1. 1. M.C. Periodontium is:
A. Complicate anatomic formation
B. Anatomic formation with conjunctive origin
C. A fissure localized between compact lamelae of dental alveolar and root.
D. Anatomic formation localized between compact lamelae of dental alveola and root
cementum.
E. Anatomic formation, localized between dental alveolar and root cementum
2.C.M. Throughout it’s extent periodontium is in directly contact with:
A. Maxilar bone
B. Through apical foramen with dental pulp
C. At the alveolar margine with gingiva and periostium
D. Oral cavity
E. Vestibul and oral cavity
3. C.S. Ending of periodontium formation is:
A. 1 month after radicular apex formation
B. Coincide with final dentinar apex formation
C. 1 year after complete root apex formation
D. 6 months after complete root apex formation
E. 3 months after complete root apex formation
4. S.C. According to E. Gofung data the width of periodontal fissure is:
A. To mandibule 0,15 – 0,22 mm, maxilla 0,2 – 0,25 mm
B. To mandible 0,2 – 0,25 mm, maxilla 0,15 – 0,22 mm
C. To mandible 0,22 – 0,27 mm, maxilla 0,25 – 0,9 mm
D. To mandible 0,1 – 0,2 mm, maxilla 0,2 – 0,3 mm
E. The width of periodontal fissure is the similar to maxilla and mandible
5. S.C. With age the width of periodontal fissure is:
A. Becomes wider in case of inflammation
B. Becomes wider after thickening of periodontium
C. Periodontal fissure is reduced till 0,1-0,15 mm
D. At the beginning is reduced, then during ageing becomes thick
E. Process of ageing is not reflected to the status of periodontal fissure.

2. 6.C.M. thickness changings of periodontium is observed in:
A. Pathological processes
B. Overload
C. Hipercimentosis
D. As a result of caries cavity preparation
E. As a result of amalgam filling
7.C.M. Fascicules of calogene fibers are classified:
A. Transeptal fibres
B. Free gingival and circular fibers
C. Pectinates and oblique fibers
D. Apical fibers group
E. Transitory fibers
8. C.M. Functions of periodontium:
A. Maintains inflammatory processes in periodontium
B. Support
C. Trophic protective
D. Distribution of plastic pressure
E. Trophic sensorial and protection
9. C.S. Basic Functions of periodontium:
A. Plastic and trophic
B. distribution of presasure
C. support
D. sensorial
E. protection
10. C.M. According to etiology of periodontitis can be distinguished:
A. Medicamentous
B. Infectious
C. Alergic
D. Traumatic
E. After incorrect treatment

3. 11.C.S. The most common Inflamatory process of periodont is conditioned by:
A. Microflora virulence
B. Endotoxine action on distroyence of gram + bacteries of periapical tissues
C. Chronic trauma
D. Attenuation of adaptive mechanisms of periodontium
E. As a result of incorrect treatment
12.C.M Pathological anatomy of acute apical periodontitis (phase I):
A. tissular infiltration by leukocytes polimorfo-nuclear
B. Predominant perivascular lympho-histiocytic infiltration
C. Tissular necrosis with abces formation
D. Widening of periostium and alveolar resorbtion
E. polynuclear solitary
13. C.M. For apical acute serous periodontitis is characteristic:
A. Localized pain
B. Uninterrupted pain
C. Painful periods pass into silent periods
D. Pain with annoying character that corresponds to affected tooth
E. Pain irradiates on trigeminus nerve route
14. M.C.Apical acute periodontitis, purulent form has the pain like:
A. Annoying
B. Intensified pain, pulsed
C. Pain on touching the tooth, to bitting, sensation of “long tooth”
D. Pain “attacks” with long lasting painless periods
E. Sometimes pain irradiates along trigeminus nerve route
15.C.S. Radiological pictures of Deviations in maxilars patients with acute apical
periodontitis
A. outbreak of osteoporosis in region of radicular apex with clear clear outline
B. unclear picture shape of braking line
C. Loosing of clarity picture
D. Clear picture of spongious substance
E. Osteolisis outbreak with clear outlines

4. 16. M.C.Clinical picture of apical acute periodontits:
A. Painful probing to localized point on pulp horn projection
B. Painful probing on all the bottom of carious cavity
C. Painless probing
D. Thermodiagnosis: the pain is intensified to cold and hot excitings
E. The reaction of tooth to thermal factors are painless
17. M.C.Percussion and electriodontodianosis of apical acute periodontitis is:
A. Paintess percussion
B. Pain to horizontal percussion
C. Pain to vertical percussion
D. Electricodontodiagnosis – reacts to 100 mkA current and more than
E. EOD – reacts-to current 60 mkA
18. M.C.Chronic fibrous periodontitis can appear:
A. After incorrect treatment of other forms of periodontitis
B. As the result of acute periodontitis
C. After pushing off the phosphate – cement out of apex
D. Healing other forms of periodontitis
E. The outcome of pulpitis treatment
19. . S.C.The diagnosis of fibrous periodontitis is based on:
A. patient acuses
B. Electric odontodiagnosis
C. Xray image
D. Probing
E. Percussion

5. 20. S.C.X-ray image in chronic fibrous periodontitis shows:
A. Deformation of periodontal space with bone tissue resorbtion of dental alveola
B. Deformation of periodontal space by its enlargement in periapical region with
preewation of cortical plate
C. Uniform dilation of space, in periapical region and resorbtion of root cementum
D. Uniform dilation of periodontal space and hypercemen tosis
E. Deformation of periodontal space in form of broken like
21.C.M. Pathological anatomy of fibrous periodontitis:
A. Movements of cellular elements and increasing of fibrous tissue with thick fascicules
B. Small focars of inflammatory infiltrate and vascular sclerosis
C. Granulous tissue and decreasing of cellular elements
D. Fibrous tissue with thick fascicules and a big amount of capilares
E. Epithelial passage and tissue with thick fascicules
22. S.C.Chronic granulous periodontitis appears:
A. Usually after pulpitis treatment
B. From granulomatous Periodontitis
C. Usualy from acute Periodontitis and it is a stage of evolution for chronic inflamtion
D. As the result of not treated in time the dental caries
E. After insufficient sanation of oral cavity
23. M.C.Patient’s complains with chronic granulomatous Periodontitis are:
A. Insignificant painful sensations
B. Insignificant pain from sweet
C. Sensation of “pressing”, swelling discomfort
D. Insignificant pain from hot food
E. Insignificant pain during pressing on teeth

6. 24. S.C.For which form of periodontitis is characteristic the for presence of fistula:
A. For chronic Fibrous Periodontitis
B. For chronic Granulomatous Periodontitis
C. For chronic Granulous
D. For acute purulent Periodontitis
E. For chronic Granulous Periodontitis in stage of aggravation
25.C.M. Which form of chronic periodontitis doesn’t need accurance of Xray
examination AND based on:
A. Chronic fibrous
B. Chronic granulous
C. Chronic granulomatous
D. Fistula
E. Gingival hiperimy of affective adiacent tissue
26. S.C.The symphom of vasoparesis is characteristic For:
A. Acute serous Periodontitis
B. Chronic Serous Periodontitis
C. Acute purulent Periodontitis
D. Chronic Granulous Periodontitis
E. Chronic Fibrous Periodontitis
27. S.C.On radiologic film in Chronic Granulous Periodontitis is appreciated:
A. Uniform dilatation at periodontal space in periapical region
B. Osteolisis focus in apical region
C. Osteolisis focus in apical region with vaque contour
D. Osteolisis focus in apical region with vaque contour and broken line
E. Osteolisis focus in round shape, with clear limmits well delimited and 0,5 cm in
diameter
28. M.C.Pathologic anatomy of chronic Granulous Periodontitis is:
A. Increase of leucocyte infiltration
B. Tissue necrosis and abscess formation
C. Increase of fibrous lissue with thick fibers
D. Formation of granulous tissue with great number of cappilaries, fibroblasts,
plasmatic cells, leucocytes.
E. Lysis of periapical tissue by osteoclasts (cortical lamellae of alveolar, dentine,
radicular cementum

7. 29. C.S. Which form of chronic periodontitis is the most active:
A. Chronic fibrous
B. Chronic granulous
C. Chronic granulomatous
D. Acutisation of chronic granulomatous periodontitis
E. Acutisation of chronic fibrous periodontitis
30. C.M. Intoxication ways in chronic granulous periodontitis:
A. consequence of resorptive process in alveola
B. Toxic products of inflammation are absorbed in blood
C. Granular proliferation in osteomedular space of maxilares, formed fistules with
purulent secretion
D. Consequence of resorbtive process in radicular cement
E. Consequence of resorbtive process in radicular dentine
31. S.C.The most reversible form of Periodontitis (high % of successful outcome)
has:
A. Chronic Fibrous Periodontitis
B. Chronic Granulomatous Periodontitis
C. Chronic Granulomatous
D. Chronic Apical Periodontitis
E. Chronic Apical Periodontitis in stage of aggravation
32..M.S.Chronic granulomatous Periodontitis in most of cases is:
A. Is clinical manifested
B. Isn’t clinically manifested
C. Absence of subjective and objective data
D. Subjective and objective data are evident manifestet
E. Rarely is created fistula, hyperemia, swelling
33. S.C.The diagnosis of chronic Granulomatous Periodontitis in most of cases is
established on:
A. probing
B. percussion
C. Radiologic data
D. Palpation
E. Electric odontometry data

8. 34. M.C.Radiologic datum indicate in chronic Granulomatous Periodontitis:
A. Focus and osteolysis in apical region with vague contour
B. Small focus of osteolysis with well expressed contour
C. Lysis focus in shape at broken line
D. Round focus or oval with near 0,5 cm in diameter
E. Focus of lysis with limits like “flame tonque”
35. C.M. The biggest amount of granulome are contained in:
A.Molars
B.incisors
C.Canines
D.Premolars
E. Incisors and canines
36. C.M. Pathological anathomy of granulomatous periodontitis:
A. A partial substitution of periodontal tissue with granulation tissue
B. Missing of wallpaper epithelium
C. Epithelial ways and epithelial wallpaper
D. Colagen elastic fibers
E. Granulous tissue is separate from adjacent bone by fibrous capsule
37. M.C.The central part of granuloma contains:
A. A cavity is formed
B. The contained plasmatic cells
C. Protein detritus and lipide detritus
D. Cells of hystogenesis and hae matogenesis
E. Crystals of cholesterin
38. M.C.Accroding to anatomical structure granuloma is devided into:
A. Simple and complicated
B. Simple and mixed
C. Collagenic granuloma
D. Elastic granuloma
E. Nonepithelial and epithelial

9. 39. M.C.In chystogranuloma and radicular chysts are formed cavities that contain:
A. Degenerated epithelial cells
B. Eosinophil exsudate, proteic detritus, lipide detritus
C. Cholesterin crystals
D. Serous exudate
E. Lymphocyte and hystiocyte infiltration
40.
41. M.C.Clinical picture chronic Periodontitis in stage of aggravation is
characteristic by following signs:
A. Constant pain, collateral edema
B. Dental mobility, pain to palpation
C. Probing and thermal probe cause pain
D. EOM – 60 mkA
E. Radiologic image – vague picture of spongy bone
42. S.C.What kind of epithelium has granuloma:
A. Epidermal
B. Reticular stratum
C. Adventitia layer
D. Malpighiam of oral cavity epithelium
E. Keratinous stratum
43. S.C.Chysto – granuloma is tapered with:
A. Epithelium
B. Granulous tissue
C. Epithelial bay
D. Plasmatic cells
E. Plasmatic cells and leucocytes
44. C.M. Characterise chisto- granuloume:
A. Clear outline
B. Unclear outline
C. Lack of structural bone in resorbtion bone focar
D. Radiologic opacity
E. Allivation of radiological picture

10. 45. S.C.Chystogranuloma dimension is:
A. 0,5 cm
B. 0,25 cm
C. 1,8 cm
D. 0,5 – 0,8 cm
E. 2,5 cm
46.C.M. The basic principles of the most effective methods of treatment of apical
periodontitis:
A. Root canal filling with minimal pushing of material above apex
B. Effective preparation of infective root canals
C. Root canal filling till the physiological norrowing
D. Treatment of periapical lesion till the exudacion absence
E. Root canal filling till apical foramen.
47. C.M. Contraindication of chronic periodontitis treatment ( can not be treated):
A. The teeth of the bell-shaped canals which form a bayonet
B. teeth with chronic periapical focars
C. teeth with foreign bodies in their roots
D. teeth with curbed and penetrable canals
E. pluriradicular teeth with false ways.
48. M.C.Radiologically in periodontitis can be appreciated:
A. Presence of foreign bodies
B. Number and root shape
C. Tooth mobility II – III degree
D. Relation of roots with different anatomic formation (mental foramen, mandibular
canal)
E. masticatory eficience of tooth

