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APPROACH TO PATIENTS WITH
DYSMENORRHEA
Reem Ahmed Alyahya
INTRODUCTION
 Dysmenorrhea is considered to be the most common gynecologic
complaint in all ages and races and one of the most common causes
of pelvic pain.
 Estimates of the prevalence of dysmenorrhea vary widely (16.8% to
81%3), and rates as high as 90% have been recorded.
 Symptoms typically begin in adolescence and may lead to school and
work absenteeism, as well as limitations on social, academic, and
sports activities.
OBJECTIVES
 Definition
 Types
 Risk factors
 Pathogenesis
 Primary dysmenorrhea
 Secondary dysmenorrhea
DEFINITION
 Its defined as 'painful menstruation'
 Derived from the Greek words: dys, meaning painful; meno, meaning
monthly; and rhoe, meaning flow.
 It is experienced as uterine cramps and can occur a few days prior to
menstruation and/or during menstruation, and usually subsides at
the end of menstruation.
TYPES
Dysmenorrhea is further defined within 2 subcategories:
1. Primary dysmenorrhea: occurs in the absence of an underlying
pelvic pathology.
2. Secondary dysmenorrhea: occurs in the presence of pelvic
pathology.
RISK FACTORS
PROTECTIVE FACTORS
 Regular exercise
 Early child birth
 Oral contraceptive use
PATHOGENESIS
PRIMARY DYSMENORRHEA
PRIMARY DYSMENORRHEA Ionset
6 to 12 months
after menarche,
with peak
prevalence
occurring in the
late teens or early
twenties. Signsand
symptoms
Recurrent, crampy,
suprapubic pain
occurring just
before or during
menses and
lasting 2 to 3
days; pain may
radiate into the
lower back and
thighs.
Associated
symptoms
Nausea or
vomiting, fatigue,
bloating, Head-
ache, diarrhea,
and general
malaise
PRIMARY DYSMENORRHEA II
Physical examination :
Normal
Investigations:
urine tests should be
ordered to rule out
pregnancy or infection
•The pain is a result of normal physiologic process
and has no underlying pathology.
Clarification
•it’s a common problem reported by young females
and the therapy is available and simple.
•It is also common for the pain to either disappear
or substantially lessen after the birth of the first
child.
Reassurance
TREATMENT
TREATMENT: PHARMACOLOGICAL
 NSAIDs is the first line treatment for primary dysmenorrhea, as it will
inhibit prostaglandin synthesis.
 The choice of NSAID should be based on effectiveness and
tolerability for the individual patient.
 Medications should be taken one to two days before the anticipated
onset of menses, and continued on a fixed schedule for two to three
days.
TREATMENT: PHARMACOLOGICAL
TREATMENT: PHARMACOLOGICAL
 Oral, intravaginal, and intrauterine hormonal contraceptives have
been recommended for management of primary dysmenorrhea, as
they will inhibit ovulation.
 However, the evidence supporting their effectiveness is limited.
 Both 28-day and extended cycle oral contraceptives are reasonable
options in women with primary dysmenorrhea.
IT IS RECOMMENDED TO ALLOW AT LEAST THREE
FULL CYCLES TO ASSESS EFFICACY OF EITHER OF
THESE APPROACHES.
TREATMENT: NON-
PHARMACOLOGICAL
Strong evidence of effectiveness:
Topical heat
Moderate exercise
Nutritional interventions
(omega-3 , vitamin B2)
Insufficient evidence of
effectiveness:
Acupuncture
Yoga
massage
TREATMENT: SURGICAL OPTIONS
 If a patient continues to have significant dysmenorrhea with this
treatment, further testing for causes of secondary dysmenorrhea
should be considered, and surgical options, such as interruption of
pelvic nerve pathways, should be explored when applicable.
 e.g. uterosacral nerve ablation and pre-sacral neurectomy
SECONDARY
DYSMENORRHEA
SECONDARY DYSMENORRHEA Ionset
older women with
recent history of
dysmenorrhea
Signsand
symptoms
Pain is continuous
dull aching lower
abdominal pain
accompanied by
backache occurring
in parous women
after many years of
relatively painless
menstruation.
Associated
symptoms
menorrhagia,
intermenstrual
bleeding,
dyspareunia, post-
coital bleeding, and
infertility.
