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By: LOKENDRA YADAV
   To recognize the normal rhythm of the heart -
    “Normal Sinus Rhythm.”
   To recognize the 13 most common heart
    arrhythmias.
   To recognize an acute myocardial infarction on
    a 12-lead ECG.
The 12-Lead ECG contains a wealth of
information. In Module V you learned that ST
segment elevation in two leads is suggestive
of an acute myocardial infarction. In this
module we will cover:
    ST Elevation and non-ST Elevation MIs
    Left Ventricular Hypertrophy
    Bundle Branch Blocks
   When myocardial blood supply is abruptly
    reduced or cut off to a region of the heart, a
    sequence of injurious events occur beginning
    with ischemia (inadequate tissue perfusion),
    followed by necrosis (infarction), and eventual
    fibrosis (scarring) if the blood supply isn't
    restored in an appropriate period of time.

   The ECG changes over time with each of these
    events…
Ways the ECG can change include:
                         ST elevation &
                            depression




                                     T-waves

                           peaked     flattened
Appearance                inverted
of pathologic
Q-waves
Non-ST Elevation
There are two
distinct patterns
of ECG change
depending if the
infarction is:                           ST Elevation




–ST Elevation (Transmural or Q-wave), or
–Non-ST Elevation (Subendocardial or non-Q-wave)
The ECG changes seen with a ST elevation infarction are:

 Before injury Normal ECG

 Ischemia      ST depression, peaked T-waves, then
               T-wave inversion

 Infarction    ST elevation & appearance of
               Q-waves
 Fibrosis      ST segments and T-waves return to
               normal, but Q-waves persist
Here’s a diagram depicting an evolving infarction:
A. Normal ECG prior to MI

B. Ischemia from coronary artery occlusion
    results in ST depression (not shown) and
    peaked T-waves

C. Infarction from ongoing ischemia results in
    marked ST elevation

D/E. Ongoing infarction with appearance of
   pathologic Q-waves and T-wave inversion

F. Fibrosis (months later) with persistent Q-
    waves, but normal ST segment and T- waves
Here’s an ECG of an inferior MI:
Look at the
inferior leads
(II, III, aVF).

Question:
What ECG
changes do
you see?
ST elevation
and Q-waves
Extra credit:
What is the
rhythm? Atrial fibrillation (irregularly irregular with narrow QRS)!
Here’s an ECG of an inferior MI later in time:
Now what do
you see in the
inferior leads?

ST elevation,
Q-waves and
T-wave
inversion
The ECG changes seen with a non-ST elevation infarction are:

Before injury Normal ECG

Ischemia        ST depression & T-wave inversion

Infarction      ST depression & T-wave inversion

Fibrosis        ST returns to baseline, but T-wave
                inversion persists
Here’s an ECG of an evolving non-ST elevation MI:
Note the ST
depression and
T-wave
inversion in
leads V2-V6.

Question:
What area of
the heart is
infarcting?

Anterolateral
Compare these two 12-lead ECGs. What stands out
 as different with the second one?




       Normal             Left Ventricular Hypertrophy

Answer: The QRS complexes are very tall
        (increased voltage)
Why is left ventricular hypertrophy characterized by tall QRS
complexes?
 As the heart muscle wall thickens there is an increase in
 electrical forces moving through the myocardium resulting
 in increased QRS voltage.




  LVH          Increased QRS voltage       ECHOcardiogram
   Criteria exists to diagnose LVH using a 12-lead ECG.
       For example:
            The R wave in V5 or V6 plus the S wave in V1 or V2 exceeds
             35 mm.

• However, for now, all
  you need to know is
  that the QRS voltage
  increases with LVH.
Turning our attention to bundle branch blocks…

                             Remember normal
                             impulse conduction is
                               SA node 
                               AV node 
                               Bundle of His 
                               Bundle Branches 
                               Purkinje fibers
Normal Impulse Conduction
Sinoatrial node

   AV node

 Bundle of His

Bundle Branches

 Purkinje fibers
So, depolarization of
the Bundle Branches
and Purkinje fibers
are seen as the QRS
complex on the ECG.

Therefore, a conduction
block of the Bundle
Branches would be          Right
reflected as a change in   BBB
the QRS complex.
With Bundle Branch Blocks you will see two
changes on the ECG.
  1.   QRS complex widens (> 0.12 sec).
  2.   QRS morphology changes (varies depending on ECG
       lead, and if it is a right vs. lef t bundle branch block) .
Why does the QRS complex widen?

When the conduction
pathway is blocked it
will take longer for
the electrical signal
to pass throughout
the ventricles.
What QRS morphology is characteristic?
For RBBB the wide QRS complex assumes a
unique, virtually diagnostic shape in those
leads overlying the right ventricle (V1 and V2).

   V1



     “Rabbit Ears”
What QRS morphology is characteristic?
For LBBB the wide QRS complex assumes a
characteristic change in shape in those leads
opposite the left ventricle (right ventricular
leads - V1 and V2).