11. 49. C.M. Treatment of chronic periodontitis is made:
A. Clinical healthy patients
B. Patients with weakened forms of pathology
C. Aggravated systemic pathology
D. non-transportable patients
E. mental disorders patients
50. C.M. Radiologicaly is possible to determine:
A. Root canals lengths compared to adjacent teeth
B. Missing of pain, edhem, fistula
C. Rests of putrified masses
D. Data about the width and shape of the tooth cavity
E. Number and shape of roots
51. M.C.Passage ways of action under microbian flora of root canal are:
A. Root canal opening
B. Physico – chemical
C. Incision on transitory fold
D. Chemical
E. Isthmus enlargement and root apex
52. M.C.The goal of medicamentous cleaning of root canal is:
A. To act under pathogenic flora from endodontic space
B. To liquidate pathologic flora from endodontic space
C. To act under degenerated produces from root canal
D. To neutralize degenerated produces of organic rests
E. To decrease the number of microorganisms from root canal
53. C.M. Sterelisation of macro and micro canals is made through:
A. Relief f periapical inflammation
B. Efficient biomechanical treatment
C. Chemical stability of antiseptic solution in root canals
D. Abundent medicamentous treatment of endodontical space
E. Stimulation of inflammatory periapical process

12. 54. M.C.Requirements to medicamentous remedies used in root canal sterilization
are:
A. To possess bactericide and fungicide effect
B. Not to be inactivated in nutritional medium
C. To differ from surrounding tissue medium
D. To differ from surrounding tissue in colour
E. To possess lasting antibacterial effect
55. M.C.Commonly used antiseptics on:
A. Chloramine
B. Antibiotics
C. Natrium hypochlorite
D. Chlorhexidine
E. Resorcin formaline
56. M.C.3% sol. of Natrium hypohlorite has the property to:
A. To dissolve organic remnants
B. To dicrease periapical inflammation
C. To sterilize root canal
D. To stimulate the regenerative processes in periapical region
E. To enlarge the root canal
57. C.S. ‘’Parcan’’ (Septodont) contain:
A. Sol.1% Hipochlorit Na
B. Clorhexidine
C. Sol.3%purificated hipochlorit Na
D. Alchool ethylic
E. Dexametazone
58. M.C.Modern antiseptics on:
A. Chlorhexidine 0,1-3%
B. H2 O2 2-3%
C. “Parcan”
D. Formaline
E. 1-3% sol carbolic acid

13. 59. C.M. infected Zone by Lucomschi in periodontitis:
A. Macrocanal zone
B. Paradontal zone
C. Microcanalicular zone
D. Marginal periodontium zone
E. Periodontal fissure zone
60. M.C.The purpose of filling the root canal is:
A. To hemetise canals all along the apex
B. To restore anatomic micro shape and dental function
C. To block microflora in dentinal canalliculi
D. To remove bad small from root canal
E. Formation of canditions for treatment the apical Pt, regeneration of apical tissue that
are pathalogic modificated
61. M.C.Requirement to root canal filling materials
A. Not to be changed in volume
B. Not to influence under healing of apical region
C. Not to be radiopaque
D. To passess bactericide effect
E. Not to be resorbatle in root canals
62. M.C.Materials based on epoxide resine are:
A. Ftorodent
B. AH26, AH +
C. Pepsodent
D. Endodent, termosil
E. Diaket
63. C.M. Paste based on zinc oxide and eugenol:
A. Forfenan
B. Zinc-eugenol
C. Endometazon
D. Biocalex
E. Rezodent

14. 64. C.M. Gutapercea poste are compound from:
A. Gutapercea 20%
B. Gutapercea 60%
C. Zn oxide 60%
D. Was 12%
E. Bariu sulphate 5%, metallic acids 3%
65. M.C.Positive properties for gutta – percha posts are:
A. Cause hermetically close of root canals
B. Are soluble and easy to be removed
C. Radiological opacity
D. Do not absorb saliva and microorganisms
E. Easy to be removed
66. C.M. more often gutapercea post are used in :
A. Mixt root canal filling ( classical method)
B. Ultrasound condensation technic
C. Inserting of gutapercea without paste
D. Vertical condensation ( hot method)
E. Vertical condensation ( cold method)
67. C.M. Silver poste are compound from:
A. Silver 99,8%
B. Silver 95,8%
C. Cooper and chromium 0,2%
D. Cooper, chromium and nichel 0,2%
E. Iron 4,2%
68. C.M. Pozitive properties of silver posts:
A. Good elasticity
B. Hard to desobturate
C. Antiseptical action
D. Out of apex irritate paradontium
E. Oligodimanic action

15. 69..M.C.Before filling the root canak with periodontitis must be asymptomatically
as:
A. Not to have painful sensations to mastication
B. In pause not to be painful
C. To vertical percussion to be painless
D. Palpation in region of root apex projection is painless
E. To be painless to horizontal percussion
70..M.C.The tooth with Periodontitis must be filled only if the following conditions
are respected:
A. Radicular turundae are dry
B. Paper points are wet
C. Turundae are un coloured
D. No smell
E. Weak smell, less coloured
71..S.C.Frequence of rotation for Lentullo during root canal filling is:
A. 30.000 rpm
B. 5000 rpm
C. 800 – 1000 rpm
D. 2000 rpm
E. 300 rpm
72. C.S. Lentullo in time of root canal filling must to rotate :
A. According to Clock pin
B. Against clock pin
C. Direction of rotation doesn’t matter
D. Initial According to Clock pin, in the end against
E. Manualy rotate lentula in time of filling.

16. 73. C.S. Root canal filling in periodontitis are made till:
A. Small pushing transapicaly
B. Radiological apex
C. Physiological isthmus
D. Anathomical apex
E. Filling level isn’t important.
74. C.M. In case if lentullo not achieve needed depth:
A. Distance till apex is filled , changing lentullo
B. Distance till apex is filled with Kerr file N.10-20
C. Distance till apex is filled with drill
D. Distance till apex is filled with miller file
E. Distance till apex is filled with dilator of canal
75. C.M. Finishing of root canal filling is when:
A. In tooth cavity no paste
B. In tooth cavity appear surplus of paste
C. Formation of con with material In mouth of entrance in canal
D. At the entrance is not formed con with material
E. Appear pain during root canal filling
76. C.S. Root canal filling with gutapercea , gutapercea is inserted with:
A. Fingers
B. Pincer
C. It is not important, important is to get the task
D. Probe
E. Smoother
77. C.S. rest of gutapercea post (extracanalar) is taken of:
A. With sharp Excavator
B. With bur
C. As patient wish
D. It is not necessary to remove it
E. Is adopted on all length of root canal till the filling.

17. 78. In which visit is applied permanent filling after filling with gutapercea:
A. In day of root canal filling
B. Next visit
C. As pacient wish
D. On bottom of the cavity is left a layer of paste 1 mm and cavity is filled with
Kavidur
E. It is not important when to aply permanent filling.
79. C.M. Correct root canal filling is made when:
A. Filling is homogeneous and continuous
B. Is made till radiological apex
C. Material is pushed transapicaly
D. Gutapercea is inserted transapicaly
E. Is made heterogeneous and fragmented.
80..S.C.The technique of lateral condensation of gutta – percha is done with:
A. With gutta condensor
B. Manuall plugger
C. Spreader
D. Needle from syringe
E. Ultrasounds in root canal together with post
81. S.C.Master cone from gutta - percha is introduced till:
A. Together with sealer
B. Physiologic apex
C. Radiologic apex
D. Anatomic apex
E. Apical foramen with creation of maximum close
82. S.C.Mechanical processing (instrumentation) of root canal in Periodontitis is
done by usage of:
A. Crown – Down technique
B. Step – Back
C. Step – Back, and Crow - Down
D. Initially Step – Back, ending with Crown – Down
E. Is not important

18. 83. M.C.Mechanical processing of root anal in apical Periodontitis is done with by:
A. With endodontic instruments
B. By thirds (1/3, ½, 1/3)
C. By thirds, begining with apical third
D. By thirds, begining with radicular isthmus
E. ABC
84..S.C.The gutta – percha condensation in vertical method is done by:
A. Spreader
B. Plugger
C. Gutta – condensor
D. Needle from syringe
E. Probe
85. M.C.Drying of root canal is done with:
A. Sterile paper points
B. Air from syringe tray
C. Turundae moisted in alcohol or ether
D. Turundae on radicular needle
E. Turundae moisted in vagothyl 36%
86. C.M. Silver posts are used:
A. Where other techinks are imposible
B. It is possible to use it in all teeth
C. In narrow canals
D. Their usage does not depend on canal state
E. In curbed canals
87. C.S. In case of presence of fractured instrument in upper molar:
A. We try to remove instrument
B. Refilling of root canal
C. In case of impossibility to fill effective root canal – we make radicular amputation
D. Hemisection
E. Tooth extraction

19. 88. C.S. In case of indication for conservative-surgical treatment, which method we
use in mandibular molars
A. Apical resection
B. Hemisection
C. Root amputation
D. Tooth extraction
E. Chiuretage
89. C.M. Complication of acute and exacerbated periodontitis:
A. Periostites
B. Acute osteomyelitis
C. Parodontitis
D. Gingivitis
E. Letal exetus
90. C.M. Perforation of root canal walls can be:
A. When instrument axe doesn’t fulfil to root canal axe
B. In time of incorrect instrumental preparation
C. In time of usage of blunt instruments
D. In time of usage of mechanical dilator
E. In time of usage of instruments with large calibre
91. M.C.The insufficient fixation or irresponsable mechanical processing of root
canal may lead to:
A. Inhalation of instruments
B. Syncope
C. Anafilactic shock
D. Trauma of soft tissue
E. Swallowing of instruments

20. 92. M.C.Inhalation or swallowing of instruments is possible after the:
A. Un conscious movements of patients tongue
B. Insufficient fixation of radicular needle during x-ray
C. The irresponsable instrumental cleaning of root canal
D. Insufficient fixation of instrument in the doctors’ hand
E. Incorrect behaviour of patent in dental chair
93. M.C.In case of swallowing the instrument is indicated:
A. Special diet (potatoe, pea )
B. Radiologic control along some days
C. Clyster
D. Surgical intervention to remove the instrument
E. Patient hospitalization in surgical department
94. C.M. With manipulation can cause appearance of subcutaneous emphizemus:
A. Large apical foramen
B. Drying of root canals with air pistols
C. Usage of solution with high concentration of hydrogen peroxide in time of root canal
working
D. Pushing of air under preasure into root canals
E. Root canal filling with liquid endodontical paste.
95.C.M. Instrumental fracture in time of root canal processing can be from:
A. Incorrect force apply on instrument
B. qualitative instrumental usage
C. The failure of the root axis by applying force to the tool
D. usage of sharp instruments
E. usage of qualitative stainless steel instruments

21. 96. C.M. To avoid instrument fracture dentist is obliged to observe the following
rules:
A. By making use of endodontic instruments in strict caliber succesivitate
B. To work with endodontic instruments only in wet canals
C. to sterilize the working part of instruments of the flame of spirtiera
D. To respect angles of curvature of pulpoextractoarelor, drill, dilators channel
E. time of instrument usage Is not important
97.C.M. in time of filling of which teeth filling material can enter in mandibular
foramen:
A. incisors
B. mandibular premolars
C. canines
D. in time of filling of distal root canals of inferior molars
E. teeth 8.
98.C.S. How many visits are needed for treatment of acute purulent periodontitis:
A. 1
B. 2
C. 3
D. 4
E. 2 or 3
99.C.S. How many visits are needed for treatment of chronic granulous
periodontitis in acutisation:
A. 5
B. 2
C. 3
D. 1
E. 1 or 2 visits
100.C.S. In how many visites is treated chronic fibrous periodontitis:
A. 1
B. 2
C. 3
D. 4
E. 1 or 2

22. 101. C.M. chronic granulomatous periodontitis of tooth 26,root canals are not
penetrable but at apical region have process , which tactics should have a
dentist:
A. Treat the tooth in 1 visit, filling the root canal till possible limit.
B. Leave in root canals of EDTA for 48 hours
C. Leave tooth open, in second visit will cross the canals
D. Remove on bandage, cross the canals, filling the canals
E. As a consecince of non filled canals tooth will be extracted.
102. C.M. periostites in region of teeth 24,25,26, chronic exacerbated periodontitis
of tooth 25. Dentist tactic:
A. Xray tooth 25
B. Patient go to surgeon for incision on transectorial plic 24,25,26
C. Phisiotherapeutical treatment
D. Drenage through Open teeth , mechanical and medicamentous processing of root
canals
E. Drenage through root canal, enlargement of apical constriction. Teeth leave open.
103. C.M. chronic granulous periodontitis tooth 36. In lingual channel presents of
fractured instrument in the bucal one filling material is only half of the length
ofchannel, the channel is curved, to pass it is impossible. Dentist action:
A. Indication for tooth Extraction
B. filling of distal root canal and permanent cavity filling
C. hemisection
D. Applying of turunda with EDTA, drenage
E. Remove the drenage and cross the root canals.
104. CM Convenţional permiabilization of the root canal is possible to divide into 3
stages:
A. Cavity preparation
B. Apical preparation of root canal (2-3mm)
C. Coronal preparation of root canal (3-5mm)
D. Central preparation of root canal
E. Preparation of trans apical zone.

