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Stroke
SUBMITTED To~ jitendra sir
Kp
SUBMITTED by~Kripendra kumar
Stroke
SUBMITTED BY~Kripendra kumar
SUBMITTED TO~jitendra sir
PHYSIOTHERAPY DIPARTMENT
INTRODUCTION
•Stroke is on acute onset of neurological
dysfunction due to an abnormality in cerebral
circulation with resultant signs and symptoms
which corresponds to involvement of
facilitation areas of the brain.
TRANSIENT ESCHIMIC ATTACK
•It is defined as the sudden onset of
neurological deficiats due to an
abnormality in cerebral circulation with
the sign and symptoms losing for less
than 24 hours.
Kp
EPIDEMIOLOGY
•Thired leading cause of death.
•The incidence of stroke is about 1.25
times greater for males Females.
•Most common cause of disability among
adults.
Kp
ETIOLOGY
• Atherosclerosis
• Cerebral thrombus
• Cerebral embolus
• Embolism from the heart cardioc Origin
• Intracranial haemorrhage
• Subarachoid haemorrhage
• Intracranial small vessel disease
• Arteries venous malformation
• Haematological disorders
} Atherothromolism
MISCELLANEOUS RARE CAUSE OF STROKE
• Infective endocarditis and HIV infection.
• Prioperative stroke (Due to hypotentionand boundary
zone infaction trauma to and dissectio of neck.
• Migrain
• Chronic meningitis
• Inflammatory bowel disease
• hypoglycemio
RUPTURE AND NARROWING ARTERY
RISK FACTORS
•Smoking
•Obesity
•Lack of physical exercise
•Infactin
•psychological factors
•Vasectomy
WARNING SINGS OF STROKE
• Sudden numbness or weakness of face Or leg On
side of body.
• Sudden canfusing traunle speaking or
understanding.
• Sudden onset of dizziness loss of balance are
coordination.
• Sudden severe headaches with no known cause.
• Other inportant but less common stroke Symptoms
include.
CLASSIFICATION
•Dipending an the cause
•haemorrhagic stroke
Introcranial haemorrhage
Subarachnaid haemorrhage
• Thrombotic: more common usually occurs in
The sleeping house.
Characterised by gradual onset of symptoms.
• Embolic: occurs in The walking hours of the
Day sudden onset of symptoms Preceded by
giddiness in most conditions.
•Dipending on the duration
. Acute stroke :to aperoid of one week or
untile spasticity develops.
. Sub acute stroke :after The development of
spasticity and last for a period of 3-12 months
. Chronic stroke : more than 12 months.
•Dipending on the symptoms
• MCA syndrome
• ACA syndrome
• PCA syndrome
• Vertebro basil Artery syndrome
• Vertebral Artery
• Basilor Artery
• Internal caroted artery
• Lacunar syndrome
MCA
•Contralateral hemiplegia(UL and face more
affected than LL)
•Ideomotor aprexia
•Ataxia of contralateral limb
•Limbleft hemisphere infaction.
PCA
• Coordination disorders such as tremor or ataxia
• Cartical blindnesss
• Congnitive impairment including memory
impairment.
• Contralateral sensory impairment
• Thalamic syndrome.
• Contralateral hemiplegia and third nerve palsy.
ACA
•Contralateral hemiplegia or monoplegia
of LL.
•Contralateral sensory loss LL.
•Urinary incontinience.
•Apraxia.
•Contralateral group Reflex.
Primary impairment
• Altered sensation
• Vision
• Weakness
• Alteration of tone
• Abnormal synergy
• Abnormal reflexes
• Altered co-ordination
• Postural control and balance
• Speech, language, and swollowing
• Emotional status
INDIRECT IMPAIRMENTS
•Musculoskeletal changes.
•Neurological signs.
•Cardiac function.
