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Lecture on Blepharitis , Stye & Chalazion For 4th Year MBBS Undergraduate Students By Prof. Dr. Hussain Ahmad Khaqan
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Ophthalmology presentation, Eye clinic residency teachings, blepharitis, nasolacrimal duct obstruction, medical residency programmes, Ear nose and throat, dacryocystitis, lacrimal sac tumors, lacrimal sac drainage, nasolacrimal duct nursing care, dacrocystorhinostomy, endoscopic DCR, Eye lid inflammation, anatomy of the eye lid, punctum, canaliculi, tear drainage channel, infections of the eye lid, criggler's massage
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Lecture on Blepharitis , Stye & Chalazion For 4th Year MBBS Undergraduate Students By Prof. Dr. Hussain Ahmad Khaqan
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Ophthalmology presentation, Eye clinic residency teachings, blepharitis, nasolacrimal duct obstruction, medical residency programmes, Ear nose and throat, dacryocystitis, lacrimal sac tumors, lacrimal sac drainage, nasolacrimal duct nursing care, dacrocystorhinostomy, endoscopic DCR, Eye lid inflammation, anatomy of the eye lid, punctum, canaliculi, tear drainage channel, infections of the eye lid, criggler's massage
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Allergic conjunctivitis is inflammation of the conjunctiva (the membrane covering the white part of the eye) due to allergy.Although allergens differ among patients, the most common cause is hay fever. Symptoms consist of redness (mainly due to vasodilation of the peripheral small blood vessels), edema (swelling) of the conjunctiva, itching, and increased lacrimation (production of tears). If this is combined with rhinitis, the condition is termed allergic rhinoconjunctivitis. The symptoms are due to release of histamine and other active substances by mast cells, which stimulate dilation of blood vessels, irritate nerve endings, and increase secretion of tears. Treatment of allergic conjunctivitis is by avoiding the allergen (e.g., avoiding grass in bloom during "hay fever season") and treatment with antihistamines, either topical (in the form of eye drops), or systemic (in the form of tablets). Antihistamines, medications that stabilize mast cells, and nonsteroidal anti-inflammatory drugs (NSAIDs) are generally safe and usually effective.
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Gloria George
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About Author: Dr. Muhammad Zeeshan Hameed MBBS,FCPS(Resident Eye Surgeon) GMC/DHQ Teachng Hosptal, Gujranwala Pakistan. About Presentation: It covers following topics Introduction Epithelial Keratitis Disciform Keratitis Necrotizing Stromal Keratitis Neurotrophic Keratopathy Herpetic Iridocyclitis Prophylaxis Complications
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With a knowledge of anatomy, diseases of eyelids cannot be dealt with. Here we submit our presentation on anatomy of eyelids which will be helpful to all ophthalmogists, and students.
Anatomy Of The Eyelids
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Ankit Punjabi
The lecture has been given on Oct. 18th, 2010 by Dr. Khalid.
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College of Medicine, Sulaymaniyah
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This presentation illustrates the disease of blepharitis
Blepharitis
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Amr Mounir
Uveitis classification and clinical features
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Uveitis classification & clinical features 1
lijulk
Glaucoma associated with ocular trauma and intraocular haemorrhage,inflammatory
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Bipin Bista
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cornea physiology
Amrit Acharya
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Diseases of sclera
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clinical case presentation on anterior uveitis
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Manish Shetty
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Allergic conjunctivitis is inflammation of the conjunctiva (the membrane covering the white part of the eye) due to allergy.Although allergens differ among patients, the most common cause is hay fever. Symptoms consist of redness (mainly due to vasodilation of the peripheral small blood vessels), edema (swelling) of the conjunctiva, itching, and increased lacrimation (production of tears). If this is combined with rhinitis, the condition is termed allergic rhinoconjunctivitis. The symptoms are due to release of histamine and other active substances by mast cells, which stimulate dilation of blood vessels, irritate nerve endings, and increase secretion of tears. Treatment of allergic conjunctivitis is by avoiding the allergen (e.g., avoiding grass in bloom during "hay fever season") and treatment with antihistamines, either topical (in the form of eye drops), or systemic (in the form of tablets). Antihistamines, medications that stabilize mast cells, and nonsteroidal anti-inflammatory drugs (NSAIDs) are generally safe and usually effective.
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With a knowledge of anatomy, diseases of eyelids cannot be dealt with. Here we submit our presentation on anatomy of eyelids which will be helpful to all ophthalmogists, and students.
Anatomy Of The Eyelids
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The lecture has been given on Oct. 18th, 2010 by Dr. Khalid.
Ophthalmology 5th year, 1st & 2nd lectures (Dr. Khalid)
Ophthalmology 5th year, 1st & 2nd lectures (Dr. Khalid)
College of Medicine, Sulaymaniyah
www.ophthalclass.blogspot.com has the complete class and MCQs on lids and adnexa for undergraduate medical students. Class 1 in the series deals with the basic anatomy of the eyelid and the eyelid margin. A few of the congenital eyelid disorders are mentioned. Special emphasis is given to blepharitis – inflammation of the eyelid margin, its types, clinical features and management. Next, common causes of eyelid swellings including hordeolum or stye and chalazion are discussed. Finally a brief mention is made about disorders of the eyelashes – trichiasis, poliosis, madarosis and distichiasis.