23. 105.C.M. Instruments for root canal enlargement are:
A. K-rimer
B. K-file
C. H-file
D. Pulpextractor
E. Radicular fiel.
106. C.S. in tooth 24 was applied arsenical paste bandage. The patient was
submitted on time. The next visit is diagnosed – toxic medicamentous
periodontitis.Doctor action:
A. Extraction of the tooth 24
B. Remove the bandage, enlargement of tooth cavity and mechanical and
medicamentous root canal treatemnets, tooth leave open
C. Opening the cavity of the tooth, mechanical and medicamentous processing of root
canals. Leave bandage with unitiol.
D. indication for phisiotherapeutical treatment till pain releave.
E. interviewing the patient. Recommended irrigation with iodinol
107. CM Pacient was diagnosed with drug-induced periodontitis from arsenical
paste.dentist action:
A. Indication for tooth extraction
B. Indicate physiotherapy procedures
C. Opening the cavity of the tooth, mechanical and medicamentos processing of root
canals
D. We indicate mouth bath with sodium carbonate and salt water
E. on the mouth of root canal foramen is applied turunda melted in iodine solution.

24. 108. CS. Dentist action in treatment of toxic periodontitis caused by arsenical
paste:
A. Insert into root canal wet turunda with iodine solution which is dressing in dentin.
B. on canal orifice is left a turunda melted in iodine solution, Bandage
C. turunde melted in iodine solution leak and leave in the tooth cavity. apply 2-3 times
heated flour. Leave tooth open.
D intracanalar electrophoresis with a solution of KI (potassium iodide).
E. baths mouth indicated for patients with iodine solution and salt
109. C.M. Chronic fibrous periodontis tooth 13. 2/3 of crown are destroyed.
Medical action:
A. radiography to determine root state
B. It is going to be extracted
C. filling of apical third with "Apexit"
D. is going to prosthetics for making inlay
E. Persuade the patient to keep the tooth
110. C.M. The patient was diagnosed with chronic granulomatous periodontitis
tooth 26. Radiography reveals granuloma located in the region of the posterior
buccal root. Medical action:
A root canal filling in 1 visit
B. root canal filling in 3-4 visits
C. root canal filling in 2 visits
D. apixectomy of posterior buccal root
E. Hemisection

25. 111. C.M. Diagnosis - radicular cyst in the region of teeth 44,45. Medical action:
A. Extraction of teeth 44, 45 with cystectomy
B. root canal filling of teeth 44.45 with "Sealapex" and gutapercea
C. cystectomy
D. apexectomy of teeth 44.45
E. Hemisection
112. CM Criterias of efficient of endodontic treatment:
A. Removing the pain in acutisation of periodontitis
B. Depends on antiseptic solution which is used
C. Restoration of bone tissue when there is presence of changes in periapical tissues
during treatment
D. Depends on root canal filling
E. Restoration of function and anatomical shape of the tooth
113. C.S. in which period of time is estimated efficacy of endodontic treatmen in
granulomatous chronic periodontitis:
A. In the nearest time after treatment
B t.ill 6 months
C. After 2 or more years
D. after two months
E. after 3 months

26. 114. CM Erors during tooth cavity preparation:
A. Opening of the tooth cavity in one or two points, being mistaken with root foramens
B. Removing of too much hard tissue which cause of the weakening tooth crown
C. Lack of direct access to the root canals
D. Removing of undermined dentine
E. cavity with direct access to the root canals
115. CS more plastic endodontic instruments are :
A. Stainless steel
B. carbonized steel
C. Ni-Ti Alloys
D. Gauze
E. Steel
116.C.M. endodontic Instrumentul Gates Gliden are used for:
A enlargement of mouth fof entarence in root canal
B. removing of rest of dentin from root canals
C. Transition coronary segment as part of a part of the root canal
D. Processing of the apical third of the canal
E. remooving of gutta-percha from the root canal
117. CS endodontic instrument Largo is used for:
A. enlargement of the root canal orifice
B. The permeability of the upper third of the root canal
C. Enlargement of the apical orifice
D. Permiabilization of narrow canals
E. enlargement of the root canal

27. 118. C.M. working Technique with K-Reamer:
A. is inserted into root canal, are made clockwise rotationby ¼ or ½ turn
B. is inserted into root canal, then is rotated counter clockwise with ¼ or ½ turn
C. in time of work is strong pushed
D. is Elastic with increased capacity for excision
E. Perform penetration, rotation, retraction
119. C.M. instruments for enlargement and shaping of root canal walls:
A. "Endosore File"
B. Pulpoextractor
C. K-file
D. rasp
E. H-file (drill Headstrom)
120. CM Instruments for widening of the root canal:
A. K-file
B K-flexofile
C. K-Rimer
D. Golden medium K-flexofile
E. Hedstrom-file
121. CMInstrumente to widen the canal:
A. K-file
B K-flexofile
C. K-Rimer
D. Golden medium K-flexofile
E. Hedstrom-file

28. 122. CM widening technic of the root canal with Hedstrom, file (H-file):
A. excised tissues affected only in one direction - the retraction
B. dentin Excision is made on rotation
C. dentin excision is made on scraper
D. 90o-180o rotation and retraction
E. 180o - 360o rotation and retraction
123. C.S.Canal Leader 2000 is:
A. A multi-functionable angle piece
B. endodontic instrument for permiabilizarea of root canal
C. endodontic instruments for root canal filling
D. endodontic instrument for root canal enlargement
E. endodontic Instrument for root lavage
124. C.M.Piesa multifunctionable in –angle allow:
A. To determine the root canal orifices
B. mecshanical preparation of root canal
C. preparation of carious cavities
D. lavage and filling of root canals
E. To perform full rotation into root canal
125. M.C.In endodontic handpiece the instrument does:
A. Rotation movements alternate in limits 30-1500
B.Rotation movements alternate simultaniously with rotary one
C. Rotation movements alternate simultaniously with those pushing 0,4 – 0,8 mm.
D. To root canal processing does vertical movements by pushing and regulated with
practising pressure
E.Vertical movements by pushing and retraction without practising pressure while canal
processing.

29. 126. C.M. ideal root canal treatment is with usage of cofferdam:
A. prevent swallowing of dental dust, instruments, the lavage fluid channel
B. Defend canal tooth cavity from the hit of saliva and microorganisms entering
C.prevent instrument fracture into root canal
D. Prevent perforation of canal walls
E. Improve access to working field and root canal
127. CM Principle for creation of access to the root canal:
A complete removal of the pulp chamber roof
B. Removing the pulp chamber roof in projection of entarance of root canals
C. Introduction in the root canal of instruments without curvature
D. Removing undermined cavity wall ,cavity should be extended by the pulp chamber
wall
E. Respect precautions to avoid peforation of the pulp chamber
128. C.M. After working length determination we do:
A. permeability
B. Widening
C. level of the final filling
D. The apical opening
E. Choosing of main cone (central)
129. CS notion'’root canal working length "
A. Distance from the radiological apex till the orifices of root canal
B. The distance from the radicular apex of the root canal up to the hole
C Distance from thephysiologica narrow till the canal opening
D. Distance from apical foramen until the root canal orifice
E. Distance from anatomical foramentill root canal orifice

30. 130. Cs in clinical situations is determined the working length of the tooth and not
of the root. Why:
A working length of the tooth is more important for clinician
B. Working length of the tooth and the root is the same thing
C. Working length of the tooth - the distance from the physiological narrowing till
oclusal surface
D. becausein clinical situations it is not possible to determine the working length of the
root
E. There is not significance to be determined
131. C.M. The working length of the tooth is determined :
A the stopper is fixed by the table length to different tooth root
B. It should be confirmed radiologically
C. It is confirmed by the electronic measure of root canal length
D. insertion of the file till it foll down and appear pain
E. stopper is fixed by tabulated average length
132. C.M. After root canal treatment the canal must match the following
requirements:
A. To keep it’s the direction
B. To be different shapes and sizes
C. to have cone shape
D. to not have staps
E. end with apical structure

31. 133. C.M. Processing of curved root canals involve :
A. Instrument is givet shape of curved root canal
B. Ni-Ti instruments with active peak (aggressive)
C. The use of flexible instruments Ni-Ti alloy with passive peak
D. instruments Movements must be propulsive
E. instruments Movements must-propelled rotary exceeding 90o-100o
134. CM Function of canal lavage solutions during mechanical preparation of root
canal are:
A. dentinal debris and gangrenous rets Evacuation
B. removing of The bad smell
C. lavage solutions have mission as lubricant
D. The creation of conditions for treatment
E. antiseptic action, removing of oiled layer (organic and inorganic compounds)
135. M.C.Basic principles of the most effective and sparing method of apical
Periodontitis treatment are:
A. Carefully mechanical processing of infected root canal
B. Removal the action of the biogenic amine
C. Treatment of transapical focus till exudate disappearance
D. Desensitization of organism
E. Ulterior filling of root canal
136. M.C.Trypsin has:
A. Bacteriostatic effect and stimulates tissue regeneration
B. Anesthetic effect
C. Stimulates phagocytosis and ihibits hyaluonidase
D. Wide antimictobial spectrum
E. Distructive effect under bacterial toxines

32. 137.M.C.Medicamentous processing of root canals with Chlorhexidine has:
A. Bactericide action
B. Pronounced antiexudative effect
C. Antiseptic action
D. Desensitizing effect
E. Bactericide effect under gram pozitive and gram negative bacteria
138.M.C.Iodinol has:
A. The property to create conditions for exudate evacuation
B. Wide antibacterial spectrum and to induce tissue regeneration
C. Distructive effect
D. Antimicotic effect
E. Is not toxic and absent of antigenic qualities
139. CM.why in treatment of apical periodontitis are used steroids:
A. Because they have pronounced anti-inflammatory action
B. Possess a large antimicrobial-spectrum
C. Possess desensitizing effect
D. accelerates tissue regeneration
E. Possess antiexudativ effect
140. M.C.Doctor’s attitude when apical acute Periodontitis has medicamentous
origin (arsenic):
A. Immediately removal of coronal pulp and that radicular one
B. Mechanic processing and medicamnentous of root canals
C. Medicamentous processing of canals by antisepetics 2% chloramine, 3% H2O2
D. Mechanic processing and medicamentous of root canals, enlargement of apical
orifice, tooth is left opened
E. To leave in root canal a turundae moisted in 5% sol Unitiol or 1% Iodinol

33. 141. Cm in pronounced exudative process toothis necessary to:
A. mechanical and medicamentos preparation and then to seal
B for few days left open
C.to trepanned dental crown
D. to enlarged apical foramen
E. infiltrative anesthesia performed and expected attenuation of the inflammatory
process
142. M.C.In case of acute inflammatory process to attenuate the pain is
necessary to:
A. To trepannize the tooth by air turbine handpiece
B. To leave the tooth opened for several days
C. For trepannation to use mechanic hand piece
D. To enlarge apical orifice
E. To realize anesthesia
143. S.C.When there are intoxication symtoms (fever, headache, weakness) is
necessary to indicate:
A. Analgetics
B. Antibiotics
C. Tooth extraction
D. To realize anesthesia to transitory fold
E. Physical – therapeutical methods
144. C.M. in acute periodontitis with periostitis complication is effective:
A. to trepanned tooth and expect pain relieve
B. to made anesthesia in transition plic
C. injection of antibiotics in the transition plic
D. horizontal incision subperiosteal not less than 2cm, pending exudate
E. Extraction of tooth

34. 145. CS Treatment of acute periodontitis is determined by the rule:
A. In the first visit
B. In the second visit
C. In the third visit
D. In the second visit, available in 5-7 days after dolore sensations
E. On the fourth visit
146. C.M. Treatment of acute periodontitis ends the rule:
A required the patient
B. After removal sensations dolore
C. The disappearance of exudate
D. When probing and thermal factors action is painless
E. When palpation and percussion are painless
147. M.C.If after filling of root canal the pain appears then:
A. Is indicated fluctuorization
B. Are indicated analgetics
C. The incision is done on transitory fold
D. Mouth washings with saline solution
E. Is recommended to apply hot – water bottle till the pain is attenuated
148. M.C.The attitude with unpermeable canals to multirooted teeth is:
A. Is applied the com bined method of treatment: ipregnation and filling with Foredent
B. Electrophoresis with ied compound
C. Tooth is extracted
D. The tooth is filled in II nd visit
E. The root canals are filled on passing length and the patinet is warning about the
possible consequences.

