burn
Clinical features-
-blistering and or loss of epithelium
-underlying dermis is pink and moist
-the capillary return is clearly visible when
blanched
-pin prick sensation is normal
Deep partial thickness burn
Clinical features-
1. the epidermis is usually lost.
2. the exposed dermis is not as moist as that in a superficial
burn.
3. the colour does not blanch with pressure.
4. sensation is reduced.
5. unable to distinguish sharp from blunt pressure.
Full thickness burn
Clinical features-
1. The whole of the dermis is destroyed in this burn .
2. They have a hard ,leathery feel.
3. There is no capillary return.
4. Often thrombosed vessels can be seen under the skin.
5. These burns are completely anaesthetised.
Difference Between Superficial
and Deep Burn
Trait Superficial Burn Deep Burn
Involvement Only Epidermis Epidermis, Dermis and
Appendages
Pain Intense pain Less or no pain
Blister Present Absent
Fluid Loss Less Fluid More Fluid
Need Of Skin Grafting No need Needed
Healing Heals completely Heals with ugly scar
Complication No Heals with formation of
Hypertrophied scar or
keloid
Causes of burn withprobable depth..
Causes of burn Probable depth of burn
1.Scald 1.Superficial but with deep
dermal patches in the absence
of good first aid.
2.Fat burn 2.Deep dermal
3.Flame burn 3.Mixed deep dermal and full
thickness.
4.Alkali burn including cement 4.Often deep dermal or full
thickness.
5.Acid burns 5.Weak concentrations -
superficial, strong
concentrations -deep dermal.
6.Electrical contract burn 6.Full thickness.
MANAGEMENT
 Pre- Hospital Care
 The principles of pre-
hospital care are:
-Ensure rescuer safety
-Stop the burning
process
-Check for other injuries
-Cool the burn wound
 Give oxygen-
If anyone
involved in a fire
in an enclosed
space or if there
is an altered
consciousness
level.
 Elevate-
sitting a patient up with
a burned airway may
life saving in the event
of a delay in transfer to
hospital care. Elevation
of burned limbs will
reduce swelling and
discomfort.
Criteria for Hospitalization:
Children with > 10%TBSA Burn
Adult with > 15%TBSA Burn
Burn around mouth, nostrils, eyes,
ears, genitalia, anus, breasts,
hands,Face.
Burn with inhalation injury
Deep burn of any percentage
Acid and other chemical burn
Electric burn
Extreme of ages
Hospital care
 The principles of managing an acute
burn injury are the same as in any
acute trauma case that is according
to ATLS .
 Assessment and resuscitation
should be carried out
simultaneously-
 A - Airway control
 B - Breathing and Ventilation
 C – Circulation
 D – Disability
- neurological status
 E – Exposure with environmental
control.
 F – Fluid resuscitation
1.. ATLS
2.. Analgesic
3.. Broad spectrum antibiotic
4.. Tetanus prophylaxis
5.. Blood transfusion (if
anaemia)
6.. IV nutrition
 Airway
 Rapid primary survey
and check airway-
Burn injury to the
airway above the larynx
by hot gases or to the
lower airway by inhaled
steam cause the
respiratory epithelium to
swell rapidly and that
may completely block the
airway.
 Breathing
Inhalational injury:
Difficulty in breathing due to
chemical pneumonitis is
caused by the minute particles
that stick to the moist lining
cause intense reaction to the
alveoli.
1.Treatment-
-nebulizers and warm
humidified oxygen,
-physiotherapy,
- sometimes continuous
or intermittent positive pressure
ventilation in an intensive care
unit may be needed.
Warning signs of burns
to the respiratory
system:
■ Burns around the
face and neck
■ A history of being
trapped in a burning
room
■ Change in voice
■ Stridor
 Circulation :
1. Intravenous fluid therapy
 Primary Requirements:
-Intravenous drip
Wide bore canula.
CVP catheter.
- Blood sample
Hb%, Haematocrit, S.Electrolyte,
S. Creatinine, Blood Urea, Blood
grouping & Cross matching.
- Urinary Catheterization.
Fig: 62% burn patient with Subclavian central venous catheter
C.V.P.
cathete
r
 Different fluids used in
resuscitation
 Crystalloid:
- Ringer’s lactate
.Widely & safely use all
over the World.
