Diabetes ppt

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Type 2 Diabetes and Genes

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Diabetes mellitus is a metabolic disease, characterized by high glucose level in blood (hyperglycaemia). Insulin-dependent diabetes mellitus (IDDM) Juvenile-onset diabetes Type1 diabetes is characterized by the presence of antibodies in blood. Glutamic Acid Decarboxylase antigen (GAD) Insulin autoantibodies (IAAs), Islet cell autoantibodies (ICAs). These are the markers of the immune destruction of the ß cell. Therefore, those with more than one autoantibody(i.e., ICA, IAA, GAD etc.) are at high risk.

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Diabetes Diabetes A disease that effects your body’s ability to produce or use insulin Insulin is a hormone produced in the pancreas that regulates the metabolism of glucose and other nutrients. Types of Diabetes Type 1 diabetes is a chronic condition in which the pancreas produces little or no insulin, a hormone needed to allow sugar (glucose) to enter cells to produce energy. Type 2 diabetes is a chronic condition that affects the way your body metabolizes sugar (glucose), your body's main source of fuel. Incidence of Diabetes in US Total: 25.8 million children and adults in the United States—8.3% of the population—have diabetes. Under 20 years of age •215,000, or 0.26% of all people in this age group have diabetes •About 1 in every 400 children and adolescents has diabetes Age 20 years or older •25.6 million, or 11.3% of all people in this age group have diabetes Age 65 years or older •10.9 million, or 26.9% of all people in this age group have diabetes Men •13.0 million, or 11.8% of all men aged 20 years or older have diabetes Women •12.6 million, or 10.8% of all women aged 20 years or older have diabetes Causes Type 1: genetic predisposition and environmental factors Viruses may trigger the disease in genetically susceptible individuals Produce no insulin in the pancreas Type 2: Obesity; poor diet Lack of physical activity High blood pressure Symptoms Type 1: Unexplained weight loss (even though you are eating and feel hungry) Insulin Shock Loss of consciousness (rare) Diabetic Coma Hypoglycemia(low blood sugar) Blurred vision Fatigue Symptoms continued Type 2: Increased thirst and frequent urination. Increased hunger/weight gain Fatigue Blurred vision Diabetic Coma Diagnosis and Tests of Diabetes Random blood sugar test Fasting Blood Glucose (FBG) Glycated hemoglobin (A1C) test Prevention Type 1 Unknown way to prevent Researchers are working on preventing the disease or further destruction of the islet cells in people who are newly diagnosed Type 2 stay at a healthy weight/Eat healthy Increase physical actives Lose weight Lower your blood sugar Treatment Type 1 Taking insulin Exercising regularly and maintaining a healthy weight Eating healthy foods Monitoring blood sugar Type 2 Blood sugar monitoring Healthy eating Home Base Business Opportunities http://freedomofsuccess.com A complete 8-Step 'Money Getting' Formula ($297 Value) In this High-Value Video Training Series, I'll learn the most effective ways to build a successful online business and the Core 'Must Have' principals to Effective Entrepreneurship in today’s market. These videos outline your BIGGEST money making principals all in one place Watch Video Here http://freedomofsuccess.com

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MODY is the name given to a collection of different types of inherited forms of diabetes that usually develop in adolescence or early adulthood. MODY stands for “Maturity-onset diabetes of the young” and was given that name in the past because it acted more like the adult type of diabetes (Type 2 Diabetes) but was found in young people. MODY limits the body’s ability to produce insulin, but is different than the juvenile type of diabetes (Type 1 Diabetes). When our bodies don’t produce enough insulin, it can increase blood glucose levels. High blood glucose levels lead to diabetes.