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Dr. Faiza delivers an insightful lecture on the distinguishing characteristics of skeletal, smooth, and cardiac muscles, offering a comprehensive understanding of their histology, physiological anatomy, electrophysiological properties, and contractile mechanisms. Through meticulous tabulation and comparison, she aims to equip learners with a detailed comprehension of the unique features and functionalities of each muscle type. Beginning with a detailed exploration of histological differences, Dr. Faiza outlines key distinctions such as multinucleation in skeletal muscle, mononucleation in smooth and cardiac muscles, and the presence of striations in skeletal and cardiac muscles. She meticulously elucidates the structural arrangement of each muscle type, highlighting their cellular morphology and organization within tissues. Transitioning to physiological anatomy, Dr. Faiza navigates through various aspects including innervation, level of control, initiation of contraction, and modification by hormones. By delineating the role of the somatic nervous system in skeletal muscle control, and contrasting it with the autonomic nervous system's influence on smooth and cardiac muscles, she offers a nuanced understanding of neural regulation in muscle physiology. Furthermore, Dr. Faiza delves into electrophysiological properties, elucidating the mechanisms underlying action potential generation, duration, and ionic basis in each muscle type. Through insightful analysis, she reveals how differences in action potential characteristics contribute to variations in muscle contraction speed, duration, and resistance to fatigue. Finally, Dr. Faiza meticulously examines the contractile mechanisms of skeletal, smooth, and cardiac muscles, shedding light on the sliding filament theory, mechanisms of calcium action, and speed of contraction. By comprehensively comparing these mechanisms, she provides learners with a deep understanding of the intricate processes governing muscle contraction and function. Through engaging presentation and meticulous analysis, Dr. Faiza's lecture serves as a valuable resource for students seeking a thorough understanding of the fundamental differences between skeletal, smooth, and cardiac muscles.
Difference Between Skeletal Smooth and Cardiac Muscles
Difference Between Skeletal Smooth and Cardiac Muscles
MedicoseAcademics
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These are simplified slides discussing the regulation of cardiac output and venous return. Learning objectives: 1. Comprehend the determinants of cardiac output and factors affecting cardiac output 2. Comprehend the factors affecting stroke volume and heart rate and total peripheral resistance 3. Identify the factors regulating venous return 4. Discuss the causes of high and low output cardiac failure 5. Enlist the functions of veins and recognise the significance of venous reservoirs Study Resources: 1. Chapter 20, Guyton and Hall Textbook of Medical Physiology, 14th edition 2. Chapter 30 and 32, Ganong’s Review of Medical Physiology, 26th edition 3. Chapter 10, Human Physiology by Lauralee Sherwood, 9th edition 4. Physiology, Cardiac Output - StatPearls https://www.ncbi.nlm.nih.gov/books/NBK470455/
Cardiac Output, Venous Return, and Their Regulation
Cardiac Output, Venous Return, and Their Regulation
MedicoseAcademics
In this lecture on circulatory shock, Dr. Faiza, an Assistant Professor of Physiology, delves into the profound implications of inadequate blood flow throughout the body, leading to tissue damage. The session begins by defining circulatory shock and elucidating its physiological causes, including decreased cardiac output, diminished blood volume, decreased vascular tone, obstruction to blood flow, excess metabolic rate, and abnormal perfusion patterns. Dr. Faiza categorizes shock into various types, such as cardiogenic shock, hypovolemic shock, neurogenic shock, obstructive shock, anaphylactic shock, and septic shock, and explores the pathophysiological basis of each. Furthermore, the lecture examines the stages of shock, from the non-progressive phase where compensatory mechanisms aim for full recovery to the progressive phase where shock worsens steadily without intervention, potentially leading to irreversible damage. Dr. Faiza discusses therapeutic interventions for shock, including replacement therapy, blood and plasma transfusion, sympathomimetic drugs, and other adjunctive treatments like head-down positioning, oxygen therapy, and glucocorticoids.
Circulatory Shock, types and stages, compensatory mechanisms
Circulatory Shock, types and stages, compensatory mechanisms
MedicoseAcademics
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ANATOMY AND PHYSIOLOGY OF RESPIRATORY SYSTEM.pptx
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Difference Between Skeletal Smooth and Cardiac Muscles
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Cardiac Output, Venous Return, and Their Regulation
Cardiac Output, Venous Return, and Their Regulation
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Circulatory Shock, types and stages, compensatory mechanisms
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The Eyelids Husam Salhab
1.
THE EYELIDS Husam
Salhab
2.
3.
Modified sebaceous gland,
oily secretions Ciliary glands (glands of moll) Modified sweat glands open b/n adjacent lashes
4.
5.
Diseases of the
eyelid Abnormal position Inflammation Lumps (masses) Abnormalities of the lashes
6.
ABNORMAL LID POSITION
Ptosis Entropion Ectropion
7.
Ptosis: Abnormal
low position of the upper eyelid
8.
9.
10.
11.
12.
13.
14.
15.
16.
17.
Blepharitis squamos debris,
inflammation of the lid margin, skin and eyelash follicles
18.
19.
20.
21.
Chalazion
22.
23.
24.
25.
26.
27.
Pale, Ulcerated, Nodule
28.
29.
Hard nodules or
scaly patches
30.
31.
THANK YOU تمّت
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