- 0.9% Sodium chloride solution
- Hypertonic saline solution
- 5% DNS
- Hartman’s solution
- 5% DA
 Colloid:
- Plasma
- Plasma substitute
e.g. Dextran, Gelatin
Parkland Formula
In 1st 24 hrs:
Total fluid – 4ml x bodyWeight in kg x
% burn. = ml Ringer’s lactate
In 1st 8 hrs – ½ of total fluid
In 2nd 8 hrs – ¼ of total fluid
In 3rd 8 hrs – ¼ of total fluid
 In children, maintenance fluid must also be given. This
is normally dextrose–saline given as follows:
• 100 ml kg–1 for 24 hours for the first 10 kg;
• 50 ml kg–1 for the next 10 kg;
• 20 ml kg–1 for 24 hours for each kilogram over 20 kg
body weight.
 Colloid resuscitation
Plasma proteins are responsible for
the inward oncotic pressure.
Without proteins, plasma volumes
would not be maintained as there
would be edema.
Proteins should be given after the first
12 hours of burn,
because before this time, the massive
fluid shifts cause proteins to leak out of
the cells.
The commonest is the Muir and
Barclay formula:
• 0.5 x percentage body surface
area burnt x weight = one portion;
• periods of 4/4/4, 6/6 and 12
hours respectively;
• one portion to be given in each
period.
FOR COLLOID
Nutrition of Burn Patient
■ Burns patients need
extra feeding
■ A nasogastric tube
should be used in all
patients with burns over
15% of TBSA
■ Removing the burn and
achieving healing stops
the catabolic drive
Monitoring
 Clinical
 Bio Chemical & Haematological
 Invasive
Clinical Monitoring
-Pulse rate : <120beats/min.
-Blood pressure
-Urine output : Reliable guide
0.5 –1.5 ml / kg / hrs in
adult,
10-20 ml / hrs. in child
-Core & shell temperature
-Thirst
-Sensorium
Biochemical & Haematological
Monitoring
- Hb % & Haematocrit
- Urine osmolality
- Serum electrolyte
- Serum creatinin & blood urea
Invasive Monitoring
- CentralVenous pressure:
-should maintain 2-8 cm of
water
- Arterial blood pressure
Management of Burn Wound
- Conservative
- Surgical
Management of burn wound has
undergone remarkable changes
since middle of the 1960’s,
when burn physician has multiple
agents to choose in controlling
bacterial growth.
 1% silver sulfadiazine
(silcream, silverzin)
 0.5% silver nitrate.
 Gentamycin cream
 Povidone iodine.
Common topical agents
Principles of dressings for burns
■Full-thickness and deep dermal
burns need antibacterial dressings
to delay colonisation prior to
surgery
■ Superficial burns will heal and
need simple dressings
■ An optimal healing environment
can make a difference to outcome
in borderline depth burns
• Dressing procedure should be
performed in operation
theatre.
• All instruments and dressing
materials used, should be
sterile and of medical grade.
Surgical treatment of deep burns
 Deep dermal burns need tangential shaving
and split-skin grafting
 All but the smallest full-thickness burns
need surgery
 All burnt tissue needs to be excised
 Stable cover, permanent or temporary,
should be applied at once to reduce burn
load
q Type of Burn suitable for excision :
- Deep partial thickness burn.
- Deep burn.
q Excision can be done by :
- Tangential excision
- Total burn excision
Burn Excision
Complications of BURN
 Immediate Complication:
 Shock : Hypovolumic and Neurogenic
 Renal Failure
 Acute respiratory Distress Syndrome (ARDS)
 Pneumonia
 Respiratory Failure
 Laryngeal Edema
 Acute GIT ulcer: Curling’s ulcer
 Hypothermia
 Multiple organ system failure (MOSF)
 Delayed Complication:
 Wound Infection
 Septicaemia
 Protein lossing enteropathy
 Cerebral damage
 Late Complication:
 Hypertrophic Scar
 Keloid
 Post burn Contracture
 Marjolin’s ulcer.
Complication
Figure: Hypertrophic scarring
following a deep dermal burn.
Figure: Keloid
Complication
Fig: Post Burn Contracture Fig: Marjolin Ulcer
Measures to Prevent
Complication Of Burn.
 I/V fluid for volume replacement.
 Adequate Dressing
 Antibiotic to prevent infection.
 Anti ulcerants to prevent Curling
ulcer.
 Escheratomy in case of deep burn
(Cont..)
 High protein diet
 Physiotherapy to prevent
contracture
 Vitamin C supplementaion
 Diuretics to prevent renal
failure
 Early skin grafting.
THANK YOU……..

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BURN ... by Dr. Rezuan .. JIMCH , Bangladesh

  • 1. Presented BY……. Dr. Rezuan Internee Doctor Jahurul Islam Medical College & Hospital, Bangladesh
  • 2. Burn It may be defined as injuries resulting from the application of dry heat or chemical substances to the external surface of the body resulting in more or less destruction of tissues.