35. 149. C.M. treatment of Acute apical periodontitis traumatic origin reduces to:
A. Introduction in the transitional plic 0.5 ml of hydrocortisone
B. perform radiography
C.fixation tooth
D. Remove the cause, symptomatic treatment indicate
E. perform electroodontometry
150. CM choosing treatment method of chronic apical periodontitis depends on:
A. Size of transapical tissue damage
B. The local manifestation of symptoms
C. to which group of teeth affected tooth is hanging
D. the patient's general status (endocarditis, nephritis, etc..)
E. Patient Visit
151. . M.C.Methods for chronic apical Periodontitis treatment are:
A. Conservative
B. Biologic
C. Conservative – surgical
D. Amputation method
E. Surgical
152. Cm in treatment of chronic apical periodontitis doctor solves the following
tasks:
A. Restoring the anatomical shape of the tooth
B. Action on flora and micro macrocanale
C. Restore function
D. Remove the action of biogenic amines
E. Remove inflammation of the periodontium

36. 153. C.M. In the treatment of chronic apical periodontitis in the doctor are the
following tasks:
A. Stimulation of periodontal tissue regeneration
B. acting on The inflammatory process of root canal
C. Mechanical Enlargement of root canal
D. body Desensitization
E. Enlargement of the apical orifice
154. CM Extension of the cavity on the lingual or oclusal surface in the treatment
of apical periodontitis provides:
A favorable access to canal
B. direct acess without obstaclesinto root canal of endodontic instruments
C. Fixing sufficient obturation
D. exudate evacuation
E. direct action on transapical tissues
155. Cm in treatment of chronic apical periodontitis is needed to respect rules of
caution:
A thorough and thoughtful performed mechanically remove debris that prevents root
trans-apical contents
B. removing of debris on thirds, from apical third
C. preventive Insertion of antiseptics into canal (2% sol. Chloramine)
D. to work attentive to avoid canal injection
E. Removing content on third root, start with third from coronary

37. 156. M.C.The narrow canals and obliterated are enlarged with EDTA, its action
is based on:
A. Formation of compounds with enamel calcium
B. Formation of compounds with dentine calcium
C. Demineralized effect followed by remineralized
D. Decalcination effect
E. Calcium solubleness
157. M.C.Method of root canal enlargement with EDTA is done in the following
way:
A. Cotton turunda moisted in EDTA sol. is introduced in the root canal for 20-30 sec.
B. A new one portion of sol. is introduced after 30 sec.
C. The already formed complex is absorbed and new one portion is introduced
D. EDTA sol. is pushed in canal with special syringe
E. The sol. is changed 2-4 times while 1-2 min.
158. C.M. Canal enlargement after EDTA action is carried out:
A. Drill
B. pulp extractor
C. Hedstrom
D. Alize
E. canal Dilator
159. C.M task for drying the root canal:
A. canal is processed with alcohol soaked tampons
B. We work with compressed air
C. The canal is processed with ether-soaked tampons
D. Puster
E. The dried paper cone

38. 160.M.C.The treatment of chronic periodontitis is reduced to:
A. Action under microflora of carious cavity
B. Evacuation of necrotized pulp
C. Action under microflora of cavity with different vedicamentous substances
D. Action under microflora of canal with different medicamentous substances
E. Mechanical dilatation of root canal and apical orifice
161. C.S. Absolute indicationin treatment of monoradicular in one visit is:
A. clean cotton turunda
B. painless percution
C. Presence of fistula
D. when the canal is completely crossable
E. satisfactory clinical evolution
162. Cs in case of acutisation of process after filling is necessary to:
A to introduced in transition plic 0.1 ml fold. Hydrocortisone
B. to introduced in transition plic 0.2 ml of hydrocortisone (dissolved in 2% floor.
Novocaine)
C. Applications on with Tantum Verde" 10 min.
D. Applications of corticosteroids ungvente
E. mouth bath with salt
163. CS. After filling of chronic periodontitis was acutisation, doctor actions:
A. Introduction in transition plic of 2% novocaine
B. Introduction in transition plic in root apx Projection 1 ml. Lincomycin 2% lidocaine
(1:1)
C. Introduction in transition plic 2 ml. 2% sol. lidocaine
D. Introduction in transition plicin papex projection sol. lincomycin
E. we indicate mouth baths "Rotocan" and analgesics

39. 164. CS Is it possible to treatm in a single visit chronic periodontitis in acut phase
in monoradicular teeth
A. Yes
B. No
C. Yes, if it indicated for soft tissue incision on the transition plic
D. No, because the result will be complicated by phlegmon
E. It is contraindicated
165. CM diferencial diagnosis criterias of acute serous periodontitis:
A. tooth pain appears only from preasure on tooth, gradually increasing
B. pain irradiation, tooth feel like "grown up"
C. Tooth mobility is not determined
D. It is possible mucosal hyperemia, pain on palpation
E. Changes in the lining surrounding mucosa of the tooth not determined
166. M.C.Criterion of differential diagnosis for acute serous Periodontitis:
A. The pain appears only to pressing, gradually increasing
B. Pain irradiation, the tooth is “prolonged”
C. Tooth mobility is not determined
D. Is possible hyperemia of mucosa, pain to palpation
E. Changes of surrounding mucosa of tooth is not determined
167. M.C.Hemisection is:
A. Is done to mandibular – molars
B. Extraction of root together with coronal part that belongs to it.
C. Is realized to maxillary molars
D. Is done to mandibular premolars
E. Root extraction without coronal part that belongs it

40. 168.M.C.Root amputation is:
A. Root extraction together with coronal part that belongs to it
B. Root extraction with out coronal part of tooth
C. Is done to maxillary molars
D. Is done to mandibulary molars
E. Is done to premolars
169. Cm in case of fracture of the instrument during root canal preparation is
necesary first to performed:
A tooth radiography
B. To inform patient
C. Determine the length of the instrument remained in hand
D. To attempted to remove instrument
E. To extract tooth
170. CM Which root canal filling material are more effectively used in the
treatment of chronic periodontitis:
A. Pasta with antibiotics
B. phosphate cement
C. Silapex or apexid
D. iodoform paste
E. Endometazon with gutta-percha cones
171.M.C.Doctor’s attitude in case of haemorrhage within root canal is:
A. To introduce cotton turundae moisted in Iodinol
B. To introduce cotton turundae in 3% sol H2O2
C. To introduce with alcohol
D. To introduce with physiologic solution
E. To introduce with Vagothyl solution

41. 172. C.M. Mechanism of action of hydrogen peroxide on the gangrenous contents of
the canal:
A. Dry
B. Thermal effect
C. dentin decalcification
D. Release of active oxygen
E. Formation of acid from contact with necrotic pulp
173. CS Presents of fistula is characteristic for:
A. Acute serous Periodontitis
B.acute purulent Periodontitis
C. Chronic granulos Periodontitis
D. Chronic granulomatous periodontitis
E. Chronic fibrous Periodontitis
174. C.M. The terms of treatment of chronic periodontitis depends on:
A. appearance of the group of tooth
B. Age of patient
C. root canals permeability
D. antibacterial activity of any medicaments
E. Form of periodontitis
175.C.S. Is it justified pushing trans-apical phosphate cement in the treatment of
chronic periodontitis. It should flow backward trans-apical root canal material:
A. Yes, because phosphate cement stimulates bone regeneration
B. No, because it is considered a foreign body
C. No, because it is considered a foreign body and prevent periodontal tissue
regeneration
D. No, because the result is acute inflammatory process
E. Yes, because it does not irritate the tissues and tooth color

42. 176. CS Basic properties of EDTA solution is:
A. antiseptic
B. dentin decalcination
C. Wetting of canal
D. Anti-inflammatory effect
E. Stimulation of regenerative processes
177. C.S. Silver postare indicated in ermethical filling of:
A large root canals
B. Canals with unformed apex
C. narrow curves canals
D. canals with perforated wall
E. All of the cases listed
178. CM Necrotic pulp from toothcanal willbe removed with pulp extractor with
the use of an preliminary antiseptical canal . for this should be used:
A. Sol. 1-2% soil. cloramică
B. Sol. metronidazole
C. Sol. 0.02% chlorhexidine
D. Sol. 5% tincture of iodine
E. Sol. 1% sol. iodinol
179. C.S. The most favorable form of chronic periodontitis is:
A. Chronic granulosa Periodontitis
B. Chronic fibrous Periodontitis
C. Chronic granulomatous periodontitis
D. Exacerbation of chronic periodontitis
E. ABC

43. 180. C.M. goals of Acute periodontitis treatment :
A. Restoring the anatomical shape
B. Removing of the inflammatory process in the periodontium
C. Prevention of destructive forms of periodontitis
D. Restoration of tooth function
E. Removing of the infection source
181. C.S. The basic method for diagnosis of chronic apical periodontitis is:
A. EOM
B. Radiography
C. probing
D. percussion
E. thermometry +
182.M.C.Lesions of hard tissue that appear in the period of development are:
A. Hypoplasia
B. Hyperplasia
C. Acidic necrosis
D. Erosion
E. Fluorosis
183.M.C.Lesions of hard tissue that appear in the period of development:
A. Dysplasia Cap-de-pont
B. Amelogenesis imperfecta
C. Dentinogenesis imperfecta
D. Wedge-form defect
E. Marble bones

44. 184. C.M.Marble bones make part from group of lesion which affect:
A. Nervous system
B. Vascular system
C. Hard dental tissue
D. Bone tissue
E. Gastro-intestinal tract
185. C.M. Hard dental tissue affection which appear in time of eruption of the
teeth:
A. Fluorosis and hipoplasy
B. Erosion and necrosis
C. Wedged defect and hiperestesy
D. Pathological abrasion and traumatism
E. Amiloginesis and dentinoginesis imperfect
186.C.M. Hard dental tissue affection which appear in time of eruption of the teeth:
A. Hipoplasy and hiperplasy
B. Fluorosis and Capdepont displasy
C. Erosion and necrosis
D. Dentinoginesy si ameloginesy imperfect
E. Marble bones
187. C.M. Hard dental tissue affection which appear after eruption of the teeth:
A. Necrosis
B. Erosion
C. Fluorosis
D. Hiperplasy
E. Hipoplasy
188.C.M. Hard dental tissue affection which appear after eruption of the teeth:
A. Hipoplasy and hiperplasy
B. Erosion and necrosis
C. Hiperestezy and abrasion
D. Dentinoginesis si ameloginesis imperfect
E. Fluorosis and marble bone

45. 189. C.M. Hard dental tissue affection which appear after eruption of the teeth:
A. Congenital lesion
B. Wedged defect and hiperestesy
C. Hiperplasy and fluorosis
D. Traumatism and necrosis
E. Apical periodontitis
190. C.M. congenital syphilis triad consist of:
A. Pfluger teeth
B. Parenchimatous keratitis
C. Congenital deafness
D. Congenital blinding
E. Hutchinson teeth
191. C.S. cause of hipoplasy appearance is :
A. Insufficient nutrition
B. Metabolic disturbances in children body
C. Kidney functional disturbance
D. Digestive system functional disturbance
E. Central nervous system functional disturbance
192. C.M. as a result of which cells functional disturbance of is developed
hipoplasy:
A. Eritrocites
B. Leucocites
C. Ameloblasts
D. Histiocites
E. In aggravated case of odontoblasts
193. S.C.Hypoplasia is classified in:
A. Acute and chronic
B. Mild, medium, heavy forms
C. Systemic, local
D. I,II,III degree
E. Temporary and permanent

