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§3.Periodontal  Diseases ,[object Object],[object Object],[object Object]
Content ,[object Object],[object Object],[object Object],[object Object],[object Object]
Definition of periodontal diseases ,[object Object]
[object Object],[object Object],Aetiology - How does periodontal disease start?
530
These bacteria possess biochemical capacities for the pathogenesis of inflammatory periodontal diseases  .
[object Object]
[object Object],[object Object]
There are two types of calculus: Supragingival calculus  is the hard deposit on top of the teeth, the kind we can see and feel.  Subgingival calculus  forms below the gums. It is just as hard and adherent as supragingival calculus, this allows the bacteria to congregate in greater numbers.  Though it is difficult to remove plaque formed on top of/within subgingival calculus, it is necessary to do because these pathogens cause more periodontal tissue breakdown.
[object Object],[object Object],[object Object]
Supragingival dental plaque, early stage in formation. Supragingival dental plaque, late stage in formation.
If  plaque is allowed to build up on the teeth,  toxin  or  enzymes  produced by the bacteria can  damage the attachment of the gums, periodontal ligament, and bone to the teeth.
[object Object]
If the inflammation is allowed to continue, the bone will begin to demineralize and dissolve. This stage is known as periodontitis . As the bone dissolves around the teeth, the teeth become unsupported and will fall out.
As more bone is destroyed, the tooth begins to loosen and, as the pockets get deeper, abscesses often occur.  Sometimes the gum will shrink as bone underneath is destroyed so that part of the root will become visible in the mouth
[object Object]
2.Local contributing  factors may increase the risk or severity of  periodontal disease: 1.Oral hygiene: 2.Food impaction 3.Tauma  from occlusion
3.Systemic factor: ,[object Object],[object Object],[object Object],[object Object],[object Object]
 
Classification of gingival disease Gingival disease Chronic gingivitis  Gingival  hyperplasia System  Acute necrotizing ulcerative gingivitis Acute  papilla gingivalista Acute multiple abscesses of the gingiva Margingnal gingvitis Hyperplastic gingvitis  Puberty  gingivitis  Pregnancy gingvitis  Drug-induced Hereditary gingival fibromatosis Epulis Gingivitis associated systemic diseases
Classification of periodontitis
[object Object],[object Object],[object Object],Acute necrotizing ulcerative  gingivitis Acute  papilla gingivalista Acute multiple abscesses of the gingiva
Eatiology Smear for fusospirochaetal bacteria  fusospirochaetal  bacteria  Some  reasons Defense  of  host  ANUG
Existing  gingivitis and periodontitis are important contributing factor   Deep pocket are suitable for bacteria growth Local or systemic factor  make bacteria grow
Clinical features ,[object Object],[object Object],[object Object],[object Object],ANUG
[object Object],[object Object],[object Object],[object Object],ANUG
[object Object],[object Object],[object Object],Acute  papilla  gingivalista Acute necrotizing ulcerative  gingivitis Acute  papilla gingivalista Acute multiple abscesses of the gingiva
Acute  papilla  gingivalista ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Clinical features ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],Acute  papilla  gingivalista
Multiple papillae Acute suppurative inflammation 3.Acute multiple abscesses of the gingivae Acute necrotizing ulcerative  gingivitis Acute  papilla gingivalista Acute multiple abscesses of the gingivae
clinical features ,[object Object],[object Object],[object Object],[object Object],[object Object]
1.Margingnal gingivitis ,[object Object],[object Object],[object Object],Chronic gingivitis  Margingnal gingvitis Hyperplastic gingvitis  Puberty  gingvitis  Pregnancy gingivitis
2. Hyperplastic gingivitis  ,[object Object],[object Object],[object Object]
3.Puberty  gingivitis  ,[object Object],[object Object],[object Object]
4.Pregnancy gingivitis  ,[object Object],[object Object],[object Object],Chronic gingivitis  Margingnal gingvitis Hyperplastic gingivitis  Puberty  gingivitis  Pregnancy gingvitis
1.Drug-induced hyperplasia ,[object Object],[object Object],[object Object],Gingival  hyperplasia Drug-induced gingival hyperplasia Hereditary  gingival fibromatosis Epulis
2.Hereditary  gingival fibromatosis
3.Epulis ,[object Object],[object Object],[object Object],Gingival  hyperplasia Drug-induced gingiva hyperplasia Hereditary  gingival fibromatosis Epulis
Epulis ,[object Object],[object Object]
Leukemia  ,[object Object],[object Object],Leukemia  Gingivitis associated systemic diseases
Treatment of gingival diseases includes ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
 
The main topic  Definition : Dental plaque Calculus Classification of periodontal diseases

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张结 3 1 1

  • 1.
  • 2.
  • 3.
  • 4.
  • 5. 530
  • 6. These bacteria possess biochemical capacities for the pathogenesis of inflammatory periodontal diseases .
  • 7.
  • 8.
  • 9. There are two types of calculus: Supragingival calculus is the hard deposit on top of the teeth, the kind we can see and feel. Subgingival calculus forms below the gums. It is just as hard and adherent as supragingival calculus, this allows the bacteria to congregate in greater numbers. Though it is difficult to remove plaque formed on top of/within subgingival calculus, it is necessary to do because these pathogens cause more periodontal tissue breakdown.
  • 10.
  • 11. Supragingival dental plaque, early stage in formation. Supragingival dental plaque, late stage in formation.
  • 12. If plaque is allowed to build up on the teeth, toxin or enzymes produced by the bacteria can damage the attachment of the gums, periodontal ligament, and bone to the teeth.
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  • 14. If the inflammation is allowed to continue, the bone will begin to demineralize and dissolve. This stage is known as periodontitis . As the bone dissolves around the teeth, the teeth become unsupported and will fall out.
  • 15. As more bone is destroyed, the tooth begins to loosen and, as the pockets get deeper, abscesses often occur. Sometimes the gum will shrink as bone underneath is destroyed so that part of the root will become visible in the mouth
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  • 17. 2.Local contributing factors may increase the risk or severity of periodontal disease: 1.Oral hygiene: 2.Food impaction 3.Tauma from occlusion
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  • 20. Classification of gingival disease Gingival disease Chronic gingivitis Gingival hyperplasia System Acute necrotizing ulcerative gingivitis Acute papilla gingivalista Acute multiple abscesses of the gingiva Margingnal gingvitis Hyperplastic gingvitis Puberty gingivitis Pregnancy gingvitis Drug-induced Hereditary gingival fibromatosis Epulis Gingivitis associated systemic diseases
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  • 23. Eatiology Smear for fusospirochaetal bacteria fusospirochaetal bacteria Some reasons Defense of host ANUG
  • 24. Existing gingivitis and periodontitis are important contributing factor Deep pocket are suitable for bacteria growth Local or systemic factor make bacteria grow
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  • 30. Multiple papillae Acute suppurative inflammation 3.Acute multiple abscesses of the gingivae Acute necrotizing ulcerative gingivitis Acute papilla gingivalista Acute multiple abscesses of the gingivae
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  • 37. 2.Hereditary gingival fibromatosis
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  • 42.  
  • 43. The main topic Definition : Dental plaque Calculus Classification of periodontal diseases