  • 3. INTRODUCTION….  Throughout the country, an estimated 1% of the population suffers from different types of burns each year. OThe incidence of burn injury varies greatly between cultures.
  • 4. Human skin can tolerate tempratures up to 42 – 44* C But above these, the higher the temperature the more severe the tissue destruction Exposure 10 seconds at 50ºC redness 10 seconds at 60ºC very superficial burn 10 seconds at 70ºC full thickness burn 1 second at > 70ºC partial thickness burn 1 minute at 55-65ºC partial thickness burn 5 minutes at 50ºC full thickness burn
  • 5. SCALD It is the injuries produced by application by moist heat (like as hot water, oil) to the body .
  • 6. TRAITS BURN (Dry Heat) SCALD (Moist Heat) Cause Flame , Hot Metal Boiling Water,milk,oil etc Site At or Above the site of contact At or Below the site of contact Singeing of hair Present Absent Depth Often deep upto skin or more Usually depth is less Healing Delayed Comperatively early Scar Thick Thin Contracture Common Not common
  • 7. The common causes of burns in Bangladesh are as follows: A. Flame burn: 75% of incidences occur due to flame injuries - CAUSES OF BURN….
  • 8. 1. Accidental:- this occurs mainly in the home during household work like cooking, particularly from gas leakage or burst stoves 2. Trapped in a burning house. It includes..
  • 9. 3. Using fire pots for warming in the winter, especially old people and children. 4. Warming the lower part of the body after delivery specially women in remote village areas.
  • 10. B. Others: About 20% burns are electric burns. The cause of electric burn is mainly accidental. 5. Homicidal and suicidal: Pouring kerosene oil, diesel, gasoline or other inflammable liquids onto a human body
  • 11.  Fluid Shift  Period of inflammatory response  Vessels adjacent to burn injury dilate > increase capillary hydrostatic pressure & increase capillary permeability Patho-phisiology…
  • 12. The increase in permeability is such that large protein molecules can also now escape with ease. The damaged collagen and these extravesated proteins increase the oncotic pressure within the burned tissue, further increasing the flow of water from the intravascular to the extravascular space.
  • 13.  Continuous leak of plasma from intravascular space  Associated imbalance of fluid, electrolytes & acid-base occur  Hemoconcentration  Lasts 24 – 36 hours
  • 14. SYSTEMIC CHANGES Cardiac  decreased cardiac output Pulmonary  Alveolar damage  Gas exchange  Can progress to respiratory failure
  • 15. Gastrointestinal  Curling ulcer formation  Blood flow to splancnic circulation  Metabolic  Hypermetabolic state  Immunologic  Increased risk of infection
  • 16. ■ Burns produce an inflammatory reaction ■ This leads to vastly increased vascular permeability ■ Water, solutes and proteins move from the intra- to the extravascular space The shock reaction after burns :
  • 17. ■ The volume of fluid lost is directly proportional to the area of the burn ■ Above 15% of surface area the loss of fluid produces shock (Cont..)
  • 18. Burn Pathology of Burn (Jackson’s Zone of Injury) Zone of Hyperaemia Zone of Stasis Zone of Coagulation Necrosis
  • 19. Severity is determined by :  Depth of burn  Extend of Burn calculated in percent of total body surface  location of burn  patient risk factor
  • 20. Assessing the area of a burn : ■ The rule of nines is adequate for a first approximation only ■ The patient’s whole hand is 1% TBSA, and is a useful guide in small burns ■ The Lund and Browder chart is useful in larger burns
  • 21. Burn size needs to be formally assessed in a controlled environment. This allows the area to be exposed and any soot or debris washed off.