46. 194. M.C.What are the characteristic signs of macula in hypoplasia:
A. Smooth
B. Soft
C. Shiny
D. Is not colored with dyes
E. Dull
195. C.S. shape changings of which teeth is named Hetchinson and Fornier teeth:
A. Canines
B. Premolars
C. Upper incisors
D. Lateral incisors
E. Central lower inicsors
196. shape changings of which teeth is named Pfluger teeth:
A. Canines
B. First Premolars
C. Second molar
D. First and second premolar
E. Incisors
197. C.M. Enamel hipoplosy is differentiated from:
A. Middle caries
B. Incipient caries
C. Enamel necrosis
D. Superficial caries
E. Enamel erosion
198.C.M. treatment methods of hipoplasy:
A. Tooth extraction
B. Restoration of defects with composite materials
C. Remineralized therapy
D. In aggravated case of prosthetic treatment
E. Orthodontic treatment

47. 199.C.S. Tetraceclinic teeth are as a result of administration in formation time and
mineralisation of hard tissue:
A. Pesticides
B. Nitrates
C. Hard metal salts
D. Steroid administration
E. Tetracecline
201. C.S. on which teeth more often is present local hipoplasy:
A. Incisors
B. Canies
C. Molars
D. Premolars
E. Wisdom teeth
202.S.C.Intoxication with fluor (fluorosis) appears after:
A. Increased consumption of tea
B. Consumption of fluoric salt
C. Increased content of fluor in potable water
D. Remineralization therapy
E. Antibiotic administration
203.S.C.Fluorosis is:
A. Endemic disease
B. Infectious disease
C. Zoosis
D. Viral provenience
E. Hereditary pathology

48. 204. C.S. primary manifestation of fluorosis is in:
A. Superior incisors and premolars
B. Rarely on inferior incisors and molars
C. Canines
D. Incisors and canines
E. Premolars and molars
205.S.C.The admitted concentration of fluor in potable water is:
A. 5 mg/l
B. 4 mg/l
C. 3,5 mg/l
D. 2,5 mg/l
E. 1,5 mg/l
206. C.M. Which concentration of flour in potable water can due to modification in
teeth:
A. 8mg/l
B. 7mg/l
C. 6mg/l
D. 5mg/l
E. 4mg/l
207. C.S. In localities with arid climate sometimes records advanced dental
fluorosis when fluoride content in drinking water is moderate (0.5-0.7 mg /
l).Which is the cause:
A. Consumption of tea
B. Consumption of fluoride salt
C. Chewing tobacco and tea leaf
D. Excessive intake of water in the body
E. Eating fish and foods fats

49. 208. S.C.What’s the effect of fluorine:
A. Cariogenic
B. Mutagenic
C. Cancerigenic
D. Cariostatic
E. Hemostatic
209. M.C.Following forms of fluorosis evolve withont tissue loss:
A. hatching form
B. Destructive
C. Erosive
D. Maculate (d)
E. Chalky – granular
210. C.M. With the loss of dental tissue forms of fluorosis develops following:
A. Distructive
B. Hashurated
C. Erosive
D. Maculated
E. Granulo-cretaceus
211. C.S. Maculated fluorosis is differentiated from:
A. Incipient caries
B. Hipoplasy
C. Acid necrosis
D. Hard tissue erosion
E. Hiperplasy

50. 212. M.C.The treatment for hatching maculate and granular forms of fluorosis
is:
A. Restoration of defects with composite materials
B. Tooth extraction
C. Prosthetic treatment
D. Discolouring treatment
E. Remineralization therapy
213. C.M. In treatment of destructive forms of fluorosis are used:
A. Composite Materials
B. prosthetical method
C. Surgical Methods
D. orthodontic method
E. complex Treatment
214. C.S.excessive formation of enamelis named:
A. hyperostosis
B. Ameloginesis
C. Dentinoginesis
D. Hypertonia
E. hyperplasia

51. 215. C.M. from hireditary disorders in development of dental tissues are:
A. Necrosis and erosion
B. imperfect Amelogenesis and dentinogenesis
C. Caria and pulpitis
D. Dysplasia Capdepont and marble bone disease
E. apical periodontitis and parodontitis
216. M.C.The syndrom Stainton – Capdepont is characterised by:
A. Modification of tooth crown colour
B. Early loss of enamel
C. Ruined abrasion
D. Hard tissue fragility
E. Undeveloped enamel and dentine
217. M.C.Dentinogesis imperfecta is characterised by:
A. Undeveloped coronal tissue
B. Absence of dental crown
C. Undeveloped roots
D. Absence of roots
E. Teeth mobility
218. S.C.Dental trauma can be:
A. Mild, medium, heavy
B. Acute, chronic
C. Acute, chronic, exacerbated
D. I, II, III degree
E. Localized, generalized

52. 219. CM. in dental luxation how should be bone tissue state for allowing the
preservation of tooth:
A. The bone tissue is intact on a stretch of more than half the length of root
B The bone tissue is intact during the whole length of the root
C. The bone tissue is intact on area of at least 1/3 of the root
D. The bone may be missing
E. may lack a wall of socket
220. C.M. What are the sequence of manipulations in tooth luxation:
A tooth extraction
B. anesthesia
C. placement of the tooth in his natural place
D. exterpation
E. fixation of The tooth
221. C.S. Reaction of teeth on which current indicate pulp necrosis:
A. 2-3 MKA
B. 20-25 MKA
C. 110 MKA and more
D. 60-70 MKA
E. 20 MKA and more
222. M.C.Treatment of complete luxation is:
A. Suture of alveola of dislocated tooth
B. The tooth is trepannized, pulp extirpation, root canal filling
C. Manufacturing of prosthetic device
D. Antispetic treatment of root and alveola
E. The tooth is left to its place and fixed

53. 223. C.M. When the fracture affect a part of crown without releasing of pulp
cavity:
A crown is restored with composite materials
B. exterpation
C. sending to the surgeon
D. sending to prosthetic
E. Restore with parapulpare post
224. C.M. on which length of root canal we apply the filling into root canal for
fixation of the post:
A 1/2
B. apical third
C. 1/4 apical
D. 1/5 apical
E. 1/6 apical
225.C.M. decisive diagnosis of root fracture is:
A. Patient Complaints
B.percution
C. Ultrasonography
D. radiography
E. Electroodontometry
226. C.M.in which shape of root fracture can not serve as support:
A. Longitudinal
B. Transversal
C. fragmentation
D. Diagonal
E. Oblique

54. 227. M.C.The treatment of chronic trauma of toth consists in:
A. Tooth extraction
B. Prosthetic treatment
C. Remineralization therapy
D. Restoration of defect
E. Removal of traumatic focus
228. C.M. primary manifestation of physiological abrasion are:
A. molars and premolars tubers
B. palatal and labial surface of the upper molars and premolars
C. The lingual and buccal surface of the lower molars and premolars
D. Edge and cusps of canines
E. The labial surface of the incisors
229. C.M. In case of right occlusion are created conditions for abrasion:
A. vestibular Surface of molars
B. incisors
C. palatal surface of the incisors
D. oclusal cusps
E. lingual surface of lower molars
230. C.M. In case of deep occlusion primarily is abrasion of:
A oclusal cusps
B. labial surface of the lower incisors
C. palatal surface of the upper incisors
D. palatal surface of canines and molars
E. lingual surface of the lower premolars

55. 231. C.S. in absence of which teeth there is a lack of intense abrasion of remaining
teeth:
A. canines
B. incisors
C. molars
D. premolars
E. wisdom teeth
232. S.C.Abrasion is classified in:
A. Acute and chronic
B. Localized and generalized
C. Mild, medium, severe
D. I, II, III degree
E. Acute, chronic, exacerbated
233. C.S. acid Necrosis is explained by direct action on the adamantine of:
A. Antibiotics
B. Basis
C. acids
D. Mercury
E. nitrates
234. M.C.Wedge – form defect is localized:
A. To teeth neck on palatal surface
B. To teeth neck on lingual surface
C. To occlusal surface
D. To teeth neck on buccal surface
E. To teeth neck on vestibular surface

56. 235. M.C.The walls of wedge – form defect are:
A. Soften (decrepited)
B. Hard shining
C. Is absent
D. Shiny
E. Smoothed
236. C.M. debut manifestation of Wedge defect are differenciate from:
A. fluorosis
B. hypoplasia
C. Careis
D. erosion
E. necrosis
237. C.M. wedged defects are differanciate from erosion by:
A. Radiography
B. their Form
C. Location
D. Responding to excitants
E. EOM
238. M.C.The treatment of beginning forms of wedge – form defect consist in:
A. Prosthetics
B. Rebuilding treatment
C. Remineralization therapy
D. Orthodontic – treatment
E. To reduce mechanical overloading

57. 239. C.M. In case of big defects of wedget defect treatment consists of:
A. filling
B. Extraction
C. Making artificial crowns
D. exterpation
E. Orthodontic Treatment
240. CS differential Diagnostic of tooth hard tissue necrosis is made with:
A wedge defect
B. hypoplasia
C. erosion
D. fluorosis
E. Amelogenesis imperfect
241. S.C.Erosion has the following form:
A. Conical
B. Rounded
C. Oval
D. Oval irregular
E. Flame
242. M.C.Erosion is differentiated from:
A. Fluorosis
B. Hypoplasia
C. Caries
D. Wedge form defect
E. Trauma of crown

58. 243. M.C.The treatment for erosion consists in:
A. Prosthetics
B. Removal of mechanical and chemical factors
C. Tooth extraction
D. Remineralization therapy
E. Filling of defects
244. C.S. Increased sensitivity to mechanical, chemical and thermal agents on
dental tissues is called:
A. Adentie
B. acute pain syndrome
C. hyperostosis
D. hyperplasia
E. Hyperesthesia
245. M.C.Hyperesthesia is registered in:
A. Fluorosis
B. Caries
C. Excessive abrasion
D. Wedge – form defect
E. Denudation of neck tooth
246. S.C.Hyperesthesia is divided into:
A. Systemic and localized
B. Acute and chronic
C. Easy, medium, severe
D. I, II, III degree
E. Permanent and periodical

59. 247. S.C.Hyperesthesia is differentiated from:
A. Caries
B. Apical periodontitis
C. Pulpitis
D. Marginal periodontitis
E. Neuralgia
248. S.C.The basic treatment of hyperestesia consists in:
A. Prosthetics
B. Filling
C. Remineralization therapy
D. Tooth extraction
E. Irradiation with ultraviolet rays
249. : Person who suffer of endemic goiter appear more often:
A. Periodontitis
B. decay
C. fluorosis
D. Abnormalities in dental arches, swelling of the mouth
E. Periodontitis
250. C.S. In hipoparatireosis there is a reduction in the blood content of:
A. Iron
B. Fluor
C. Calcium
D. Vit. B6
E. Magnesium

60. 251. C.S. In case of partial or total absence of parathyroid glands in children are
observed:
A. Adentition
B. sistemic enamel hypoplasia till its definitive absence (aplasia)
C. Underdevelopment of roots
D. roots apsences
E. absence of some groups of teeth
252. C.M. In case of acromegaly is found:
A. teeth volume enlarged
B. deposits of secondary dentine
C. A storage of secondary cement at root apex, which cause hipercimentosis
D. excessive storage of enamel
E. disproportionate skeletal growth
253. C.M. In gigantism is determined:
A. Hyperplasia
B. Hipercimentosis
C. aplasia
D. teeth enlargement
E. Increase the size of the roots
254. C.M. In cases of congenital hypothyroidism is noted:
A. early teeth eruption
B. changing of temporar with permanent teeth
C. absences of dental buds
D. Retention of tooth eruption
E. Retention of exchange of deciduous teeth with permanent

61. 255. S.C.Stainton – Capdepont syndrome is:
A. Infectious disease
B. Endemic
C. Avitaminosis
D. Hereditary
E. Oncologic
256. S.C.In Stainton – Capdepont syndrome are involved:
A. Milk teeth and those permanent
B. Incisors and canines
C. Premolars
D. Molars
E. Incisors and premolars
257. C.S. Central Simptome of Stainton-Capdepont syndrome is:
A. Underdevelopment enamel
B. Changing of teeth colour
C. Underdevelopment dentin
D. spontaneous nocturnal pain
E. pains caused by physical and chemical factors
258. S.C.In Stainton – Capdepont syndrome the “gigantic” dentinal conalliculi
are filled with:
A. Lymph
B. Blood
C. Water
D. Exsudate
E. Transudate

