This document summarizes a case study of a 40-year-old man presenting with syncope and angina. On examination, he had a bisferiens pulse and a harsh mid-systolic murmur. An echocardiogram revealed hypertrophic cardiomyopathy with a resting left ventricular outflow tract gradient of 27 mmHg. He was diagnosed with hypertrophic obstructive cardiomyopathy and started on atenolol and verapamil to alleviate his symptoms and prevent complications like sudden cardiac death.
11. WHAT ARE THE CAUSES OF CHEST PAIN WITH SYNCOPE ?
12. LOSS OF BLOOD UPPER GE BLEED HYPOVOLEMIA DUE TO CHEMICAL PERITOITIS, SEVERE PAIN ACUTE PANCREATITIS CARDIORESPIRATORY EMBARRASSMENT PLEURAL HEMORRHAGE CARDIORESPIRATORY EMBARRASSMENT TENSION PNEUMOTHORAX CARDIAC COMPRESSION PERICARDIAL TAMPONADE OBSTRUCTION TO CIRCULATION ACUTE PULMONARY EMBOLISM LARGE AREA OF MYOCARDIUM AT RISK, ARRHYTHMIAS ANGINA PECTORIS ARRHYTHMIAS(VT/VF) VASOSPASTIC ANGINA LARGE AREA OF MYOCARDIUM AT RISK, ARRHYTHMIAS ACUTE MYOCARDIAL INFARCTION MECHANISM CONDITION
33. LBBB RV PACING RV ECTOPY DELAYED ELECTRICAL ACTIVATION OF LV EARLY ELECTRICAL ACTIVATION OF RV AS IN TYPE B WPW SYNDROME. IN WPW SYNDROME, REVERSED SPLIT OCCURS ONLY WHEN THERE IS SIGNIFICANT PRE-EXCITATION SEVERE TRICUSPID REGURGITATION EARLY PULMONIC CLOSURE ANEURYSM OF ASCENDING AORTA POST-STENOTIC DILATION IN AS INCREASE OF HANGOUT INTERVAL ON THE AORTIC SIDE SEVERE AS SEVERE SYSTEMIC HYPERTENSION ACUTE MYOCARDIAL INFARCTION DURING AN EPISODE OF ANGINA CARDIOMYOPATHY SEVERE AR LARGE PATENT DUCTUS ARTERIOSIS PROLONGED LV MECHANICAL SYSTOLE CAUSES MECHANISM
38. INVESTIGATION NORMAL SINUS RHYTHM AXIS : (– 30°) LEFT ATRIAL ENLARGEMENT POOR PROGRESSION OF R WAVE LVH PRESENT LARGE NEGATIVE PRECORDIAL T WAVES ECG