•Pulmonary function
TESTS AND MEASURES
•Urine analysis
•Cumplite blood count
•Blood sugar level
•Blood cholesterol and lipid profile
•Cardiac evaluation
•IMAGNIG
• CT scan
• MRI (magnetic resonance imaging
• Cerebral angiograbhy
PHYSIOTHERAPY
ASSESSMENT
HISTORY
•Abruptly onset with rapid coma is
suggestive of cerebral haemorrhage.
•Embolus also occur rapidly with no wroning
and Is frequently associated with heart
disease or heart complications.
PAST HISTORY
Include head trauma, presence of
manjar or mina risk factors,Medication ,
positive family history, and Alterations in
patient function.
OBSERVATION
•May have Abnormal posturing of limbs.
•Synergist patterns in the upper limb and
lower limb.
•Facial abnormality
•May Use a walking aid,s
•Abnormal gait pattern may also be
observed.
JOINT INTERGRITY AND MOBILIYY
• Glunohumeral subluxation
•Shoulder impingement syndrome.
•Athesive capsulitis
•Complex regional pain syndrome and
shoulder – hand syndrome
RANGE OF MOTION
•Soft tissue shortening and contractures
•Increased muscle stiffness
•Joint immobility
MOTOR FUNCTION
•Synergistis patterns of movement.
•Hypertonicity
•Weakness
•Associated movements or synkinesis.
•Aproxia including motor and verbal
aprexia.
GAIT AND LOCOMOTION
•Decreased extention of hip and
hyperextention Of knee.
•Decrextention of knee and hip during
swing phase.
•Decreased ankle dorsiflation at initial
contact and during stance resulting in
hip circumduction.
BALANCE
•Compromised static as well as dynamic
balence.
•pusher’s syndrome may be present
resulting in fall on the Affected side.
POSTURE
•Spastic patterns can involve flextion and
abduction of ArmFlextion of elbow and
supination of elbow with finger flextion.
•Hip and knee extention with ankle
plantarflexion and inversion.
•Protracted and dispersed shoulder, scoliosis
and hip hiking.
PROBLEM LIST
• Tonal abnormalities.
• Muscular weakness.
• Synergistic pattern.
• Tightness and contractures.
• Imbalance and inco-ordination.
• Gait abnormalities.
• Postural abnormalities.
• Functional disability.
PHYSIOTHERAPY
MANAGEMENT
ACUTE STAGE
•Positioning strategies
•Improve respiratory and circulatary
function.
•Privent pressure sores
•Privent from deconditioning.
POSITIONING STRATEGIES
• In supaine
• In side lying on normal side
• In side lying on affected side
INPROVE RESPIRATORY AND CIRCULATARY
FUNCTION
•Breathing exercise
•Chest expansion exercise
•Postural drainage
•Huffing and coughing techniques
•Possive exercise
PRIVENT PRESSURE SORES
•Proper Positioning
•Relieve pressure by dpadding
•Frequent turning and changing Position
•Prevent from moisture.
PRIVENT FROM DISLOCATION
• Early mobilization in the bed (Active turning
supine to side, side to supine, sitting, sit to
stand)
• Pelvic bridging exercise.
• Early propped up Positioning sitting and then
Laer to standing.
• Moving around the bed
• Facilitate movement of functioning Limbs.
PAST ACUTE STAGE
•5days a week for a minimum of 3hours of
Active rehabilitation per day.
•Intensive rehabilitation it vital ar stable.
IMPROVE SENSORY FUNCTION
•Presentation of Repeated sensory stimulus.
•Streching, stroking, suparficial and deep
pressure, icing, vibration.
•Waight bearing exercise.
•Improve other senses like use of visual and
audiotary.
•PNF technic.
FLEXIBILITY AND JOINT INTERGRITY
•Soft tissue joint mobilization and ROM
exercise.
•AROM and PROM With end range stretch.
•Effective Positioning and edema reduction.
•Streching programme and splitting.
MANAGE SPASTICITY
•Sustained stretch and slow iceing of spastic
muscle.
•Rhythmic rotation
•Weight bearing exercise
•Slow rocking movement
•Positianig in anti synergistic pattern.