62. 259. M.C.In dysplasia Capdepont radiography shows:
A. Undeveloped roots
B. Normal formation of roots, that are commonly thin and short
C. Pathologic resorbtion of roots
D. Tooth cavity from coronal and radicular side is wide
E. Tooth cavity from canal and radicular side is narrow
260. S.C.Hypoplasia is localised in dependence on:
A. Number of afected ameloblasts
B. Number of afected odontoblasts
C. Patient’s systemic disease
D. Age of child when has supported the disease
E. Place of chemical factors action
261. C.S. Expresare degree of hypoplasia depends on:
A. Number of ameloblaşti involved in the process
B. Number of dentinoblaşti involved in the process
C. Chronic conditions that the patient has
D. The severity of illness incurred during tooth bud development
E. diseases borne by the mother during lactation
262. C.S. Local Hipoplazia more often occurs in premolars because:
A. During child development supports most diseases
B. action of Exogenous factors on premolars is more aggressive
C. Cleaning and self-cleaning of premolars is difficult
D. their primordial are located between roots of milk molars
E. Supports excessive masticatory effort

63. 263. C.M. Addition of which nutritional elements reduce fluorosis manifestation:
A. Vitamins C, D
B. Calcium gluconate
C. Nitrate
D. Pisticides
E. Vitamins B6, B12
264. M.C.Marble bones disease is also called:
A. Osteosclerosis
B. Osteoporosis
C. Osteopetrosis
D. Osteogenesis
E. Neo-osteogenesis
265. C.S. high electrical conductivity Possess:
A. muscle tissue
B. skin
C. dental hard tissues
D. Saliva
E. The bone tissue
266. C.S. Lower electrical Conductubility possess:
A. muscle tissue
B. skin
C. Hard Dental Tissues
D. Saliva
E. The bone tissue

64. 267. C.S. Electrical Conductibility of human body tissues through to:
A. presence of liquid solution
B. ions presence
C. Presence of negative charges
D. presence ofs positive charge
E. Presence of biopotentail in cell
268. CS Destination of hydrophilic bandage contribute to:
A. uniform distribution of electriccurent
B. Protect skin from burnings
C. decrease skin resistance to electricity
D. decrease tissue heating
E. lowing action of oxidized Electricity
269. C.M. Hydrophilic bandage is not be used in case of:
A. galvanization
B. Diatermy
C. ultrasonic treatment
D. diadinamoterapiei
E. fluctuarization
270. C.S. hydrophilic bandage is wetted :
A. In all of the following
B. cold tap water
C. Distilled water
D. warm solution containing inorganic salts or physiologic solution
E. Hot tap water

65. 271. Exist different action of different poles of electric continuous current
A. No, because through both electrodes cross same current flows
B. Yes, it is
C. No, because the direct current is distributed uniformly, the largest accumulates at the
cathode (-)
D. Yes, because the direct current is distributed uniformly in tissues with different
electroconductibility
E. No, because the biggest part pass through the anode (+)
272. S.C.Electrophoresis represents:
A. Treatment with electric current
B. Treatment with electric current of high frequence
C. Administration of medicamentous substances with continuous current
D. Administration of medicamentous substance with alternating current
E. With ultrasounds
273. CS. In Transcanalar electrophoresis carious cavity is better to close:
A. with dentin paste because it removes easily after the procedure
B. with cement, because tooth cavity is closed hermeticaly
C. with sticky wax
D. with normal wax
E. with compressed cotton swab because it keeps the electrode in carious cavity
274. C.S. Electrophoresis withpotassium iodide solution is often prescribed:
A. In case of severe periodontitis
B. For all forms of periodontitis
C. In case of chronic periodontitis with non-penetrating canal
D. In case of toxic periodontitis (Arsenic)
E. in The teeth which resists to electric current tightness

66. 275. C.S. Physiotherapy treatment method with ultra high frequency electric field
(UVC) is prescribed:
A. In case of chronic periodontitis
B. In cases of severe periodontitis
C. In case of chronic periodontitis with non-penetrating canals
D. In case of toxic periodontitis (Arsenic)
E. The teeth, which are not resistant to electric current tightness
276. C.S. In case of pain appearance after root canal filling(the presence of edema,
hiperemy) is reasonable to administrate:
A. Ultra high frequency electric field (UVC)
B. Microwave
C. Fluctorization
D. electrophoresis with novocaine
E. darsonvalization
277. S.C.Duration of electrophoresis cure is:
A. 20-30 min.
B. 6-8 sec.
C. 20-60 min.
D. 2-4 min.
E. 5-10 min.
278. C.S. the exhibition timeof canalar diathermocoaulation of Granulation tissue
are:
A. 2-3 sec.
B. 6-8 sec.
C. 60 sec.
D. 20 sec.
E. 1-2 min.

67. 279. S.C.Diathermal coagulation of root canal is administered in:
A. For root canal sterilization
B. Tooth devitalization
C. For diathermal coagulation of granular tissue and radicular pulp
D. For anesthesia
E. All previously mentioned
280. C.S. Electrophoresis with sol. 10% potassium iodide is:
A. In case of severe periodontitis
B. For all forms of periodontitis
C. In case of chronic periodontitis withnon-penetrating canal
D. In case of toxic periodontitis (arsenical)
E. The teeth that resists electric current tightness
281. M.C.Electroodontometry is used for:
A. To appreciate the state periferical nerve termination of pulp
B. To appreciate the state of periodontal nerve termination
C. For differential diagnosis of pulpitis and periodontitis
D. For differential diagnosis of caries and pulpitis
E. To appreciate excitability of periferical nerv – termination of periodontium
282. In electroodontometry are used electric current:
A. from 0 to 150 MCA
B. from 0 to 150 A
C. From 10 to 50 A
D. from 10 to 50 mcA
E. from 50 to 100 mcA

68. 283. M.C.In the treatment of chronic periodontitis is used:
A. Electrophoresis
B. Diatermal coagulation of granulations within root canal
C. Microwaves
D. Fluctuorization
E. d’Arsonval current
284. C.S. 150 mcA Electrical excitability of the pulp corresponds to disease:
A. Acute Pulpitis
B. Deep caries cavity .
C. Chronic parodontitis
D. gangrenous pulpitis
E. Chronic Periodontitis
285. C.S. In the absence of tooth reaction on power 100 mcA is assumed on the
following :
A. medium Caries
B. intact Tooth
C. Chronic Periodontitis
D. gangrenous pulpitis
E. profound caries
286. . S.C.Fluctuorization is recommended in:
A. Mulpiple caries
B. Chronic periodontitis
C. Chronic pulpitis
D. Acute periodontitis, marginal periodontitis
E. In all situations previously mentioned

69. 287. S.C.d’Arsonval current is recommended in:
A. Multiple caries
B. Chronic periodontitis
C. Chronic gingivitis
D. Neuralgia of trigeminus
E. Neuritis
288. S.C.In acute periodontitis is administered:
A. Electrophoresis
B. d’Arsonval current
C. Diathermal coagulation
D. Microwaves, fluctuorization
E. Electric field with ultrahigh frequence
289. CM morpho-functional Complex of periodontium involve:
A. Gingiva
B. Periodontium
C. alveolar bone tissue
D. Cement
E. enamel
290. M.C.Gingiva is composed from:
A. Interdental papilla
B. Marginal gingiva
C. Alveolar gingiva
D. Periodontal space
E. Interdental septum

70. 291. Which is the insertion place of adherent gum:
A. The space between adjacent teeth
B. alveolar bone
C. neck region of the teeth
D. hard palate
E. all named above
292. M.C.Morphologicaly the gingiva is composed from:
A. Epithelium
B. Submucous layer
C. Connective tissue or lamina propria
D. Fat tissue
E. Glandular elements
293 S.C.Marginal gingiva is localized:
A. In the space between the neighbouring teeth
B. On alveolar bone
C. Arround teeth neck
D. To radicular apex
E. To radicular bifurcation
294. CM Which are peculiarities of gingival epithelium structure:
A. It is a multi-layered tissue
B. possess keratinization properties
C. regenerates continuously
D. Contribute to the secretion of saliva
E. The presence of abundant glycogen in epithelial cells

71. 295. CS Celules of which gingival epithelium form contact with enamel apatite
crystals:
A. Oral Epithelium
B. basal epithelium
C. sulcular Epithelium
D. adherent Epithelium
E. The cells of each of these layers connect together with texture organic enamel apatite
crystals
296. M.C.The rose-pale shade of gingiva is because of:
A. Absence of submucous layer
B. Melanine concentration
C. Translucence of gingival epithelium due to blood vessels
D. Kind of nutrition
E. Concentration of fluor in potable water
297. CM Which is the concentration of glycogen in gingival epithelial cells in norm
and in case of inflammation of the gingive:
A. in norm gingival epithelial cells don’t contain glycogen or can be find traces of
glycogen
B. The amount of glycogen increases with inflammation
C. The amount of glycogen decrease in inflammation
D. gingival epithelial cells normally contain a significant concentration of glycogen
E. The concentration of glycogen in gingival epithelial cells is not subject to change in
case of inflammatory gingival process

72. 298. M.C.What is the gingival sulcus:
A. Its the space between tooth root and alveolar bone
B. Is the space between tooth surface and the gingiva that adhere to it
C. It is a pathologic formation
D. It is a physiologic formation
E. Is the synonim for “periodontal pocket”
299. CS At which level is the bottom of the gingival sulcus:
A. In the cervical region of enamel
B. In the dintino-enamel junction
C. In the anatomical neck
D. In the cervicalregion of root
E. varies depending on the age of the patient but without damaging the tooth circular
ligament
300. S.C.The depth of gingival sulcus in norm is:
A. 1,0 – 1,5 mm
B. 1,5 – 2,0 mm
C. 2,0 – 2,5 mm
D. 2,5 – 3,0 mm
E. 3,0 – 3,5 mm
301. C.S.gingival fluid is formed as a result of:
A.glandular secretions of gingival ephithelium
B. Increased permeability of the blood vessels in the gingival sulcus
C. local inflammatory processes
D. Some endocrine changes
E. Hypersecretion of salivary gland

73. 302. M.C.Gingival liquid has the following characteristics:
A. Has a similar composition with blood serum
B. Contains aminoacids, fibrinolytic factors, gamaglobuline
C. Ha protective function of surrounding periodontal tissue
D. The quantily of gingival liquid is increased in gingival inflammation
E. All the counting properties are characteristics of gingival liquid
303. C.S.gingival fluid can be collected from:
A. The spaces between the tooth root and the alveolar bone
B. salivary gland ducts
C. gingival sulcus
D. periodontal area
E. tooth cavity
304CS .which are defense mechanisms that determine the function of the gingive:
A gingival epithelium keratinization response as a response to mechanical pressure
B. Ability of lysozyme to depolymerize polysaccharides cell membrane of
microorganisms
C. Production of antibodies lymphoid cells and plasma cells
D. The ability of phagocytosis
E. protection function is performed by the above-named properties
305.. CS What type of fibers form circular ligament:
A. Elastic fibers
B. argirofile fibers
C. reticular fibers
D. collagen fibers
E. myelinated fibers

74. 306. CS By chemical structure and composition cement resembles with:
A. enamel Tissue
B. The bone tissue
C. dentinal tissue
D. pulp tissue
E. None of these tissues
307. M.C.How is called the cementum localized to the top of root:
A. Acellular
B. Cellular
C. Primary
D. Secondary
E. Pericementum
308. M.C.How is called the cementum localized to the root bifurcation:
A. Acellular
B. Cellular
C. Primary
D. Secondary
E. Pericementum
309. CM. continuous formation of cementoid tissue is carried out by:
A. acellular Cement
B. cellular cement
C. primary cement
D. secondary Cement
E. Periciment

75. 310. CS How is named cells that secrete the organic matrix of cement:
A. Odontoblaste
B. Cimentoblaste
C. Cimentoclaste
D. Fibroblasts
E. osteoclast
311. CM Which is the width of the periodontal space along the tooth root in time:
A. The largest dimension is recorded at the edge of the root apex and tooth socket
B. The largest dimension of the periodontal space is the middle third of the root
C. At the apex of the root periodontal area is narrower
D. In the middle third of the root periodontal space narrows
E. periodontal space has the same dimensions along the tooth root
312. M.C. What are structural elements that form periodontium:
A. Collagen fibers
B. Fibroblasts
C. Mastocytes
D. Blood vessels
E. Elastic fibers
313. CM periodontal Fascicule of collagen fiber orientation are divided as:
A transseptal fibers
B. parallel fibers
C. oblique Fibre
D circular Fibre
E. perpendicular fibers

76. 314. CM Periodontium is formed following cells:
A. Fibroblasts
B. Erythrocytes
C. mastocytes
D. plasmacytes
E. Histocytes
315. CS Which typeof cell under certain conditions can cause a cyst:
A. Fibroblasts
B. Mast cells
C. Osteoblasts
D. Epithelial Cells
E. Cementoblast
316. CS Which structural elements of periodontium constitute the support base of
the tooth:
A collagen fibers
B.celular elements
C. Elastic fibers
D. Blood vessels
E. Nerves
317. M.C.What are the main functions of periodontum:
A. Keeping the tooth in alveola
B. Force spreading in mastication process
C. Insurance of cementum nutrition
D. Sensory
E. Regeneration