  • 22. The best measurement is to cut a piece of clean paper as the size of the patient’s whole hand (digits and palm), which represents 1% TBSA, and match this to the area. In the case of smaller burns
  • 23. which states that –  each upper limb is 9% TBSA,  each lower limb 18%,  the torso 18% each side  the head & neck 9%,  Perineum 1% rule of nines
  • 24. Before going to the Classification, 1st we take a look about the layers of skin….. The human skin is an intricate organ. It has two layers-dermis and epidermis.The outer most, known as the epidermis, Below the epidermis lies the dermis. Classification Of Burn:
  • 25. Layers Of Skin:  Epidermis : ○ Stratum Corneum ○ Stratum Lucidum ○ Stratum Granulosum ○ Stratum Spinosum ○ Stratum Basale  Dermis ○ Papillary Dermis ○ Reticular Dermis
  • 26. Nowthe Classification of Burn……….  A. According to Depth Of burn:  Superficial Partial Thickness Burn: Involve epidermis + Papillary Dermis  Deep Partial Thickness Burn: Involve epidermis + Upto Reticular Dermis  Full thickness Burn: Involve the whole dermis
  • 27. B. Traditionally :  1St Degree Burn : Only Epidermis  2nd Degree Burn: Epidermis and part of Dermis  3rd Degree Burn: Full thickness Skin
  • 28. Superficial partial thickness burn Clinical features- -blistering and or loss of epithelium -underlying dermis is pink and moist -the capillary return is clearly visible when blanched -pin prick sensation is normal
  • 29. Deep partial thickness burn Clinical features- 1. the epidermis is usually lost. 2. the exposed dermis is not as moist as that in a superficial burn. 3. the colour does not blanch with pressure. 4. sensation is reduced. 5. unable to distinguish sharp from blunt pressure.
  • 30. Full thickness burn Clinical features- 1. The whole of the dermis is destroyed in this burn . 2. They have a hard ,leathery feel. 3. There is no capillary return. 4. Often thrombosed vessels can be seen under the skin. 5. These burns are completely anaesthetised.
  • 31. Difference Between Superficial and Deep Burn Trait Superficial Burn Deep Burn Involvement Only Epidermis Epidermis, Dermis and Appendages Pain Intense pain Less or no pain Blister Present Absent Fluid Loss Less Fluid More Fluid Need Of Skin Grafting No need Needed Healing Heals completely Heals with ugly scar Complication No Heals with formation of Hypertrophied scar or keloid
  • 32. Causes of burn withprobable depth.. Causes of burn Probable depth of burn 1.Scald 1.Superficial but with deep dermal patches in the absence of good first aid. 2.Fat burn 2.Deep dermal 3.Flame burn 3.Mixed deep dermal and full thickness. 4.Alkali burn including cement 4.Often deep dermal or full thickness. 5.Acid burns 5.Weak concentrations - superficial, strong concentrations -deep dermal. 6.Electrical contract burn 6.Full thickness.
  • 33. MANAGEMENT  Pre- Hospital Care  The principles of pre- hospital care are: -Ensure rescuer safety -Stop the burning process -Check for other injuries -Cool the burn wound
  • 34.  Give oxygen- If anyone involved in a fire in an enclosed space or if there is an altered consciousness level.
  • 35.  Elevate- sitting a patient up with a burned airway may life saving in the event of a delay in transfer to hospital care. Elevation of burned limbs will reduce swelling and discomfort.
  • 36. Criteria for Hospitalization: Children with > 10%TBSA Burn Adult with > 15%TBSA Burn Burn around mouth, nostrils, eyes, ears, genitalia, anus, breasts, hands,Face. Burn with inhalation injury Deep burn of any percentage Acid and other chemical burn Electric burn Extreme of ages
  • 37. Hospital care  The principles of managing an acute burn injury are the same as in any acute trauma case that is according to ATLS .  Assessment and resuscitation should be carried out simultaneously-
  • 38.  A - Airway control  B - Breathing and Ventilation  C – Circulation  D – Disability - neurological status  E – Exposure with environmental control.  F – Fluid resuscitation 1.. ATLS
  • 39. 2.. Analgesic 3.. Broad spectrum antibiotic 4.. Tetanus prophylaxis 5.. Blood transfusion (if anaemia) 6.. IV nutrition
  • 40.  Airway  Rapid primary survey and check airway- Burn injury to the airway above the larynx by hot gases or to the lower airway by inhaled steam cause the respiratory epithelium to swell rapidly and that may completely block the airway.
  • 41.  Breathing Inhalational injury: Difficulty in breathing due to chemical pneumonitis is caused by the minute particles that stick to the moist lining cause intense reaction to the alveoli.
  • 42. 1.Treatment- -nebulizers and warm humidified oxygen, -physiotherapy, - sometimes continuous or intermittent positive pressure ventilation in an intensive care unit may be needed.
  • 43. Warning signs of burns to the respiratory system: ■ Burns around the face and neck ■ A history of being trapped in a burning room ■ Change in voice ■ Stridor
  • 44.  Circulation : 1. Intravenous fluid therapy  Primary Requirements: -Intravenous drip Wide bore canula. CVP catheter. - Blood sample Hb%, Haematocrit, S.Electrolyte, S. Creatinine, Blood Urea, Blood grouping & Cross matching. - Urinary Catheterization.
  • 45. Fig: 62% burn patient with Subclavian central venous catheter C.V.P. cathete r
  • 46.
