Gap Acidosis

THE CASE ,[object Object],[object Object],[object Object],[object Object]

THE ANION GAP ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]

Increased lactate production ,[object Object],[object Object],[object Object],[object Object]

Increased lactate production ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]

Increased lactate production ,[object Object],[object Object],[object Object],[object Object],[object Object]

Primary decrease in lactate utilization ,[object Object],[object Object],[object Object]

Mechanism uncertain ,[object Object],[object Object],[object Object],[object Object],[object Object]

Toxins Isopropanol is primarily metabolized via alcohol dehydrogenase to acetone

Clinical course in acute ethylene glycol intoxication ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]

Clinical course in acute ethylene glycol intoxication 12 to 24 hours Cardiovascular Mild hypertension, tachycardia, and shock Pulmonary Tachypnea adult respiratory distress syndrome pulmonary edema pneumonitis Metabolic Metabolic acidosis with elevated anion gap decreased osmolal gap possible tetany from hypocalcemia, and hyperventilation

[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]

Conclusion ,[object Object],[object Object],[object Object]

Gap Acidosis

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Gap Acidosis