•Electrical stimulation.
Thank you

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stroke

  • 1. Stroke SUBMITTED To~ jitendra sir Kp SUBMITTED by~Kripendra kumar Stroke SUBMITTED BY~Kripendra kumar SUBMITTED TO~jitendra sir PHYSIOTHERAPY DIPARTMENT
  • 2. INTRODUCTION •Stroke is on acute onset of neurological dysfunction due to an abnormality in cerebral circulation with resultant signs and symptoms which corresponds to involvement of facilitation areas of the brain.
  • 3. TRANSIENT ESCHIMIC ATTACK •It is defined as the sudden onset of neurological deficiats due to an abnormality in cerebral circulation with the sign and symptoms losing for less than 24 hours. Kp
  • 4. EPIDEMIOLOGY •Thired leading cause of death. •The incidence of stroke is about 1.25 times greater for males Females. •Most common cause of disability among adults. Kp
  • 5. ETIOLOGY • Atherosclerosis • Cerebral thrombus • Cerebral embolus • Embolism from the heart cardioc Origin • Intracranial haemorrhage • Subarachoid haemorrhage • Intracranial small vessel disease • Arteries venous malformation • Haematological disorders } Atherothromolism
  • 6. MISCELLANEOUS RARE CAUSE OF STROKE • Infective endocarditis and HIV infection. • Prioperative stroke (Due to hypotentionand boundary zone infaction trauma to and dissectio of neck. • Migrain • Chronic meningitis • Inflammatory bowel disease • hypoglycemio
  • 8. RISK FACTORS •Smoking •Obesity •Lack of physical exercise •Infactin •psychological factors •Vasectomy
  • 9. WARNING SINGS OF STROKE • Sudden numbness or weakness of face Or leg On side of body. • Sudden canfusing traunle speaking or understanding. • Sudden onset of dizziness loss of balance are coordination. • Sudden severe headaches with no known cause. • Other inportant but less common stroke Symptoms include.
  • 10. CLASSIFICATION •Dipending an the cause •haemorrhagic stroke Introcranial haemorrhage Subarachnaid haemorrhage
  • 11. • Thrombotic: more common usually occurs in The sleeping house. Characterised by gradual onset of symptoms. • Embolic: occurs in The walking hours of the Day sudden onset of symptoms Preceded by giddiness in most conditions.
  • 12. •Dipending on the duration . Acute stroke :to aperoid of one week or untile spasticity develops. . Sub acute stroke :after The development of spasticity and last for a period of 3-12 months . Chronic stroke : more than 12 months.
  • 13. •Dipending on the symptoms • MCA syndrome • ACA syndrome • PCA syndrome • Vertebro basil Artery syndrome • Vertebral Artery • Basilor Artery • Internal caroted artery • Lacunar syndrome
  • 14. MCA •Contralateral hemiplegia(UL and face more affected than LL) •Ideomotor aprexia •Ataxia of contralateral limb •Limbleft hemisphere infaction.
  • 15. PCA • Coordination disorders such as tremor or ataxia • Cartical blindnesss • Congnitive impairment including memory impairment. • Contralateral sensory impairment • Thalamic syndrome. • Contralateral hemiplegia and third nerve palsy.
  • 16. ACA •Contralateral hemiplegia or monoplegia of LL. •Contralateral sensory loss LL. •Urinary incontinience. •Apraxia. •Contralateral group Reflex.
  • 17.
  • 18.
  • 19. Primary impairment • Altered sensation • Vision • Weakness • Alteration of tone • Abnormal synergy • Abnormal reflexes • Altered co-ordination • Postural control and balance • Speech, language, and swollowing • Emotional status
  • 20. INDIRECT IMPAIRMENTS •Musculoskeletal changes. •Neurological signs. •Cardiac function. •Pulmonary function
  • 21. TESTS AND MEASURES •Urine analysis •Cumplite blood count •Blood sugar level •Blood cholesterol and lipid profile •Cardiac evaluation
  • 22. •IMAGNIG • CT scan • MRI (magnetic resonance imaging • Cerebral angiograbhy
  • 24. HISTORY •Abruptly onset with rapid coma is suggestive of cerebral haemorrhage. •Embolus also occur rapidly with no wroning and Is frequently associated with heart disease or heart complications.