77. 318. CS which fiber participate in periodontal regeneration in case of orthodontic
movement:
A. Elastic fibers
B. collagen fibers
C.argirofile fibers
D. Reticular fibers
E. myelinated fibers
319. CS How vary periodontal space size with age:
A. increase
B. decrease
C. stay unchanged
D. decrease only in the middle third of the tooth root
E decrease only at the apical part
320. CM which cells are basis of alveolar bone structure and root cementum:
A. Osteoblasts
B. Odontoblasts
C. Lymphocytes
D. Cementoblasts
E. epithelial cells
321. CM Which are the particularities of periodontal spaces sizes:
A. The wide portion of the periodontal space is in the neck region of the tooth
B. In the molar periodontal space is narrower than in the frontal
C. periodontal Spaces of tooth on the upper jaw are narrower than those of the lower jaw
D. The wider portion of the periodontal space is in the middle third of the root
E. molars has periodontal spaces wider than the frontal one

78. 322. CS Formation of alveolar bone is performed by:
A. Odontoblast
B. Cementoblast
C. Cementoclast
D. Osteoblasts
E. mastocytes
323. C.M.l alveolar bone tisue consists of:
A compact substance
B. muscular fibers
C. spongy substance
D. Odontoblaste
E. Bone marrow
324. CM radiological image of periodontium allows us to see:
A. alveolar bone tissue
B. dental pulp
C. enamel-dentin junction
D. dentoalveolar Ligaments
E. periodontal phant
325. CM What image presents us contact radiography (performed inside the
mouth):
A. periodontal tissues Status in the region of 3-4 teeth
B. Characteristics of one jaw
C. structural features of both jaws
D. Relationship between jaws
E. Structural changes in the tooth root apex 3-4 teeth

79. 326. CM Which is radiological aspect of interdental septa the rule:
A. It has a conical shape
B. unclear shape of septa top
C. Possess a pyramid shape
D. interdental septa have the appearance of an area of osteoporosis
E. interdental septa tops are rounded
327. CM how is the picture of the upper jaw bone trabeculae:
A are arranged horizontal
B. predominant network design
C. predominant vertical orientation
D. Are oblique
E. a uniform loop
328. C.M.Which are periodontal functions:
A. Defense
B. trophic
C. Plastic
D. Depreciation
E. The secretion
329. CS How is explained plastic function of periodontium :
A. continuous formation of periodontal tissues
B. The ability of keratinization
C. The uniform distribution of masticatory pressure
D. formation of gingival fluid
E. The presence of capillaries and nerves

80. 330. CM Which cell types release periodontal plastic function:
A. Cimentoblasts
B. Osteoblasts
C. Odontoblasts
D. Lymphocytes
E. Fibroblasts
331. CM which factors condition development of localized periodontal disease:
A. The incorrect application of fillings
B. Making the wrong prosthesis
C. Reducing reactivity body
D. Pulpites
E. Blood disorders
332. CM which factors condition development of generalised periodontal disease:
A. Endocrine Disorders
B. Gastrointestinal Disorders
C. Infectious Diseases
D. cardiovascular disease
E. atherosclerotic Changes of vessels
333. M.C.According to form of manifestation gingivitis can be:
A. Catarrhal
B. Granulant
C. Ulcerative
D. Erosive
E. Hypertrophic

81. 334 M.C.According to extension gingivitis can be:
A. Exacerbated
B. Cattarhal
C. Localized
D. Chronic
E. Generalized
335. M.C.According to form of evolution gingivitis can be:
A. Acute
B. Chronic
C. Exacerbated
D. Progressive
E. Tardy
336. M.C.According to form of manifestation marginal periodontitis can be:
A. Ulcerative
B. Mild
C. Abscessed
D. Severe
E. Generalized
337 M.C.According to evolution character are distinguished the following forms of
marginal periodontitis:
A. Acute
B. Chronic
C. Aggravated
D. Abscess
E. Remission

82. 338 M.C.According to form of extension marginal periodontitis can be:
A. Localized
B. Generalized
C. Rapidly
D. Tardy
E. Progressive
339. M.C.According to type of evolution periodonthosis is classified in:
A. Chronic
B. Acute
C. Remission
D. Exacerbated
E. Abscessed
340. M.C.According to form of manifestation periodontitis can be:
A. Mild
B. Medium
C. Severe
D. Catarrhal
E. Hypertrophic
341. M.C.What are the local factors that determine development of periodontal
desease:
A. Bacterial plaque
B. Occlusion anomalies
C. Diabetus mellitus
D. Insufficient hygiene of oral cavity
E. Diseases of gastro – intestinal system

83. 342. M.C.What are the general factors that determine development of
peridontal disease:
A. Anomalies of teeth position
B. Carious cavities
C. Endocrine diseases
D. Somatic diseases
E. Disorders of nervous system
343. M.C.What are the specific regions for bacterial plaque localization:
A. On proximal surfaces of teeth
B. To neck region
C. On occlusal surfaces
D. In fissures, pits of crown
E. On incisal margin
344. M.C.What are the causes of bacterial plaque
A. Particularities of anatomic structure and tooth position
B. Insufficient oral hygiene
C. Incorrect brushing of teeth
D. Qualitative and quantitative modifications of saliva and buccal liquid
E. Glucide prevailing and soft deposits in nutrition
345. S.C.What is the bacterial plaque:
A. Epithelial membrane that covers errupted tooth
B. Product of saliva composed from aminoacids and glucides
C. A crowd of bacteria and produces of vital activity that are fixed on tooth surface
D. A membrane of tooth protection
E. A produce of gingival liquid

84. 346. CM microbial plaque matrix is composed of:
A. Lactobacterii
B Streptococci
C. Protein
D. Sucrose
E. Polysaccharides
347. CM inorganic Components of microbial plaque:
A. Magnesium
B. Potassium - K
C. Iodine
D. Phosphorus
E. Zinc
348. CM Mark the correct order of the stages of dental plaque formation:
A. Formation of extracellular structure
B. Formation of film on the surface of the tooth
C. Growth of bacteria and plaque
D. Fixing bacteria on film
E. increase the pH of the dental plaque with accumulation of calcium salts
349. M.C.What the quantitative changes of saliva that cause development of
bacterial plaque:
A. Dicrease of saliva volume
B. Hypersalivation
C. Increase of lipase
D. Reduction of saliva secretion
E. High concentration of imunoglobulins

85. 350. M.C.What are the qualitative changes of saliva that cause development of
bacterial plaque:
A. Hyposalivation
B. Reduction of lipase quantitaty
C. Low concentration of imunoglobulins
D. Reduction of saliva secretion rhythm
E. Increase of lysozyme content
351. CM Which are clinical methods of examination and diagnosis of periodontal
disease:
A. Interrogation
B. Inspection exobucal
C. Inspection endobucal
D. Blood analises
E. Radiography
352. M.C.What are the auxilliary methods of periodontal examination:
A. Radiologic
B. Analyses
C. Electroodontometry
D. Shiller – Pisarev probe
E. Functional methods
353 S.C.Schiller – Pisarev probe permits us to determine:
A. Concentration of glycogen in gingiva
B. Situation of cappilaries in gingiva
C. Mobility degree of teeth
D. Manifestation of distructive changes of periodontium
E. Profoundness of periodontal pockets

86. 354. CS dental pathological mobility I degree corresponds to:
A. tooth Movements vestiblo-oral sense of maximum 1mm
B. tooth Movements vstibulo-oral sensemore than 2mm
C. tooth Movements vestiblo-oral and mesio-distal more than 1-2 mm
D. tooth Movements in every sense
E. tooth Movements in the vertical plan
355. dental pathological mobility II degree corresponds to
A. tooth Movements vestiblo-oral sense of maximum 1mm
B. . tooth Movements vstibulo-oral sensemore than 2mm
C. tooth Movements vestiblo-oral and mesio-distal more than 1-2 mm
D. tooth Movements in every sense
E. tooth Movements in the vertical plan
356. S.C.The profoundthess of gingival pocket is appreciated with:
A. Probe
B. Tweezer
C. Excavator
D. Endodontic needle
E. Plugger
357. M.C.Kulajenko probe will allow us to:
A. Appreciate cappilary stability to vacuum
B. Speed of haematoma formation
C. Concentration of glycogen in gingiva
D. Determination of hygienic index
E. Determination of periodontal pocket content

87. 358. CM Gravity of periodontitis by periodontal code is estimated as follows:
A. 0.1-1.0 - mild periodontitis
B. 1.0-4.0 - mild periodontitis
C. 1,5-4,0 - average periodontitis
D. 4.0-5,0 - average periodontitis
E. 4.0 to 8.0 - severe periodontitis
359.. CM gingivitis Gravity by gingival code is estimated as follows:
A. 0.1-1.0 - mild gingivitis
B. 1.0-4.0 - mild gingivitis
C. 1.1 to 2.0 - Average gingivitis
D. 4.0-5,0 - gingivitis average
E. 2.1 to 3.0 - severe gingivitis
360 S.C.PMA index permits us to appreciate the:
A. State of marginal periodontum
B. State of oral cavity hygiene
C. Degree of gingival retraction
D. Degree of gingival bleeding
E. Composition of periodontal pachet content
361 C.S. in norm hygienic index corresponds to:
A. till 1 ball
B. Over 1 ball
C. Up to 2 balls
D. Up to 0.5 balls
E. Up to 2.5 balls

88. 362. C.S.Which is the aim of the CPITN index apreciation:
A. Determination of the manifestation of clinical signs of periodontal disease
B. oral mucosa state
C. regional lymph nodes state
D. The bone resorbtion
E. Determination of gingival fluid
363. C.M. PI index is estimated as follows:
A. 0,1 - 1,0 - early stage or the degree of damage
B. 1.5 to 4.0 - II degree of damage
C. 4.0 -8.0 - III degree of damage
D. 1.0 to 4.0 - the degree of damage
E. 4.0 to 8.0 - II degree of damage
364. S.C.PI index will permit us to appreciate the:
A. Intensity and spreading of periodontal disease
B. State of oral hygiene
C. Degree of teeth mobility
D. Degree of gingival bleeding
E. Profoundness of periodontal pocket
365. S.C.Feodor – Volotkina index permits us to determine:
A. State of oral cavity hygiene
B. State of marginal periodontium
C. Intensity of marginal periodontium lesion
D. Degree of inflammation of gingiva
E. Profoundness of periodontal pocket

89. 366.C.S for assessment of Fyodorov- Volodchina index is used:
A.Solution Siller-Pisarev
B. Solution Parm
C. The solution Greene
D. Solution Wermillion
D. Solution Kulajenco
367. C.S.What determine the term "gingivitis"
A periodontal tissue inflammation with progressive destruction of periodontal and
alveolar bone
B. A process of periodontal dystrophic
C. Inflammation of the gingiva, accompanied by gingival tooth ligament damage
D. Inflammation of the gingiva, which evoluete without affecting gingival tooth ligament
E. A process of progressive destruction of periodontal tissues
368. M.C.What are the local factors that contribute to gingivitis development:
A. Microbial plaque
B. Crowded teeth
C. Diastema
D. Trema
E. Pulp inflammation
369. M.C.Clinical picture and differential diagnosis of gingivitis are:
A. Is manifested in old-age
B. Bone tissue resorbtion is absent
C. Tooth mobility
D. Absence of pockets
E. Purulent eliminations from periodontal pockets

90. 370. C.M Chronic catarrhal Gingivitis develop as a result of:
A. acute respiratory infections
B. dental plaque
C. Disturbance of the endocrine system
D. Action of long professional action
E. Inflammation of the dental pulp
371. M.C.Clinical signs for chronic catarrhal gingivitis are:
A. Absence of pain
B. Distructive process of interdental septum
C. Gingival bleeding during tooth brushing
D. Gingival papilla is hypertrophic
E. There is gingival hyperemia with cyanotic shade
372. CM Which are the clinical features of catarrhal gingivitis exacerbation stage:
A. Pain in time of eating
B. insignificant gingival Bleeding
C. gingiva is edematous, reddish color
D. Lack of dental plaque deposits
E. The presence of periodontal pockets
373. CM catarrhal symptomatic Gingivitis can occur :
A. infectious affection
B. Some allergies
C. generalized periodontitis
D. deep cavity
E. pulpitis