SECONDARY DYSMENORRHEA II
Suspected condition Physical examination
findings
Investigations required
Endometriosis:
deep dyspareunia, dysuria,
dyschezia, and subfertility
fixed or retroverted uterus
or reduced uterine
mobility, adnexal masses,
and uterosacral nodularity.
 Transvaginal and pelvic
ultrasonography
 laparoscopy with biopsy
and histology is the
preferred diagnostic test
Pelvic inflammatory
disease: History of lower
abdominal pain in sexually
active patients
cervical motion
tenderness, uterine
tenderness, and/or
adnexal tenderness, fever
and abnormal cervical or
vaginal mucopurulent
discharge.
 Saline microscopy of
vaginal fluid may show
organism.
 elevated inflammatory
markers
SECONDARY DYSMENORRHEA III
Suspected condition Physical examination
findings
Investigations required
Adenomyosis:
Usually associated with
menorrhagia and
intermenstrual bleeding.
enlarged, tender, boggy
uterus
 Transvaginal
ultrasonography will
detect endometrial
tissue within the
myometrium.
Leiomyomata “ fibroid “ :
Cyclic pelvic pain with
menorrhagia and
dyspareunia
firm, enlarged, and
irregularly shaped uterus
 Transvaginal
ultrasonography
TREATMENT
Treatment of the underlying
pathology.
CONCLUSION
1. Dysmenorrhea is one of the most common causes of pelvic pain,
and It negatively affects patients’ quality of life and results in
activity restriction.
2. A history and physical examination is usually the only things
needed to diagnose primary dysmenorrhea.
3. The presence of menorrhagia, intermenstrual bleeding,
dyspareunia, post- coital bleeding, and infertility should raise the
physician’s suspicion to the presence of a secondary cause.
REFERENCES
 American and family physician: Diagnosis and Initial Management
of Dysmenorrhea
 Current diagnosis and treatment in family medicine, 4th edition
 BMJ : Assessment of dysmenorrhea
THANK YOU FOR
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clinical approach to patients with dysmenorrhea

  • 1. APPROACH TO PATIENTS WITH DYSMENORRHEA Reem Ahmed Alyahya
  • 2. INTRODUCTION  Dysmenorrhea is considered to be the most common gynecologic complaint in all ages and races and one of the most common causes of pelvic pain.  Estimates of the prevalence of dysmenorrhea vary widely (16.8% to 81%3), and rates as high as 90% have been recorded.  Symptoms typically begin in adolescence and may lead to school and work absenteeism, as well as limitations on social, academic, and sports activities.
  • 3. OBJECTIVES  Definition  Types  Risk factors  Pathogenesis  Primary dysmenorrhea  Secondary dysmenorrhea
  • 4. DEFINITION  Its defined as 'painful menstruation'  Derived from the Greek words: dys, meaning painful; meno, meaning monthly; and rhoe, meaning flow.  It is experienced as uterine cramps and can occur a few days prior to menstruation and/or during menstruation, and usually subsides at the end of menstruation.
  • 5. TYPES Dysmenorrhea is further defined within 2 subcategories: 1. Primary dysmenorrhea: occurs in the absence of an underlying pelvic pathology. 2. Secondary dysmenorrhea: occurs in the presence of pelvic pathology.
  • 6. RISK FACTORS PROTECTIVE FACTORS  Regular exercise  Early child birth  Oral contraceptive use
  • 9. PRIMARY DYSMENORRHEA Ionset 6 to 12 months after menarche, with peak prevalence occurring in the late teens or early twenties. Signsand symptoms Recurrent, crampy, suprapubic pain occurring just before or during menses and lasting 2 to 3 days; pain may radiate into the lower back and thighs. Associated symptoms Nausea or vomiting, fatigue, bloating, Head- ache, diarrhea, and general malaise
  • 10. PRIMARY DYSMENORRHEA II Physical examination : Normal Investigations: urine tests should be ordered to rule out pregnancy or infection
  • 11. •The pain is a result of normal physiologic process and has no underlying pathology. Clarification •it’s a common problem reported by young females and the therapy is available and simple. •It is also common for the pain to either disappear or substantially lessen after the birth of the first child. Reassurance TREATMENT
  • 12. TREATMENT: PHARMACOLOGICAL  NSAIDs is the first line treatment for primary dysmenorrhea, as it will inhibit prostaglandin synthesis.  The choice of NSAID should be based on effectiveness and tolerability for the individual patient.  Medications should be taken one to two days before the anticipated onset of menses, and continued on a fixed schedule for two to three days.