                                        Broad,
Normal                                  deep S
                                        waves
THANK YOU!

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ECG

  • 2. To recognize the normal rhythm of the heart - “Normal Sinus Rhythm.”  To recognize the 13 most common heart arrhythmias.  To recognize an acute myocardial infarction on a 12-lead ECG.
  • 3. The 12-Lead ECG contains a wealth of information. In Module V you learned that ST segment elevation in two leads is suggestive of an acute myocardial infarction. In this module we will cover:  ST Elevation and non-ST Elevation MIs  Left Ventricular Hypertrophy  Bundle Branch Blocks
  • 4.
  • 5. When myocardial blood supply is abruptly reduced or cut off to a region of the heart, a sequence of injurious events occur beginning with ischemia (inadequate tissue perfusion), followed by necrosis (infarction), and eventual fibrosis (scarring) if the blood supply isn't restored in an appropriate period of time.  The ECG changes over time with each of these events…
  • 6. Ways the ECG can change include: ST elevation & depression T-waves peaked flattened Appearance inverted of pathologic Q-waves
  • 7. Non-ST Elevation There are two distinct patterns of ECG change depending if the infarction is: ST Elevation –ST Elevation (Transmural or Q-wave), or –Non-ST Elevation (Subendocardial or non-Q-wave)
  • 8. The ECG changes seen with a ST elevation infarction are: Before injury Normal ECG Ischemia ST depression, peaked T-waves, then T-wave inversion Infarction ST elevation & appearance of Q-waves Fibrosis ST segments and T-waves return to normal, but Q-waves persist
  • 9. Here’s a diagram depicting an evolving infarction: A. Normal ECG prior to MI B. Ischemia from coronary artery occlusion results in ST depression (not shown) and peaked T-waves C. Infarction from ongoing ischemia results in marked ST elevation D/E. Ongoing infarction with appearance of pathologic Q-waves and T-wave inversion F. Fibrosis (months later) with persistent Q- waves, but normal ST segment and T- waves
  • 10. Here’s an ECG of an inferior MI: Look at the inferior leads (II, III, aVF). Question: What ECG changes do you see? ST elevation and Q-waves Extra credit: What is the rhythm? Atrial fibrillation (irregularly irregular with narrow QRS)!
  • 11. Here’s an ECG of an inferior MI later in time: Now what do you see in the inferior leads? ST elevation, Q-waves and T-wave inversion
  • 12. The ECG changes seen with a non-ST elevation infarction are: Before injury Normal ECG Ischemia ST depression & T-wave inversion Infarction ST depression & T-wave inversion Fibrosis ST returns to baseline, but T-wave inversion persists
  • 13. Here’s an ECG of an evolving non-ST elevation MI: Note the ST depression and T-wave inversion in leads V2-V6. Question: What area of the heart is infarcting? Anterolateral
  • 14.
  • 15. Compare these two 12-lead ECGs. What stands out as different with the second one? Normal Left Ventricular Hypertrophy Answer: The QRS complexes are very tall (increased voltage)
  • 16. Why is left ventricular hypertrophy characterized by tall QRS complexes? As the heart muscle wall thickens there is an increase in electrical forces moving through the myocardium resulting in increased QRS voltage. LVH Increased QRS voltage ECHOcardiogram
  • 17. Criteria exists to diagnose LVH using a 12-lead ECG.  For example:  The R wave in V5 or V6 plus the S wave in V1 or V2 exceeds 35 mm. • However, for now, all you need to know is that the QRS voltage increases with LVH.
  • 18.
  • 19. Turning our attention to bundle branch blocks… Remember normal impulse conduction is SA node  AV node  Bundle of His  Bundle Branches  Purkinje fibers
  • 20. Normal Impulse Conduction Sinoatrial node AV node Bundle of His Bundle Branches Purkinje fibers
  • 21. So, depolarization of the Bundle Branches and Purkinje fibers are seen as the QRS complex on the ECG. Therefore, a conduction block of the Bundle Branches would be Right reflected as a change in BBB the QRS complex.
  • 22. With Bundle Branch Blocks you will see two changes on the ECG. 1. QRS complex widens (> 0.12 sec). 2. QRS morphology changes (varies depending on ECG lead, and if it is a right vs. lef t bundle branch block) .
  • 23. Why does the QRS complex widen? When the conduction pathway is blocked it will take longer for the electrical signal to pass throughout the ventricles.
  • 24. What QRS morphology is characteristic? For RBBB the wide QRS complex assumes a unique, virtually diagnostic shape in those leads overlying the right ventricle (V1 and V2). V1 “Rabbit Ears”
  • 25. What QRS morphology is characteristic? For LBBB the wide QRS complex assumes a characteristic change in shape in those leads opposite the left ventricle (right ventricular leads - V1 and V2). Broad, Normal deep S waves