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HEAD AND NECK IMAGING
EHAB ABOU ELFOTOUH. MD.
 Orbital Varices.
 Increased antero-grade flow, can result from vascular
malformations of the face and scalp.
 Obstruction of distal venous drainage, as cavernous sinus
thrombosis.
 Ab-normal retro-grade flow, as carotid–cavernous fistula.
 Increased intracranial pressure.
 Normal variant.
 Most common cause of spontaneous orbital
hemorrhage.
 Lesions result from congenital weakness in
post-capillary venous wall.
 Have a large communication with the venous
system and distend during maneuvers that
increase venous pressure.
 Have only a small
communication with
the venous system
and do not distend.
 Patients with usually
manifest stress or
painful proptosis.
 CT images:
 Normal
appearance, axial on
supine position.
 Or mild enlargement of
the involved veins.
 Increases venous
pressure is required to
demonstrate lesion
distensibility.
 Varices may be
smooth club-like,
triangular, or
segmental dilatation
of Opth. veins.
 Orbital mass of
vessels.
 At MR imaging:
 Hypo- to hyper-
intense signal on T1.
 Hyper-intense signal
on T2.
 Intense enhancing
pattern.
 Fed by ophthalmic artery branches.
 Consist of multiple congenital micro-vascular
connections between arteries and veins.
 Manifest with peri-orbital swelling, dilated
retinal veins, visible or palpable pulsations, an
audible bruit, glaucoma, and, visual field
defects.
 Oculocerebro-cutaneous
syndrome.
 Consists of unilateral
AVM of the visual
pathways and midbrain.
 Facial vascular nevi or
telangiectasias.
 Patients present with
intracranial hemorrhage,
optic atrophy and
pulsatile exophthalmos.
 Abnormal communication between the
cavernous sinus and one or more branches of
the internal or external carotid artery.
 Causes direct trauma, surgery, dural sinus
thrombosis, or spontaneously.
 Spontaneous with Ehlers-Danlos syndrome
and osteogenesis imperfecta.
 Manifest with the
classic triad of pulsatile
exophthalmos,
conjunctival chemosis,
and an auscultatory
bruit.
 Gradual decrease in
visual acuity.
 Palsy of cranial nerves
III, IV, V, and VI.
 CT or MR imaging:
 Proptosis.
 Extraocular muscle
enlargement.
 Superior ophthalmic
vein distention.
 Cavernous sinus
dilatation .
 Intracanalicular ophthalmic artery aneurysms are
extremely rare.
 More common carotid-ophthalmic artery
aneurysms.
 Arise at origin of the ophthalmic artery.
 May extend intra-cranially above the sella and
sometimes extend into or through optic canal.
 Lesion withdiameter of 2–3 mm are usually
asymptomatic.
 More than 3 mm may compress the artery or optic
nerve or rupture.
Orbital lesions ii
Orbital lesions ii

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Orbital lesions ii

  • 1. HEAD AND NECK IMAGING EHAB ABOU ELFOTOUH. MD.
  • 2.  Orbital Varices.  Increased antero-grade flow, can result from vascular malformations of the face and scalp.  Obstruction of distal venous drainage, as cavernous sinus thrombosis.  Ab-normal retro-grade flow, as carotid–cavernous fistula.  Increased intracranial pressure.  Normal variant.
  • 3.  Most common cause of spontaneous orbital hemorrhage.  Lesions result from congenital weakness in post-capillary venous wall.  Have a large communication with the venous system and distend during maneuvers that increase venous pressure.
  • 4.  Have only a small communication with the venous system and do not distend.  Patients with usually manifest stress or painful proptosis.
  • 5.  CT images:  Normal appearance, axial on supine position.  Or mild enlargement of the involved veins.  Increases venous pressure is required to demonstrate lesion distensibility.
  • 6.  Varices may be smooth club-like, triangular, or segmental dilatation of Opth. veins.  Orbital mass of vessels.
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  • 9.  At MR imaging:  Hypo- to hyper- intense signal on T1.  Hyper-intense signal on T2.  Intense enhancing pattern.
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  • 11.  Fed by ophthalmic artery branches.  Consist of multiple congenital micro-vascular connections between arteries and veins.  Manifest with peri-orbital swelling, dilated retinal veins, visible or palpable pulsations, an audible bruit, glaucoma, and, visual field defects.
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  • 14.  Oculocerebro-cutaneous syndrome.  Consists of unilateral AVM of the visual pathways and midbrain.  Facial vascular nevi or telangiectasias.  Patients present with intracranial hemorrhage, optic atrophy and pulsatile exophthalmos.
  • 15.
  • 16.  Abnormal communication between the cavernous sinus and one or more branches of the internal or external carotid artery.  Causes direct trauma, surgery, dural sinus thrombosis, or spontaneously.  Spontaneous with Ehlers-Danlos syndrome and osteogenesis imperfecta.
  • 17.  Manifest with the classic triad of pulsatile exophthalmos, conjunctival chemosis, and an auscultatory bruit.  Gradual decrease in visual acuity.  Palsy of cranial nerves III, IV, V, and VI.
  • 18.  CT or MR imaging:  Proptosis.  Extraocular muscle enlargement.  Superior ophthalmic vein distention.  Cavernous sinus dilatation .
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  • 26.  Intracanalicular ophthalmic artery aneurysms are extremely rare.  More common carotid-ophthalmic artery aneurysms.  Arise at origin of the ophthalmic artery.  May extend intra-cranially above the sella and sometimes extend into or through optic canal.  Lesion withdiameter of 2–3 mm are usually asymptomatic.  More than 3 mm may compress the artery or optic nerve or rupture.