  • 25. PAST HISTORY Include head trauma, presence of manjar or mina risk factors,Medication , positive family history, and Alterations in patient function.
  • 26. OBSERVATION •May have Abnormal posturing of limbs. •Synergist patterns in the upper limb and lower limb. •Facial abnormality •May Use a walking aid,s •Abnormal gait pattern may also be observed.
  • 27. JOINT INTERGRITY AND MOBILIYY • Glunohumeral subluxation •Shoulder impingement syndrome. •Athesive capsulitis •Complex regional pain syndrome and shoulder – hand syndrome
  • 28. RANGE OF MOTION •Soft tissue shortening and contractures •Increased muscle stiffness •Joint immobility
  • 29. MOTOR FUNCTION •Synergistis patterns of movement. •Hypertonicity •Weakness •Associated movements or synkinesis. •Aproxia including motor and verbal aprexia.
  • 30. GAIT AND LOCOMOTION •Decreased extention of hip and hyperextention Of knee. •Decrextention of knee and hip during swing phase. •Decreased ankle dorsiflation at initial contact and during stance resulting in hip circumduction.
  • 31. BALANCE •Compromised static as well as dynamic balence. •pusher’s syndrome may be present resulting in fall on the Affected side.
  • 32. POSTURE •Spastic patterns can involve flextion and abduction of ArmFlextion of elbow and supination of elbow with finger flextion. •Hip and knee extention with ankle plantarflexion and inversion. •Protracted and dispersed shoulder, scoliosis and hip hiking.
  • 33. PROBLEM LIST • Tonal abnormalities. • Muscular weakness. • Synergistic pattern. • Tightness and contractures. • Imbalance and inco-ordination. • Gait abnormalities. • Postural abnormalities. • Functional disability.
  • 35. ACUTE STAGE •Positioning strategies •Improve respiratory and circulatary function. •Privent pressure sores •Privent from deconditioning.
  • 36. POSITIONING STRATEGIES • In supaine • In side lying on normal side • In side lying on affected side
  • 37.
  • 38. INPROVE RESPIRATORY AND CIRCULATARY FUNCTION •Breathing exercise •Chest expansion exercise •Postural drainage •Huffing and coughing techniques •Possive exercise
  • 39. PRIVENT PRESSURE SORES •Proper Positioning •Relieve pressure by dpadding •Frequent turning and changing Position •Prevent from moisture.
  • 40. PRIVENT FROM DISLOCATION • Early mobilization in the bed (Active turning supine to side, side to supine, sitting, sit to stand) • Pelvic bridging exercise. • Early propped up Positioning sitting and then Laer to standing. • Moving around the bed • Facilitate movement of functioning Limbs.
  • 41. PAST ACUTE STAGE •5days a week for a minimum of 3hours of Active rehabilitation per day. •Intensive rehabilitation it vital ar stable.
  • 42. IMPROVE SENSORY FUNCTION •Presentation of Repeated sensory stimulus. •Streching, stroking, suparficial and deep pressure, icing, vibration. •Waight bearing exercise. •Improve other senses like use of visual and audiotary. •PNF technic.
  • 43. FLEXIBILITY AND JOINT INTERGRITY •Soft tissue joint mobilization and ROM exercise. •AROM and PROM With end range stretch. •Effective Positioning and edema reduction. •Streching programme and splitting.
  • 44. MANAGE SPASTICITY •Sustained stretch and slow iceing of spastic muscle. •Rhythmic rotation •Weight bearing exercise •Slow rocking movement •Positianig in anti synergistic pattern. •Electrical stimulation.