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Diabetes is a serious condition when your body cannot make or effectively use a hormone called insulin. Since there is insufficient insulin, your body cannot move glucose from your bloodstream into your muscle, fat, and liver cells. It leads to a glucose surplus in your bloodstream. Type 1 and type 2 diabetes are the most common forms. Still, there are other forms, such as prediabetes and gestational diabetes. If you’re living with diabetes, you probably wondered how you developed diabetes or whether your children will develop it, too. Sometimes you inherit a predisposition to diabetes, or something in your environment triggers it. While the exact cause of most types of diabetes is unknown, age, gender, weight, genetic makeup, family medical history, ethnicity, and environmental factors can influence the risk of developing diabetes. Therefore, there is no common cause of diabetes that fits every diabetes subgroup, as it varies depending on the individual and the type. Type 1 diabetes is an autoimmune condition where the body has auto-antibodies that destroy the insulin-producing pancreatic cells. As these cells decrease, the body’s ability to create insulin decreases. The little to no insulin causes glucose to build up in your blood. Because the destroyed pancreatic cells cannot supply the body with sufficient insulin, type 1 diabetes leaves you insulin-dependent for life. Therefore, it’s also known as insulin-dependent diabetes. Studies show that about 5%–10% of diabetics are type 1. ==> Simple 1 Minute “Diet Hack” REVERSES Type 2 Diabetes For decades, there was a common misconception of type 1 diabetes being a juvenile disease that typically appears in early childhood or adolescence. However, type 1 diabetes can develop at any age but often gets diagnosed at a younger age. The destruction of the insulin-producing pancreatic beta cells varies from person to person. Some people experience a gradual decrease in beta cells, while some lose the functioning beta cells exceptionally quickly. During the onset, the pancreas can still produce some insulin. Therefore, an outside source of insulin is necessary during this phase. Still, as the body continues to reduce the amount of insulin produced, insulin levels from an external source must get adjusted. Keywords best medicine for sugar patient impaired fasting glucose management management of impaired glucose tolerance diabetes mellitus management ppt type 1 diabetes management plan herbs to lower sugar remedies to reduce blood sugar home remedies to lower cholesterol and blood sugar acog intrapartum management of diabetes impaired glucose tolerance in pregnancy management management of hyperglycemia in icu hypoglycemia management nhs helps manage blood sugar blood sugar reduction using natural medicine management of diabetic patient in dental clinic ppt home remedies to reduce sugar level in blood diabetes management plan 2019 sick day plan for type 1 diabetes diasend uploader omnipod wound manageme

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Diabetes is the condition in which the body does not properly process food for use as energy. Most of the food we eat is turned into glucose, or sugar, for our bodies to use for energy. The pancreas, an organ that lies near the stomach, makes a hormone called insulin to help glucose get into the cells of our bodies. When you have diabetes, your body either doesn't make enough insulin or can't use its own insulin as well as it should. This causes sugars to build up in your blood. This is why many people refer to diabetes as ―sugar‖ Diabetes can cause serious health complications including, heart disease, blindness, kidney failure, and lower-extremity amputations.

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Dr. Faiza delivers an insightful lecture on the distinguishing characteristics of skeletal, smooth, and cardiac muscles, offering a comprehensive understanding of their histology, physiological anatomy, electrophysiological properties, and contractile mechanisms. Through meticulous tabulation and comparison, she aims to equip learners with a detailed comprehension of the unique features and functionalities of each muscle type. Beginning with a detailed exploration of histological differences, Dr. Faiza outlines key distinctions such as multinucleation in skeletal muscle, mononucleation in smooth and cardiac muscles, and the presence of striations in skeletal and cardiac muscles. She meticulously elucidates the structural arrangement of each muscle type, highlighting their cellular morphology and organization within tissues. Transitioning to physiological anatomy, Dr. Faiza navigates through various aspects including innervation, level of control, initiation of contraction, and modification by hormones. By delineating the role of the somatic nervous system in skeletal muscle control, and contrasting it with the autonomic nervous system's influence on smooth and cardiac muscles, she offers a nuanced understanding of neural regulation in muscle physiology. Furthermore, Dr. Faiza delves into electrophysiological properties, elucidating the mechanisms underlying action potential generation, duration, and ionic basis in each muscle type. Through insightful analysis, she reveals how differences in action potential characteristics contribute to variations in muscle contraction speed, duration, and resistance to fatigue. Finally, Dr. Faiza meticulously examines the contractile mechanisms of skeletal, smooth, and cardiac muscles, shedding light on the sliding filament theory, mechanisms of calcium action, and speed of contraction. By comprehensively comparing these mechanisms, she provides learners with a deep understanding of the intricate processes governing muscle contraction and function. Through engaging presentation and meticulous analysis, Dr. Faiza's lecture serves as a valuable resource for students seeking a thorough understanding of the fundamental differences between skeletal, smooth, and cardiac muscles.

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