Presentation on utility of ultrasound in the arena of prehospital and retrieval medicine.
I intentionally strayed away from e-FAST & focussed more on the ongoing resuscitation of a medically shocked patient.
2. Disclaimer
I have not worked in retrieval.
I have not had formal ECHO training.
I have scanned lots of hearts.
I have asked lots of questions & read lots of answers.
I love ultrasound...
10. RUSH protocol
Primary US-assessment of undifferentiated shock
leading to a mechanical cause of obstructive shock. Second, the left ventricle can be
analyzed for global contractility. Determination of the size and contractility status of
the left ventricle will allow for those patients with a cardiogenic cause of shock to
be rapidly identified.10,11
The third goal-directed examination of the heart focuses
on determining the relative size of the left ventricle to the right ventricle. A heart that
has an increased size of the right ventricle relative to the left ventricle may be a sign
of acute right ventricular strain from a massive pulmonary embolus in the hypotensive
patient.12,13
The second part of the RUSH shock ultrasound protocol focuses on the determina-
tion of effective intravascular volume status, which will be referred to as ‘‘the tank.’’
Placement of the probe in the subxiphoid position, along both the long and short
axis of the inferior vena cava (IVC), will allow correct determination of the size of the
vessel. Looking at the respiratory dynamics of the IVC will provide an assessment
of the patient’s volume status to answer the clinical question, ‘‘how full is the
Table 1
Rapid Ultrasound in SHock (RUSH) protocol: ultrasonographic findings seen with classic
shock states
RUSH
Evaluation
Hypovolemic
Shock Cardiogenic Shock Obstructive Shock Distributive Shock
Pump Hypercontractile
heart
Small chamber
size
Hypocontractile
heart
Dilated heart
Hypercontractile
heart
Pericardial effusion
Cardiac
tamponade
RV strain
Cardiac thrombus
Hypercontractile
heart (early sepsis)
Hypocontractile
heart (late sepsis)
Tank Flat IVC
Flat jugular
veins
Peritoneal fluid
(fluid loss)
Pleural fluid
(fluid loss)
Distended IVC
Distended jugular
veins
Lung rockets
(pulmonary
edema)
Pleural fluid
Peritoneal fluid
(ascites)
Distended IVC
Distended jugular
veins
Absent lung sliding
(pneumothorax)
Normal or small IVC
(early sepsis)
Peritoneal fluid
(sepsis source)
Pleural fluid (sepsis
source)
Pipes Abdominal
aneurysm
Aortic
dissection
Normal DVT Normal
Abbreviations: DVT, deep venous thrombosis; IVC, inferior vena cava; RV, right ventricle.
32. RV strain on ECHO
RV:LV > 0.9
RV >30 mm
RV (free-wall) hypokinesis
McConnell’s sign
Paradoxical septal motion
Editor’s Capsule Summary
What is already known on this topic
Right ventricular dysfunction on echocardiography is
associated with a worse prognosis in pulmonary
embolism. Prompt treatment of pulmonary
embolism appears to be associated with better
outcomes.
What question this study addressed
Can emergency department bedside
echocardiography in patients with suspected
pulmonary embolism reliably identify those who have
right ventricular dysfunction, potentially leading to
earlier diagnosis and treatment?
What this study adds to our knowledge
This observational study of a convenience sample of
146 patients with moderate to high risk or confirmed
pulmonary embolism found that right ventricular
dysfunction was highly specific for pulmonary
embolism.
How this is relevant to clinical practice
Bedside echocardiography may be a useful addition
disposition could be expedited, resulting in a possible reduction
in morbidity and mortality.
Figure 1. Right ventricular dilatation (right ventricle:left
ventricle ratio >1:1) in this apical 4-chamber image of a
patient with an acute pulmonary embolism. RV, right ventricle;
LV, left ventricle; RA, right atrium; LA, left atrium.
Dresden et al Bedside Echocardiography for Diagnosis of Pulmonary Embolism
44. Lung rockets [aka. B-lines]
1. A comet tail
2. Arises from pleural line
3. Moves in concert with lung sliding
4. Does NOT fade
5. Laser-like, ie. well defined
6. Hyperechoic
7. Obliterates the A-lines
144 of acute circulatory failure are competing:
hypovolemic shock and distributive shock.
In this context distributive shock is assim-
ilated to septic shock not simply for sake
of simplicity, but also because the other
causes (anaphyllactic, spinal shock) are
infrequent and easy to be diagnosed. The
A-profile is correlated with a pulmonary ar-
tery occlusion pressure (PAOP) equal to or
lower than 18 mmHg with a 93% specific-
ity and 97% positive predictive value (2). A
shocked patient who displays the A-profile,
at this step, is called a FALLS-responder.
This patient can, and needs to receive fluid.
The FALLS-protocol is a therapeutic test.
It administers fluid with strict monitoring
of the clinical parameters of circulation
an
plo
of
pro
eve
cir
(w
sho
cas
wo
he
peu
Di
If
id
ove
lun
ter
cha
the
mm
an
Th
det
an
ste
is d
nis
cau
the
ram
“false-positives” makes little sense when another sign
is added: the A-line sign (i.e., no B-line seen), with
60% sensitivity but 100% specificity, a logical finding
interlobular septa come only from visceral pleura [23]
One motionless B-line discounts pneumothorax. Too
superficial linear probes make it difficult to distinguish
B-lines from other comet-tail artifacts (Figure 5)
Abolished lung-sliding plus absence of B-lines, at the
anterior area, in supine patients, is called A’-profile in
the BLUE-protocol (Figure 6). The third step—the
Figure 5 Interstitial syndrome and the lung rockets. Two examples
the text) are visible, called lung rockets (here septal rockets correlating w
called ground-glass rockets. Two examples of pulmonary edema (with gr
These parasites are ill-defined, short, and do not erase A-lines (arrows), am
ill” (2010 Ed, Chapter 17), with kind permission of Springer Science.