  • 47.  Different fluids used in resuscitation  Crystalloid: - Ringer’s lactate .Widely & safely use all over the World. - 0.9% Sodium chloride solution - Hypertonic saline solution - 5% DNS - Hartman’s solution - 5% DA  Colloid: - Plasma - Plasma substitute e.g. Dextran, Gelatin
  • 48. Parkland Formula In 1st 24 hrs: Total fluid – 4ml x bodyWeight in kg x % burn. = ml Ringer’s lactate In 1st 8 hrs – ½ of total fluid In 2nd 8 hrs – ¼ of total fluid In 3rd 8 hrs – ¼ of total fluid  In children, maintenance fluid must also be given. This is normally dextrose–saline given as follows: • 100 ml kg–1 for 24 hours for the first 10 kg; • 50 ml kg–1 for the next 10 kg; • 20 ml kg–1 for 24 hours for each kilogram over 20 kg body weight.
  • 49.  Colloid resuscitation Plasma proteins are responsible for the inward oncotic pressure. Without proteins, plasma volumes would not be maintained as there would be edema. Proteins should be given after the first 12 hours of burn, because before this time, the massive fluid shifts cause proteins to leak out of the cells.
  • 50. The commonest is the Muir and Barclay formula: • 0.5 x percentage body surface area burnt x weight = one portion; • periods of 4/4/4, 6/6 and 12 hours respectively; • one portion to be given in each period. FOR COLLOID
  • 51. Nutrition of Burn Patient ■ Burns patients need extra feeding ■ A nasogastric tube should be used in all patients with burns over 15% of TBSA ■ Removing the burn and achieving healing stops the catabolic drive
  • 52. Monitoring  Clinical  Bio Chemical & Haematological  Invasive
  • 53. Clinical Monitoring -Pulse rate : <120beats/min. -Blood pressure -Urine output : Reliable guide 0.5 –1.5 ml / kg / hrs in adult, 10-20 ml / hrs. in child -Core & shell temperature -Thirst -Sensorium
  • 54. Biochemical & Haematological Monitoring - Hb % & Haematocrit - Urine osmolality - Serum electrolyte - Serum creatinin & blood urea Invasive Monitoring - CentralVenous pressure: -should maintain 2-8 cm of water - Arterial blood pressure
  • 55. Management of Burn Wound - Conservative - Surgical
  • 56. Management of burn wound has undergone remarkable changes since middle of the 1960’s, when burn physician has multiple agents to choose in controlling bacterial growth.  1% silver sulfadiazine (silcream, silverzin)  0.5% silver nitrate.  Gentamycin cream  Povidone iodine. Common topical agents
  • 57. Principles of dressings for burns ■Full-thickness and deep dermal burns need antibacterial dressings to delay colonisation prior to surgery ■ Superficial burns will heal and need simple dressings ■ An optimal healing environment can make a difference to outcome in borderline depth burns
  • 58. • Dressing procedure should be performed in operation theatre. • All instruments and dressing materials used, should be sterile and of medical grade.
  • 59. Surgical treatment of deep burns  Deep dermal burns need tangential shaving and split-skin grafting  All but the smallest full-thickness burns need surgery  All burnt tissue needs to be excised  Stable cover, permanent or temporary, should be applied at once to reduce burn load
  • 60. q Type of Burn suitable for excision : - Deep partial thickness burn. - Deep burn. q Excision can be done by : - Tangential excision - Total burn excision Burn Excision
  • 61. Complications of BURN  Immediate Complication:  Shock : Hypovolumic and Neurogenic  Renal Failure  Acute respiratory Distress Syndrome (ARDS)  Pneumonia  Respiratory Failure  Laryngeal Edema  Acute GIT ulcer: Curling’s ulcer  Hypothermia  Multiple organ system failure (MOSF)
  • 62.  Delayed Complication:  Wound Infection  Septicaemia  Protein lossing enteropathy  Cerebral damage  Late Complication:  Hypertrophic Scar  Keloid  Post burn Contracture  Marjolin’s ulcer.
  • 63. Complication Figure: Hypertrophic scarring following a deep dermal burn. Figure: Keloid
  • 64. Complication Fig: Post Burn Contracture Fig: Marjolin Ulcer
  • 65. Measures to Prevent Complication Of Burn.  I/V fluid for volume replacement.  Adequate Dressing  Antibiotic to prevent infection.  Anti ulcerants to prevent Curling ulcer.  Escheratomy in case of deep burn
  • 66. (Cont..)  High protein diet  Physiotherapy to prevent contracture  Vitamin C supplementaion  Diuretics to prevent renal failure  Early skin grafting.