  • 14. TREATMENT: PHARMACOLOGICAL  Oral, intravaginal, and intrauterine hormonal contraceptives have been recommended for management of primary dysmenorrhea, as they will inhibit ovulation.  However, the evidence supporting their effectiveness is limited.  Both 28-day and extended cycle oral contraceptives are reasonable options in women with primary dysmenorrhea.
  • 15. IT IS RECOMMENDED TO ALLOW AT LEAST THREE FULL CYCLES TO ASSESS EFFICACY OF EITHER OF THESE APPROACHES.
  • 16. TREATMENT: NON- PHARMACOLOGICAL Strong evidence of effectiveness: Topical heat Moderate exercise Nutritional interventions (omega-3 , vitamin B2) Insufficient evidence of effectiveness: Acupuncture Yoga massage
  • 17. TREATMENT: SURGICAL OPTIONS  If a patient continues to have significant dysmenorrhea with this treatment, further testing for causes of secondary dysmenorrhea should be considered, and surgical options, such as interruption of pelvic nerve pathways, should be explored when applicable.  e.g. uterosacral nerve ablation and pre-sacral neurectomy
  • 19. SECONDARY DYSMENORRHEA Ionset older women with recent history of dysmenorrhea Signsand symptoms Pain is continuous dull aching lower abdominal pain accompanied by backache occurring in parous women after many years of relatively painless menstruation. Associated symptoms menorrhagia, intermenstrual bleeding, dyspareunia, post- coital bleeding, and infertility.
  • 20. SECONDARY DYSMENORRHEA II Suspected condition Physical examination findings Investigations required Endometriosis: deep dyspareunia, dysuria, dyschezia, and subfertility fixed or retroverted uterus or reduced uterine mobility, adnexal masses, and uterosacral nodularity.  Transvaginal and pelvic ultrasonography  laparoscopy with biopsy and histology is the preferred diagnostic test Pelvic inflammatory disease: History of lower abdominal pain in sexually active patients cervical motion tenderness, uterine tenderness, and/or adnexal tenderness, fever and abnormal cervical or vaginal mucopurulent discharge.  Saline microscopy of vaginal fluid may show organism.  elevated inflammatory markers
  • 21. SECONDARY DYSMENORRHEA III Suspected condition Physical examination findings Investigations required Adenomyosis: Usually associated with menorrhagia and intermenstrual bleeding. enlarged, tender, boggy uterus  Transvaginal ultrasonography will detect endometrial tissue within the myometrium. Leiomyomata “ fibroid “ : Cyclic pelvic pain with menorrhagia and dyspareunia firm, enlarged, and irregularly shaped uterus  Transvaginal ultrasonography
  • 22. TREATMENT Treatment of the underlying pathology.
  • 23. CONCLUSION 1. Dysmenorrhea is one of the most common causes of pelvic pain, and It negatively affects patients’ quality of life and results in activity restriction. 2. A history and physical examination is usually the only things needed to diagnose primary dysmenorrhea. 3. The presence of menorrhagia, intermenstrual bleeding, dyspareunia, post- coital bleeding, and infertility should raise the physician’s suspicion to the presence of a secondary cause.
  • 24. REFERENCES  American and family physician: Diagnosis and Initial Management of Dysmenorrhea  Current diagnosis and treatment in family medicine, 4th edition  BMJ : Assessment of dysmenorrhea

Notas del editor

  1. primary dysmenorrhea is caused by the release of prostaglandins from the endometrium at the time of menstruation, which induce smooth muscle contraction in the uterus, causing pressure within the uterus to exceed that of the systemic circulation. Ischemia ensues, causing an anginal equivalent in the uterus. The cause of secondary dysmenorrhea varies with the underlying disease.
  2. A pelvic examination is not essential for adolescents with symp- toms of primary dysmenorrhea who have never had vaginal intercourse. The diagnosis is clinical
  3. reassurance are an essential part of management. The intensity of pain may be aggravated by apprehension and fear, and reassurance that the pain does not indicate any serious disorder may lessen the symptoms.