Lichtenstein Annals of Intensive Care 2014, 4:1
http://www.annalsofintensivecare.com/content/4/1/1
45. Lung rockets [aka. B-lines]
1. A comet tail
2. Arises from pleural line
3. Moves in concert with lung sliding
4. Does NOT fade
5. Laser-like, ie. well defined
6. Hyperechoic
7. Obliterates the A-lines
144 of acute circulatory failure are competing:
hypovolemic shock and distributive shock.
In this context distributive shock is assim-
ilated to septic shock not simply for sake
of simplicity, but also because the other
causes (anaphyllactic, spinal shock) are
infrequent and easy to be diagnosed. The
A-profile is correlated with a pulmonary ar-
tery occlusion pressure (PAOP) equal to or
lower than 18 mmHg with a 93% specific-
ity and 97% positive predictive value (2). A
shocked patient who displays the A-profile,
at this step, is called a FALLS-responder.
This patient can, and needs to receive fluid.
The FALLS-protocol is a therapeutic test.
It administers fluid with strict monitoring
of the clinical parameters of circulation
an
plo
of
pro
eve
cir
(w
sho
cas
wo
he
peu
Di
If
id
ove
lun
ter
cha
the
mm
an
Th
det
an
ste
is d
nis
cau
the
ram
“false-positives” makes little sense when another sign
is added: the A-line sign (i.e., no B-line seen), with
60% sensitivity but 100% specificity, a logical finding
interlobular septa come only from visceral pleura [23]
One motionless B-line discounts pneumothorax. Too
superficial linear probes make it difficult to distinguish
B-lines from other comet-tail artifacts (Figure 5)
Abolished lung-sliding plus absence of B-lines, at the
anterior area, in supine patients, is called A’-profile in
the BLUE-protocol (Figure 6). The third step—the
Figure 5 Interstitial syndrome and the lung rockets. Two examples
the text) are visible, called lung rockets (here septal rockets correlating w
called ground-glass rockets. Two examples of pulmonary edema (with gr
These parasites are ill-defined, short, and do not erase A-lines (arrows), am
ill” (2010 Ed, Chapter 17), with kind permission of Springer Science.
Lichtenstein Annals of Intensive Care 2014, 4:1
http://www.annalsofintensivecare.com/content/4/1/1
Dx = acute cardiogenic pulmonary oedema
Sn 97%, Sp 95%
53. References
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medicine clinics of North America, 28(1), 29–56– vii. doi:10.1016/j.emc.2009.09.010
Seif, D., Perera, P., Mailhot, T., Riley, D., & Mandavia, D. (2012). Bedside Ultrasound in Resuscitation and the Rapid Ultrasound in Shock Protocol.
Critical Care Research and Practice, 2012(3), 1–14. doi:10.1097/01.CCM.0000260680.16213.26
Lichtenstein, D. (2013). FALLS-protocol: lung ultrasound in hemodynamic assessment of shock. Heart, lung and vessels, 5(3), 142–147.
Lichtenstein, D. A. (2014). Lung ultrasound in the critically ill. Annals of intensive care, 4(1), 1. doi:10.1186/2110-5820-4-1
Lichtenstein, D. A., & Mezière, G. A. (2008). Relevance of lung ultrasound in the diagnosis of acute respiratory failure: the BLUE protocol. Chest, 134
(1), 117–125. doi:10.1378/chest.07-2800
MD, S. D., et al. (2014). Right Ventricular Dilatation on Bedside Echocardiography Performed by Emergency Physicians Aids in the Diagnosis of
Pulmonary Embolism. Annals of Emergency Medicine, 63(1), 16–24. doi:10.1016/j.annemergmed.2013.08.016
Nagdev, A. D., Merchant, R. C., Tirado-Gonzalez, A., Sisson, C. A., & Murphy, M. C. (2010). Emergency department bedside ultrasonographic
measurement of the caval index for noninvasive determination of low central venous pressure. Annals of Emergency Medicine, 55(3), 290–295. doi:
10.1016/j.annemergmed.2009.04.021
Meyer, G., Vicaut, E., Danays, T., Agnelli, G., Becattini, C., Beyer-Westendorf, J., et al. (2014). Fibrinolysis for Patients with Intermediate-Risk
Pulmonary Embolism. The New England journal of medicine, 370(15), 1402–1411. doi:10.1056/NEJMoa1302097
Sosland, R. P., & Gupta, K. (2008). Images in cardiovascular medicine: McConnell's Sign. Circulation, 118(15), e517–8. doi:10.1161/
CIRCULATIONAHA.107.746602
Rudski, L. G.., et al. (2010). Guidelines for the echocardiographic assessment of the right heart in adults: a report from the American Society of
Echocardiography endorsed by the European Association of Echocardiography, a registered branch of the European Society of Cardiology, and the
Canadian Society of Echocardiography. Journal of the American Society of Echocardiography : official publication of the American Society of
Echocardiography, 23(7), 685–713– quiz 786–8. doi:10.1016/j.echo.2010.05.010
http://resus.me/prehospital-ultrasound/