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Cap 18 Parte V
- 1. M édula adrenal Liberacion continua: tono a bajos niveles ([hormona]) Dos poblaciones de celulas Epi y NEpi
- 3. Activacion de la medula adrenal Músculo esquelético Glucogeno se hidroliza Glucosa ATP Tejido Adiposo TG FFAs Otros tejidos: ATP Hígado Glucógeno se hidroliza Corazón Receptores β 1 ↑ HR, ↑dP/dt
- 4. Glandula Pineal Pinealocitos Melatonina precursor = serotonina Podria jugar un rol Relojes biologicos niveles bajos durante el dia Comienzo de madurez sexual Niveles de Melatonina bajan en la pubertad
- 8. prepro / proinsulina / insulina
- 17. Increased rate of glucose transport into target cell Increased rate of glucose utilization and ATP generation Increased conversion of glucose to glycogen (liver, skeletal muscle) Increased amino acid absorption and protein synthesis Increased triglyceride synthesis (adipose tissue) Beta cells secrete insulin Rising blood glucose levels Normal glucose levels (70 110 mg/dL) Declining blood glucose levels Increased breakdown of glycogen to glucose (liver, skeletal muscle) Increased breakdown of fats to fatty acids (adipose tissue) Increased synthesis and release of glucose (liver) Alpha cells secrete glucagon Blood glucose concentration rises Blood glucose concentration declines
- 19. Diabetes Mellitus Insulina = Receptor de insulina Donde podría estar el problema?
- 31. Riñón From Liver Lung capillaries Adrenal Cortex Hyporhalamus Hyporhalamus
- 35. Fuente: Molecular Biology of the Cell, Alberts et al
Notas del editor
- Compelling evidence was gathered that signified that this factor was probably acting in the hypothalamus to modulate metabolism. In 1994, a breakthrough discovery revealed that the obese (ob) gene was encoding a small protein produced mainly by fat tissue, named leptin (Zhang et al., 1994).