2. Orbital Varices.
Increased antero-grade flow, can result from vascular
malformations of the face and scalp.
Obstruction of distal venous drainage, as cavernous sinus
thrombosis.
Ab-normal retro-grade flow, as carotid–cavernous fistula.
Increased intracranial pressure.
Normal variant.
3. Most common cause of spontaneous orbital
hemorrhage.
Lesions result from congenital weakness in
post-capillary venous wall.
Have a large communication with the venous
system and distend during maneuvers that
increase venous pressure.
4. Have only a small
communication with
the venous system
and do not distend.
Patients with usually
manifest stress or
painful proptosis.
5. CT images:
Normal
appearance, axial on
supine position.
Or mild enlargement of
the involved veins.
Increases venous
pressure is required to
demonstrate lesion
distensibility.
6. Varices may be
smooth club-like,
triangular, or
segmental dilatation
of Opth. veins.
Orbital mass of
vessels.
7.
8.
9. At MR imaging:
Hypo- to hyper-
intense signal on T1.
Hyper-intense signal
on T2.
Intense enhancing
pattern.
10.
11. Fed by ophthalmic artery branches.
Consist of multiple congenital micro-vascular
connections between arteries and veins.
Manifest with peri-orbital swelling, dilated
retinal veins, visible or palpable pulsations, an
audible bruit, glaucoma, and, visual field
defects.
12.
13.
14. Oculocerebro-cutaneous
syndrome.
Consists of unilateral
AVM of the visual
pathways and midbrain.
Facial vascular nevi or
telangiectasias.
Patients present with
intracranial hemorrhage,
optic atrophy and
pulsatile exophthalmos.
15.
16. Abnormal communication between the
cavernous sinus and one or more branches of
the internal or external carotid artery.
Causes direct trauma, surgery, dural sinus
thrombosis, or spontaneously.
Spontaneous with Ehlers-Danlos syndrome
and osteogenesis imperfecta.
17. Manifest with the
classic triad of pulsatile
exophthalmos,
conjunctival chemosis,
and an auscultatory
bruit.
Gradual decrease in
visual acuity.
Palsy of cranial nerves
III, IV, V, and VI.
26. Intracanalicular ophthalmic artery aneurysms are
extremely rare.
More common carotid-ophthalmic artery
aneurysms.
Arise at origin of the ophthalmic artery.
May extend intra-cranially above the sella and
sometimes extend into or through optic canal.
Lesion withdiameter of 2–3 mm are usually
asymptomatic.
More than 3 mm may compress the artery or optic
nerve or rupture.