91. 374. M.C. Physiotherapy Treatment of catarrhal gingivitis include:
A. hydromassage;
B. Diatermocoagulation;
C. Vacuum massage;
D. Dynamic current;
E. Gingivotomy
375. CM which radiological methods can be applied to study periodontal disease:
A. thermometry
B. panoramic radiography
C. Sialografia
D. Electroodontometria
E. Orthopantomography
376. CM which laboratory methods allow us to establish the diagnosis and selection
of appropriate treatment of periodontal disease:
A blood analises
B. cytological method
C. thermometry
D. Radiography
E. Bacterial examination
377. CS Which is the result obtained from the method V.Kulajenco in catarrhal
gingivitis:
A. Accumulation of glycogen in epithelial cells
B. The occurrence in short time of hematoma as a result of the decrease in capillary
vacuum resistance
C. Increase the number of leucocytes and epithelial cells migration
D. Change the quantity and quality of gingival fluid
E. Increased collagenase activity

92. 378. M.C.What is the clinical picture of acute catarrhal gingivitis:
A. Gingival bleeding to easy touch with probe
B. Halitosis
C. Hyperplasia of interdental papilla
D. Pain during alimentation
E. Intoxication state of organism
379. CM chronic catarrhal gingivitis is manifested by:
A red gingiva
B. pocket depth 3.5-4.5 mm
C. Bleeding in time of brushing
D. gingivalenlargement
E. gingival cyanotic hiperemy
380. CM untreated chronic catharal gingivitis may be cause of:
A. hipertophic gingivitis
B. ulcerative gingivitis
C. wedged Defect
D. generalized periodontitis
E. dental caries
381. CS How could be called ulcerative gingivitis:
A. ulcerative Periodontitis
B. Papillon-Lefevre syndrome
C. Vincent necrotizing ulcerative gingivitis
D. Vincent necrotizing ulcerative periodontitis
E. Vincent necrotizing ulcerative Periodontotis

93. 382.C.S. which id Radiological picture of catarrhal gingivitis exacerbation stage:
A. absence of bone distruction
B. outbreaks of osteoporosis in the interdental septa
C. Detection of focals of osteosclerosis in the interalveolar septa
D. The presence of osteodistruction in the medium third of interdental septa
E. resorptionof interalveolar septa
383. M.C.What are the measures for treatment the chronic catarrhal gingivitis:
A. Removal of soft and hard deposits
B. Administration of antipyretics and antibiotics
C. Indications of physical – therapeutical treatment
D. Local application of antiinflammatory remedies and keraroplastic one
E. Application of sclerozing therapy
384. C.S which specific clinical features of mild catarrhal gingivitis:
A. Inflammation of the interdental gingiva
B. inflammation of marginal gingiva
C. inflammatory Processes of alveolar gingiva
D. The presence of gingival pocket
E. inflammatory of The gingival-dental ligaments
385. M.C.What are the causal factors in hypertrophic gingivitis development:
A. Pregnance
B. Addaministration of difenine
C. Blood system disease
D. Pubertary period
E. Hypovitaminosis C

94. 386. S.C.What is the degree of gingival hyperplasia in hypertrophic gingivitis of
medium gravity:
A. Covers ¼ from tooth surface
B. Covers 1/3 from tooth surface
C. Covers ½ from tooth surface
D. Covers more than ½ of tooth surface
E. Covers all the tooth surface
387. S.C.What is the degree of gingival hyperplasia in hypertrophic gingivitis of
mild gravity:
A. Covers till 1/3 from tooth crown
B. Covers more than ½ of tooth crown
C. Covers more than 2/3 of tooth crown
D. Covers all coronal surface
E. Covers till ½ from coronal surface
388. M.C.What is the degree of gingival hypertrophia in hypertrophic gingivitis
of severe gravity:
A. Covers 1/3 from tooth surface
B. Covers till ½ of tooth crown
C. Covers 2/3 from coronal surface
D. Covers all the coronal surface
E. Covers till 2/3 of coronal surface
389. CM . which are the results of laboratory tests for chronic catarrhal gingivitis:
A. Reduce the number of leukocytes
B. Increase in gingival fluid immunoglobulin
C. Increased collagenase activity
-lymphocytes
E. Increasing the concentration of erythrocytes in the blood

95. 390. CM which are anatomo-pathological changes in catarrhal gingivitis:
A. Disturbance of normal epithelium keratinisation
B. absence of phenomena paracheratosis
C. Reducing the amount of glycogen in squamous cell layer
D. thickening and cross-linking of collagen fibers
E. Increase the number of mastocytis
391. CM which factors could contribute to the development of necrotic ulcerative
gingivitis:
A. acute respiratory affection
B. Using the abundance of carbohydrates
C. Psychological stress and emotional
D. III molar eruption difficulty
E. increased concentration of fluoride in the drinking water
392. CM which are patient complaints if necrotic ulcerative gingivitis:
A pronounced. Pain in the gingiva during eating
B. Pain at night
C. gingival bleeding while brushing teeth
D. Mobility of teeth
E. bad smal, putrid mouth
393. CM Which are objective examination results of ulcero-necrotic gingivitis:
A. Poor oral hygiene
B. hyperemy of gingiva
C. gingival hypertrophy
D. gingiva at the periphery is covered by a necrotic membrane
E. The II degree of tooth mobility

96. 394. M.C.Clinical signs of ulcerative gingivitis are:
A. Acute beginning
B. Slow beginning
C. Gingival hyperplasia
D. Gingival haemorrhage
E. Prononced pain durring brushing and nutrition
395. M.C.The objective examination in ulcerative – necrotic gingivitis will
notice:
A. Limphatic submandibular nodules are made greater
B. General state is affected
C. Gingiva is covered with necrotic membrane of grey colour
D. Root exposure with 2-3 mm
E. Hypertrophic gingiva
396. CM bacterioscopic examination ofthe product extracted from the focar in
ulcero-necrotic gingivitis allows detection:
A. Fusobactery
B. Fungis
C. Treponema pallidum
D. Koch bacillus
E. spirochetes
397. CM which morphological changes occur in ulcero-necrotic gingivitis:
A. acanthosis epithelium
B. decrease vascular permeability
C. The increase in the number of collagen fiber
D. leukocyte infiltration
E. This phenomenon of stasis in blood and lymphatic vessels

97. 398. M.C.What particularities are characteristic for gingivitis:
A. Is often met to persons in age
B. Frequent connection of gingival inflammatory processes with demineralization
focuses (caries in stage of stain to the tooth neck)
C. Gingival bleeding to probing
D. Presence of periodontal pockets
E. Presence of ostedistructive processes on x-ray film
399. CM catharal Gingivitis must be distinguished from:
A. symptomatic catharal Gingivitis in case of infection s and allergies
B. symptomatic tatharal Gingivitis periodontitis
C. pulpitis
D. osteomyelitis
E. neuralgia
400. CS Which pathological process predominates in hipertrofic gingivitis:
A. The proliferation
B. ulceration
C. atrophy
D. sclerosis
E. destruction
401. C.M. evolution of hipetrofic gingivitis :
A. Acute
B. Chronic
C. acutisation
D. Progressive
E. Fast

98. 402. CM Which clinical forms are characteristic for hipetrofic gingivitis:
A. edematous
B. ulcerous
C. fibrous
D. Gangrenous
E. proliferative
403. CM Which conditional factors may make the process of gingival proliferation:
A. overflowing edges of fillings
B. Endocrine Disorders
C. Anomalies of tooth position
D. Hiposalivation
E. dental plaque
404. CM which are patients complaints in hipetrofic gingivitis:
A. The unusual aspect of gingiva
B. gingival Bleeding during brushing
C. nocturnal pain
D. Facial Asymmetry
E. Pain with irradiation in the head
405. CM fibrous hypertrophic Gingivitis is characterized by:
A. gingiva is cyanotic
B. The presence of dental deposits
C. gingival bleed at the slightest touch
D. lack of periodontal pockets
E. resorbtion ofinterdental septum

99. 406. M.C.Clinical signs of hypertrophic gingivitis are:
A. Presence of periodontal pockets
B. Teeth mobility
C. False pocket
D. Esthetic disorders
E. Gingiva dicreased in volume
407C.M. . Clinical Sign of ulcerative gingivitis are:
A dental calculus
B. bad smal
C. dental Mobility
D. Pain during eating
E. pockets
408. CM laboratory examination of gingivitis are:
A. Electroodontometry
B. Bacterial examination
C. general blood analises
D. surgeon Consultation
E. Sialografy
409. S.C.In gingivitis on radiogram determined the following changes:
A. Changes are not determined
B. Resorbtion interalveolar septum to 1/3 from root length
C. Absence of interalveolar septum
D. Resorbtion with osseouss pocket
E. Osteoporosis of interalveolar septum

100. 410. CM which are the principles of local treatment of catarrhal gingivitis:
A. Removing of calculusr and dental deposits
B. Removing of incorrectly made fillings and prosthesis
C. Application of anti-inflammatory remedies
D. Implementation of sclerosing remedies
E. Implementation of keratoplastic remedies
411 M.C.General treatment of catarrhal gingivitis includes:
A. Administration of vitamins C, P, B1, A, E
B. Oral administration of Vicasol
C. Gingival massage
D. Orthodontic treatment
E. Antibiotics administration.
412.C.M.Treatment ofcatarrhal gingivitis consists of:
A. mouth asanation
B. Removal of dental calculus
C. instillation of periodontal pockets
D. Applications of local inflammatory
E. vitamin therapy
413. M.C.Treatment for ulcerative gingivitis consists in:
A. Removal of necrotic pellicle
B. Removal of subgingival and supragingival calculus
C. Applications with antibiotics, glucocorticoids
D. Instillations in periodontal pocket
E. Gingivectomy

101. 414.. C.M. General Treatament of ulcerative gingivitis include:
A. cardiac remedies
B vitamin therapy
C. desensitized remedies
D. metronidazole per os
E. Bathes with antiseptic
415. M.C.The principles for hypertrophic gingivitis treatment are:
A. Removal of calculus
B. Sclerozing therapy
C. Electrophoresis with Heparine
D. Orthodontic treatment
E. Rinsing with antiseptics
416. CM Treatment of hypertrophic gingivitis consists of:
A. intrapapilar injections sol. 50-60% glucose
B. Applications of corticosteroid gels
C. Gingivoectomy
D. Administration of antipyretic remedies
E. The use of tranquilizers
417. CS Which is radiological picture of chronic catarrhal gingivitis:
A. Alveolar bone structure is normal
B. focar Bone resorption
C. Diffuse bone resorption
D. The presence of bone pockets
E. focars of osteodistrucţion of peaks of intralveolare septa

102. 418. CS Which is radiological picture ofacute catarrhal gingivitis:
A. Alveolar bone structureis normal
B. Diffuse bone resorption
C. outbreaks of osteoporosis and osteosclerosis
D. The presence of bone pockets
E. resorption on the third of intralveolar septa
419. CMwhich are changesin chronic catarrhal gingivitis :
A edematiation and hyperemia ofdental papillae
B. is hyperemy and cyanotic of gingival margin
C. The gingival margin is covered with gray deposits
D. interdentalpapillae are ulcerated
E. determination in gingival pockets of granulation tissue
420. C.M. which are obiective sign in Vincent ulcero-necrotic gingivitis:
A. papillae are edematous, hyperemous
B. The gingival margin is covered by a necrotic membrane which easily removable
C. Hypertrophy of gingival line
D. ulcers at the margins of gingiva
E. soft deposits on gingival margine
421.C.M. Local Tratamentul of Vincent ulcero-necrotic gingivitis include:
A. sclerosing therapy
B. Irrigation with antiseptic solutions and subsequent implementation of the solution
mixture with metronidazole chlorhexidin
C. Gingivoectomy
D. Application of antibiotic unguent
E. gingival anesthesia for removing of necroticdeposits

103. 422.C.M. General Tratamentul of Vincent ulcero-necrotic gingivitis include:
A. vitamin A. Therapy
B. Antibiotic therapy
C. Surgical treatment
D. desensibilization remedies
E. Physiotherapy
423. CM treatment of fibrous hypertrophic gingivitis is:
A. Suppression of traumatic factors and sclerosing therapy
B. Physical therapy
C. Application of anesthetic remedies
D. Administration of antibiotics per os
E. mouthirigation with antiseptic solutions
424. CM in Which age is more prevalent periodontal destructive changes:
A. Young people
B. In adolescents
C. In people over 40 years old
D. In people under 30
E. The old people
425. CM which ar local risk factors in the development of inflammatory
periodontal diseases:
A. Anomalies and deformation of maxilars
B. Diabetes
C. neuropathies
D. gingivolabial frenulum hypertrophy
E. oral vestibule reduced in volume