The Heart See!

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The Heart SEE! Katharine Ferrante Grade 7 Holy Name School

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Ashley I’ve always been interested in what happens to the body/heart when someone has a stroke. I never really understood why it was always just one side of the body and how and why it affected one’s speech and movement. So, my question is, what happens during a stroke? A lot of what happens during a stroke is related to the nervous system, but the causes of a stroke are from the lack of blood supply to the brain. The lack of blood supply can be caused by a few different reasons; blood clot in an artery of the brain or rupture of a blood vessel in the brain, also called a cerebral hemorrhage. (The Macmillian Encylopedia, 2003) Because of the lack of blood/oxygen, portions of the brain start to die which causes paralysis and sometimes death. (Mader, 2015) Some of the signs someone is having a stroke is slurred speech, one side of the face is droppy, numbness or temporary blindness. (Mader, 2015) The reason that someone may get a blood clot or rupture a blood vessel is because of hypertension (high blood pressure) and other diseases related to the heart valves that is left untreated. High blood pressure is very much so preventable by taking care of yourself. Eating right, exercising and keeping stress levels down. Our textbook states that almost one in five young adults have high blood pressure. (Mader, 2015) The recovery of a stroke victim is usually good. Most have to go through therapy to regain any function lost but that is minute to what it could’ve been. You could say that a stroke is preventable; maintain a healthy lifestyle to prevent hypertension (high blood pressure) which in turn will prevent strokes. A healthy heart = a healthy life! Cassie After reading about the cardiovascular system this week I found many interesting facts. One interesting topic that caught my attention was what the heart specifically does. Each side of the heart has a job and responsibility when it comes to pumping blood. What does each side of the heart specifically do? The heart has two sides, separated by an inner wall called the septum.(Institute 2011) The right side of the heart pumps blood to the lungs. Blood is carried away from the heart through arteries. When the blood arrives at the lungs it then receives oxygen. The oxygenated blood is then pumped back to the heart where the left side distributes it to the rest of the body. Blood that comes back to the heart is carried through veins. · The pulmonary artery carries blood from the right side of the heart to the lungs to pick up a fresh supply of oxygen. (Institute 2011) · The pulmonary veins carry oxygen-rich blood from the lungs to the left side of the heart so it can be pumped to the body. (Institute 2011) Finding information in regards to the heart and how each side functions was pretty easy. The APUS library had several different articles in the database. Also, for other sources I made sure to look for certified sources ending in .gov or .net. James I chose to discuss the card.

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Sheep Heart: Circulatory/Respiratory Dissection Lab - Sheep Heart Circulatory and Respiratory systems are critical adaptations that allow animals to 1) take in oxygen from the external environment to make energy in cell respiration, 2) to get rid of carbon dioxide waste produced in cell respiration, and 3) cycle nutrients throughout the body for use by the cells. The respiratory system allows animals to do gas exchange, moving oxygen from the external environment into the blood and CO2 from the blood into the external environment. The circulatory system pumps the blood throughout the body to move oxygen, CO2, and nutrients to their final destinations. There are two types of circulatory systems in animals: closed and open. Both types have at least one pump (i.e. heart) to push the circulatory fluid throughout the body. In closed systems, the blood is ALWAYS contained within blood vessels, while in open systems, the fluid -- called Ifinph -- is pumped into open sinuses instead of blood vessels. Humans have a CLOSED circulatory system, so we will focus on that model today. Sheep Sheep are vertebrate chordate mammals like humans, so their circulatory anatomy is almost identical to ours. They have a 4-chambered heart and blood vessels that contain blood.Heart Diagram - External The sheep heart has 4 chambers -- a left and right atrium on top (the external surface of the atria is called an auricle), and a left and right ventricle on bottom. Note that whenever we refer to anatomical positions, we refer to left and right from the position of the organism we're looking at. So the "right" is on our left, and vice versa. You can see the pulmonary trunk/artery in blue on top of the heart, and the aorta in red. These two arteries carry blood away from the heart after it has been pumped out of the ventricles. The pulmonary veins and vena cava return blood to the atria. The cells of the heart are fed oxygen via blood in the coronary arteries, which are visible on the surface of the heart. Blockage of these arteries starves the heart of oxygen, resulting in a heart attack.The basic layout of the mammalian heart. Find the four chambers of the heart: the left and right atria on top, and the left and right ventricles on bottom. Remember that the heart's purpose is to pump blood throughout the body. Which chambers of the heart beat first? Notice the valves separating them. Which valve is on the right side of the heart? The left? What is the purpose of these valves? Do you see any other valves in the heart?.

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Step into the captivating realm of cardiovascular physiology as Dr. Faiza, an esteemed Assistant Professor of Physiology, unravels the intricacies of recording an electrocardiogram (ECG). In this illuminating lecture, students are guided through the fundamental principles of ECG interpretation, from lead systems and electrode placement to the analysis of waves, segments, and intervals. Beginning with an exploration of the twelve-lead system, Dr. Faiza elucidates the significance of bipolar limb leads (I, II, III), unipolar limb leads (aVR, aVL, aVF), and unipolar chest leads (V1-V6). Through mnemonic aids and clinical anecdotes, students gain a comprehensive understanding of lead placement techniques, enabling them to navigate the complexities of ECG recording with confidence and precision. As the lecture unfolds, Dr. Faiza delves into the nuances of ECG interpretation, empowering students to correlate ECG findings with electrophysiological events in the cardiac cycle. With a focus on waveform morphology and rhythm analysis, students learn to determine heart rate, rhythm, and axis deviation, deciphering the electrocardiographic manifestations of various cardiac pathologies and physiological states. Moreover, the lecture explores the normal waves, segments, and intervals of an ECG, providing students with a detailed roadmap for interpreting these essential components. From the P wave to the T wave, students gain insight into the voltage, duration, and clinical significance of each waveform, equipping them with the skills to identify aberrations and anomalies in ECG tracings. Enriched with clinical pearls and practical tips, this lecture serves as a cornerstone for students embarking on their journey into cardiovascular medicine. By demystifying the art of ECG interpretation, Dr. Faiza empowers students to harness the power of this diagnostic tool in clinical practice, fostering a deeper appreciation for the intricate interplay between electrical activity and cardiac function.

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In this lecture on circulatory shock, Dr. Faiza, an Assistant Professor of Physiology, delves into the profound implications of inadequate blood flow throughout the body, leading to tissue damage. The session begins by defining circulatory shock and elucidating its physiological causes, including decreased cardiac output, diminished blood volume, decreased vascular tone, obstruction to blood flow, excess metabolic rate, and abnormal perfusion patterns. Dr. Faiza categorizes shock into various types, such as cardiogenic shock, hypovolemic shock, neurogenic shock, obstructive shock, anaphylactic shock, and septic shock, and explores the pathophysiological basis of each. Furthermore, the lecture examines the stages of shock, from the non-progressive phase where compensatory mechanisms aim for full recovery to the progressive phase where shock worsens steadily without intervention, potentially leading to irreversible damage. Dr. Faiza discusses therapeutic interventions for shock, including replacement therapy, blood and plasma transfusion, sympathomimetic drugs, and other adjunctive treatments like head-down positioning, oxygen therapy, and glucocorticoids.

Circulatory Shock, types and stages, compensatory mechanisms

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In this captivating lecture, Dr. Faiza, an esteemed Assistant Professor of Physiology, embarks on a comprehensive journey into the multifaceted realm of local blood flow regulation. With meticulous detail and clarity, she unravels the intricate mechanisms that govern the dynamic balance between tissue perfusion and vascular resistance, shedding light on both acute and chronic aspects of blood flow control. Beginning with an exploration of acute influences, Dr. Faiza elucidates how physiological factors such as metabolic demand, myogenic responses, and autoregulatory mechanisms orchestrate rapid adjustments in local blood flow. Students gain insight into the vasodilator theory, which elucidates the role of metabolic byproducts and vasoactive metabolites in promoting vasodilation, alongside the oxygen demand theory, which underscores the interplay between tissue oxygenation and nutrient demand. Delving deeper, the lecture delves into the fascinating realm of humoral control, where hormones and vasoactive substances wield significant influence over vascular tone. Dr. Faiza navigates through the intricacies of endothelial-derived factors like nitric oxide (NO) and histamine, alongside hormonal regulators such as angiotensin II and vasopressin. Through vivid explanations and illustrative examples, she unveils the intricate dance of vasodilation and vasoconstriction orchestrated by these biochemical messengers. Moreover, the lecture extends its gaze to the extrinsic control of blood flow, where sympathetic innervation exerts a profound impact on vascular dynamics. Dr. Faiza elucidates the role of sympathetic tone in modulating vascular resistance, highlighting its implications in diverse physiological and pathological scenarios. From skin perfusion to coronary circulation, students gain a nuanced understanding of how neural inputs shape local blood flow patterns. Enriched with clinical insights and contemporary research findings, this lecture equips students with a holistic understanding of blood flow regulation. By exploring concepts like active and reactive hyperemia, vascular remodeling, and collateral circulation, Dr. Faiza fosters a deeper appreciation for the adaptive responses of the cardiovascular system to changing physiological demands and pathological insults.

Control of Local Blood Flow: acute and chronic

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